ライフサイエンスコーパス: Brodmann

1 Brodmann area (BA) 3a of the primary somatosensory corte

2 Brodmann area (BA)35 structurally covaried within the AT

3 Brodmann showed areas 26, 29, 30, 23, and 31 on the huma

4 Brodmann's 100-year-old summary map has been widely used

5 Brodmann's area 10 is one of the largest cytoarchitectur

6 Brodmann's area 44 delineates part of Broca's area withi

7 read with the prefrontal cortex (9 out of 10 Brodmann's areas in the left hemisphere) and temporal lo

8 hemisphere) and temporal lobe (10 out of 11 Brodmann's areas in the left hemisphere) while the pulvi

9 ntify regions of hypoperfusion/infarct of 16 Brodmann areas.

10 metimes bridged to other columns in layer 4 (Brodmann layer 4C).

11 (i) CUD with the largest rostroventral ACC [Brodmann Area (BA) 10, 11, implicated in default brain f

12 ivity of the anterior cingulate cortex (ACC; Brodmann's areas 24 and 32), whereas no ACC response to

13 ing syntactic processing, patients activated Brodmann areas 45/47 bilaterally and right middle tempor

14 he enzyme were compared for 18 samples of AD Brodmann area 9/10 frontal cortex with 11 age-matched co

15 ma-power time courses were estimated for all Brodmann areas by combing magnetoencephalographic and MR

16 the putamen, caudate, nucleus accumbens, and Brodmann area 9) and 5 brain cell types (excitatory and

17 The effects of age and Brodmann areas were analyzed with a nested multiple anal

18 at connects lateral orbitofrontal cortex and Brodmann area 10 with the anterior temporal lobes.

19 ling revealed that the entorhinal cortex and Brodmann area 35 show the earliest signs of tau accumula

20 uantitative histology in the hippocampus and Brodmann area 9 in 72 clinically matched individuals wit

21 rder in the CA region of the hippocampus and Brodmann's area 9 of the prefrontal cortex.

22 h at the junction of the anterior insula and Brodmann area 47, in Brodmann area 37, and unilaterally

23 the medial surface of the temporal lobe and Brodmann's area 21 of the middle temporal gyrus.

24 ing the neocortex into regions approximating Brodmann areas (BAs).

25 ortex (mPFC) centered on the prelimbic area (Brodmann's area 32), at five different intervals after t

26 The ipsilateral premotor area (Brodmann area 6), bilateral posterior parietal areas (Br

27 nce for the crucial role of Wernicke's area (Brodmann's area 22) in word comprehension and indicate t

28 d 42) and auditory-visual association areas (Brodmann areas 20 and 37) but were rarely found in somat

29 misphere, and surrounding association areas (Brodmann's areas 10, 11, 12, and 32).

30 homotopic posterior parietal cortical areas (Brodmann areas 39 and surroundings) via the posterior an

31 area 6), bilateral posterior parietal areas (Brodmann area 7) and precuneus showed an increase in rCB

32 ried out a deep RNA-seq analysis of the BA4 (Brodmann area 4) motor cortex from seven human HD brains

33 posterior parietal cortex (PPC) bilaterally [Brodmann area (BA) 40, extending into BA 7] and dorsolat

34 erebral cortex, such as those made famous by Brodmann and von Economo, are invaluable for understandi

35 marmosets at the start of the 20th century (Brodmann, 1909) and refined more recently (Paxinos et al

36 is known about whether individual cingulate Brodmann areas show gender-specific patterns of age-rela

37 cts, an rCBF increase in subgenual cingulate Brodmann's area 25 and a decrease in right prefrontal co

38 iated with metabolism in anterior cingulate (Brodmann area 24/25) and orbitofrontal (Brodmann area 11

39 activity) in the rostral anterior cingulate (Brodmann's area 24/32).

40 ith abnormalities in the anterior cingulate (Brodmann's area 32) and dorsolateral prefrontal cortex (

41 Brodmann area 39), left posterior cingulate (Brodmann area 31) and left nucleus accumbens/caudate.

42 ients in the left dorsal anterior cingulate [Brodmann area (BA) 32] but decreased in the right fronta

43 th the layer classification of the classical Brodmann scheme, and provide additional insight into the

44 Across species, Broca's area comprises Brodmann's areas 44 and 45.

45 work involving Broca's area, which comprises Brodmann Areas 44 and 45 (BA44 and BA45).

