ライフサイエンスコーパス: BPI

1 BPI (bactericidal/permeability-increasing) is a potent a

2 BPI and BFI were completed for 7 consecutive days (days

3 BPI and LBP(N)-BPI(C) promote apparently CD14-independen

4 BPI binding to the bacterial surface rapidly triggers po

5 BPI could thus induce a p53-like response even in the pr

6 BPI gene transcript and protein were detected in airway

7 BPI has two domains with the same fold, but with little

8 BPI is based on the formulation of the original BrainPhy

9 BPI levels at 15 min of dialysis with CTA (10.91 +/- 3.6

10 BPI mRNA was detected in bovine retina; retinal pigment

11 BPI produced a dose-dependent increase (up to 3-fold) in

12 BPI was previously shown to retain aspects of its own fu

13 BPI, but not control protein thaumatin, activated extrac

14 BPI-CETP chimeras are inhibited by LPS but cannot be inh

15 BPI-derived peptide P2 rapidly halted oxygen consumption

16 BPI-enhanced delivery of the blebs to MDDC did not incre

17 BPI-inducible protein A (BipA) is a member of the family

18 BPI-reported pain reduction from baseline to 4 weeks aft

19 BPIs occurred in less than 1% of patients.

20 all imaging tractography study comparing 118 BPI patients and 86 healthy control individuals.

21 a 1.02-point lower (95% CI, -1.58 to -0.47) BPI score at 12 months (3.57 vs 4.59).

22 itiation factor (eIF) 4E-binding protein (4E-BPI), sensitizes fibroblasts to apoptosis in a manner st

23 domain of BPI, thus creating the notion of a BPI/PLUNC structural superfamily.

24 all-bowel pathogen that causes cholera, to a BPI-derived peptide, P2.

25 xhibited IgG antibody autoreactivity against BPI in 64.7% and 46.7% of patients with positive blood c

26 how disrupted Shh signaling protects against BPI could uncover variants in the Shh pathway that cause

27 ectively demonstrated seropositivity against BPI fold containing family A member 1 (BPIFA1), BPI fold

28 Baseline FACT-G (P = .0016) and BPI (P = .0001) scores were significantly associated wit

29 Both, fluorescently labeled blebs and BPI were internalized by MDDC under these conditions.

30 in of LBP, COOH-terminal domain of BPI), and BPI(N)-LBP(C) with purified (3)H-LPS and, subsequently,

31 detailed clinical observations that EvC and BPI do not occur in the same individuals, led us to hypo

32 ctivities of combinations of cathepsin G and BPI were additive, as were those of combinations of cath

33 factors, along with elongation factor G and BPI-inducible protein A.

34 Thus, defensins, cathepsin G, and BPI are the major anti-H. capsulatum effector molecules

35 n the presence of Ca(2+) and Mg(2+), LBP and BPI each promote aggregation of LPS to protein-LPS aggre

36 human monocyte response to LPS, both LBP and BPI inhibited LPS-stimulated TNF-alpha production in mou

37 udy presented here, plasma levels of LBP and BPI were measured, predialysis, 15 min into dialysis and

38 6) Da), but only LPS associated with LBP and BPI(N)-LBP(C) is disaggregated in the presence of CD14.

39 ther, the association between LBP levels and BPI release during hemodialysis and clinical and laborat

40 nces, the physical properties of LBP-LPS and BPI-LPS complexes have been compared in this study by se

41 t structural differences between SPLUNC1 and BPI.

42 rt of bacteremic patient sera exhibited anti-BPI IgG responses in 23/154 (14.9%) patients with autore

43 status models (ASMs) were considered: ASM1 (BPI and schizoaffective disorder, BP cases (SABP) only),

44 with myeloid differentiation, such as AZU1, BPI, CTSG, LYZ and RNASE2 as well as of antiproliferativ

45 eptor, endothelin type A receptor, lamin B1, BPI fold-containing family B member 1, peroxisomal trans

46 Overall, mean (SD) baseline BPI scores in the intervention and control groups were 5

47 ier after Mg2+-induced displacement of bound BPI.

