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1 eatment of intracellular pathogens including Brucella melitensis.
2 I that is important for the pathogenicity of Brucella melitensis.
3 rB2, and asp24, in both Brucella abortus and Brucella melitensis.
4 cal to Escherichia coli and 77% identical to Brucella melitensis.
5 teremias and bacteremia, that were caused by Brucella melitensis.
6 he metabolic profile of mice challenged with Brucella melitensis.
7 he development of neurobrucellosis caused by Brucella melitensis.
8  pathogenesis of focal brucellosis caused by Brucella melitensis.
9 e hypothesized that the znuA gene present in Brucella melitensis 16 M would be similar to znuA in B.
10  regulator previously linked to virulence in Brucella melitensis 16 M.
11 ompare it to those of Brucella suis 1330 and Brucella melitensis 16 M.
12 the sensitivity to serum of smooth, virulent Brucella melitensis 16M and B. abortus 2308, rough wboA
13                       The recently completed Brucella melitensis 16M and Brucella suis 1330 genomes h
14                                          The Brucella melitensis 16M genome was examined for proteins
15  28-kDa protein from a lambdagt11 library of Brucella melitensis 16M genomic DNA.
16 rucellosis, Balb/c mice were challenged with Brucella melitensis 16M in a nose-only aerosol exposure
17 showed that a purE mutant (delta purE201) of Brucella melitensis 16M is attenuated for growth in cult
18  cultured murine peritoneal macrophages with Brucella melitensis 16M treated with complement- and/or
19 ming by the injection of heat-killed or live Brucella melitensis 16M.
20 1 (58.4% identity), and the OMP31 porin from Brucella melitensis (31.7% identity).
21               The pathophysiology of chronic Brucella melitensis aerosol infection was monitored in t
22  caused primarily by the bacterial pathogens Brucella melitensis and B. abortus.
23 phages that are lytic mainly for B. abortus, Brucella melitensis and Brucella suis; Group III was com
24      A similar mutation has been adapted for Brucella melitensis and tested to determine whether oral
25 es are present in Agrobacterium tumefaciens, Brucella melitensis, and all sequenced strains of Rhizob
26 he wboA gene in smooth, virulent B. abortus, Brucella melitensis, and Brucella suis results in rough,
27 pathogens, including Francisella tularensis, Brucella melitensis, and Legionella pneumophila.
28    Mice were infected intraperitoneally with Brucella melitensis, and the clinical course of disease,
29 tandard tube tests with Brucella abortus and Brucella melitensis antigens (SATA and SATM), serum enzy
30 he HMG-CoA lyases from Bacillus subtilis and Brucella melitensis at 2.7 and 2.3 A resolution, respect
31 telline protein B [VpB]) and containing live Brucella melitensis attenuated mutant vjbR::Tn5 (BMEII11
32 as developed first time for the detection of Brucella melitensis (B. melitensis) based on the screeni
33 dically relevant pathogens Brucella abortus, Brucella melitensis, Bartonella henselae, and Legionella
34 ial genera, and the recombinant homolog from Brucella melitensis bound heme and catalyzed its degrada
35 olderia pseudomallei, Clostridium botulinum, Brucella melitensis, Brucella abortus, Brucella suis, an
36 on of the four species pathogenic in humans (Brucella melitensis, Brucella abortus, Brucella suis, an
37      We have discovered that the prokaryotes Brucella melitensis, Brucella abortus, Erythrobacter lit
38 ive Gram-negative disease-causing organisms: Brucella melitensis, Brucella suis, Vibrio cholera, Yers
39 ing four clades as follows: Brucella abortus-Brucella melitensis, Brucella suis-Brucella canis, Bruce
40 efaciens (AGRTU), and the mammalian pathogen Brucella melitensis (BRUME).
41    Here, we demonstrate that inactivation of Brucella melitensis by gamma-irradiation inhibited its r
42                                   Except for Brucella melitensis, cgMLST schemes for Brucella species
43                After intranasal inoculation, Brucella melitensis chronically infects the mononuclear
44 is, Yersinia pestis, Francisella tularensis, Brucella melitensis, Clostridium botulinum, Vaccinia vir
45 onsisting of the lipopolysaccharide (LPS) of Brucella melitensis complexed with the outer membrane pr
46                   Accidentally infected with Brucella melitensis during her laboratory research, Evan
47                                              Brucella melitensis encounters a variety of conditions a
48 iagnosis was established by the isolation of Brucella melitensis from abscess fluid.
49                   Comparison of B. suis with Brucella melitensis has defined a finite set of differen
50 unusually, a closely related human pathogen, Brucella melitensis, has four putative BioR-binding site
51  take advantage of the BALB/c mouse model of Brucella melitensis infection will help to clarify the c
52                                              Brucella melitensis is a facultative intracellular bacte
53                                              Brucella melitensis is a major livestock bacterial patho
54                                              Brucella melitensis is a well-adapted zoonotic pathogen
55                                              Brucella melitensis is an intracellular pathogen that es
56        Among the different Brucella species, Brucella melitensis is considered the most virulent.
57                                              Brucella melitensis is endemic among livestock populatio
58 gnant women who become acutely infected with Brucella melitensis is spontaneous pregnancy loss or ver
59                                              Brucella melitensis is the cause of brucellosis in sheep
60 VA) subtyping methods on a collection of 101 Brucella melitensis isolates from sporadic human cases o
61 ntranasal immunization of mice with purified Brucella melitensis lipopolysaccharide (LPS) as a noncov
62 subcutaneously or intranasally with purified Brucella melitensis lipopolysaccharide (LPS) or with LPS
63 sruption of Tat increases the sensitivity of Brucella melitensis M28 to the membrane stressor sodium
64  a genetic deletion mutant (deltapurE201) of Brucella melitensis (n = 6), its virulent parental strai
65 (n=13, 4.2%), Brucella spp (n=12, 3.9%), and Brucella melitensis (n=11, 3.6%).
66 be P41, similar to a hypothetical protein of Brucella melitensis, occurred among 41% of middle ear is
67                                              Brucella melitensis, one of the causative agents of huma
68 escence imaging to gain greater insight into Brucella melitensis pathogenesis.
69                            The response to a Brucella melitensis purEK deletion mutant, delta purE201
70                                    The three Brucella melitensis ribosomal RNA operons rrnA, rrnB, an
71 ants by mariner transposon mutagenesis using Brucella melitensis rough mutant 16M delta manBA as the
72 J774.A1 cell line to infection with virulent Brucella melitensis strain 16M.
73 e equally common to all Brucella abortus and Brucella melitensis strains but unique to Brucella.
74 ribed the TIR domain-containing protein from Brucella melitensis, TcpB (BtpA/Btp1), to be involved in
75 ells alone confers marked protection against Brucella melitensis that is abrogated by cotransfer of B
76 oup classical Brucella species as biovars of Brucella melitensis, the commonly recognized Brucella sp
77 potential role for regulation of flagella in Brucella melitensis via cyclic-di-GMP.
78                                              Brucella melitensis was identified in 80% of patients fo
79                                              Brucella melitensis was the causative agent in 80% (135/
80        Human brucellosis is caused mainly by Brucella melitensis, which is often acquired by ingestin
81 enuated, purine-auxotrophic mutant strain of Brucella melitensis, WR201, for its ability to elicit ce
82 l other bacteria including Brucella abortus, Brucella melitensis, Yersinia enterocolitica O9, Escheri