ライフサイエンスコーパス: Bordetella pertussis

1 ory illness caused by the bacterial pathogen Bordetella pertussis.

2 ory illness caused by the bacterial pathogen Bordetella pertussis.

3 ainst the endemic human respiratory pathogen Bordetella pertussis.

4 e phenomenology of infection and immunity to Bordetella pertussis.

5 rotection against both B. bronchiseptica and Bordetella pertussis.

6 respiratory disease caused by the bacterium Bordetella pertussis.

7 rom an IncP-1b plasmid, pBP136 isolated from Bordetella pertussis.

8 ry disease of humans caused by the bacterium Bordetella pertussis.

9 is an important virulence factor produced by Bordetella pertussis.

10 n AB(5) toxin produced by the human pathogen Bordetella pertussis.

11 ssis toxin (PT) across the outer membrane of Bordetella pertussis.

12 in, an autotransporter passenger domain from Bordetella pertussis.

13 nserved in both Bordetella parapertussis and Bordetella pertussis.

14 isease caused by the obligate human pathogen Bordetella pertussis.

15 regulates expression of virulence factors in Bordetella pertussis.

16 chiseptica and the expression of vrg loci in Bordetella pertussis.

17 ence-associated filamentous hemagglutinin of Bordetella pertussis.

18 in homologues of the wlbA and wlbL genes of Bordetella pertussis.

19 ly related CyaC acyltransferase expressed by Bordetella pertussis.

20 on and virulence of the whooping cough agent Bordetella pertussis.

21 ines may not protect against transmission of Bordetella pertussis.

22 irulence regulon of the whooping-cough agent Bordetella pertussis.

23 (CyaA) plays a key role in the virulence of Bordetella pertussis.

24 ory illness caused by the bacterial pathogen Bordetella pertussis.

25 The limits of detection were as follows: for Bordetella pertussis, 2 CFU/ml; for Legionella pneumophi

26 Bordetella pertussis, a causative agent of whooping coug

27 We have used Bordetella pertussis, a common neonatal respiratory trac

28 tion and failure to induce direct killing of Bordetella pertussis, a synthetic scheme was devised for

29 Bordetella pertussis adenylate cyclase (AC) toxin belong

30 f YipB and the enzymatic active component of Bordetella pertussis adenylate cyclase (Cya) was translo

31 ESAT-6 or CFP10 with genetically detoxified Bordetella pertussis adenylate cyclase (CyaA) are recogn

32 Bordetella pertussis adenylate cyclase (CyaA) toxin has

33 rmational behavior of an RTX domain from the Bordetella pertussis adenylate cyclase consisting of nin

34 in-dependent adenylate cyclase domain of the Bordetella pertussis adenylate cyclase protein was trans

35 The catalytic domain of Bordetella pertussis adenylate cyclase toxin (ACT) trans

36 Epinephrine and Bordetella pertussis adenylate cyclase toxin (ACT), cAMP

37 The use of genetically detoxified Bordetella pertussis adenylate cyclase toxin (CyaA) as a

38 f bound acyl chains govern the activities of Bordetella pertussis adenylate cyclase toxin (CyaA), Esc

39 -residue receptor-binding domain (RD) of the Bordetella pertussis adenylate cyclase toxin CyaA fused

40 and T25 fragments of the catalytic domain of Bordetella pertussis adenylate cyclase were fused to CTA

41 sts involving C-terminal fusions with a Cya (Bordetella pertussis adenylate cyclase) reporter indicat

42 CR3 serves as the receptor for the Bordetella pertussis adhesin filamentous hemagglutinin (

43 (FHA) is a dominant cell surface-associated Bordetella pertussis adhesin.

44 s toxin (PT), a secreted virulence factor of Bordetella pertussis, ADP ribosylates mammalian G(i) pro

45 Antibody-dependent complement killing of Bordetella pertussis after immunization with a three-com

46 ells stimulated with LPS or heat-inactivated Bordetella pertussis Ag.

47 Temporal expression patterns of the Bordetella pertussis alcaligin, enterobactin and haem ir

48 Pathogenic microorganisms such as Bordetella pertussis also express proteins with lectin-l

49 Bordetella pertussis, an obligate human pathogen and the

50 The persistence of Bordetella pertussis and B. parapertussis within vaccina

51 Two highly infectious bordetellae, Bordetella pertussis and B. parapertussis, have emerged

52 be caused by two closely related pathogens, Bordetella pertussis and B. parapertussis.