48 nd functional interference scale of the BPI (BPI-FI).

49 fold containing family A member 1 (BPIFA1), BPI fold containing family A member 2 (BPIFA2)/parotid s

50 y meaningful pain progression as assessed by BPI-SF pain severity was longer with enzalutamide than w

51 ses of E. coli to sublethal injury caused by BPI.

52 isolate sensitive to synergistic killing by BPI and defensins.

53 ort that a specific inhibitor of BCL6 called BPI can trigger a p53 response in DLBCL cells.

54 Compared with controls, BPI patients had significant reductions in mean generali

55 helium, sites where the previously described BPI family members are not expressed.

56 e-N-propoxy bacteriopurpurinimide (dipropoxy-BPI), which was readily oxidized in oxygen atmosphere yi

57 a proband affected with bipolar I disorder (BPI) and at least one other member affected with BPI or

58 n euthymic subjects with bipolar I disorder (BPI) and healthy control subjects.

59 most linkage studies of Bipolar I disorder (BPI) have used relatively small samples.

60 hrough a proband who had bipolar I disorder (BPI) were interviewed by a psychiatrist, assigned an all

61 studies of biomarkers of bipolar I disorder (BPI).

62 corresponding disulfide analogue (disulfide-BPI).

63 ell line was treated in vitro with disulfide-BPI, yielding a CC50 value of 0.05 +/- 0.005 muM.

64 ive bearing 3,4-ethylenedioxythiophene (EDOT-BPI) and its palladium complex (EDOT-PdBPI) were synthes

65 ns but produce profoundly different effects: BPI and a bioactive N-terminal fragment BPI-21 exert a s

66 Epithelial BPI was, in part, responsible for killing a commensal st

67 Neutrophil extract BPI content on post-BMT days +20, +30, and +100 (169+/-3

68 , the Cohen kappa coefficients were 0.99 for BPI, 0.99 for BPII, and 0.98 for recurrent unipolar diso

69 28); there was no significant difference for BPI-SF item 3 or pain interference.

70 ier in blood and exudate cells than mRNA for BPI, consistent with release of progressively less matur

71 that GPC4 is a specific binding protein for BPI on RPE to mediate the activation of ERK1/2, Akt, and

72 Risk for BPI was more weakly predicted by deviations from the FCA

73 A possible role for BPI in clearance of cell-free endotoxin has also been su

74 0 using the Brief Pain Inventory-Short Form (BPI-SF) were randomly assigned to receive either O3-FAs

75 nd were the Brief Pain Inventory-Short Form (BPI-SF), Brief Fatigue Inventory (BFI), Functional Asses

76 ales of the Brief Pain Inventory-short form (BPI-sf), length of Intensive Care Unit stay and postoper

77 ncluded the Brief Pain Inventory-Short Form (BPI-SF), Western Ontario and McMaster Universities Osteo

78 25 with the Brief Pain Inventory Short Form (BPI-SF).

79 pain score (Brief Pain Inventory-Short Form [BPI-SF] question 3 worst pain, score </=3 vs >/=4).

80 ssed by the Brief Pain Inventory Short Form [BPI-SF] questionnaire) and health-related quality of lif

81 hree of the Brief Pain Inventory Short Form [BPI-SF] questionnaire) or mildly symptomatic (score of 2

82 em 3 on the Brief Pain Inventory-Short Form [BPI-SF]) and symptomatic skeletal events, alongside prep

83 cts: BPI and a bioactive N-terminal fragment BPI-21 exert a selective and potent antibacterial effect

84 Cationic peptides derived from BPI exhibit anti-inflammatory activity.

85 n, and a peptidomimetic (XMP.Z) derived from BPI were examined.

86 n even when over 40% of the sequence is from BPI.

87 ing EvC in the Amish confers protection from BPI.

