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1                                              C. diphtheriae and C. ulcerans mutants defective in haem
2                                              C. diphtheriae C7(-) acquired iron from heme, hemoglobin
3                                              C. diphtheriae ciuE deletion mutants exhibited a defect
4                                              C. diphtheriae secretes ChtA and HtaA hemophore proteins
5                                              C. diphtheriae was also able to use human serum albumin
6 re, we describe the genomic variation of 502 C. diphtheriae isolates across 16 countries and territor
7                                            A C. diphtheriae mntA mutant grew as well as the wild type
8                                            A C. diphtheriae zur mutant was more sensitive to peroxide
9    In this study, two clones isolated from a C. diphtheriae chromosomal library were shown to activat
10                           Four clones from a C. diphtheriae genomic library complemented several of t
11 23, 44 patients (median age, 44 years) had a C. diphtheriae-positive clinical culture, with most dete
12 gene complemented the mutant phenotypes of a C. diphtheriae DeltadtxR strain.
13          Two (9.5%) cases died, one due to a C. diphtheriae infection and one due to C. ulcerans.
14 e the source of galactan length variation, a C. diphtheriae ortholog of the M. tuberculosis carbohydr
15                                     Although C. diphtheriae does not depend on other actinomycetal ge
16 mbrane when expressed in Escherichi coli and C. diphtheriae.
17 rences from the mammalian HO-1 and bacterial C. diphtheriae HO structures, which suggests a structura
18 ome corynephages, and DT is only produced by C. diphtheriae isolates that harbor tox+ phages.
19 the utilization of heme as an iron source by C. diphtheriae and that the heme oxygenase activity of H
20 se of the Hb-Hp complex as an iron source by C. diphtheriae requires multiple iron-regulated surface
21 ional host iron sources that are utilized by C. diphtheriae.
22 oter-lacZ fusion was dependent on the cloned C. diphtheriae chrA and chrS genes (chrAS), which encode
23          A search of the partially completed C. diphtheriae genome identified a gene, mntR, whose pre
24 briae, FimA, is expressed in corynebacteria, C. diphtheriae strain NCTC13129 polymerized FimA to form
25 x RT-PCR assay to detect tox and distinguish C. diphtheriae from the closely related species C. ulcer
26                            None of the eight C. diphtheriae cases were fully immunized.
27 htheriae IE, required hospital admission for C. diphtheriae or a related condition.
28 ened the need for laboratories to screen for C. diphtheriae.
29  A new IRP, designated IRP6, was cloned from C. diphtheriae by a SELEX-like procedure.
30             Total cellular RNA isolated from C. diphtheriae was used to identify the transcriptional
31 ional DtxR-regulated promoter/operators from C. diphtheriae designated IRP3, IRP4, and IRP5.
32 epidemic of 1993 to 1998 and 13 non-Georgian C. diphtheriae strains (10 Russian and 3 reference isola
33 esults of this work provide insight into how C. diphtheriae and other pathogenic and commensal coryne
34                                           In C. diphtheriae C7, optimal expression from the hmuO prom
35                            Pilus assembly in C. diphtheriae requires the pilin motif and the C-termin
36 ompare and contrast galactan biosynthesis in C. diphtheriae and M. tuberculosis In each species, the
37 mined the precise amount of DtxR per cell in C. diphtheriae.
38 that safeguards oxidative protein folding in C. diphtheriae against thermal stress.
39            Transcription of the hmuO gene in C. diphtheriae is controlled under a dual regulatory mec
40 irst time that dtxR is a dispensable gene in C. diphtheriae.
41               Deletion of the hrtAB genes in C. diphtheriae produced increased sensitivity to hemin,
42                      Inactivation of hmuT in C. diphtheriae by site-specific recombination had no eff
43 ism of the regulation of heme homeostasis in C. diphtheriae.
44 talloregulatory proteins to be identified in C. diphtheriae.
45                     The use of hemin iron in C. diphtheriae involves the dtxR- and iron-regulated hmu
46  and characterized two novel genetic loci in C. diphtheriae that encode factors that bind hemin and H
47  of the general oxidative folding machine in C. diphtheriae.
48 indings demonstrated that the irp6 operon in C. diphtheriae encodes a putative ABC transporter, that
49 l Mn(2+) transport systems may be present in C. diphtheriae.
50 dependent activation at the hmuO promoter in C. diphtheriae; however, it was observed that significan
51 n and may function as the haemin receptor in C. diphtheriae.
52  function as cell surface hemin receptors in C. diphtheriae.
53  insights into transcriptional regulation in C. diphtheriae.
