ライフサイエンスコーパス: CBF

1 CBF and cerebrovascular control were measured using midd

2 CBF and CMRO2 were quantified before, during and after d

3 CBF and relative CBF in the most perfused area of each n

4 CBF at rest was quantified on a voxelwise basis using ar

5 CBF change in the bilateral parietal cortices also corre

6 CBF differences have been detected in MDD, and may revea

7 CBF is correlated with tumor microvascular density.

8 CBF reduction required protein kinase C but was not asso

9 CBF responses to neural activity (functional hyperemia),

10 CBF transcriptional complex directly bound to Il13 and V

11 CBF was significantly restored with ECFCs and almost tot

12 CBF was strongly correlated with microvascular density (

13 CBF-tau associations were related to cognition and drive

14 CBFs and CBF regulon genes were down-regulated in rdm4 b

15 (27,467 vs. 20,841 mm Hg/min(-1); p = 0.02), CBF was similar in both groups.

16 soquinolines (THIQs) in the presence of Ph(3)CBF(4) as an oxidant to afford 1-substituted THIQs.

17 he best estimator of the penumbra (group A), CBF relative to the contralateral hemisphere (AUC 0.80)

18 In addition, CBF-independent classes of BR-regulated COR genes are id

19 e ameliorated white matter lesions, although CBF responses were unchanged.

20 eported together as core binding factor AML (CBF-AML).

21 iation analyses assessed relationships among CBF/sPDGFRbeta, tau, and cognition.

22 Decreased amygdala CBF correlated with reduced depressive symptoms.

23 t on BBB disruption, cerebral apoptosis, and CBF were evaluated by SPECT/CT up to 14 d after MCAO.

24 ial impact on BBB disruption, apoptosis, and CBF.

25 CBFs and CBF regulon genes were down-regulated in rdm4 but not nr

26 peat/DREB binding factor (CBF)-dependent and CBF-independent cold signaling pathways to tolerate cold

27 C-REPEAT BINDING FACTOR (CBF)-dependent and CBF-independent pathways.

28 ransfer function analysis of BP as input and CBF velocity as output can express dynamic CA through it

29 r adults had lower cognitive performance and CBF, but similar cerebrovascular reactivity to CO(2) and

30 show both resting-state field potentials and CBF were depressed after cocaine administration (19.8+/-

31 Correlations between LFP power and CBF, used to estimate NVC, were enhanced under high ACh

32 -skull electroencephalography recordings and CBF with laser Doppler flowmetry in the rat's somatosens

33 FA in the SN and CBF in the caudate nucleus were inversely correlated wit

34 genotype and a Hfd on capillary stalling and CBF.

35 d artery (ICA) and vertebral artery (VA) and CBF velocity at the middle cerebral artery (MCA).

36 a real-time visualization of vasculature and CBF was available in high spatial resolution through the

37 in areas of vasoconstriction (angiogenesis), CBF remained reduced even after 1 month of detoxificatio

38 been investigated The present study assessed CBF regulation and neurovascular coupling during submaxi

39 induced CBF increases than to the background CBF should be considered when interpreting functional MR

40 of understanding the interplay between basal CBF and resting neuronal activity.

41 The correlation between CBF and microvascular density was analyzed in specimens

42 tudies that reported no relationship between CBF changes and the occurrence of AMS.

43 With CVR = BP/CBF, Slope-CVRICA , Slope-CVRVA and Slope-CVRiMCA were d

44 There was no difference in whole-brain CBF among the 3 cohorts (P = .148).

45 ed by rdm4 overlapped with those affected by CBFs.

46 n diminishes global and individual capillary CBF responses to neuronal stimuli, resulting in neurovas

47 terial CO2 are particularly potent to change CBF (1 mmHg variation in arterial CO2 changes CBF by 3%-

48 BF (1 mmHg variation in arterial CO2 changes CBF by 3%-4%), the coupling mechanism is incompletely un

49 G E3 ligase 5 and the transcription cofactor CBF-beta.

50 ed that exchange transfusions would decrease CBF and OEF by increasing CaO2, thereby relieving cerebr

51 e vertex ECT electrode, as well as decreased CBF within lateral frontoparietal regions.

52 An acute hypoxic challenge decreased CBF (P < 0.05) in untrained VEGF(f/f) , untrained VEGF(H

53 Our results show significantly decreased CBF and CMRO2 during hypothermic CPB.

54 Cold stress decreased CBF by 20-30% and CDO(2) by 12-19% (both P < 0.01) irres

55 he ability of this approach to differentiate CBF in different locations of the oviduct at different p

56 his study demonstrate common and dissociable CBF abnormalities across neuropsychiatric disorders in y

57 tary to PANX1, in ATP release and downstream CBF modulation following a mechanical stimulus in airway

58 Several DREB/CBF TFs directly promote transcription of the IAA5 and I

59 8 families, including 26 members of the DREB/CBF family.

