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1 monas syringae and the necrotrophic pathogen Botrytis cinerea.
2 larvae and the necrotrophic fungal pathogen Botrytis cinerea.
3 susceptibility to the necrotrophic pathogen Botrytis cinerea.
4 onset of disease symptoms when infected with Botrytis cinerea.
5 ces foliar resistance to the fungal pathogen Botrytis cinerea.
6 ptibility to necrotrophic pathogens, such as Botrytis cinerea.
7 resistant to a necrotrophic fungal pathogen, Botrytis cinerea.
8 yringae and the necrotrophic fungal pathogen Botrytis cinerea.
9 protection against the fungal phytopathogen Botrytis cinerea.
10 nfection by the necrotrophic fungal pathogen Botrytis cinerea.
11 esponses to the necrotrophic fungal pathogen Botrytis cinerea.
12 d in resistance to the necrotrophic pathogen Botrytis cinerea.
13 noculation sites of Penicillium expansum and Botrytis cinerea.
14 ) bacteria as well as phytopathogenic fungus Botrytis cinerea.
15 ular vesicles (EVs) into the fungal pathogen Botrytis cinerea.
16 However, its production is limited by Botrytis cinerea.
17 patens) against the important plant pathogen Botrytis cinerea.
18 resistance in the phylloplane to the fungus Botrytis cinerea.
19 fense responses to the necrotrophic pathogen Botrytis cinerea.
20 capacity, and antimicrobial activity against Botrytis cinerea.
21 tants show resistance to the fungal pathogen Botrytis cinerea.
22 asitic interaction against the phytopathogen Botrytis cinerea.
23 al immunity against the fungal phytopathogen Botrytis cinerea.
24 behaviour of the inclusion complexes against Botrytis cinerea.
25 cearum, but not to the necrotrophic pathogen Botrytis cinerea.
26 ns of ripe grape (Vitis vinifera) berries by Botrytis cinerea.
27 ibility to the necrotrophic fungal pathogen, Botrytis cinerea.
28 are made with berries infected by the fungus Botrytis cinerea.
29 opsis mutant wrky33 is highly susceptible to Botrytis cinerea.
30 SsPV1 can infect and cause hypovirulence in Botrytis cinerea.
31 irulence of the necrotrophic fungal pathogen Botrytis cinerea.
32 d protection against the necrotrophic fungus Botrytis cinerea.
33 s with its plant host and the plant pathogen Botrytis cinerea.
34 sistance to the necrotrophic fungal pathogen Botrytis cinerea.
35 ceptible than wild-type plants to the fungus Botrytis cinerea.
36 le to S. sclerotiorum and the related fungus Botrytis cinerea.
37 Aspergillus niger, Trichoderma harzianum and Botrytis cinerea.
38 nd against the necrotrophic fungal pathogen, Botrytis cinerea.
39 a arabidopsidis, and the necrotrophic fungus Botrytis cinerea.
40 when seedlings were infected with the fungus Botrytis cinerea.
41 nfection by the necrotrophic fungal pathogen Botrytis cinerea.
42 l for defense toward the necrotrophic fungus Botrytis cinerea.
43 ae pv. tomato DC3000 and the fungal pathogen Botrytis cinerea.
44 ance toward the necrotrophic fungal pathogen Botrytis cinerea.
45 trongly induced by the necrotrophic pathogen Botrytis cinerea.
46 bidopsis by the necrotrophic fungal pathogen Botrytis cinerea.
47 antly increased resistance toward gray mold (Botrytis cinerea), a pathogen responsible for major loss
49 a multifaceted mechanism of action (MOA) in Botrytis cinerea, a multiresistant phytopathogenic fungu
51 Crh1 protein from the phytopathogenic fungus Botrytis cinerea acts as a cytoplasmic effector and elic
52 rthe oryzae Inhibition of EXO70 by ES2-14 in Botrytis cinerea also reduces its virulence in Arabidops
53 ified SsPV1/WF-1 virions into strain KY-1 of Botrytis cinerea also resulted in reductions in virulenc
54 esistance to several plant pathogens (namely Botrytis cinerea, Alternaria brassicicola and Golovinomy
55 ereus, but not Escherichia coli or the fungi Botrytis cinerea and Alternaria alternata, likely due to
56 ced susceptibility to the necrotrophic fungi Botrytis cinerea and Alternaria brassicicola as well as
57 ibility to the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola concomitant
58 istance to the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola in the Noss
59 ibility to the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola, whereas HU
63 eased susceptibility to the pathogenic fungi Botrytis cinerea and Alternaria brassisicola Both PAMPs
64 ined for responses to the fungal necrotrophs Botrytis cinerea and Alternaria solani, bacterial pathog
65 ed susceptibility to the necrotrophic fungus Botrytis cinerea and an increased tolerance to the biotr
66 increased resistance to the fungal pathogens Botrytis cinerea and Bipolaris sorokiniana but not to th
67 ease resistance to the necrotrophic pathogen Botrytis cinerea and contributes to basal defense induce
69 d defenses against the necrotrophic pathogen Botrytis cinerea and for the shade-triggered increased s
71 )-one (PT) as inhibitors of fungal pathogens Botrytis cinerea and Magnaporthe oryzae ATG4-mediated AT
72 , Penicillium italicum, Rhizopus stolonifer, Botrytis cinerea and Monilinia fructicola, all of which
73 the rescued virions, we further transfected Botrytis cinerea and Monilinia fructicola, two economica
74 ybees from pathogens (phytopathogenic fungus Botrytis cinerea and pathogenic bacteria, respectively).