46 ortex Brodmann area 36 and entorhinal cortex Brodmann area 28.

47 ity involved the medial orbitofrontal cortex Brodmann area 13, which is implicated in reward, and whi

48 nectivity of the medial orbitofrontal cortex Brodmann area 13.

49 Second, the lateral orbitofrontal cortex Brodmann area 47/12 had increased functional connectivit

50 ectivity of the lateral orbitofrontal cortex Brodmann area 47/12 is related to depression.

51 ectivity of the lateral orbitofrontal cortex Brodmann area 47/12 with these three brain areas was low

52 The lateral orbitofrontal cortex Brodmann area 47/12, involved in non-reward and punishin

53 BF) decreases in medial orbitofrontal cortex Brodmann's area 10/11, which were absent in the healthy

54 , especially involving the perirhinal cortex Brodmann area 36 and entorhinal cortex Brodmann area 28.

55 ocannabinoid system in the prefrontal cortex Brodmann's area 9 of 42 schizophrenia subjects and match

56 25 and a decrease in right prefrontal cortex Brodmann's area 9, were not present in the depressed gro

57 ngular gyrus, and the temporal visual cortex Brodmann area 21.

58 diminished in the visual association cortex (Brodmann area [BA] 18; -20.0% vs. control, F((2,22)) = 8

59 investigated in the primary auditory cortex (Brodmann area 41), a site of conserved pathology in Sz.

60 e observed in the anterior cingulate cortex (Brodmann area 24), and they persisted after recovery sle

61 d cingulate gyrus/anterior cingulate cortex (Brodmann area 24).

62 activation in the anterior cingulate cortex (Brodmann area 32) and the orbitofrontal cortex.

63 tex and bilateral anterior cingulate cortex (Brodmann area 32), and enhanced connectivity between DN

64 Brodmann's area 32) or the cingulate cortex (Brodmann's area 24).

65 i was low in the subgenual cingulate cortex (Brodmann's area 25) and high in the amygdala displayed t

66 In contrast, the anterior cingulate cortex (Brodmann's areas 24 and 32) was more active when respond

67 44) and the left anterior cingulate cortex (Brodmann's areas 24/32).

68 n area 38), the ventromedial frontal cortex (Brodmann area 11/32) bilaterally, and the amygdaloid com

69 of arteriolosclerosis in the frontal cortex (Brodmann area 9) was strongly associated with hippocampa

70 right superior dorsolateral frontal cortex (Brodmann's area 8), the right inferior frontal pole (Bro

71 receptors were found in the frontal cortex (Brodmann's areas 6, 7, 8, 9, 10, 11, 44, 45, 47), anteri

72 in the entorhinal area of the human cortex (Brodmann's area 28).

73 y to a site within the primary motor cortex (Brodmann area 4) located in the vicinity of the arm func

74 n the left parieto-temporo-occipital cortex (Brodmann area 37) in reading-epilepsy patients compared

75 eactivity were detected in occipital cortex (Brodmann's area 18) in either group, or in the hippocamp

76 erior flow deficits in the occipital cortex (Brodmann's areas 18 and 19), usually symmetric, and best

77 as demonstrated in the orbitofrontal cortex (Brodmann area 11) with a right-sided predominance.

78 sions); namely lateral orbitofrontal cortex (Brodmann area 47) and medial prefrontal cortex.

79 l motor cortex and inferior parietal cortex (Brodmann area 40), the lateral premotor cortex and bilat

80 teral prefrontal cortex and parietal cortex (Brodmann areas 9 and 40) was related to task performance

81 recorded from the anterior parietal cortex (Brodmann's areas 3a, 3b, 1, and 2) of monkeys performing

82 y in the anterior rostral prefrontal cortex (Brodmann area 10) and temporal poles (Brodmann area 20/3

83 stimulation to the medial prefrontal cortex (Brodmann area 10) and the dorsolateral prefrontal cortex

84 s within the ventromedial prefrontal cortex (Brodmann area 10) during punished reversal errors compar

85 diminished engagement of prefrontal cortex (Brodmann area 10) when viewing angry faces during functi

86 eas in right dorsolateral prefrontal cortex (Brodmann area 10).