53 in two closely related respiratory pathogens Bordetella pertussis and Bordetella bronchiseptica Altho

54 pproximately 10% of the annotated genomes of Bordetella pertussis and Bordetella bronchiseptica and c

55 Bordetella pertussis and Bordetella bronchiseptica are c

56 Bordetella pertussis and Bordetella bronchiseptica are t

57 Bordetella pertussis and Bordetella bronchiseptica estab

58 ium at pH values ranging from pH 6.0 to 7.6, Bordetella pertussis and Bordetella bronchiseptica FtrAB

59 tin siderophore by the respiratory pathogens Bordetella pertussis and Bordetella bronchiseptica is de

60 We investigated Bordetella pertussis and Bordetella bronchiseptica LPS-d

61 Bordetella pertussis and Bordetella bronchiseptica rely

62 Bordetella pertussis and Bordetella bronchiseptica, gram

63 Bordetella pertussis and Bordetella bronchiseptica, whic

64 erophore biosynthesis and transport genes in Bordetella pertussis and Bordetella bronchiseptica.

65 Bordetella pertussis and Bordetella parapertussis are cl

66 Of the three Bordetella species, Bordetella pertussis and Bordetella parapertussis are no

67 dy confirmed that Bordetella bronchiseptica, Bordetella pertussis and Bordetella parapertussis have t

68 PCR assay for detecting and differentiating Bordetella pertussis and Bordetella parapertussis in nas

69 und time from days to hours for detection of Bordetella pertussis and Bordetella parapertussis In thi

70 nce for the detection and differentiation of Bordetella pertussis and Bordetella parapertussis nuclei

71 Detection of Bordetella pertussis and Bordetella parapertussis using

72 fections, we challenged IL-1R(-/-) mice with Bordetella pertussis and Bordetella parapertussis, the c

73 ates during 2008-2010 were tested by PCR for Bordetella pertussis and Bordetella parapertussis.

74 ipA homologues are expressed in Bvg(i) phase Bordetella pertussis and Bordetella parapertussis.

75 the activities of adenylyl cyclase toxin of Bordetella pertussis and edema toxin of Bacillus anthrac

76 ci is identical to the tracheal cytotoxin of Bordetella pertussis and has been shown to kill ciliated

77 ne-containing compounds to support growth of Bordetella pertussis and influence pertussis toxin trans

78 tes the expression of the virulence genes of Bordetella pertussis and negatively regulates a second s

79 nostic PCR target for discriminating between Bordetella pertussis and other Bordetella species that a

80 PTx) is a major virulence factor produced by Bordetella pertussis and, in its detoxified form PTd, is

81 within the inactivated adenylate cyclase of Bordetella pertussis), and purified protein derivative o

82 romoter region prevent batB transcription in Bordetella pertussis, and although expressed, the batB g

83 ARGs in the manures were Bacillus anthracis, Bordetella pertussis, and B. anthracis (sulfonamide resi

84 mologous to the filamentous hemagglutinin of Bordetella pertussis, and MchB has homology to other Tps

85 Intestinal parasites, Bordetella pertussis, and other infectious disease agent

86 ogenic species, such as Helicobacter pylori, Bordetella pertussis, and Pseudomonas aeruginosa.

87 e of pertactin, an AT virulence protein from Bordetella pertussis, and show that OM secretion of the

88 BvgS sensor kinase in its phosphorylation in Bordetella pertussis, and the effect of the T194M mutati

89 from infection of the respiratory tract with Bordetella pertussis, and the secreted adenylate cyclase

90 , Shigella flexneri, Pseudomonas aeruginosa, Bordetella pertussis, and Yersinia pestis), has prompted

91 sms, performs haem ligation to a coexpressed Bordetella pertussis apocytochrome c in an Escherichia c

92 Bordetella parapertussis and Bordetella pertussis are closely related species that ca

93 Current acellular vaccines against Bordetella pertussis are effective in preventing severe

94 Lipopolysaccharides (LPS) of Bordetella pertussis are important modulators of the imm

95 ases of pertussis (whooping cough) caused by Bordetella pertussis are on the rise, and understanding

96 ssis toxin (Ptx) expression and secretion in Bordetella pertussis are regulated by a two-component si

97 s of antimicrobial susceptibility testing of Bordetella pertussis are time consuming and require spec

98 usely adherent Escherichia coli, and BrkA of Bordetella pertussis, are localized to the bacterial pol

99 Environmental factors, such as Bordetella pertussis, are thought to sensitize central e

100 of IS481, a frequent target for diagnosis of Bordetella pertussis, as approximately 5%.

