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1 ay contribute to the aggressive virulence of C. psittaci.
2 solates of C. pneumoniae, and 20 isolates of C. psittaci.
3 er C. pneumoniae strains, C. trachomatis, or C. psittaci.
4 thogen C. pneumoniae, and the zoonotic agent C. psittaci.
5 ead birds from pet stores for infection with C. psittaci.
6 strains of C. trachomatis and two strains of C. psittaci.
7 ig inclusion conjunctivitis (GPIC) strain of C. psittaci.
8 d within 1 h of infection of host cells with C. psittaci 6BC but that protein quantity peaks during t
9                                              C. psittaci 6BC infectious particles were electroporated
10 the 1000-fold difference in lethal dose of 2 C. psittaci 6BC strains in mice.
11            We report the genome sequences of C. psittaci 6BC, the prototype strain of the species, an
12 homologous recombination was investigated in C. psittaci 6BC.
13 nd C. trachomatis, is partially truncated in C. psittaci 6BC.
14 d 169 animal specimens; 98 were positive for C. psittaci (71.4% genotype A, 3.1% genotype B, 4.1% gen
15 rted; however, two other chlamydial species, C. psittaci and C. abortus, are known zoonotic pathogens
16 ng and weak homologies were also detected in C. psittaci avian and feline pneumonitis strains, respec
17  plates, the synthetic peptides reacted with C. psittaci B577 antisera, but not with sera from specif
18  VD 2 region were strongly reactive with all C. psittaci B577 antisera.
19 As were compared to those from the reference C. psittaci B577 elementary body (EB) ELISA and the Chla
20 blems, were screened for antibodies by these C. psittaci B577 peptide ELISAs and an ELISA with recomb
21                                          The C. psittaci B577 peptide ELISAs, the LPS ELISA, and the
22  protein of C. psittaci serovar 1 (omp1 type C. psittaci B577).
23        Peptide antigens were identified with C. psittaci B577-immune sera by solid-phase scanning of
24               These results suggest that the C. psittaci B577-peptide and Chlamydia LPS ELISAs are su
25 een developed to rapidly detect and genotype C. psittaci by light-upon-extension chemistry and high-r
26 being most similar to those originating from C. psittaci, C. felis and C. caviae.
27 BC, the prototype strain of the species, and C. psittaci Cal10, a widely used laboratory strain.
28 PCR to avian specimens increased the rate of C. psittaci detection.
29 emic chlamydial species in chickens, whereas C. psittaci dominates only in pigeons.
30 s that intranasal delivery of UV-inactivated C. psittaci EB formulated in Vibrio cholerae ghosts (VCG
31 Srp, whose sequence is 64% identical between C. psittaci GPIC and C. trachomatis, is partially trunca
32 uring outbreaks of psittacosis to infer that C. psittaci had been transmitted from birds purchased in
33 ts receptor are critical for defense against C. psittaci in mouse lung infection.
34  trachomatis IncA is structurally similar to C. psittaci IncA and is also localized to the inclusion
35              Infection of HEK 293 cells with C. psittaci increased IFN-gammaR expression only in cell
36 ci, uniquely targets the nuclear envelope of C. psittaci-infected cells and uninfected neighboring ce
37                                 The lysis of C. psittaci-infected cells was characterized further and
38                                     Notably, C. psittaci-infected cells were lysed with greater effic
39 e cells migrated identically to that seen in C. psittaci-infected cells, indicating the host cell was
40 ted lysis differ between C. trachomatis- and C. psittaci-infected cells.
41 wo additional higher M(r) forms are found in C. psittaci-infected cells.
42 onstrated that these proteins are present in C. psittaci-infected HeLa cells but are absent or below
43 rescently labelled anti-IncA antibodies into C. psittaci-infected HeLa cells resulted in immunostaini
44                                              C. psittaci is currently grouped into seven avian genoty
45  the interpretation of results obtained with C. psittaci models of infection and immune resolution, p
46                                              C. psittaci modulates virulence by alteration of host im
47        Overall our findings demonstrate that C. psittaci-negative ocular adnexal EMZL exhibit biased
48 ith Chlamydia trachomatis inclusions but not C. psittaci or C. pneumoniae inclusions.
49                                          The C. psittaci plasmid was also sequenced.
50  three species of Chlamydiaceae; LcrH-2 from C. psittaci reacted with LcrE from C. pneumoniae but not
51 (VDs) of the major outer membrane protein of C. psittaci serovar 1 (omp1 type C. psittaci B577).
52  of EUO, a previously described ORF of avian C. psittaci strain 6BC which is preferentially transcrib
53 antiserum raised against a recombinant ovine C. psittaci strain POMP, and two possessed surface-expos
54         None of the C. trachomatis serovars, C. psittaci strains, other organisms, or human DNAs test
55 on at the periphery of aberrant RB, while in C. psittaci, treatment causes SEP to localize to distinc
56 ment of the 16S rRNA gene were observed when C. psittaci was electroporated with the recombination su
57     Additionally, target cells infected with C. psittaci were lysed by C. trachomatis-elicited immune
58                                 TETR-PCR for C. psittaci with primer set CPS 100-CPS 101 showed subst