87 activation in left medial prefrontal cortex (Brodmann area 10, 32), right hippocampus, bilateral angu

88 ation in the dorsolateral prefrontal cortex (Brodmann area 46/9) in both the control and Parkinson's

89 =9) in the dorsal lateral prefrontal cortex (Brodmann Area 9) of sudden death medication-free individ

90 ruited right dorsolateral prefrontal cortex (Brodmann areas 45 and 46) to a significantly greater ext

91 d lesions in premotor and prefrontal cortex (Brodmann areas 6, 8, 9, and 46), and 100% with posterior

92 ected within dorsolateral prefrontal cortex (Brodmann areas 9 and 46) and amygdala.

93 ostmortem tissue from the prefrontal cortex (Brodmann's area 46) of 14 matched triads of subjects wit

94 of the right dorsolateral prefrontal cortex (Brodmann's area 46/9) in the context of normal task-depe

95 area 32) and dorsolateral prefrontal cortex (Brodmann's area 9) in MDD.

96 eas damage to the lateral prefrontal cortex (Brodmann's area 9) in monkeys causes a loss of inhibitor

97 volumes were found in the prefrontal cortex (Brodmann's area 9) of PTSD patients than in comparison s

98 92--287%) in dorsolateral prefrontal cortex (Brodmann's area 9) of schizophrenic and bipolar subjects

99 on, the left dorsolateral prefrontal cortex (Brodmann's area 9) was more active for color naming than

100 n the thalamus and medial prefrontal cortex (Brodmann's area 9).

101 ter in the left and right prefrontal cortex (Brodmann's areas 11, 10, 8, and 44) and the left anterio

102 mPFC lesions of prelimbic cortex (Brodmann's Area 32) retarded EB conditioning in the trac

103 , involving rostral ventral premotor cortex (Brodmann area 44) and intraparietal sulcus.

104 The right dorsal premotor cortex (Brodmann area 6) and the right precuneus (Brodmann area

105 n responses in the left sensorimotor cortex (Brodmann area [BA] 4), bilaterally in the supplementary

106 cular dementia, in superior temporal cortex (Brodmann area 22) from Alzheimer's disease patients (n =

107 ects activated the superior temporal cortex (Brodmann area [BA] 22) bilaterally, the precentral gyrus

108 We investigated primary visual cortex (Brodmann area 17) from the Stanley Neuropathology Consor

109 ity of neurons in the primary visual cortex (Brodmann's area 17, BA17).

110 , cerebellum, prefrontal association cortex [Brodmann's area 9 (BA9)] and motor cortex [Brodmann's ar

111 ea 6, 10] and the anterior cingulate cortex [Brodmann's area 32]).

112 ipsilesional posterior primary motor cortex [Brodmann area (BA) 4p], contralesional anterior primary

113 [Brodmann's area 9 (BA9)] and motor cortex [Brodmann's area 4 (BA4)].

114 nclude right dorsolateral prefrontal cortex [Brodmann's area (BA 9)], bilateral parietal (BA 40/7), a

115 the right medial orbital prefrontal cortex [Brodmann's area (BA) 25 and medial BA 11], where methylp

116 n expression in the human prefrontal cortex [Brodmann's area 11 (BA11) and BA47].

117 the left posterior inferior temporal cortex [Brodmann area (BA) 37, or Wernicke's Wortschatz], left c

118 d to local atrophy in the entorhinal cortex, Brodmann area 35 and the anterior hippocampus and tau lo

119 ) and cortical thickness (entorhinal cortex, Brodmann area 35) were obtained using Automated Segmenta

120 cy range to be in the primary visual cortex, Brodmann areas 17, 18 and 19 with minor contribution fro

121 al cortical regions (viz., ectorhinal cortex=Brodmann's area 36, perirhinal cortex=area 35, lateral e

122 vinar nuclei of the thalamus and 39 cortical Brodmann's areas.

123 in auditory and speech association cortices (Brodmann areas 22, 39, and 42) and auditory-visual assoc

124 10 and 24) and posterior cingulate cortices (Brodmann's area 31), and post hoc analyses indicated tha

125 emotor, and the anterior cingulate cortices (Brodmann's areas 4, 6, and 32).