101 reted autotransporter virulence protein from Bordetella pertussis, as measured by small angle X-ray s

102 e the incidence and clinical presentation of Bordetella pertussis-associated hospitalization in perin

103 With the infection rate of Bordetella pertussis at a 60-year high, there is an urge

104 riaceae (SPATEs) and the pertactin family of Bordetella pertussis autotransporters is released from t

105 ular pertussis vaccines and the evolution of Bordetella pertussis away from vaccine-mediated immunity

106 A comparative analysis of the Bordetella pertussis, B. bronchiseptica, and B. parapert

107 Bordetella pertussis, B. parapertussis, and B. bronchise

108 T-PCR) assay for the rapid identification of Bordetella pertussis, B. parapertussis, and B. holmesii

109 vant, and treated simultaneously with killed Bordetella pertussis bacteria, as indicated.

110 the fimbrial subunit genes fim2 and fim3 of Bordetella pertussis behave differently from each other

111 FhaC is an outer membrane transporter from Bordetella pertussis belonging to the two-partner secret

112 arison of real-time PCR positivity rates for Bordetella pertussis between specimens collected with ra

113 he discovery that the adenylate cyclase from Bordetella pertussis binds to the CD11b/CD18 integrin, w

114 nt diagnostic tool for the identification of Bordetella pertussis, Bordetella holmesii, and Bordetell

115 Bordetella pertussis, Bordetella parapertussis and Borde

116 Bordetella pertussis, Bordetella parapertussis, and Bord

117 h is caused by infection of the airways with Bordetella pertussis (Bp).

118 or nasal administration of highly attenuated Bordetella pertussis BPZE1 provides effective and sustai

119 pathogens such as Agrobacterium tumefaciens, Bordetella pertussis, Brucella spp., Bartonella henselae

120 n established role in protective immunity to Bordetella pertussis, but this evidence is based largely

121 Bordetella pertussis BvgA is a global response regulator

122 Pertussis toxin (PT) is secreted from Bordetella pertussis by a type IV secretion system, know

123 died of respiratory illness were tested for Bordetella pertussis by conventional and real-time PCR a

124 d for the ability to promote phagocytosis of Bordetella pertussis by human neutrophils.

125 is transported across the outer membrane of Bordetella pertussis by the type IV secretion system kno

126 In infants, Bordetella pertussis can cause severe disease, manifeste

127 Bordetella pertussis causes a profound inflammatory resp

128 Bordetella pertussis causes severe respiratory illness a

129 Bordetella pertussis causes whooping cough, an endemic r

130 T), a monomer of DAP-type peptidoglycan from Bordetella pertussis, causes cytopathology in the respir

131 ovirus, respiratory syncytial virus A and B, Bordetella pertussis, Chlamydophila pneumoniae, and Myco

132 Bordetella pertussis colonization can be deliberately in

133 thogenic bacteria Pseudomonas aeruginosa and Bordetella pertussis contain in their outer membranes th

134 Bordetella pertussis contributed to a modest proportion

135 s toxin (PT), a virulence factor secreted by Bordetella pertussis, contributes to respiratory tract i

136 ependent adenylate cyclase gene derived from Bordetella pertussis (cyaA').

137 endent adenylate cyclase domain (Cya) of the Bordetella pertussis cyclolysin was used as a reporter p

138 severe respiratory disease mainly caused by Bordetella pertussis Despite wide global vaccination cov

139 f vaccinated adults are being diagnosed with Bordetella pertussis disease.

140 ite widespread childhood vaccination against Bordetella pertussis, disease remains prevalent.

141 office tested positive for low quantities of Bordetella pertussis DNA, we suspected prelaboratory con

142 The adenylate cyclase toxin (ACT) of Bordetella pertussis does not require a receptor to gene

143 rtussis toxin, we identified and mutated the Bordetella pertussis dsbA, dsbB, and dsbC homologues.

