ライフサイエンスコーパス: BP

1 BP (ND) levels were similar for (11)C-FEKAP and (11)C-GR

2 BP as a partial domesticate.

3 BP did not interfere with CAR assembly or the rate of CA

4 BP may differ considerably when measured in the office a

5 BP reduction after RDN was similar for patients with var

6 BP reduction in patients with measurements at 6, 12, 24,

7 BP was directly measured in conscious animals at the end

8 BP(ND) in the ES group was negatively correlated with po

9 BP) from El Gigante rock shelter, Honduras, that are clo

10 BP, promoted the commensal behaviour of the house mouse,

11 BP-3 and PP exposure caused R-loop formation in a normal

12 sT-1 from 60 to 90 min matched best with 1TC BP (ND) Conclusion: The novel synaptic vesicle glycoprot

13 eduction in post-amphetamine [(11)C]CIMBI-36 BP(ND)(frontal).

14 (55.6%) MR OA-using participants, 25 (69.4%) BP OA-using participants, and 16 (44.4%) participants us

15 pothesized that ketamine activates mTORC1-4E-BP signalling in pyramidal excitatory cells of the corte

16 Accordingly, interfering with the mTORC1/4E-BP/eIF4E axis inhibited the growth potential endowed by

17 Moreover, cells with 4E-BP knockdown were resistant to BBR-induced cell death an

18 ically disordered eIF4E binding proteins (4E-BPs) regulates cap-dependent translation by weakening th

19 nological innovation, and from ca. 6400-5900 BP, underpinned a ~four-fold human population growth.

20 Between ~7600-5900 BP, intense exploitation of a warmer, more productive ma

21 geo-cultural sequence dating between 20.5kyr BP to 6.5kyr BP and represents an ideal location in whic

22 sequence dating between 20.5kyr BP to 6.5kyr BP and represents an ideal location in which to explore

23 his case suggests that (18)F-alpha(v)beta(6)-BP PET/CT is a promising noninvasive approach to identif

24 genetic structure began to develop by 5,800 BP, followed by bi-directional gene flow between the Nor

25 conversion efficiency of 19.83% from 16.95% (BP-free).

26 fects of black phosphorus (BP) nanosheets, a BP-PAO fiber with enhanced uranium extraction capacity a

27 s treated by MT, 576 (64%) were handled on a BP system.

28 red to all other participants, [(11)C]ABP688 BP(ND) values were lower in the striatum, OFC, and insul

29 In these regions, low [(11)C]ABP688 BP(ND) values were only seen in the high risk group that

30 5, P = 0.02) and in external cohorts (ACCORD-BP, ORIGIN, and TRIUMPH, total N = 3059, P = 0.005).

31 oup and 2% and 0% from the MT group achieved BP less than 140/90 mm Hg and less than 130/80 mm Hg wit

32 decrease (P<0.001), while TPR did not affect BP.

33 it is unknown whether this receptor affects BP under type 1 diabetes.

34 ndividuals dating from ~9,000-500 years ago (BP), with a particular focus on the period of the rise a

35 has no overall effect on daytime ambulatory BP compared with a dairy-free diet.

36 with CKD, BP metrics derived from ambulatory BP monitoring are associated with cardiovascular outcome

37 ese data support the wider use of ambulatory BP monitoring in the evaluation of hypertension in patie

38 (N(cases)/N(controls) = 59,674/316,078) and BP (N = 757,601), we find positive genetic correlations

39 -retest variability for 1TC V (T) (<=5%) and BP (ND) (<=10%).

40 parison of the 2 sequence groups' (MR-BP and BP-MR) REI showed the median differences between T0 and

41 role in modulation of vascular function and BP in experimental models in vivo and in humans.

42 le of known mediators (diabetes mellitus and BP) by adjusting for measured values (conventional analy

43 est was measured as the change in BP(ND) and BP(P) after amphetamine.

44 moderate relation between cardiac output and BP responses after placebo administration (r = 0.53, P =

45 line D(2/3) receptor availability (BP(P) and BP(ND)) and amphetamine-induced dopamine release (DeltaB

46 ive medications, hypertension remission, and BP control according to current guidelines (<130/80 mm H

47 was significantly lower in SP than in RF and BP.

48 cs were assessed and compared between SP and BP.