88 is [(BPI)Cu(O2CPh)] (1-O2CPh), formed from [(BPI)Cu(PPh3)2], oxidant, and benzoic acid.

89 ecombinant 21-kDa modified fragment of human BPI (rBPI21), containing the active antimicrobial and en

90 fragments and the crystal structure of human BPI have established that BPI consists of two functional

91 .4 angstroms, the crystal structure of human BPI shows a boomerang-shaped molecule formed by two simi

92 Further, while human BPI also suppressed LPS-dependent NO secretion in mouse

93 etic studies of BP disorder since bipolar I (BPI) and BPII disorders often cluster in the same famili

94 gation of high numbers of EvC and Bipolar I (BPI) cases in several large Amish families descending fr

95 reatment outcome in patients with bipolar I (BPI) disorder.

96 We previously identified BPI fold-containing family B member 3 (BPIFB3) as a regu

97 Risk for SZ and bipolar illness (BPI) are predicted by school achievement (SA) at age 16

98 a neuromedium called BrainPhys(TM) Imaging (BPI) in which we optimize the concentrations of fluoresc

99 results localize the site of LPS binding in BPI to a region no larger than the amino terminal 155 am

100 rical pain responder (> or = 30% decrease in BPI) than the 137 patients in the usual-care group (P <

101 The relative deficiency in BPI of newborns raised the possibility that supplementin

102 ved from newborn cord blood are deficient in BPI.

103 existence of an anatomic disconnectivity in BPI and further underscore a role for interhemispheric d

104 tervention group had greater improvements in BPI pain severity over the 12 months of the trial whethe

105 , and subcortical structures, are present in BPI subjects, even while euthymic.

106 Median time to progression in BPI-SF pain at its worst was 5.7 months (95% CI 5.6-5.7)

107 pathophysiological features of psychosis in BPI.

108 Secondary pain outcomes included BPI interference and severity, global pain improvement,

109 with known antibacterial activity including BPI, EPX, MPO and AZU1.

110 The bactericidal/permeability increasing (BPI) and lipopolysaccharide (LPS)-binding (LBP) proteins

111 er of the bacterial/permeability increasing (BPI) fold-containing protein family, which shares struct

112 er of the bacterial/permeability-increasing (BPI) family of antimicrobial proteins but was first iden

113 of the bactericidal/permeability-increasing (BPI) fold-containing (BPIF) protein family, sharing stru

114 own as bactericidal/permeability-increasing (BPI) fold-containing protein, family A, member 1 (BPIFA1

115 of the bactericidal/permeability-increasing (BPI) gene with pulmonary function in a cohort of patient

116 donor bactericidal/permeability-increasing (BPI) haplotypes were associated with a 2-fold to 3-fold

117 Bactericidal/permeability-increasing (BPI) protein has been shown to play an important role in

118 to the bactericidal/permeability-increasing (BPI) protein.

119 ne effects on the Behavioral Problems Index (BPI) and Home Observation Measurement of the Environment

120 index [BDI] versus bronchoprotection index [BPI]: 1.62 +/- 0.21 versus 2.02 +/- 0.40 [mean +/- SEM],

121 We defined 2 bone PET indices (BPIs) that incorporate anatomic information from CT and

122 ndelian disease loci in bipolar individuals (BPI and BPII, p = 0.06).

123 Moreover, heparitinase also inhibited BPI actions on VEGF and PDGF-B mRNA expression induced b

124 hly sensitive to the BCL6 peptide inhibitor, BPI.

125 nal 10% time on boosted protease inhibitors (BPIs) was associated with reduced KS incidence in the th

126 seroma, paresthesia, brachial plexus injury (BPI), and lymphedema was available for 821 patients.

127 otein-protein interactions similar to intact BPI.

128 fter treatment, by the Brief Pain Inventory (BPI) and the M D Anderson Symptom Inventory (MDASI).

