ライフサイエンスコーパス: Brodmann's area

1 vinar nuclei of the thalamus and 39 cortical Brodmann's areas.

2 Brodmann's area 10 is one of the largest cytoarchitectur

3 Counts of reelin mRNA-positive neurons in Brodmann's area 10 of either nonpsychiatric subjects or

4 1) of the left prefrontal cortex (maximal in Brodmann's area 10) was seen.

5 's area 8), the right inferior frontal pole (Brodmann's area 10), and the right lateral orbitofrontal

6 BF) decreases in medial orbitofrontal cortex Brodmann's area 10/11, which were absent in the healthy

7 rons in four cortical regions (frontal pole [Brodmann's area 10], primary motor [area 4], primary som

8 sis were detected in the ventral prefrontal (Brodmann's areas 10 and 24) and posterior cingulate cort

9 Postmortem prefrontal cortices (PFC) (Brodmann's areas 10 and 46), temporal cortices (Brodmann

10 misphere, and surrounding association areas (Brodmann's areas 10, 11, 12, and 32).

11 n expression in the human prefrontal cortex [Brodmann's area 11 (BA11) and BA47].

12 ter in the left and right prefrontal cortex (Brodmann's areas 11, 10, 8, and 44) and the left anterio

13 We describe an area centered in Brodmann's area 13m of the medial OFC (mOFC) where taste

14 half of the occipital pole (corresponding to Brodmann's area 17 and serving as control) to examine th

15 as primary visual cortex, striate cortex, or Brodmann's area 17) was defined in each subject by using

16 ity of neurons in the primary visual cortex (Brodmann's area 17, BA17).

17 eactivity were detected in occipital cortex (Brodmann's area 18) in either group, or in the hippocamp

18 activation in both visual cortex, including Brodmann's areas 18 and 19 and the fusiform gyrus, and s

19 erior flow deficits in the occipital cortex (Brodmann's areas 18 and 19), usually symmetric, and best

20 rodmann's areas 7B and 39 and left occipital Brodmann's area 19.

21 y was carried out in Patas monkey neocortex (Brodmann's areas 2, 3, 4, 6, 9, 17, 18, and 24).

22 measured by semiquantitative Western blot in Brodmann's area 21 (middle temporal gyrus) of postmortem

23 the medial surface of the temporal lobe and Brodmann's area 21 of the middle temporal gyrus.

24 ndividual posterotemporal and inferoparietal Brodmann's areas (21, 22 and 39, 40, respectively) the c

25 nce for the crucial role of Wernicke's area (Brodmann's area 22) in word comprehension and indicate t

26 t sections of the medial temporal neocortex (Brodmann's area 22) of 5 male AD patients aged 60-88 yea

27 rtical region, the planum temporale (part of Brodmann's area 22), has a cytoarchitectural homolog, ar

28 dmann's areas 10 and 46), temporal cortices (Brodmann's area 22), hippocampi, caudate nuclei, and cer

29 nificantly lower metabolic activity in right Brodmann's areas 22 and 21 of the superior and middle te

30 und that the volume of the subgenual part of Brodmann's area 24 (sg24) is reduced in familial forms o

31 ht anterior cingulate gyrus, specifically in Brodmann's area 24'.

32 Brodmann's area 32) or the cingulate cortex (Brodmann's area 24).

33 activity) in the rostral anterior cingulate (Brodmann's area 24/32).

34 In contrast, the anterior cingulate cortex (Brodmann's areas 24 and 32) was more active when respond

35 ivity of the anterior cingulate cortex (ACC; Brodmann's areas 24 and 32), whereas no ACC response to

36 44) and the left anterior cingulate cortex (Brodmann's areas 24/32).

37 cts, an rCBF increase in subgenual cingulate Brodmann's area 25 and a decrease in right prefrontal co

38 i was low in the subgenual cingulate cortex (Brodmann's area 25) and high in the amygdala displayed t

39 in the entorhinal area of the human cortex (Brodmann's area 28).

40 10 and 24) and posterior cingulate cortices (Brodmann's area 31), and post hoc analyses indicated tha

41 ith abnormalities in the anterior cingulate (Brodmann's area 32) and dorsolateral prefrontal cortex (

42 n the mPFC centered on the prelimbic region (Brodmann's area 32) or the cingulate cortex (Brodmann's

43 mPFC lesions of prelimbic cortex (Brodmann's Area 32) retarded EB conditioning in the trac

44 ortex (mPFC) centered on the prelimbic area (Brodmann's area 32), at five different intervals after t

45 Within Brodmann's area 32, a glucose metabolism deficit in the

46 ea 6, 10] and the anterior cingulate cortex [Brodmann's area 32]).

47 al cortical regions (viz., ectorhinal cortex=Brodmann's area 36, perirhinal cortex=area 35, lateral e

48 retrieval and phonological processing (e.g., Brodmann's areas 37 and 39) were less likely to show tre

49 bilateral superior anterior temporal lobes (Brodmann's area 38) are selectively activated when parti

50 recorded from the anterior parietal cortex (Brodmann's areas 3a, 3b, 1, and 2) of monkeys performing

51 acaques and recorded the responses evoked in Brodmann's areas 3b, 1, and 2.