49 in scan was sufficient to quantify V (T) and BP (ND) The test-retest study showed excellent absolute

50 ation in the regulation of vascular tone and BP and suggest a novel mechanism, and therapeutic target

51 C) facilitating the establishment of an anti-BP CD4 T cell-dependent adaptive immune response leading

52 e D2/3 receptor availability was measured as BP(ND) using [(11)C]raclopride PET after antipsychotic d

53 pressure (BP) was measured with an automatic BP monitor.

54 Baseline D(2/3) receptor availability (BP(P) and BP(ND)) and amphetamine-induced dopamine relea

55 ivity (MSNA; microneurography), beat-to-beat BP (photoplethysmography) and heart rate (electrocardiog

56 Benzophenone (BP) is present in a variety of bioactive molecules.

57 HR, 4.10 [CI, 3.68 to 4.55]) and with better BP control over time.

58 nters not necessarily equipped with biplane (BP) angiosuites are performing mechanical thrombectomy (

59 We test for association with both BP traits and expression within these tissues or cell ty

60 The primary outcome was control of BP (BP < 140/90 mm Hg), analysed using mixed effects regress

61 UCAST, and investigational CLSI breakpoints (BPs).

62 e by H. sapiens, which extends to 34,000 cal BP.

63 t radiocarbon date between 2,440 and 100 cal BP, from two ancestral Ohlone sites in Central Californi

64 gesting a peak between 30,425 and 29,772 cal BP (2sigma error) which matches more depleted delta(18)O

65 tratigraphic layer dated 41.1 to 38.1 ka cal BP, documenting a modern human presence on the western m

66 lbenzylamine or phenylglycine-derived chiral BP synthons incorporating a photolabile protecting group

67 In this cohort of participants with CKD, BP metrics derived from ambulatory BP monitoring are ass

68 tcomes, and mortality, independent of clinic BP.

69 ed a small incremental advantage over clinic BP in predicting cardiovascular/renal events, which was

70 long-term cardiovascular events than clinic BP.

71 lue of 24-h BP phenotypes compared to clinic BP in predicting the subsequent fatal and non-fatal card

72 ouling activity is fabricated by compositing BP nanosheets into polyacrylamidoxime (PAO).

73 tial relative to total plasma concentration (BP(P)) were derived using an arterial input function-bas

74 ity previously observed for dyads containing BP covalently linked to thymidine, the aim of this work

75 Controlled BP was more likely among those with private insurance (4

76 Compared with having controlled BP, the presence of masked uncontrolled hypertension ind

77 US population, the prevalence of controlled BP increased between 1999-2000 and 2007-2008, did not si

78 It was estimated that controlled BP was less likely among non-Hispanic Black adults vs no

79 ing intervention is effective in controlling BP and is potentially scalable in resource-poor settings

80 The four waveforms and manual cuff BP were recorded before and after slow breathing, mental

81 Cumulative BP was calculated as the area under the curve (mm Hgxyea

82 Low-salt diets significantly decreased BP levels in normotensive Afro-Caribbean people and in h

83 s underestimate mediating roles of diabetes, BP, and their correlates.

84 h was also the case for ambulatory diastolic BP (P = 0.45).

85 its for both systolic BP (SBP) and diastolic BP (DBP), and further assessed the direction of the vari

86 SD -0.11; 95% CI -0.19, -0.02) and diastolic BP (SD -0.11; 95% CI -0.19, -0.04).

87 e effects of elevated systolic and diastolic BP on large artery stroke.

88 phocyte count with systolic BP and diastolic BP.

89 alue = 4.72 x 10-3), and automated diastolic BP measurement (beta 0.09, 95% CI, 0.03-0.16, P value =

90 7 ACC/AHA (systolic BP <130 mm Hg, diastolic BP >=80 mm Hg) and by JNC7 (systolic BP <140 mm Hg, dias

91 d by JNC7 (systolic BP <140 mm Hg, diastolic BP >=90 mm Hg) definitions.

92 < 0.001) and a greater decline in diastolic BP (-2.1 mm Hg, 95% CI -3.6 to -0.6; P < 0.006), but no

93 by 6.55 mm Hg (SD 15.17), and mean diastolic BP by 4.23 mm Hg (SD 8.68).

94 c, central-diastolic, and systemic-diastolic BP was lower at 6 mo in the 1000-IU group [-2.66 (95% CI

95 etic correlations of migraine with diastolic BP (DBP, r(g) = 0.11, P = 3.56 x 10(-06)) and systolic B

96 Higher cumulative systolic and diastolic BPs were associated with slower walking speed (both P=0.

97 The earliest BP decrease occurred 9 minutes before syncope.

98 Elevated BP variation was associated with a wide range of subclin

99 after adjusting for age, sex, BMIZ, elevated BP, and hypercholesterolemia (RR, 1.43; P = .02).