129 utcome measure was the Brief Pain Inventory (BPI) average pain severity score (range 0-10, where 0=no

130 bscale (NTX subscale), Brief Pain Inventory (BPI), and UNISCALE (UNI).

131 The Brief Pain Inventory (BPI), Functional Assessment of Cancer Therapy-Hepatobili

132 al (FACT-G) scale, the Brief Pain Inventory (BPI), the Medical Outcomes Study (MOS) questionnaire, an

133 d by question 3 of the Brief Pain Inventory (BPI).

134 st moderate intensity (Brief Pain Inventory [BPI] score >/=5).

135 , cancer-related pain (Brief Pain Inventory [BPI] worst pain score > or = 6), or both.

136 scale) and sustained (Brief Pain Inventory [BPI], 0- to 10-point scale) change in pain.

137 that the resting state of the catalyst is [(BPI)Cu(O2CPh)] (1-O2CPh), formed from [(BPI)Cu(PPh3)2],

138 The tridentate bipyridylimino isoindoline (BPI) ligands of these complexes were designed to enable

139 ptors, we have compared interactions of LBP, BPI, LBP(N)-BPI(C) (NH(2)-terminal domain of LBP, COOH-t

140 Lower KS incidence was observed with longer BPI use, after accounting for potential IRIS and other f

141 primary end point was the difference in mean BPI-SF worst pain scores at 6 weeks, which was lower for

142 et of N-substituted benzoperylene monoimide (BPI) fluorophores was synthesized and characterized stru

143 udy examines the feasibility of using murine BPI (mBPI) expressed on halophilic Archaeal gas vesicle

144 ve compared interactions of LBP, BPI, LBP(N)-BPI(C) (NH(2)-terminal domain of LBP, COOH-terminal doma

145 BPI and LBP(N)-BPI(C) promote apparently CD14-independent LPS associati

146 respectively) was similar to the neutrophil BPI content of normal controls (163+/-35 ng per 106 neut

147 studies led to the identification of nickel BPI complexes that could undergo stable charge-discharge

148 Compared with baseline, the mean observed BPI-SF score decreased by 1.74 points at 12 weeks and 2.

149 played synergy with CAP18p in the absence of BPI.

150 roup analyses revealed similar activation of BPI and control subjects during passive viewing but sign

151 Analysis of BPI fragments and the crystal structure of human BPI hav

152 and at least one post-baseline assessment of BPI-SF item 3, and patient-reported outcomes (PROs) were

153 We also show the superior capacity of BPI for optogenetics and calcium imaging of human neuron

154 The 972 kindreds include 2284 cases of BPI disorder, 498 individuals with bipolar II disorder (

155 uthors have extended the characterization of BPI interaction with membrane proteins from bovine RPE.

156 rophil extracts were analyzed for content of BPI by Western blotting and ELISA and for defensin pepti

157 minal domain of LBP, COOH-terminal domain of BPI), and BPI(N)-LBP(C) with purified (3)H-LPS and, subs

158 omain, corresponding to the active domain of BPI, thus creating the notion of a BPI/PLUNC structural

159 The N-terminal domains of BPI and LBP bound lipid A with their characteristic appa

160 ability of E. coli to tolerate low doses of BPI and escape the progression of sublethal to lethal da

161 esicles ("blebs"), we examined the effect of BPI on interactions of metabolically labeled ([(14)C]-ac

162 racterized the previously unknown effects of BPI in the eye and the molecular mechanisms involved in

163 Early (<15 min) reversible effects of BPI on bacterial colony-forming ability and outer membra

164 The constitutive expression of BPI was restricted to cell-bound protein and unaffected

165 inant 21-kDa modified N-terminal fragment of BPI (rBPI(21)), which has the biological properties of t

166 t 21 kDa modified amino-terminal fragment of BPI (rBPI21) reduced H2O2-induced apoptosis in RPE and i

167 n a recombinant 21-kDa bioactive fragment of BPI, rBPI21, and the gram-positive pathogen Streptococcu

168 Because the interaction of BPI with cell-free endotoxin, during infection, occurs m

169 possibility that supplementing the levels of BPI in plasma might enhance newborn antibacterial defens

170 nome-wide significant evidence of linkage of BPI to a candidate region of approximately 10 cM in 5q31

171 ngrafted marrow contain normal quantities of BPI and defensins.