60 and stroke, will give rise to dysfunctional CBF regulation and may result in subsequent neuronal dam

61 SIL, producing divergent influences on early CBF deficits and later white matter lesions.

62 ereas heating did not reliably change either CBF or CDO(2) (both P > 0.08).

63 Reduction of elevated CBF and OEF, both globally and regionally, suggests that

64 optimal phenotypic and functional endpoints (CBF, ASL, ion channel function, presence of differentiat

65 ading to MYB15 degradation and thus enhanced CBF expression under cold stress.

66 r astrocytes in the regulation of CO2-evoked CBF responses.

67 onic ACh deprivation hindered whisker-evoked CBF responses and the amplitude and power in most freque

68 one significantly potentiated whisker-evoked CBF responses through muscarinic ACh receptors and concu

69 ECT responders exhibited CBF increases in the dorsomedial thalamus and motor cort

70 The C-REPEAT-BINDING FACTOR (CBF) pathway has important roles in plant responses to c

71 Expression of the C-repeat-binding factor (CBF) transcription factors is induced by cold stress, wh

72 angements involving the core-binding factor (CBF) transcriptional complex.

73 HYDRATION-RESPONSIVE ELEMENT BINDING FACTOR (CBF) transcriptional regulators that control cold respon

74 ptation in the C-repeat/DREB binding factor (CBF)-dependent and CBF-independent cold signaling pathwa

75 nder cold stress by C-REPEAT BINDING FACTOR (CBF)-dependent and CBF-independent pathways.

76 The C-repeat binding factor (CBF)/dehydration-responsive element binding (DREB1) prot

77 (16.1%) of aberrant non-core-binding-factor (CBF) karyotype patients.

78 The transcription-factor-CBF-dependent cold signaling pathway plays a key role in

79 Core Binding Factors (CBFs) are a small group of heterodimeric transcription f

80 cold acclimation, C-REPEAT BINDING FACTORs (CBFs), interact with PHYTOCHROME-INTERACTING FACTOR 3 (P

81 nscription factors C-REPEAT BINDING FACTORS (CBFs).

82 s, 36 women), recordings of MAP (Finometer), CBF velocity (CBFV; transcranial Doppler ultrasound), en

83 Resting coronary blood flow (CBF) (24.6 +/- 2.0 cm/s vs. 16.6 +/- 3.9 cm/s vs. 15.1 +

84 ention has been paid to cerebral blood flow (CBF) alterations in IGE detected by arterial spin labell

85 Here, cerebral blood flow (CBF) and blood oxygen-level dependent (BOLD) resting-sta

86 rel cortex, measured by cerebral blood flow (CBF) and neurophysiological recordings (local field pote

87 es in the regulation of cerebral blood flow (CBF) and neurovascular coupling remains, however, under

88 Cerebral blood flow (CBF) and oxygen extraction fraction (OEF) are elevated i

89 study aims to evaluate cerebral blood flow (CBF) and oxygen metabolism (CMRO2) intraoperatively in n

90 al correlations between cerebral blood flow (CBF) and quantitative histologic microvascular data.

91 r ultrasound to measure cerebral blood flow (CBF) and reactivity.

92 We determined whether cerebral blood flow (CBF) and regional brain volumes were associated with PIM

93 ise comparisons between cerebral blood flow (CBF) and tau positron emission tomography (PET) images i

94 y 1.0 degrees C reduced cerebral blood flow (CBF) by 20-30% and cerebral oxygen delivery (CDO(2) ) by

95 r response and improves cerebral blood flow (CBF) by activating the rostral ventrolateral medulla.

96 Resting-state regional cerebral blood flow (CBF) can be measured noninvasively with magnetic resonan

97 d phenotypes, including cerebral blood flow (CBF) deficits, white matter lesions, and Notch3(ECD) dep

98 enuates the increase in cerebral blood flow (CBF) evoked by neural activity (functional hyperemia), a

99 whether acutely-reduced cerebral blood flow (CBF) impairs cognition in healthy adults.