54 a zinc specific transcriptional regulator in C. diphtheriae and give new insights into the intricate
55        To further define the DtxR regulon in C. diphtheriae, a DtxR repressor titration assay (DRTA)
56 e propose that the pilus-specific sortase in C. diphtheriae uses a latch mechanism to select K190 on
57 ered by a lack of molecular genetic tools in C. diphtheriae and related Coryneform species.
58 with a proposed mechanism of hemin uptake in C. diphtheriae in which hemin is initially obtained from
59 B had no effect on hemin iron utilization in C. diphtheriae.
60 n in hemoglobin-iron utilization, whereas in C. diphtheriae strains, deletion of hmuO caused no or on
61  delivered to the cytoplasm of non-lysogenic C. diphtheriae, they integrated into either the attB1 or
62                                         Most C. diphtheriae-positive cultures were polymicrobial, inc
63                   A total of 26 nontoxigenic C. diphtheriae strains isolated in the United Kingdom du
64 acterium diphtheriae strains, 9 nontoxigenic C. diphtheriae strains, and 44 strains representing the
65 esting classified the strain as nontoxigenic C. diphtheriae biotype Gravis.
66 udied the predominant strain of nontoxigenic C. diphtheriae circulating in the United Kingdom to see
67 ses, 1 was toxigenic and 3 were nontoxigenic C. diphtheriae by culture and Elek, 6 were culture-negat
68 monstrated that the majority (87.5%; 7/8) of C. diphtheriae strains represented new sequence types (S
69 he chtC gene has no affect on the ability of C. diphtheriae to use hemin or Hb as iron sources; howev
70 morphism [AFLP]) for the characterization of C. diphtheriae strains.
71  genes were constructed on the chromosome of C. diphtheriae.
72  The distribution of each genetic cluster of C. diphtheriae isolates across multiple countries in Eur
73         Numerous, highly diverse clusters of C. diphtheriae are observed across the phylogeny, each c
74 Here, we characterized a large collection of C. diphtheriae clinical isolates for their pilin gene po
75  attP site and the DIP0182 integrase gene of C. diphtheriae NCTC13129.
76 e six sortase genes encoded in the genome of C. diphtheriae are required for precursor processing, pi
77 he organism showed that several genotypes of C. diphtheriae circulated on different continents of the
78 ntly was caused by one major clonal group of C. diphtheriae (PFGE type A, ribotype R1), which was ide
79                   A distinct clonal group of C. diphtheriae isolates (ET 8 complex) emerged in Russia
80 upported the improbability of importation of C. diphtheriae into this area and rather strongly sugges
81       Therefore, recent clinical isolates of C. diphtheriae produce a single antigenic type of DT, an
82 nd dtxR genes in recent clinical isolates of C. diphtheriae revealed several tox alleles that encode
83 pand the systems for genetic manipulation of C. diphtheriae, we constructed plasmid vectors capable o
84 a dtxR mutant of C7, and in a hmuO mutant of C. diphtheriae HC1 provided further evidence that transc
85    We report that an htaA deletion mutant of C. diphtheriae strain 1737 is unable to use the Hb-Hp co
86                                   Mutants of C. diphtheriae and Corynebacterium ulcerans that are def
87                          The large number of C. diphtheriae infections among migrants is a cause for
88  play a crucial role in the pathogenicity of C. diphtheriae.
89 nd antigenically distinct from other pili of C. diphtheriae.
90 m the SpaABC pili and possibly other pili of C. diphtheriae.
91 BC transporter involved in the protection of C. diphtheriae from hemin toxicity.
92 quencing to determine the genome sequence of C. diphtheriae BQ11 and mechanism of beta-lactam resista
93 quencing to determine the genome sequence of C. diphtheriae BQ11 and the mechanism of B-lactam resist
94 infection caused by a nontoxigenic strain of C. diphtheriae and discuss the epidemiology, possible so
95    Here, we solved the solution structure of C. diphtheriae MsrB (Cd-MsrB) and unraveled its catalyti
96 acted laboratory data on susceptibilities of C. diphtheriae isolates and on other pathogens detected
97  all eight tox regions identical to those of C. diphtheriae Park-Williams No. 8 (tox type 1).
98 d on other pathogens detected at the time of C. diphtheriae identification.