172 structure of PSP and known active regions of BPI.

173 To evaluate the therapeutic role of BPI in ischemic retinopathies, the antiangiogenic activi

174 These findings suggest a novel role of BPI in the interaction of bacterial outer membrane vesic

175 were found in the SA and IQs of siblings of BPI vs matched control probands.

176 electron microscopy confirmed the storage of BPI in primary granules and showed that p15s are stored

177 n 524 subjects in a genetic linkage study of BPI disorder.

178 We assessed the utility as such of BPI fold-containing family A member 2 (BPIFA2), also kno

179 terminus of CETP and the carboxy terminus of BPI did not retain any observable CETP function.

180 rmeability, the mechanistic understanding of BPI's action may provide novel therapeutic opportunities

181 36 (IRR = 0.14; 95% CI, .02-1.00) months on BPIs compared with <6 months.

182 Months on BPIs was associated with lower KS incidence (P = .02).

183 (LOD) 3.4 for rs722642 at 78 cM) using only BPI and schizoaffective (SA), BP cases.

184 were those of combinations of cathepsin G or BPI with HNP-1, HNP-2, and HNP-3.

185 phorylation was not reduced by either LBP or BPI under conditions that result in selective TNF-alpha

186 o LPS was also not modified by either LBP or BPI.

187 han that of CAP18p ( approximately 20 nM) or BPI ( approximately 50 nM).

188 Diagnoses of SZ or BPI in the Swedish Hospital Discharge Register and the S

189 Secondary outcomes were improvement in other BPI subscales and at other time points, FACT-Hep and EOR

190 rences occurred over time in sustained pain (BPI mean pain, 0.07 point [CI, -0.23 to 0.37 points]; BP

191 content of the antimicrobial (poly)peptides BPI and defensins.

192 pain, 0.07 point [CI, -0.23 to 0.37 points]; BPI worst pain, -0.14 point [CI, -0.59 to 0.31 points]),

193 s itself related to the host defense protein BPI (bactericidal/permeability-increasing protein).

194 actericidal-permeability-increasing protein (BPI) also mediated fungistasis against H. capsulatum in

195 actericidal permeability-increasing protein (BPI) and lipopolysaccharide (LPS)-binding protein, have

196 actericidal/permeability-increasing protein (BPI) and lipopolysaccharide-binding protein (LBP), which

197 actericidal permeability increasing protein (BPI) differs from LBP by inhibiting LPS-induced human mo

198 actericidal/permeability-increasing protein (BPI) fold-containing family B, member 3 (BPIFB3) is an e

199 actericidal/permeability-increasing protein (BPI) fold-containing family B, member 6 (BPIFB6), whose

200 actericidal/permeability-increasing protein (BPI) had been shown to possess anti-inflammatory and end

201 actericidal/permeability-increasing protein (BPI) has been shown to induce various effects in retinal

202 actericidal/permeability-increasing protein (BPI) is a natural protein, stored within the neutrophil

203 actericidal/permeability-increasing protein (BPI) is found in the primary granules of adult neutrophi

204 actericidal/permeability-increasing protein (BPI) is strongly associated with Pseudomonas aeruginosa

205 actericidal/permeability-increasing protein (BPI) is thought to play an important role in killing and

206 actericidal/permeability-increasing protein (BPI) of neutrophils is a lipopolysaccharide (LPS)-bindin

207 actericidal/permeability-increasing protein (BPI) released from polymorphonuclear leukocytes (PMNs) i

208 actericidal/permeability-increasing protein (BPI) to 1.7 A.