144 y of recombinant adenylate cyclase (CyaA) of Bordetella pertussis (EC ) by conductimetric determinati

145 ry acylation catalyzed by LpxL proteins from Bordetella pertussis, Escherichia coli, and Haemophilus

146 iverse microbes, including Candida glabrata, Bordetella pertussis, Escherichia coli, and L. pneumophi

147 Bordetella pertussis establishes infection by attaching

148 better-characterized TPS systems specify the Bordetella pertussis FHA and Haemophilus influenzae HMW1

149 Comparison of HMW1-PP and the Bordetella pertussis FHA secretion domain (Fha30) reveal

150 t homology with the N-terminal region of the Bordetella pertussis filamentous hemagglutinin (FHA), wh

151 ter-membrane, beta-barrel transporter of the Bordetella pertussis filamentous hemagglutinin adhesin.

152 A prominent feature of the promoters of Bordetella pertussis fimbrial subunit genes fim2, fim3 a

153 of recombinant adenylate cyclase toxin from Bordetella pertussis for the development of protective a

154 respiratory disease caused by the bacterium Bordetella pertussis, for which humans are the only know

155 Bordetella bronchiseptica and Bordetella pertussis form biofilms on abiotic surfaces a

156 ells in immunity to the respiratory pathogen Bordetella pertussis gammadelta T cells, predominantly V

157 )-neutral (Escherichia coli) and (G+C)-rich (Bordetella pertussis) genomes.

158 n in vitro reactions, or in crude lysates of Bordetella pertussis grown under varying laboratory cond

159 inst Bordetella pertussis was measured using Bordetella pertussis growth inhibition assay.

160 n of growth phase-related gene regulation in Bordetella pertussis has been reported previously.

161 Infection of mice and pigs with Bordetella pertussis has many features of the infection

162 eBABE-conjugated BvgA to the fha promoter of Bordetella pertussis has revealed that three dimers, for

163 ter membrane receptor proteins, and although Bordetella pertussis has the bfrD and bfrE genes, the ro

164 he respiratory tract caused by the bacterium Bordetella pertussis, has reached levels not seen since

165 reatment failures and in vitro resistance of Bordetella pertussis have been reported on several occas

166 nts and mammals - Agrobacterium tumefaciens, Bordetella pertussis, Helicobacter pylori and Legionella

167 The Bordetella pertussis heme utilization gene cluster hurIR

168 Bordetella pertussis identification was common among you

169 ory techniques are changing the landscape of Bordetella pertussis illness diagnosis.

170 rom living asymptomatic infected mice and of Bordetella pertussis in 1 microl of nasal aspirate from

171 is a paucity of data regarding the burden of Bordetella pertussis in African women and young infants,

172 thromycin resistance was first recognized in Bordetella pertussis in Arizona in 1994, and since then,

173 disease similar to whooping cough caused by Bordetella pertussis in children.

174 aterials performed similarly for recovery of Bordetella pertussis in culture.

175 tion and transmission of the etiologic agent Bordetella pertussis In response to this escalating publ

176 a provide evidence of ongoing circulation of Bordetella pertussis in the region.

177 is the most common method used for detecting Bordetella pertussis in the United States, most laborato

178 olonization factor A, TcfA) for detection of Bordetella pertussis infection by lateral flow immunoass

179 Diagnosis of Bordetella pertussis infection has been difficult due to

180 ulted in higher childhood mortality than did Bordetella pertussis infection in 2003-2004.

181 vestigated lung gene expression responses to Bordetella pertussis infection in adult mice, revealing

182 hown to reduce pulmonary inflammation during Bordetella pertussis infection in mouse models.

183 Bordetella pertussis infection may be a more common caus

184 Bordetella pertussis infection of the airways causes the

185 A previous study used a murine model of Bordetella pertussis infection to demonstrate that treat

186 human monocytes to mediate the clearance of Bordetella pertussis infection was examined.

187 Bordetella pertussis infections and illnesses were prosp

188 reased the ability of laboratories to detect Bordetella pertussis infections, it has also been associ

189 The BrkA protein of Bordetella pertussis inhibits killing by the antibody-de

190 such as the secretion of pertussis toxin by Bordetella pertussis into human cells and the delivery o

191 The adenylate cyclase toxin (ACT) of Bordetella pertussis intoxicates target cells by generat

192 Bordetella pertussis is a human pathogen that can infect

193 The filamentous hemagglutinin/FhaC pair of Bordetella pertussis is a model two-partner secretion sy

194 Bordetella pertussis is a strictly human pathogen.

195 Bordetella pertussis is among the leading causes of vacc

196 Bordetella pertussis is an important cause of infection

197 ial and sufficient to prevent infection with Bordetella pertussis is derived from data from animal an

198 Bordetella pertussis is highly infectious among humans b

199 that asymptomatic nasopharyngeal carriage of Bordetella pertussis is inducible in humans and to defin