126 and right ventrolateral prefrontal cortices (Brodmann's area [BA] 47) and the right amygdala, a prior

127 in the mesial and lateral premotor cortices (Brodmann area 6).

128 dmann's areas 10 and 46), temporal cortices (Brodmann's area 22), hippocampi, caudate nuclei, and cer

129 alone recruited "core" regions of deduction [Brodmann area (BA) 10p and 8m], whereas linguistic infer

130 ded cytoarchitectonically into four distinct Brodmann areas (3a, 3b, 1, and 2), but these areas have

131 Tissue samples containing the DLPFC (Brodmann area 46) were Golgi-stained, and basilar dendri

132 ging results suggests that posterior-dorsal (Brodmann area, BA, 44) and anterior-ventral parts (BA 45

133 d (individually and by averages of estimated Brodmann's areas and brain regions) using linear regress

134 motor (Brodmann area 46) and verbal fluency (Brodmann area 45) tasks.

135 een the mirrored representations in the four Brodmann areas, as predicted from electrophysiology meas

136 ostmortem brain tissues (N = 61 samples from Brodmann Area 10, BA10).

137 ated on synaptosome fractionated tissue from Brodmann area 28 (BA28; n = 58 samples).

138 les were evaluated for the anterior frontal (Brodmann area [BA] 10), posterior frontal (BA 6), pariet

139 nvergent evidence indicates that frontopolar Brodmann area 10, and more generally the anterior prefro

140 retrieval and phonological processing (e.g., Brodmann's areas 37 and 39) were less likely to show tre

141 right hippocampus, bilateral angular gyrus (Brodmann area 39), left posterior cingulate (Brodmann ar

142 erved in the right anterior cingulate gyrus (Brodmann area 24), in the intraparietal sulcus of right

143 of the left anterior middle temporal gyrus (Brodmann area 21, r = 0.41, P < 0.001), along with a pre

144 In the inferior frontal gyrus [Brodmann's area 44]), receptor up-regulation was correla

145 [Brodmann's area 9], inferior frontal gyrus [Brodmann's area 47], medial frontal gyrus [Brodmann's ar

146 [Brodmann's area 47], medial frontal gyrus [Brodmann's area 6, 10] and the anterior cingulate cortex

147 t prefrontal cortex (right precentral gyrus [Brodmann's area 9], inferior frontal gyrus [Brodmann's a

148 ctic processing co-activated left hemisphere Brodmann areas 45/47 and posterior middle temporal gyrus

149 d by the anterior and posterior hippocampus, Brodmann area 36 and the parahippocampal cortex.

150 2%) but some common regions of high hubness (Brodmann areas 10, 11, and 21, cerebellum, and thalamus)

151 In Brodmann area (BA) 3b, interactions predominantly occurr

152 In Brodmann area 9, cognitively active individuals had sign

153 In Brodmann areas 25, 32, and 32', neuronal cell bodies are

154 to the dorsal anterior cingulate area 32 in Brodmann's human brain map, is anterior and dorsal to th

155 hich corresponds to the prelimbic area 32 in Brodmann's monkey brain map, caudal and ventral to the g

156 the anterior insula and Brodmann area 47, in Brodmann area 37, and unilaterally in the left middle te

157 owed the most rapid increases in activity in Brodmann area 10 and the right dorsolateral prefrontal c

158 measured by semiquantitative Western blot in Brodmann's area 21 (middle temporal gyrus) of postmortem

159 We describe an area centered in Brodmann's area 13m of the medial OFC (mOFC) where taste

160 udies were performed in prefrontal cortex in Brodmann area 9 and hippocampus obtained in 27 suicide s

161 mic acid decarboxylase (GAD) is decreased in Brodmann area 9 (BA9) of the dorsolateral prefrontal cor

162 yer II and III pyramidal neuron dendrites in Brodmann area 46 dorsolateral prefrontal cortex using th

163 acaques and recorded the responses evoked in Brodmann's areas 3b, 1, and 2.