209 actericidal/permeability-increasing protein (BPI) was originally identified as a lipopolysaccharide (

210 actericidal/permeability-increasing protein (BPI), a member of a genomically conserved lipid-interact

211 actericidal/permeability-increasing protein (BPI), an antibacterial and lipopolysaccharide-neutralizi

212 actericidal/permeability-increasing protein (BPI), which neutralizes LPS.

213 actericidal/permeability-increasing protein (BPI)-derived anti-microbial peptide.

214 actericidal/permeability-increasing protein (BPI)-derived peptide P2 and the murine alpha-defensin cr

215 actericidal/permeability-increasing protein (BPI).

216 actericidal/permeability increasing protein (BPI).

217 actericidal/permeability-increasing protein (BPI).

218 actericidal/permeability-increasing protein (BPI).

219 actericidal/permeability-increasing protein (BPI).

220 acteridical/permeability-increasing protein (BPI).

221 bactericidal/permeability inducing protein (BPI), and lipopolysaccharide binding protein (LBP) are m

222 otein glypican 4 (GPC4) serves as a putative BPI-binding protein.

223 These artificial fusions retain BPI functions such as lipopolysaccharide (LPS) binding a

224 RI-BPI showed superior duration of tissue penetration and c

225 Finally, RI-BPI could kill primary human DLBCL cells but had no effe

226 ide, retroinverso BCL6 peptide inhibitor (RI-BPI) selectively killed BCR rather than OxPhos-type DLBC

227 Notably, peptide (RI-BPI) and small molecule (FX1) BCL6 inhibitors derepresse

228 The RI-BPI could recapitulate the failure to form germinal cent

229 In this multicenter sample, BPI patients had reduced WM integrity in interhemispheri

230 t (ICE) and Brief Pain Inventory pain score (BPI-PS), is associated with patient perception of benefi

231 Mean (SE) BPI neuropathic pain severity score declined from 6.2 (0

232 Since BPI has unusual dual properties of promoting RPC and RPE

233 n though the chimera was able to retain some BPI-like properties.

234 Peak and steady state BPI concentrations were comparable with pharmacokinetic

235 olipase C, and anti-GPC4 antibody suppressed BPI-induced ERK and Akt phosphorylation in bovine RPE.

236 The synthesized BPIs exhibit positive solvachromatic emission (lambda(em

237 come was associated with improved short-term BPI scores (per $1,000, beta = -0.47; P = 0.01) and medi

238 nts were associated with improved short-term BPI scores (per $1,000, beta = -0.57; P = 0.04).

239 of addition of recombinant 21-kDa N-terminal BPI fragment (rBPI(21)) on the growth and tumor necrosis

240 Previously, we demonstrated that BPI autoreactivity in CF patient sera exhibits high avid

241 structure of human BPI have established that BPI consists of two functionally distinct domains: a pot

242 The authors recently reported that BPI can induce ERK1/2 and Akt activity and that it incre

243 Scanning electron microscopy revealed that BPI treatment produced greater membrane perturbations in

244 hmark available neuronal media and show that BPI enhances fluorescence signals, reduces phototoxicity

245 Altogether, our study shows that BPI improves the quality of a wide range of fluorescence

246 he results of the present study suggest that BPI is tightly regulated and functionally expressed by e

247 These data suggest that BPI-derived compounds may have unique therapeutic potent

248 The BPI, Western Ontario and McMaster Universities Osteoarth

249 sis of the two indices indicated that as the BPI increased, the BDI reached a plateau.

250 ain from bone metastases, as measured by the BPI, increased from 39 of 149 (26%) before SBRT to 55 of

251 with placebo also significantly improved the BPI average pain interference score, the Fibromyalgia Im

252 d a significantly greater improvement in the BPI average pain severity score (P=0.015; estimated diff

253 These results suggest mutations in the BPI gene significantly influence the risk of developing

254 nically significant change at 1 month in the BPI subscale of symptom on average in the past 24 hours.