200 Bordetella pertussis is one of the leading causes of vac

201 Pertussis toxin of Bordetella pertussis is secreted by a type IV secretion

202 Bordetella pertussis is the causative agent of whooping

203 The Gram-negative bacterium Bordetella pertussis is the causative agent of whooping

204 While the human pathogen Bordetella pertussis is the etiological agent of the acu

205 Natural infection with Bordetella pertussis is thought to result in 4-20 years

206 cough, caused by the obligate human pathogen Bordetella pertussis is undergoing a worldwide resurgenc

207 Adenylate cyclase (AC) toxin from Bordetella pertussis is unusual in that, unlike most oth

208 del toxin, the adenylate cyclase (CyaA) from Bordetella pertussis, is able to invade eukaryotic cells

209 xin virulence factor produced exclusively by Bordetella pertussis, is important for colonization of t

210 Pertussis (whooping cough), caused by Bordetella pertussis, is resurging in the United States

211 A total of 194 Bordetella pertussis isolates collected from 2008 throug

212 Bordetella pertussis isolates collected in Cincinnati fr

213 Bordetella pertussis isolation by culture has low detect

214 Bordetella pertussis lacks O antigen and is sensitive to

215 Other pathogens, including Bordetella pertussis, Legionella pneumophila, Brucellasp

216 Bordetella pertussis LPS has a branched core structure a

217 )), Bordetella parapertussis (lpxA(Pa)), and Bordetella pertussis (lpxA(Pe)) was cloned and expressed

218 We used the murine Bordetella pertussis model to test the hypothesis that t

219 effect of adenylate cyclase (AC) toxin from Bordetella pertussis on host cells has been attributed t

220 lase (AC) toxin is present on the surface of Bordetella pertussis organisms and their addition to euk

221 say on the BD Max system versus our in-house Bordetella pertussis PCR.

222 oding for two important virulence factors of Bordetella pertussis, pertactin and pertussis toxin, and

223 own ADP-ribosyltransferases (ADPRTs) such as Bordetella pertussis pertussis toxin and Mycoplasma pneu

224 (e.g., Bacillus anthracis, Yersinia pestis, Bordetella pertussis, Plasmodium falciparum, and Mycobac

225 T), an exotoxin virulence factor produced by Bordetella pertussis, plays an important early role in c

226 al swabs and induced sputum (cases only) for Bordetella pertussis polymerase chain reaction.

227 Our results indicate that the Bordetella pertussis population causing this epidemic wa

228 virulence factor of the whooping cough agent Bordetella pertussis, preferentially binds an inactive f

229 Bordetella pertussis produces a 73-kDa protein, BrkA (Bo

230 Bordetella pertussis produces multiple virulence factors

231 ertussis and the identification of antigenic Bordetella pertussis proteins support the hypothesis tha

232 ussis vaccines containing 1 or more purified Bordetella pertussis proteins.

233 umefaciens VirB/VirD4, E. coli R388 Trw, and Bordetella pertussis Ptl systems support conjugative DNA

234 The whooping cough agent Bordetella pertussis regulates the production of its vir

235 es of research and vaccination, infection by Bordetella pertussis remains a serious disease with no s

236 pathway for c-type cytochrome biogenesis in Bordetella pertussis requires at least four biogenesis p

237 utilization by Bordetella bronchiseptica and Bordetella pertussis requires the BfeA outer membrane re

238 tbreaks with documented cases of B. holmesii/Bordetella pertussis respiratory coinfection; whether th

239 mortality in non-vaccinated young children, Bordetella pertussis results in milder and prolonged cou

240 n complexes made with RNA polymerase and the Bordetella pertussis RR, BvgA, in its nonphosphorylated

241 ce of the whooping cough causative bacterium Bordetella pertussis Secreted as soluble protein, it tar

242 Legionella pneumophila, Legionella micdadei, Bordetella pertussis, Staphylococcus aureus, and Strepto

243 d intranasally with nonattenuated, wild-type Bordetella pertussis strain B1917.

244 intranasally with non-attenuated, wild type Bordetella pertussis strain B1917.

245 Bordetella pertussis strains lacking expression of perta

246 lities of the entire PFGE profiles for three Bordetella pertussis strains.