100 han 500 adults with hypertension or elevated BP and that were 6 months or longer in duration.

101 n vegetables on BP in subjects with elevated BP, with the aim of elucidating if any such effect is re

102 For DD methods, EUCAST BPs demonstrated lower susceptibility at 65% and 66%, co

103 Among these factors, BP aggregates have been suggested to promote immunogenic

104 harply decrease the size of as-exfoliated FL-BP flakes.

105 , and in the clinical processes relevant for BP control.

106 tobehavior of a series of dyads derived from BP and Thy, separated by linear linkers of different len

107 showed similar reductions in office and 24-h BP for patients with varying baseline ASCVD risk scores,

108 In AA, 24-h BP monitoring provides limited added value as a predicto

109 250-1,000 predictions depending on the 24-h BP phenotype.

110 study is to evaluate the added value of 24-h BP phenotypes compared to clinic BP in predicting the su

111 P was no more predictive than the other 24-h BP phenotypes.

112 ate which could not be isolated, the heavier BP-doped analogue exhibits remarkable solution and solid

113 2% of patients with hypertension with a high BP measurement during an ambulatory visit received an or

114 with high BP in one site tended to have high BP in another site.

115 s correlated to show that patients with high BP in one site tended to have high BP in another site.

116 he associated mechanisms that lead to higher BP are unknown.

117 Exposure to higher BP levels from young to midlife is associated with worse

118 d uncontrolled hypertension and mean 24-hour BP associated with high risk of cardiovascular disease a

119 Twenty-four-hour BP phenotypes conferred a small incremental advantage ov

120 However, BP-3 and PP had limited transactivation of target genes

121 if blockade of the TLR4-MD2 complex impacts BP and vascular function in diabetic rats.

122 ram innovation has high potential to improve BP but may be expensive and burdensome for patients, cli

123 This report describes the 4-decade change in BP levels for the population in a high disease risk sout

124 ns of interest was measured as the change in BP(ND) and BP(P) after amphetamine.

125 measures were intervention uptake, change in BP, change in clinician appointment use, and participant

126 e associated with BP (N = 16) and changes in BP over time (N = 6).

127 ell tolerated without significant changes in BP, weight, or serum potassium.

128 ssed variation as the absolute difference in BP divided by the mean over 2 sequential visits for both

129 ed results or reduce existing disparities in BP control.

130 onstrate substantial room for improvement in BP control (<140/90 mm Hg), which was 58% overall, and i

131 ic layers, whereas this effect is limited in BP lattices due to their spring-shaped space structure.

132 nician workload and results in reductions in BP similar to those in large UK trials.

133 Alterations of diurnal variation in BP are associated with high risk of progression of kidne

134 nalysis, predictors of incident CKD included BP >140/90 mm Hg, higher glycated hemoglobin, lower base

135 In conclusions, increased BP status or the fasting serum glucose level status were

136 e is significantly associated with increased BP in middle-aged men.

137 CM components and that laminin 211 increases BP proliferation in embryonic mouse neocortex.

138 here it normally is not expressed, increases BP proliferation, reduces Tbr2 levels and induces Olig2

139 w BP-associated loci and 55 were independent BP-associated single-nucleotide variants within known BP

140 k on clinical event reduction by RDN-induced BP changes could be evaluated in further studies.

141 Intensive BP-lowering treatment decreases risk for OH.

142 f BP pharmacologic treatment (more intensive BP goal or active agent) that involved more than 500 adu

143 , before or in the setting of more intensive BP treatment, should not be viewed as a reason to avoid

144 rrent clinical literature supports intensive BP lowering in patients with hypertension for improving

145 Finally, we provide insight into BP-independent biological pathways underlying SVD and su

146 the spacer could modulate the intramolecular BP/Thy photoreactivity, resulting in an enhanced selecti

147 CUDs showed significantly lower [(11)C]UCB-J BP(ND) in the hippocampus (~10%, p = 0.008, effect size

148 jor maximum in the gradient from 48 to 40 ka BP is a new feature of the IntCal20 calibration curve, w

149 geomagnetic excursion centered around 41 ka BP.

150 imately 14 thousand years before present (ka BP).

151 ated single-nucleotide variants within known BP-associated regions.

152 s could be the underlying mechanisms linking BP variation to dementia and stroke.