100 ers exhibited increased cerebral blood flow (CBF) in bilateral anterior hippocampus, while responders

101 regional reductions in cerebral blood flow (CBF) in response to decreased oxygen supply (hypoxia) at

102 nd marked reductions in cerebral blood flow (CBF) in the PFC, which were exacerbated with chronic exp

103 sly been shown to evoke cerebral blood flow (CBF) increases via the release of the vasodilator PgE2 W

104 Cerebral blood flow (CBF) is controlled by arterial blood pressure, arterial

105 Cessation of cerebral blood flow (CBF) leads to cell death in the infarct core, but tissue

106 cks absolute changes in cerebral blood flow (CBF) linked with brain function and offers a potentially

107 Changes in cerebral blood flow (CBF) may occur with acute exposure to high altitude; how

108 , fiber number (FN) and cerebral blood flow (CBF) measurements.

109 the border zone, where cerebral blood flow (CBF) nadirs; OEF in this region was reduced after CTT.

110 nfluenced by changes in cerebral blood flow (CBF) or radiotracer clearance.

111 the temporal pattern of cerebral blood flow (CBF) recovery following a sudden change in arterial bloo

112 Cerebral blood flow (CBF) reductions are an early feature of AD and are also

113 Cerebral blood flow (CBF) reductions in Alzheimer's disease patients and rela

114 aine-induced changes in cerebral blood flow (CBF) reflect neuronal activation or its vasoactive effec

115 al blood pressure (MAP)-cerebral blood flow (CBF) relationship, with little attention given to the dy

116 d TIMP3 and compromised cerebral blood flow (CBF) remains unknown.

117 of CVR, to explain the cerebral blood flow (CBF) response to a sudden change in mean arterial blood

118 play a critical role in cerebral blood flow (CBF) restoration following ischemic stroke.

119 Cerebral blood flow (CBF) significantly improved in IRL-1620-treated animals

120 to an increase in local cerebral blood flow (CBF) to allow adequate supply of oxygen and nutrients to

121 Cerebral blood flow (CBF) was measured at the internal carotid artery (ICA) a

122 ing period, hippocampal cerebral blood flow (CBF) was measured by functional magnetic resonance imagi

123 Cerebral blood flow (CBF) was measured using colour-coded duplex ultrasonogra

124 al and regional resting cerebral blood flow (CBF), (2) oxygen extraction fraction (OEF), and (3) cere

125 of associations between cerebral blood flow (CBF), an MRI measure of vascular health, and tau patholo

126 tructural connectivity, cerebral blood flow (CBF), and corticospinal excitability, respectively, befo

127 ood density, lower mean cerebral blood flow (CBF), and significant cerebral circulatory delay compare

128 se elicits increases in cerebral blood flow (CBF), as well as core body temperature; however, the iso

129 Increased visual cortex cerebral blood flow (CBF), decreased visual cortex alpha power, and a greatly

130 s defined by (15) O-PET cerebral blood flow (CBF), oxygen extraction fraction, and oxygen metabolism

131 e (Pbto2), and regional cerebral blood flow (CBF).

132 ontinuous monitoring of cerebral blood flow (CBF).

133 cerebral apoptosis, and cerebral blood flow (CBF).

134 celerate and decelerate coronary blood flow (CBF).

135 ter lesions (WMLs), and cerebral blood flow (CBF).

136 We measured cerebral blood flow (CBF, duplex ultrasound), cerebral oxygen delivery (CDO(2

137 such exercise increases cerebral blood flow (CBF; +10-20%) mediated via small elevations in arterial

138 chnique used to measure cerebral blood flow (CBF; perfusion) to understand brain function and detect

139 performed, in which 3D cerebral-blood-flow (CBF) networks in mouse brain over a large field-of-view

140 ancy may be attributed to the use of ASL for CBF measurement at both sea level and high altitude in t

141 ated gene loci, indicating a requirement for CBF heterodimers in EBNA3 recruitment during target-gene

142 ATP, which increases ciliary beat frequency (CBF) and speeds up mucociliary clearance.

143 No change in ciliary beat frequency (CBF) or airway surface liquid (ASL) meniscus length was

144 ial cells, we imaged ciliary beat frequency (CBF), intracellular calcium, and nitric oxide (NO).

145 d responses, such as ciliary beat frequency (CBF).

146 Patients with lower baseline global CBF were more likely to respond to ECT.

147 reases in Q(VA) contributed to higher global CBF throughout both EX and HS (EX: Delta12 +/- 20% vs.

148 lling provided serial measurements of global CBF (gCBF), first during exposure to 21% inspired oxygen

149 -grade pediatric brain tumors display higher CBF than do low-grade tumors, and they may be accurately

150 Regionally, we found higher CBF in the right pallidum/putamen of the cannabis users

151 However, hippocampal CBF increases postindex were more pronounced in nonrespo

152 Hyperemic CBF was less in AS compared with controls (2,170 vs. 2,7

153 ing by blocking neutrophil adhesion improved CBF and short-term memory function in APP/PS1 mice, even

154 ated the use of electrical TNS for improving CBF and brain oxygen tension (PbrO2), with the goal of d

155 er's disease and demonstrated that improving CBF rapidly enhanced short-term memory function.