247 ella bronchiseptica) and human-adapted (e.g. Bordetella pertussis) strains produce a surface-exposed

248 onent of the two-partner secretion system in Bordetella pertussis, suggests that the BamA beta-barrel

249 ate cyclase from the whooping cough pathogen Bordetella pertussis, synthesizes the second messenger b

250 , S2, and/or S4 subunits of PT in strains of Bordetella pertussis that either did or did not produce

251 g awarded to Jules Bordet, the discoverer of Bordetella pertussis, the 12th International Bordetella

252 nsidered the "master virulence regulator" in Bordetella pertussis, the causal agent of pertussis, and

253 Bordetella pertussis, the causative agent of human whoop

254 Bordetella pertussis, the causative agent of the acute c

255 The source of the muramyl peptide is Bordetella pertussis, the causative agent of the respira

256 In the human respiratory pathogen Bordetella pertussis, the causative agent of whooping co

257 been observed in other organisms, including Bordetella pertussis, the causative agent of whooping co

258 Lipid A of Bordetella pertussis, the causative agent of whooping co

259 Bordetella pertussis, the causative agent of whooping co

260 interaction with wild-type and mutant LPS of Bordetella pertussis, the causative agent of whooping co

261 Bordetella pertussis, the causative agent of whooping co

262 Bordetella pertussis, the causative agent of whooping co

263 Bordetella pertussis, the causative agent of whooping co

264 Bordetella pertussis, the causative agent of whooping co

265 Bordetella pertussis, the causative agent of whooping co

266 owed that strain BP338 of the human pathogen Bordetella pertussis, the causative agent of whooping co

267 e of the major virulence factors produced by Bordetella pertussis, the causative agent of whooping co

268 AB5-type exotoxin produced by the bacterium Bordetella pertussis, the causative agent of whooping co

269 Bordetella pertussis, the etiologic agent of whooping co

270 CyaA is crucial for colonization by Bordetella pertussis, the etiologic agent of whooping co

271 T cell response relative to that induced by Bordetella pertussis, the more common cause of pertussis

272 in, one of the virulence factors secreted by Bordetella pertussis, the pathogenic bacteria responsibl

273 Bordetella pertussis, the pathogenic bacteria responsibl

274 es to prevent nasopharyngeal colonization by Bordetella pertussis, the principal causative agent of w

275 These interventions result in elevated anti-Bordetella pertussis titers, but there have been no stud

276 of infant morbidity and mortality caused by Bordetella pertussis To better inform such interventions

277 (AC) toxin is an essential toxin that allows Bordetella pertussis to invade eukaryotic cells, where i

278 roteins encoded by the bipA genes present in Bordetella pertussis Tohama I and Bordetella bronchisept

279 nd that dendritic cell-activating adjuvants [Bordetella pertussis toxin (PT) or CpG ODN or a squalene

280 In vivo intoxication with Bordetella pertussis toxin (PTX) elicits a variety of ph

281 Bordetella pertussis toxin (PTX) promotes insulin secret

282 e role of VWF and/or Weibel-Palade bodies in Bordetella pertussis toxin-induced hypersensitivity to h

283 ur observations with HMW1B, examination of a Bordetella pertussis TpsB protein called FhaC revealed t

284 zed syndromic case definition and tested for Bordetella pertussis using real-time polymerase chain re

285 that are required for efficient export from Bordetella pertussis via the Ptl system, a member of the

286 e role of pertussis toxin (PT), an important Bordetella pertussis virulence factor, in lung transcrip

287 Bordetella pertussis was detected in 2.1% (n = 26/1257)

288 Bordetella pertussis was detected in 53 of 4200 (1.3%) c

289 No shedding of Bordetella pertussis was detected in systematically coll

290 Bordetella pertussis was diagnosed in a human immunodefi

291 d mortality after established infection with Bordetella pertussis was explored.

292 Functionality of antibodies against Bordetella pertussis was measured using Bordetella pertu

293 A PCR test for Bordetella pertussis was primarily used by a private lab

294 Transposon mutagenesis of Bordetella pertussis was used to discover mutations in t

295 fundamental nature of protective immunity to Bordetella pertussis, we studied intranasal immunization

296 tested with multitarget PCR; B. holmesii and Bordetella pertussis were exclusively detected among 48

297 a 188-kDa adenylyl cyclase toxin secreted by Bordetella pertussis, which is the etiologic agent for w

298 Pertussis toxin is secreted from Bordetella pertussis with the assistance of the Ptl tran

299 n has recently been demonstrated to occur in Bordetella pertussis, with many transcripts, including k

300 A recent increase in Bordetella pertussis without the pertactin protein, an a