255 30) and functional interference scale of the BPI (BPI-FI).

256 a combination of systematic variation of the BPI ligand and the metal center along with mechanistic i

257 ents had superior scores on all items of the BPI-FI (ie, general activity, mood, walking ability, nor

258 nically meaningful (mean 3.4 [SD 2.9] on the BPI pain-at-its-worst item at baseline, 2.1 [2.4] at 4 w

259 were mostly asymptomatic at baseline: on the BPI-SF pain severity scale of 0-10, median pain scores (

260 s remains elusive, our data suggest that the BPI peptide sensitivity of OmpU-deficient V. cholerae is

261 for response using the ICE and 605 using the BPI-PS.

262 Similar results were obtained using the BPI-PS; observed improvements were typically of lesser m

263 Primary outcome was the BPI total score, which ranges from 0 ("no pain") to 10 (

264 Whereas the BPI family are predicted to share very closely similar t

265 larity searches have shown homology with the BPI-like family of innate immune genes, particularly wit

266 Pain was assessed with the BPI-SF questionnaire, and health-related quality of life

267 Thirty BPI and 26 control subjects underwent functional magneti

268 nal portion that imparts opsonic activity to BPI.

269 Antibodies to BPI and bacterial antigens were measured in sera by ELIS

270 is autoreactivity by examining antibodies to BPI in bacteremia patients.

271 related LPS-binding protein, in contrast to BPI, did not increase association of the blebs with MDDC

272 suggest that the fate of E. coli exposed to BPI depends on both OmpR-independent mechanisms engaged

273 y depends on the high molar ratio of p15s to BPI in the extracellular fluid (approximately 50:1), whi

274 = 0.29; P < .001), and negatively related to BPI (r = -0.41; P < .0001).

275 a indicate that low-avidity IgG responses to BPI can arise acutely in response to bacteremia and that

276 Two BPI-derived compounds, rBPI(21) and XMP.Z, significantly

277 A structural alignment between the two BPI domains was used to compare the 3D-1D environments o

278 ction was significantly stronger (BDI versus BPI: 3.40 +/- 0.43 versus 6.98 +/- 1.42, p = 0.02).

279 al properties of LPS complexed to LBP versus BPI.

280 oarthritis, and taxane use, adjusted 12-week BPI-SF scores did not differ by arm (P = .58).

281 d proteins in CP compared with controls were BPI, ITGAM, CAP37, PCM1, MMP-9, MZB1, UGTT1, PLG, RAB1B,

282 es (RPC), and endothelial cells (REC); while BPI protein was measured in human vitreous and plasma.

283 ts with BP (13 males and 26 females; 28 with BPI and 11 with BPII) and 35 healthy controls were inves

284 and at least one other member affected with BPI or bipolar II disorder (BPII), we identified four re

285 rees limited to relative pairs affected with BPI.

286 infection with S. aureus was associated with BPI antibody autoreactivity in 2/15 patients (13.3%).

287 res used in a genetic study of families with BPI disorder.

288 to estimate the associations of income with BPI and HOME scores.

289 A sample of 66 patients with BPI disorder completed a self-report measure of lifetime

290 rum symptoms in this sample of patients with BPI disorder was associated with greater levels of depre

291 ment strategies are needed for patients with BPI disorder who endorse lifetime panic spectrum feature

292 Patients with BPI disorder who reported high lifetime panic-agoraphobi

293 o EvC individual has ever been reported with BPI.

294 family, sharing structural similarities with BPI-like proteins.

295 y, which shares structural similarities with BPI-like proteins.

296 ne the association of EITC payment size with BPI and HOME scores; in the second, we used EITC payment

297 ring emotion downregulation in subjects with BPI.

298 bilateral vlPFC compared with subjects with BPI.

299 oCAP18, exhibited antibacterial synergy with BPI, and the p15s also displayed synergy with CAP18p in

300 On the basis of its homology with BPIs and restricted expression of SPLUNC1 in serous cell