156 In CBF velocity (CBFV) recordings with transcranial Doppler

157 ound no difference between treatment arms in CBF or regional volumes.

158 and duration of hyperoxia-induced changes in CBF, cortical electrical activity and cognition in 30 he

159 nalyses revealed post-treatment decreases in CBF in the temporal cortex, including the amygdala.

160 med to discover and replicate differences in CBF between MDD participants and healthy controls (HC) a

161 es, leading to both an immediate increase in CBF and rapidly improved performance in spatial and work

162 An increase in CBF was observed in wild-type ALIs following mechanical

163 The increases in CBF with acute hypoxia during thermal stress were approp

164 findings reinforce the key role of PaCO2 in CBF regulation.

165 viously unappreciated cooperating pathway in CBF-AML.

166 roadly resilient against a ~31% reduction in CBF per se in healthy young and older adults.

167 significantly associated with reductions in CBF (~31%).

168 potential to recover if local reductions in CBF are restored.

169 Reductions in CBF were seen in brain regions typically associated with

170 Regional reductions in CBF, and associated vasoconstriction, within the default

171 ly compensated without an additional rise in CBF and associated with brain tissue hypoxia, or higher-

172 support that AMS may be related to increased CBF rather than vasodilation; these results contradict m

173 wing cerebral ischemia in rats by increasing CBF and reducing apoptosis.

174 t and vasodilator) prevented cocaine-induced CBF decreases and neuronal Ca(2+) changes in the PFC, an

175 port for the hypothesis that cocaine-induced CBF reductions lead to neuronal deficits that contribute

176 ctions by cocaine to the stimulation-induced CBF increases than to the background CBF should be consi

177 diverse cooperating mutations may influence CBF-AML pathophysiology as well as clinical behavior and

178 To investigate CBF changes common across psychiatric disorders, we capi

179 Moreover, translocations involving CBFs are not sufficient to induce AML on its own and the

180 n analysis showed that twelve of the lettuce CBF genes were responsive to low temperature (4 degrees

181 respond only to a particular stress, lettuce CBFs provide wider protection from combinations of abiot

182 rain microvascular network to increase local CBF.

183 These effects were not related to local CBF changes in response to PIDs.

184 fine motor performance correlated with lower CBF in the caudate nucleus (P = .01), thalamus (P = .04)

185 ION: Thrombectomy salvaged tissue with lower CBF, likely attributed to earlier reperfusion.

186 ges in cerebrovascular resistance (CVR = MAP/CBF) and MAP relative to baseline values.

187 Results Mean CBF was significantly higher for high-grade than for low

188 The mean CBF of IGE patients was significantly increased in the l

189 and asymmetric spin echo sequences measured CBF and OEF, respectively, which were compared pre- and

190 Tau PET was a significant mediator CBF/sPDGFRbeta-MoCA relationships in numerous regions.

191 lance of TIMP3 and ADAM17 activity modulates CBF through regulation of myocyte KV channel number.

192 Here we show that cold induction of most CBF regulon genes is lower in IT plants compared with SW

193 Negative CBF-tau correlations were observed predominantly in temp

194 In the replication cohort, early negative CBF-tau correlations increased in spatial extent and in

195 h a poorer prognosis compared with other non-CBF aberrant karyotypes and led to lower remission rates

196 collected at 11 h post-hCG unveiled numerous CBFs-downstream genes that are associated with inflammat

197 r transcranial Doppler for the assessment of CBF.

198 2 genomic segment in the proximal cluster of CBF elements, a negative role of HvCBF3 in the distal cl

199 hts into the pathogenesis and development of CBF-AML, while highlighting dramatic differences in the

200 tions were performed to assess the effect of CBF and radiotracer clearance changes on SUVRs and nonin

201 mpk6 mutants display increased expression of CBF genes and enhanced freezing tolerance, whereas const

202 scade in plants causes reduced expression of CBF genes and hypersensitivity to freezing, suggesting t

203 tion factor that regulates the expression of CBF genes, and the phosphorylation promotes the degradat

204 A family of CBF transcription factors plays a major role in reconfig

205 iphoton microscopy, laser speckle imaging of CBF, and electrophysiological recordings in a mouse mode