164 delta1, and gamma1 isozymes were examined in Brodmann's areas 8 and 9 of postmortem brains obtained f

165 detected at alpha frequencies (10-14 Hz) in Brodmann area 6, but did not covary with the number of r

166 d stimulation coordinates were identified in Brodmann area 46.

167 ) changes in thickness of cortical layers in Brodmann areas 11, 10, 24a and 4 differed; and (iii) dif

168 e Peg Test correlated with GM volume loss in Brodmann area 44 (Broca area; P = .02).

169 1) of the left prefrontal cortex (maximal in Brodmann's area 10) was seen.

170 Counts of reelin mRNA-positive neurons in Brodmann's area 10 of either nonpsychiatric subjects or

171 up-regulated, we studied both parameters in Brodmann's area (BA) 9 from the McLean 66 Cohort Collect

172 reduced grey-matter volumes, particularly in Brodmann's area 48 on the medial surface of the temporal

173 These studies were performed in Brodmann area (BA) 9 and hippocampus obtained from 26 su

174 f 10722 neuronal and 19913 glial profiles in Brodmann areas 9 and 17.

175 ing (lnc) RNAs as well as 213 DE proteins in Brodmann Area 9 of OUD subjects.

176 ere, we show that VGF levels were reduced in Brodmann area 25 (a portion of human vmPFC) of MDD patie

177 pared in the DLPFC, as well as separately in Brodmann areas 9 and 46.

178 ht anterior cingulate gyrus, specifically in Brodmann's area 24'.

179 cognition were partially mediated via tau in Brodmann area 35, even when including Abeta-PET as covar

180 These included Brodmann areas 19/37, the inferior (Brodmann Area 39), a

181 ere network of correlated activity including Brodmann areas 45/47 and posterior middle temporal gyrus

182 activation in both visual cortex, including Brodmann's areas 18 and 19 and the fusiform gyrus, and s

183 A cortical analysis by individual Brodmann's areas was performed.

184 included Brodmann areas 19/37, the inferior (Brodmann Area 39), and superior parietal lobule (Brodman

185 ndividual posterotemporal and inferoparietal Brodmann's areas (21, 22 and 39, 40, respectively) the c

186 apes, we found that neurons in posterior IT (Brodmann's areas TEO and posterior TE) integrate informa

187 eft posterior inferior temporal gyrus (iTG) (Brodmann area 37/19).

188 the dorsolateral prefrontal cortex (lateral Brodmann 9) while participants rested in the MRI scanner

189 ight dorsolateral prefrontal cortex and left Brodmann area 10 at the time of this deception.

190 d to the right intraparietal lobule and left Brodmann area 9 (BA9).

191 ght the functional relationship between left Brodmann area 45 and the left posterior middle temporal

192 egrity and neural activity-primarily in left Brodmann area 45 and posterior middle temporal gyrus-wer

193 d performance; poor tissue integrity in left Brodmann area 45 was associated with reduced functional

194 es showed that only tissue integrity in left Brodmann areas 45/47 was correlated with activity and pe

195 d network of regions including parts of left Brodmann areas 37 and 40 is necessary for reading and sp

196 alysis demonstrated that dysfunction of left Brodmann areas 40 (supramarginal gyrus) and 37 (posterio

197 motor adaptation task demonstrated that left Brodmann area 44 (BA44) played a key role in adaptation,

198 by MEG in the right superior parietal lobe (Brodmann's Area 7).

199 so involved the inferolateral temporal lobe (Brodmann area 20/21) and fusiform gyrus.

200 nt hypometabolism in the left temporal lobe (Brodmann areas [BAs] 20, 36, and 38), in the right front

201 bilateral superior anterior temporal lobes (Brodmann's area 38) are selectively activated when parti

202 mann Area 39), and superior parietal lobule (Brodmann Area 7).

203 ssive, controls) x anteroposterior location (Brodmann areas 25, 24, 31, 29) x hemisphere (right, left

204 nterior cingulate cortex (ACC) and/or medial Brodmann area (BA) 9 were, in some cases, impaired on vo

205 ions were done for sulci, gyri, and modified Brodmann areas to link macroscopic anatomical and micros

206 sting condition, in two regions of the MPFC (Brodmann Areas 10/32 and 24/25).