75 xpression of defense genes and resistance to Botrytis cinerea and Pectobacterium carotovorum infectio
76 n of plant defenses against foliar pathogens Botrytis cinerea and Pseudomonas syringae, which normall
79 tes JA-mediated resistance to the necrotroph Botrytis cinerea and susceptibility to the hemibiotroph
80 response to the necrotrophic foliar pathogen Botrytis cinerea and the biotrophic bacterial pathogen X
81 reased resistance to the necrotrophic fungus Botrytis cinerea and the caterpillar Mamestra brassicae
82 genes in the foliar and postharvest pathogen Botrytis cinerea and the soilborne pathogen Verticillium
83 sistance to the necrotrophic fungal pathogen Botrytis cinerea and their genetic control are poorly un
84 cular against the devastating plant pathogen Botrytis cinerea and they drastically inhibit the infect
86 to susceptibility to the necrotrophic fungus Botrytis cinerea and to feeding by larvae of tobacco hor
88 ultiple strains of the generalist necrotroph Botrytis cinerea, and have decreased camalexin productio
89 ed susceptibility to the necrotrophic fungus Botrytis cinerea, and increased sensitivity to salt and
91 The pectin matrix is the main CW target of Botrytis cinerea, and pectin methylesterification status
93 esponse to BcNEP2, a representative NLP from Botrytis cinerea, and showed that it contributes to dise
94 iting the growth of Penicillium expansum and Botrytis cinerea, and their effectivity depended on the
95 efense responses against the fungal pathogen Botrytis cinerea, and thus we conclude that the regulati
97 v. tomato DC3000 and the necrotrophic fungus Botrytis cinerea As pldgamma1 mutant plants responded wi
98 cognizes several PGs from the plant pathogen Botrytis cinerea as well as one from the saprotroph Aspe
100 d that SlDQD/SDH2 confers resistance against Botrytis cinerea attack in post-harvest tomato fruit.
101 ays a critical role in fruit defense against Botrytis cinerea attack, but the underlying mechanisms r
105 chitinase activity and induced expression of Botrytis cinerea BOT genes, although their total antagon
106 exhibit tolerance to the necrotrophic fungus Botrytis cinerea but susceptibility to the hemibiotrophi
107 tibility to the necrotrophic fungal pathogen Botrytis cinerea, but showed normal responses to virulen
108 -enolide (1) was assigned to a metabolite of Botrytis cinerea, but the spectra of several synthetic a
109 Small RNAs (sRNAs) of the fungal pathogen Botrytis cinerea can enter plant cells and hijack host A
110 T2 gene from the necrotrophic plant pathogen Botrytis cinerea, catalyzes the multistep cyclization of
111 including Pst-AvrRpt2, Dickeya dadantii, and Botrytis cinerea Characterization of the redox status de
112 ption of SEP4 in the plant grey mould fungus Botrytis cinerea completely blocked IFS formation and ab
113 dopsis resistance to the necrotrophic fungus Botrytis cinerea, consistent with substantial upregulati
114 N. crassa and the phytopathogenic gray mold Botrytis cinerea coordinate their behavior over a spatia
116 pv. tomato, Pectobacterium carotovorum, and Botrytis cinerea depending on the microbial capacity to
117 Such sRNA effectors are mostly produced by Botrytis cinerea Dicer-like protein 1 (Bc-DCL1) and Bc-D
118 hogen Pseudomonas syringae and to the fungus Botrytis cinerea Furthermore, bsk5 mutant plants were im
119 9 and 3310 mg/L against Rhizopus stolonifer, Botrytis cinerea, Fusarium oxysporum and Colletotrichum
120 grape berries (Vitis vinifera) by the fungus Botrytis cinerea (grey mould) frequently occurs in viney
121 may play an inhibitory role in resistance to Botrytis cinerea, group II (JAZ10)/III (JAZ11/12) in JA-
123 tion of systemic acquired resistance against Botrytis cinerea, (ii) the activation of the expression
126 is to delayed the postharvest development of Botrytis cinerea in tomatoes by releasing allyl-isothioc
127 e markers induced by the necrotrophic fungus Botrytis cinerea, including the genes that encode the tr
128 tion of Arabidopsis thaliana with the fungus Botrytis cinerea Indeed, in contrast to previous reports
129 the biotroph H. schachtii and the necrotroph Botrytis cinerea, indicating a potential suppression of
130 quired for resistance to the fungal pathogen Botrytis cinerea, indicating that NHO1 is not limited to
131 oligosaccharides (PDOs) in three regions of Botrytis cinerea-infected tomato fruit tissue is describ
132 ge, grape variety, region of production, and Botrytis cinerea infection affect the concentration of t