153 The 2 major factors lowering BP in tilt-induced vasovagal syncope were reduced SV and

154 ney injury in knockout mice without lowering BP.

155 tihypertensive medications while maintaining BP less than 140/90 mm Hg.

156 fetal growth on pregnancy outcomes, maternal BP, and glucose levels during pregnancy.

157 Mean BP was computed using 3 measurements.

158 group allocation, maximal reductions in mean BP were observed at 28.7 ng/mL of achieved serum 25-hydr

159 After adjusting for diabetes and measured BP, chi-squared values for associations for waist-to-hip

160 trials examining the impact of self-measured BP monitoring on cardiovascular outcomes are needed.

161 Self-measured BP monitoring, the measurement of BP by an individual ou

162 Conditional Sox9 expression in the mouse BP lineage, where it normally is not expressed, increase

163 Comparison of the 2 sequence groups' (MR-BP and BP-MR) REI showed the median differences between

164 ty is experimentally reported for multilayer BP along the armchair direction.

165 Concentrations of 1 muM and 5 muM BP-3 or PP increased DNA damage similar to that of E2 tr

166 million participants, we discovered 106 new BP-associated genomic regions and 87 rare (minor allele

167 ns (P < 5 x 10(-8)), of which 32 were in new BP-associated loci and 55 were independent BP-associated

168 Nocturnal BP was no more predictive than the other 24-h BP phenoty

169 1 and 2, we identified 84 known and 18 novel BP loci at genome-wide significance level (P < 5 x 10(-8

170 Further analysis of BP variants establishes that variants at MECOM/3q26, FGF

171 The primary outcome was control of BP (BP < 140/90 mm Hg), analysed using mixed effects reg

172 Control of BP improved from baseline to follow-up more in the inter

173 The detection of BP with thickening of the peripapillary vitreous by SD-O

174 We investigated the effect of BP-3 and PP on estrogen receptor-dependent transactivati

175 The mechanisms underpinning the impact of BP exposure on brain structure and function must be inve

176 , we report an experimental investigation of BP membrane in osmotic energy conversion and reveal how

177 f-measured BP monitoring, the measurement of BP by an individual outside of the office at home, is a

178 sociated with migraine and the mechanisms of BP-lowering medications in migraine prophylaxis are unkn

179 y conversion and reveal how the oxidation of BP influences power generation.

180 minated, with the current 75th percentile of BP generally less than the 25th percentile of BP in 1960

181 P generally less than the 25th percentile of BP in 1960.

182 ction, rendered by concurrent positioning of BP, delivered a progressively enhanced power conversion

183 on efficiency (BPE), defined as the ratio of BP to photosynthesis, and its variation across and withi

184 Randomized trials of BP pharmacologic treatment (more intensive BP goal or ac

185 , but no detectable difference in the use of BP-lowering medications between groups (OR 1.2, 95% CI 0

186 ed cardiovascular risk independent of office BP.

187 the office setting, and higher out-of-office BP is associated with increased cardiovascular risk inde

188 e, is a validated approach for out-of-office BP measurement.

189 supplementation has a differential effect on BP control remains unclear.

190 hat time, SV had a strong negative effect on BP, TPR a lesser negative effect, while HR had increased

191 d the relative effects of SV, HR, and TPR on BP to be assessed separately, although all act together.

192 ated the effect of leafy green vegetables on BP in subjects with elevated BP, with the aim of elucida

193 MI, body composition or lipid parameters, or BP between the 3 groups at 24 wk.

194 weight, body composition, lipid profile, or BP.

195 r incidence of diabetes in people with SZ or BP and (2) higher incidence of major mental illnesses in

196 a unilateral case of Bergmeister's papilla (BP) in a young female patient suffering from type 1 sial

197 n-butyl phthalate (DnBP), and butyl paraben (BP) and diluted to make creams with concentrations rangi

198 [(11)C]-(+)-PHNO BP(ND), the binding potential relative to the nondisplac

199 Black phosphorus (BP) is a monoelemental 2D material predicted to be piezo

200 tion-related defects in 2D black phosphorus (BP) is exploited to achieve visual memory, wavelength-se

201 photocatalytic effects of black phosphorus (BP) nanosheets, a BP-PAO fiber with enhanced uranium ext

202 Black phosphorus (BP), an emerging layered material, has recently been exp

203 measured (LOQ 40 copies/mL) in blood plasma (BP), seminal plasma (SP), rectal fluid (RF), and cervico

204 del-based nondisplaceable binding potential (BP (ND)) to select the optimal time window in healthy an

205 bution volume (V (T)) and binding potential (BP (ND)).