207 d circuitry, medial prefrontal cortex (mPFC; Brodmann area 9/10/32), to reward outcome (p(corrected)

208 nterior left inferior prefrontal cortex near Brodmann's Area (BA) 45/47 and more posterior and dorsal

209 y was carried out in Patas monkey neocortex (Brodmann's areas 2, 3, 4, 6, 9, 17, 18, and 24).

210 t sections of the medial temporal neocortex (Brodmann's area 22) of 5 male AD patients aged 60-88 yea

211 rodmann's areas 7B and 39 and left occipital Brodmann's area 19.

212 a centered just posteriorly on the border of Brodmann areas 4a and 6, which we distinguished from a m

213 teral prefrontal cortex (DLPFC, comprised of Brodmann areas 9 and 46) from 19 individuals with a prem

214 osteromedial cortex (PMC), which consists of Brodmann areas 23, 29, 30, 31, and 7m.

215 me in both membrane and cytosol fractions of Brodmann's areas 8 and 9 combined (prefrontal cortex).

216 al cortex (ILPFC)-that is, rodent homolog of Brodmann area 25 (BA25), and the lateral habenula (LHb)

217 tremely large, confirming the observation of Brodmann, who found large somata for these neurons in ca

218 rtical region, the planum temporale (part of Brodmann's area 22), has a cytoarchitectural homolog, ar

219 und that the volume of the subgenual part of Brodmann's area 24 (sg24) is reduced in familial forms o

220 eft inferior frontal gyrus, in the region of Brodmann's area 45.

221 ons into distal digit tip representations of Brodmann area 3b in the squirrel monkey.

222 transcranial magnetic stimulation (rTMS) of Brodmann Area (BA) nine of the left dorsolateral prefron

223 as primary visual cortex, striate cortex, or Brodmann's area 17) was defined in each subject by using

224 ate (Brodmann area 24/25) and orbitofrontal (Brodmann area 11) and prefrontal (Brodmann area 9/10) co

225 significantly higher metabolism in parietal Brodmann's areas 7B and 39 and left occipital Brodmann's

226 ich the orbitofrontal cortex (in particular: Brodmann area 11) encodes gains and expected value also

227 ilaterally in the visual cortex in patients [Brodmann area (BA) 17].

228 ng an error predicted left dorsolateral PFC (Brodmann area 8/9) activation 472 milliseconds after com

229 activated a region in the right rostral PFC (Brodmann area 10).

230 ex (ACC) and medial prefrontal cortex (PFC) (Brodmann area 10/32) 80 milliseconds after committing an

231 Postmortem prefrontal cortices (PFC) (Brodmann's areas 10 and 46), temporal cortices (Brodmann

232 e contralateral dorsal premotor cortex [PMd, Brodmann area (BA) 6] in multiple sclerosis patients tha

233 's area 8), the right inferior frontal pole (Brodmann's area 10), and the right lateral orbitofrontal

234 ns of neuronal loss, the left temporal pole (Brodmann area 38) was the most significantly and consist

235 In addition, the right temporal pole (Brodmann area 38), the ventromedial frontal cortex (Brod

236 rons in four cortical regions (frontal pole [Brodmann's area 10], primary motor [area 4], primary som

237 ortex (Brodmann area 10) and temporal poles (Brodmann area 20/38) relative to offenders with ASPD-P a

238 tex (SI) and posterior parietal cortex (PPC; Brodmann areas 7/40).

239 The precuneus (PreC; Brodmann area 7), a key hub within the default mode netw

240 x (Brodmann area 6) and the right precuneus (Brodmann area 7) showed a linear increase of rCBF as seq

241 tofrontal (Brodmann area 11) and prefrontal (Brodmann area 9/10) cortices, and with personality score

242 sis were detected in the ventral prefrontal (Brodmann's areas 10 and 24) and posterior cingulate cort

243 ss, surface area, and volume of the primary (Brodmann area 17/V1) and secondary (Brodmann area 18/V2)

244 eft dorsolateral prefrontal cortical region (Brodmann areas 44, 45, and 46), where the average global

245 and the right lateral orbitofrontal region (Brodmann's area 47).