133 WRKY33 is rapidly SUMOylated in response to Botrytis cinerea infection and flg22 elicitor treatment.
135 lly regulated during abiotic stresses during Botrytis cinerea infection or after benzothiadiazole and
136 Transgenic plants were more resistant to Botrytis cinerea infection than wild type, possibly as a
137 are upregulated coordinately in response to Botrytis cinerea infection, but through separate signal
138 with AA exhibited reduced susceptibility to Botrytis cinerea infection, confirming AA signaling in o
139 to explore how vintage, variety, region, and Botrytis cinerea infection, influence both thiol and pre
144 a lettuce time-series experiment (mock- and Botrytis cinerea-inoculated) and enables detection of ge
148 opsis, resistance to the necrotrophic fungus Botrytis cinerea is conferred by ethylene via poorly und
149 nt defense against the necrotrophic pathogen Botrytis cinerea is primarily quantitative and genetical
155 hocyanin accumulation, and susceptibility to Botrytis cinerea, one of the most important postharvest
156 freshly harvested roots were inoculated with Botrytis cinerea or Penicillium vulpinum on the day of h
157 ter are more resistant to the phytopathogens Botrytis cinerea, Pectobacterium carotovorum, and Pseudo
158 ndividual grape berries were inoculated with Botrytis cinerea, Penicillium expansum, Aspergillus nige
161 ee agriculturally important plant pathogens (Botrytis cinerea, Pseudomonas syringae, and Fusarium oxy
162 netic analysis revealed flg22-induced PTI to Botrytis cinerea requires BIK1, EIN2, and HUB1 but not g
163 r mechanisms by which PP2A-B'gamma regulates Botrytis cinerea resistance and leaf senescence in Arabi
164 tered in response to the necrotrophic fungus Botrytis cinerea revealed decreases in the levels of pho
165 esponses across diverse pathogens, including Botrytis cinerea, Sclerotinia sclerotiorum, and Pseudomo
167 ghest 1-hydroxyoctan-3-one producer, while a Botrytis cinerea strain led to a decrease of octane-1,3-
168 resistance against the necrotrophic fungus, Botrytis cinerea The induced resistance was enhanced in
169 tial for immunity to the necrotrophic fungus Botrytis cinerea The mos7-1 mutation, causing a four-ami
171 After infection with Pseudomonas syringae or Botrytis cinerea, the expression of genes regulated by b
173 ighlighted by an increased susceptibility to Botrytis cinerea This process was accompanied by an over
174 , the circadian system of the plant pathogen Botrytis cinerea to assess if such oscillatory machinery
176 t donor plants infected by the phytopathogen Botrytis cinerea transfer jasmonic acid via CMNs, which
177 dopsis thaliana with the necrotrophic fungus Botrytis cinerea using millicell culture insert, that en
178 We studied the mechanisms that the fungus Botrytis cinerea utilizes to be tolerant to well-charact
180 iverse population of the generalist pathogen Botrytis cinerea We quantified variation in lesion size
183 Small RNAs of the fungal plant pathogen Botrytis cinerea were previously shown to translocate in
185 hytopathogenic fungi, Fusarium oxysporum and Botrytis cinerea, were chosen to examine the antifungal
186 tibility to infection by the fungal pathogen Botrytis cinerea, which was associated with much stronge