206 The frontal cortex binding potential (BP(ND)(frontal)) was calculated as (V(T)(frontal)/V(T)(c

207 The primary outcome was binding potential (BP(ND)) in the anterior cingulate cortex (ACC).

208 ffness and neural control of blood pressure (BP) among older adults.

209 s on the association between blood pressure (BP) and dementia traits.

210 diet has been found to lower blood pressure (BP) and low-density lipoprotein cholesterol levels.

211 nfluences daytime ambulatory blood pressure (BP) and other cardiometabolic risk factors.

212 sly established variants for blood pressure (BP) and the FTO variant has also been associated with bo

213 Whereas blood pressure (BP) control in the population and subsequent reduced hyp

214 for maintenance of arterial blood pressure (BP) during haemorrhage in humans.

215 ive medications, global mean blood pressure (BP) has remained constant or has decreased slightly over

216 nting in the child, and high blood pressure (BP) in the older adult woman.

217 Blood pressure (BP) is a cardiovascular parameter applied to detect card

218 High blood pressure (BP) is a risk factor for cardiovascular morbidity and mo

219 icacy of dietary patterns on blood pressure (BP) lowering but their findings are largely conflicting.

220 Blood pressure (BP) management is a crucial part of critical care that d

221 x 10-10), automated systolic blood pressure (BP) measurement (beta 0.11, 95% CI, 0.03-0.18, P value =

222 of this study was to assess blood pressure (BP) reduction and event rates after RDN in patients with

223 A greater blood pressure (BP) response to contraction was observed in PAD rats.

224 Genetic studies of blood pressure (BP) to date have mainly analyzed common variants (minor

225 Large blood pressure (BP) variability may contribute to stroke and dementia, b

226 Blood pressure (BP) was inconsistently associated with migraine and the

227 Blood pressure (BP) was measured with an automatic BP monitor.

228 associated with lowering of blood pressure (BP), but the nutrient(s) responsible for these effects r

229 ffness, central and systemic blood pressure (BP), insulin sensitivity (1/fasting insulin concentratio

230 ge in systolic and diastolic blood pressure (BP), Short Physical Performance Battery (SPPB), Montreal

231 modulates vascular tone and blood pressure (BP), the role of its accessory protein phospholemman has

232 etween urinary compounds and blood pressure (BP).

233 or for hypertension and high blood pressure (BP).

234 ecordings), and beat-to-beat blood pressure (BP; photoplethysmography) during the pre- and post-drug

235 evidence suggests that 24-h blood pressures (BP) are more predictive of long-term cardiovascular even

236 cally derived carbon for biomass production (BP).

237 oned into plants organs (biomass production, BP) or-more generally-for the production of organic matt

238 drug Abs in response to biological products (BP) is a major drawback in the treatment of patients.

239 their segregation into bipotent progenitors (BPs) and unipotent pro-acinar cells (PACs).

240 ults and prevalence of US adults recommended BP pharmacotherapy by the 2017 ACC/AHA guideline based s

241 dic, and portfolio diets effectively reduced BP.

242 or to dietary sodium restriction in reducing BP in patients with CKD stage G3 or G4 and hypertension,

243 Here, engineered antigen removed BP (ARBP) that was cross-linked with 0.2% and 0.6% gluta

244 amine has been demonstrated to raise resting BP, although it has not been studied with the concomitan

245 Initial examination revealed BP 90/60 mm Hg and tachycardia.

246 in Drosophila that RIM-binding protein (RIM-BP) connects release sites physically and functionally t

247 The RIM-BP N-terminal domain, while dispensable for SV release s

248 RNA-binding proteins (RNA-BPs) play critical roles in development and disease to r

249 Severe BP elevations commonly observed in the 1960s have been n

250 ht (SD -0.12; 95% CI -0.14, -0.09), systolic BP (SD -0.11; 95% CI -0.19, -0.02) and diastolic BP (SD

251 IDH, by 2017 ACC/AHA (systolic BP <130 mm Hg, diastolic BP >=80 mm Hg) and by JNC7 (sys

252 Comparison of postdiet ambulatory systolic BP revealed no difference (P = 0.34), which was also the

253 (g) = 0.11, P = 3.56 x 10(-06)) and systolic BP (SBP, r(g) = 0.06, P = 0.01), but not pulse pressure

254 n over 2 sequential visits for both systolic BP (SBP) and diastolic BP (DBP), and further assessed th

255 uced a greater reduction in 24-hour systolic BP (SBP; from 138 to 124 mm Hg) compared with sodium res

256 However, systolic BP variability was significantly reduced with 2000 IU (a

257 rmine the magnitude of the shift in systolic BP (SBP) among Blacks and Whites from the Southeast betw

258 ses, there was a greater decline in systolic BP in the intervention than UC group (-5.0 mm Hg, 95% CI

259 astolic BP >=80 mm Hg) and by JNC7 (systolic BP <140 mm Hg, diastolic BP >=90 mm Hg) definitions.