246 n the mPFC centered on the prelimbic region (Brodmann's area 32) or the cingulate cortex (Brodmann's

247 predominantly left medial prefrontal region [Brodmann area (BA) 8/9/10] was more active during the ta

248 found in somatosensory association regions (Brodmann area 21).

249 A modifications in five human brain regions (Brodmann areas 9 and 24, and the caudate, hippocampus an

250 Twelve cortical regions (Brodmann's areas 8, 10, 44, 46, 23/31, 24/32, 20, 21, 22

251 ivation of bilateral frontostriatal regions (Brodmann's areas 9/46, 45/46; lenticular nucleus; and th

252 activity in early visual processing regions (Brodmann area (BA)17, BA18).

253 cterized the proteomes of two brain regions, Brodmann area 19 (BA19) and posterior inferior cerebellu

254 ne atlas-based grey or white matter regions: Brodmann areas 44 and 45 (together known as Broca's area

255 d gene expression data of two other regions: Brodmann area 19 (occipital cortex) and cerebellar corte

256 nificantly lower metabolic activity in right Brodmann's areas 22 and 21 of the superior and middle te

257 riptome analysis on cortical tissue samples (Brodmann areas 20 and 21) from 86 patients with mesial t

258 primary (Brodmann area 17/V1) and secondary (Brodmann area 18/V2) visual areas and the middle tempora

259 eral prefrontal cortex for the sensorimotor (Brodmann area 46) and verbal fluency (Brodmann area 45)

260 subgenual anterior cingulate cortex (sgACC; Brodmann area 25) predicts outcome in CT for depression,

261 and nonhuman area 32 has been impaired since Brodmann said he could not homologize with certainty hum

262 early tau and/or Abeta pathology (subiculum/Brodmann area 35/precuneus/posterior cingulate).

263 ctivity of the non-reward/punishment system (Brodmann area 47/12) with the precuneus (involved in the

264 lear evidence that the PPC site we targeted (Brodmann areas 7/40) contributes to tactile direction pe

265 sted that our findings support the view that Brodmann area 45 is involved in verbal response generati

266 The Brodmann area-based approach also facilitates comparison

267 medial anterior PFC (aPFC, encompassing the Brodmann area 10) on threat memory and generalization.

268 anscriptional and cellular signatures in the Brodmann area 9 (BA9) of the frontal cortex and the hipp

269 in the lingual and fusiform gyri and in the Brodmann areas 22 and 38 in superior temporal sulcus (ST

270 ed pathological changes were observed in the Brodmann areas 44 (Broca's area) and 45.

271 somatotopic maps defining the extent of the Brodmann areas could be directly observed on the cortica

272 Thus, Brodmann's map understates the rostral extent of retrosp

273 The tissue types were assigned to Brodmann's areas by using the Perry postmortem histologi

274 the marginal sulcus, likely corresponding to Brodmann area 31.

275 ortex (DLPFC; approximately corresponding to Brodmann areas 9 and 46) has demonstrable roles in diver

276 half of the occipital pole (corresponding to Brodmann's area 17 and serving as control) to examine th

277 the right auditory cortex, corresponding to Brodmann's areas 42 and 22, as well as in area 41 (prima

278 were, with only a few exceptions, limited to Brodmann area 9, suggesting regional specificity of path

279 to the D3 and D4 receptors, and localized to Brodmann area 11 (orbitofrontal cortex).

280 gnant left frontal meningioma impinging upon Brodmann area 45, presented a 'pure' dynamic aphasia.

281 using the degree of hypoperfusion in various Brodmann's areas--BA 22 (including Wernicke's area), BA

282 refrontal cortex, including anterior-ventral Brodmann's Area (BA) 45/47 and more dorsal BA 44, increa

283 Results showed that bilateral ventral [Brodmann's areas (BA) 44, 45, and 47] and right dorsal (

284 Finally, in agreement with Brodmann, gigantopyramidal neurons in both the morpholog

285 ial temporal lobe that largely overlaps with Brodmann area 35, and the entorhinal cortex.

286 Within Brodmann's area 32, a glucose metabolism deficit in the

287 nding recurrent excitation of neurons within Brodmann's Area 46 of the dorsolateral prefrontal cortex

288 creased rCBF in two mesial prefrontal zones (Brodmann's areas 8 and 10), inferior orbital frontal lob