52 [Brodmann's area 9 (BA9)] and motor cortex [Brodmann's area 4 (BA4)].

53 emotor, and the anterior cingulate cortices (Brodmann's areas 4, 6, and 32).

54 the right auditory cortex, corresponding to Brodmann's areas 42 and 22, as well as in area 41 (prima

55 Brodmann's area 44 delineates part of Broca's area withi

56 In the inferior frontal gyrus [Brodmann's area 44]), receptor up-regulation was correla

57 Across species, Broca's area comprises Brodmann's areas 44 and 45.

58 eft inferior frontal gyrus, in the region of Brodmann's area 45.

59 nding recurrent excitation of neurons within Brodmann's Area 46 of the dorsolateral prefrontal cortex

60 ostmortem tissue from the prefrontal cortex (Brodmann's area 46) of 14 matched triads of subjects wit

61 of the right dorsolateral prefrontal cortex (Brodmann's area 46/9) in the context of normal task-depe

62 and the right lateral orbitofrontal region (Brodmann's area 47).

63 [Brodmann's area 9], inferior frontal gyrus [Brodmann's area 47], medial frontal gyrus [Brodmann's ar

64 reduced grey-matter volumes, particularly in Brodmann's area 48 on the medial surface of the temporal

65 [Brodmann's area 47], medial frontal gyrus [Brodmann's area 6, 10] and the anterior cingulate cortex

66 receptors were found in the frontal cortex (Brodmann's areas 6, 7, 8, 9, 10, 11, 44, 45, 47), anteri

67 by MEG in the right superior parietal lobe (Brodmann's Area 7).

68 significantly higher metabolism in parietal Brodmann's areas 7B and 39 and left occipital Brodmann's

69 right superior dorsolateral frontal cortex (Brodmann's area 8), the right inferior frontal pole (Bro

70 creased rCBF in two mesial prefrontal zones (Brodmann's areas 8 and 10), inferior orbital frontal lob

71 me in both membrane and cytosol fractions of Brodmann's areas 8 and 9 combined (prefrontal cortex).

72 delta1, and gamma1 isozymes were examined in Brodmann's areas 8 and 9 of postmortem brains obtained f

73 Twelve cortical regions (Brodmann's areas 8, 10, 44, 46, 23/31, 24/32, 20, 21, 22

74 , cerebellum, prefrontal association cortex [Brodmann's area 9 (BA9)] and motor cortex [Brodmann's ar

75 ocannabinoid system in the prefrontal cortex Brodmann's area 9 of 42 schizophrenia subjects and match

76 rder in the CA region of the hippocampus and Brodmann's area 9 of the prefrontal cortex.

77 area 32) and dorsolateral prefrontal cortex (Brodmann's area 9) in MDD.

78 eas damage to the lateral prefrontal cortex (Brodmann's area 9) in monkeys causes a loss of inhibitor

79 volumes were found in the prefrontal cortex (Brodmann's area 9) of PTSD patients than in comparison s

80 92--287%) in dorsolateral prefrontal cortex (Brodmann's area 9) of schizophrenic and bipolar subjects

81 on, the left dorsolateral prefrontal cortex (Brodmann's area 9) was more active for color naming than

82 n the thalamus and medial prefrontal cortex (Brodmann's area 9).

83 25 and a decrease in right prefrontal cortex Brodmann's area 9, were not present in the depressed gro

84 t prefrontal cortex (right precentral gyrus [Brodmann's area 9], inferior frontal gyrus [Brodmann's a

85 ivation of bilateral frontostriatal regions (Brodmann's areas 9/46, 45/46; lenticular nucleus; and th

86 d (individually and by averages of estimated Brodmann's areas and brain regions) using linear regress

87 nclude right dorsolateral prefrontal cortex [Brodmann's area (BA 9)], bilateral parietal (BA 40/7), a

88 the right medial orbital prefrontal cortex [Brodmann's area (BA) 25 and medial BA 11], where methylp

89 refrontal cortex, including anterior-ventral Brodmann's Area (BA) 45/47 and more dorsal BA 44, increa

90 nterior left inferior prefrontal cortex near Brodmann's Area (BA) 45/47 and more posterior and dorsal

91 up-regulated, we studied both parameters in Brodmann's area (BA) 9 from the McLean 66 Cohort Collect

92 Results showed that bilateral ventral [Brodmann's areas (BA) 44, 45, and 47] and right dorsal (

93 and right ventrolateral prefrontal cortices (Brodmann's area [BA] 47) and the right amygdala, a prior

94 using the degree of hypoperfusion in various Brodmann's areas--BA 22 (including Wernicke's area), BA

95 The tissue types were assigned to Brodmann's areas by using the Perry postmortem histologi

96 read with the prefrontal cortex (9 out of 10 Brodmann's areas in the left hemisphere) and temporal lo

97 hemisphere) and temporal lobe (10 out of 11 Brodmann's areas in the left hemisphere) while the pulvi

98 apes, we found that neurons in posterior IT (Brodmann's areas TEO and posterior TE) integrate informa

99 A cortical analysis by individual Brodmann's areas was performed.