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1  month of antibiotic therapy for vectorborne Borrelia burgdorferi infection.
2 on are involved in the arthritic response to Borrelia burgdorferi infection.
3 isease is a multisystemic disorder caused by Borrelia burgdorferi infection.
4  produce protective class-switched Abs after Borrelia burgdorferi infection.
5 e skin at the tick bite site could eradicate Borrelia burgdorferi infection.
6 educe the severity of Lyme disease following Borrelia burgdorferi infection.
7  understanding of Lyme borreliosis by giving Borrelia burgdorferi infection a mythical aura of its ow
8 shown an exponential increase in the risk of Borrelia burgdorferi infection after 48-72 h of deer tic
9 ion between neuroborreliosis (nervous system Borrelia burgdorferi infection) and Lyme encephalopathy
10                             In nature, mixed Borrelia burgdorferi infections are common and possibly
11 ) has a protective effect in host defense to Borrelia burgdorferi infection, both in limiting the sev
12 thritis is surely critical in the control of Borrelia burgdorferi infection but may also have patholo
13                                              Borrelia burgdorferi infection causes an initial skin le
14 ls or patients with Ehrlichia chaffeensis or Borrelia burgdorferi infection did not recognize HGE-44.
15                                              Borrelia burgdorferi infection in beagle dogs was studie
16                                  By studying Borrelia burgdorferi infection in humanized mice reconst
17 yme disease by determining the prevalence of Borrelia burgdorferi infection in Ixodes scapularis tick
18                 T-helper responses following Borrelia burgdorferi infection in mice determine suscept
19 M immunoblotting is a useful test to confirm Borrelia burgdorferi infection in patients with acute fa
20 e antigen for the prevention of experimental Borrelia burgdorferi infection in the mouse model of Lym
21                         The establishment of Borrelia burgdorferi infection involves numerous interac
22 irst to demonstrate that the Th2 response to Borrelia burgdorferi infection of BALB/c mice is precede
23 previously established an in vitro model for Borrelia burgdorferi infection of human neuroglial cells
24                   Whether acute disseminated Borrelia burgdorferi infection should be treated differe
25 was coordinately up-regulated in response to Borrelia burgdorferi infection under both in-vitro and i
26                         We demonstrated that Borrelia burgdorferi infection upregulated the surface e
27 agnetic resonance imaging and muscle biopsy; Borrelia burgdorferi infection was confirmed by Lyme ser
28 ning tests and immunoblots for Lyme disease (Borrelia burgdorferi infection) were also performed.
29 ptoms attributed to Lyme disease but without Borrelia burgdorferi infection) were compared with 2 gro