99       Diagnostic tests for toxinogenicity of C. diphtheriae are based either on immunoassays or on bi
100 ve significant implications for treatment of C. diphtheriae infection and may lead to clinical failur
101 ignificant implications for the treatment of C. diphtheriae infection, and may lead to clinical failu
102  RAPD typing, ribotyping, and PFGE typing of C. diphtheriae strains were improved to enable rapid and
103 exotoxin produced by the causative organism, C. diphtheriae; this detection is the definitive test fo
104 istently, cell wall extracts of a particular C. diphtheriae strain (DSM43989) lacking mycolic acid es
105                            We reviewed prior C. diphtheriae-positive cultures to determine if detecti
106                         We show that several C. diphtheriae strains use the hemoglobin-haptoglobin (H
107 e to immune system-induced oxidative stress, C. diphtheriae expresses antioxidant enzymes, among whic
108   The findings from this study indicate that C. diphtheriae contains at least 18 DtxR binding sites a
109 , NMR, and in situ binding measurements that C. diphtheriae selectively captures iron-loaded hemoglob
110 ct on hemin utilization, which suggests that C. diphtheriae has an additional system for transporting
111 rom heme and hemoglobin, which suggests that C. diphtheriae possesses a novel mechanism for utilizing
112                                          The C. diphtheriae GlfT2 gave rise to shorter polysaccharide
113                                          The C. diphtheriae hmuO gene encodes a heme oxygenase that i
114 his report, we identify and characterize the C. diphtheriae hrtAB genes, which encode a putative ABC
115 or each of these systems was cloned from the C. diphtheriae chromosome, and constructs each carrying
116 iron and heme whereas transcription from the C. diphtheriae hmuO promoter shows both significant iron
117 sid operon contained a large deletion in the C. diphtheriae C7 strain, but the sid genes were unaffec
118 tial alpha-meso-hydroxylation of heme in the C. diphtheriae heme oxygenase-catalyzed reaction.
119 rynephages are capable of inserting into the C. diphtheriae chromosome at two specific sites, attB1 a
120 sidered to be non-pathogenic, outside of the C. diphtheriae complex.
121                        A BLAST search of the C. diphtheriae genome identified a seven-gene cluster th
122                        A BLAST search of the C. diphtheriae genome sequence revealed a second two-com
123  of the C. ulcerans mutants and three of the C. diphtheriae mutants.
124 ect on iron uptake or the utilization of the C. diphtheriae siderophore as an iron source.
125 volved in the synthesis and transport of the C. diphtheriae siderophore.
126           The hrtAB genes are located on the C. diphtheriae genome upstream from the chrSA operon, wh
127 nding receptors collectively function on the C. diphtheriae surface to capture hemoglobin and its spo
128 re closely related to each other than to the C. diphtheriae strains isolated in other parts of the Un
129 n iron uptake and reduced ability to use the C. diphtheriae siderophore as an iron source.
130 wed significant genetic diversity within the C. diphtheriae species, and ribotyping and MEE data gene
131                                        Thus, C. diphtheriae employs a common sortase-catalysed mechan
132 ron-regulated promoters in vivo and binds to C. diphtheriae operators in a metal-dependent manner in
133 addresses the clone's origin and relation to C. diphtheriae from throughout Russia.
134 LST scheme, which is currently restricted to C. diphtheriae.
135                                    Toxigenic C. diphtheriae was isolated from five members of four ho
136                     A total of 363 toxigenic C. diphtheriae isolates were identified among 362 patien
137 l respiratory diphtheria caused by toxigenic C. diphtheriae resistant to penicillin and all other B-l
138 l respiratory diphtheria caused by toxigenic C. diphtheriae resistant to penicillin and all other bet
139 epresent a potential reservoir for toxigenic C. diphtheriae.
140 es, 1 was toxigenic and 3 were non-toxigenic C. diphtheriae by culture and Elek, 6 were culture-negat
141               We assessed cases of toxigenic C. diphtheriae infection that were reported in 10 Europe
142 ial reservoir for the emergence of toxigenic C. diphtheriae strains if they possessed functional diph
143                                The toxigenic C. diphtheriae isolate NCTC13129 produces three distinct
144 ssed under high-iron conditions in wild-type C. diphtheriae C7(beta), but they were expressed constit
145 s secretion of diphtheria toxin by wild-type C. diphtheriae.
146 is currently the method of choice for typing C. diphtheriae.
147  Hemin iron utilization assays using various C. diphtheriae mutants indicate that deletion of the cht
148            In a blinded test of 209 verified C. diphtheriae isolates, a 99.5% agreement with the stan
149 utative composite transposon associated with C. diphtheriae isolates that dominated the diphtheria ou
150 eme release measurements are compatible with C. diphtheriae acquiring heme passively released from he
151 tants and on the results of experiments with C. diphtheriae genes cloned in Escherichia coli or analy
152                           Five patients with C. diphtheriae IE were identified within 12 months at a
153 atients (77%), including all 5 patients with C. diphtheriae IE, required hospital admission for C. di
154            Protein localization studies with C. diphtheriae showed that HtaA is associated predominan
155 Tn5-based mutagenesis technique for use with C. diphtheriae, and we used it to construct the first tr
156 o identify all patients aged >=18 years with C. diphtheriae detected in a clinical specimen (ie, woun
157 nstrate that the cohort of CR domains within C. diphtheriae's hemin-uptake system have dissociation c

 
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