260 agonist (MRA) and with longitudinal systolic BP.

261 1.8 x 10-4) and increased maternal systolic BP during pregnancy (p = 2.2 x 10-2).

262 2012 decile of 8 [IQR 6-10]), mean systolic BP fell by 6.55 mm Hg (SD 15.17), and mean diastolic BP

263 and 800 IU vitamin D3 reduced mean systolic BP over 2 y to a small and similar extent, 2000 IU reduc

264 imilar extent, 2000 IU reduced mean systolic BP variability significantly more compared with 800 IU.

265 5% CI], 1.23 to 1.83), preoperative systolic BP (aOR, 1.16 per 10-mm Hg increase; 95% CI, 1.05 to 1.2

266 CI, 1.10 to 1.66) and preoperative systolic BP (aOR, 1.17; 95% CI, 1.06 to 1.30).

267 compared intensive versus standard systolic BP lowering (targeting <120 mm Hg versus <140 mm Hg, res

268 ction, arterial stiffness, systemic-systolic BP, lipids, and inflammatory markers did not differ betw

269 rs divided into groups according to systolic BP (SBP): G1 (n = 16), resting SBP <110 mmHg and G2 (n =

270 HF drugs were assessed according to systolic BP categories (<95, 95-109, 110-129, and >=130 mm Hg).

271 lationship of lymphocyte count with systolic BP and diastolic BP.

272 iscussion is warranted regarding what target BP is acceptable and what should be limiting factors in

273 The BP measured at the four most common sites (left upper ar

274 ment effect of sacubitril/valsartan, and the BP-lowering effects of sacubitril/valsartan did not acco

275 l with the rate constants estimated from the BP triplet lifetimes.

276 ed that HT attenuates the enhancement of the BP response induced by stimulation of P2X in muscle affe

277 cope, SV and HR contributed similarly to the BP decrease (P<0.001), while TPR did not affect BP.

278 e interventions, it is unclear whether these BP changes can be generalized to diverse and high-risk p

279 ubitril/valsartan on outcomes are related to BP lowering, particularly among women who derive greater

280 ions for these and other tissues relevant to BP.

281 susceptibility and shared biology underlying BP and migraine.

282 otential relative to nondisplaceable uptake (BP(ND)) and binding potential relative to total plasma c

283 are (minor allele frequency <= 0.01) variant BP associations (P < 5 x 10(-8)), of which 32 were in ne

284 In vivo BP and regional blood flow were assessed using Doppler f

285 While BP is regulated by the function of kidney, vasculature,

286 food-related compounds were associated with BP (N = 16) and changes in BP over time (N = 6).

287 led foods were significantly associated with BP, other endogenous and food-related compounds were ass

288 ) were compared in terms of correlation with BP in humans.

289 icity with similar numbers for patients with BP (0.89, 76%, and 88%, respectively).

290 n 150 patients with SZ and 151 patients with BP and compared with 200 control subjects.

291 Patients with SZ and patients with BP could be differentiated with an accuracy of 0.86 (who

292 y for improved photovoltaic performance with BP incorporation.

293 ased liberation of "old" (6855 +/- 120 years BP) refractory carbon as dissolved organic carbon (DOC).

294 the Dark Age Cold Period (1550 to 1250 years BP) and the Little Ice Age (700 to 200 years BP) are sug

295 of the Roman Warm Period (1950 to 1550 years BP) indicate a wetter ISM.

296 BP) and the Little Ice Age (700 to 200 years BP) are suggestive of reduced ISM rainfall, whereas lowe

297 the Medieval Warm Period (1200 to 800 years BP) and the major portion of the Roman Warm Period (1950

298 yr BP and maize (Zea mays) at about 6,850 cal.

299 eolithic (MP) occupation dates to >51,000 yr BP.

300 te to an active convective regime at 9000 yr BP and has remained strong thereafter.