206 Simulations showed a limited impact of CBF and radiotracer clearance changes on multilinear ref

207 plicity and, second, to assess the impact of CBF changes and radiotracer clearance on SUVRs and nonin

208 Inducer of CBF expression 1 (ICE1) is a master regulator of CBFs, a

209 that the cold signaling activator INDUCER OF CBF EXPRESSION 1 (ICE1), FLC and the floral promoter SUP

210 cription factors ZHOUPI (ZOU) and INDUCER OF CBF EXPRESSION1 (ICE1) play a role in determining the de

211 The influence of CBF and radiotracer clearance changes on amyloid-beta lo

212 o better understand the genomic landscape of CBF-AMLs, we analyzed both pediatric (n = 87) and adult

213 maging scans with voxel-wise measurements of CBF and OEF in 84 participants with SCA who were grouped

214 gulatory element present in the promoters of CBF-regulated genes.

215 ia could lead to dysfunctional regulation of CBF and subsequent neuronal damage.SIGNIFICANCE STATEMEN

216 signal thereby contributes to the rhythm of CBF expression and the downstream COLD RESPONSIVE expres

217 EBNA3C-conditional LCL confirmed the role of CBF in the regulation of EBNA3C-induced and -repressed g

218 and FLT3) were frequent in both subtypes of CBF-AML.

219 Cold-stabilized phyB acts downstream of CBFs to positively regulate freezing tolerance by modula

220 4 constitutively increased the expression of CBFs and regulon genes and decreased cold-induced membra

221 expression 1 (ICE1) is a master regulator of CBFs, and ICE1 stability is crucial for its function.

222 nvestigate the physiological significance of CBFs, we generated a conditional mutant mouse model in w

223 med to isolate the role of T(c) from PCO2 on CBF regulation during submaximal exercise.

224 er, the isolated influence of temperature on CBF regulation during exercise has not been investigated

225 ent the first in vivo mapping of the oviduct CBF in its native context, and demonstrate the ability o

226 4,678 beats/min/mm Hg; p = 0.004), but peak CBF was lowest in patients with structural CMD compared

227 dysfunction, which leads to diminished peak CBF augmentation and increased demand during exercise.

228 siological cost: reduced cerebral perfusion (CBF).

229 e-induced changes to monolayer permeability, CBF and transepithelial resistance.

230 numbra as defined by quantitative (15) O-PET CBF was performed for comparative reasons in 23 patients

231 tified FA in the SN(FA-SNAv), CBF in the PFC(CBF-PFCAv) and FA in the parietal white matter(FA-PWMAv)

232 Some Poplar CBF homologs exhibited patterns consistent with historic

233 temperature-mediated elevations in posterior CBF during exercise that are independent of changes in P

234 sodilators, and decreases in blood pressure (CBF autoregulation) were similarly reduced in TgNotch3(R

235 ificantly increased systemic blood pressure, CBF and PbrO2 at the hyperacute phase of TBI.

236 ing by 1.5 degrees C did not reliably reduce CBF or CDO(2) Oxygen content in arterial blood was fully

237 to test the hypothesis that acutely-reduced CBF (using a pharmacological aid; indomethacin) would im

238 e tested the hypothesis that acutely-reduced CBF (using indomethacin [1.2 mg kg(-1) oral dose]) would

239 . niger, and A. flavus cultures also reduced CBF by ~10% after 60 min exposure, but effects were bloc

240 As reduced CBF may increase vulnerability to ever-present physiolog

241 de the first prospective evidence of reduced CBF in human concussion and subsequent recovery.

242 o 4330 m, but concurrent cold stress reduced CBF and CDO(2) Gross indices of cognition were not impai

243 Regional CBF increased in the right anterior hippocampus in all p

244 Regional CBF may also provide a useful biological marker across d

245 nce imaging, we measured global and regional CBF associated with clinically prescribed ECT and therap

246 Global resting CBF and regional CBF of right superior frontal cortex correlated positive

247 imaging (MRI) and abnormalities in regional CBF are present in many neuropsychiatric disorders.

248 Increased regional CBF of 21.0 mL/100 g/min for 0 Hz, 25.9 mL/100 g/min for

249 destabilize ICE1, which negatively regulates CBF expression and freezing tolerance in plants.

250 Relative CBF (rCBF) was calculated and compared between 106 MDD a

251 CBF and relative CBF in the most perfused area of each neoplasm and contr

252 or suppressor of pial collateral remodeling, CBF, and functional recovery following permanent middle

253 light (PIF3, HY5) and cold stress response (CBF).

254 ease and upregulation of the cold-responsive CBF and COR genes.

255 Resting CBF and normalized volumes were calculated for brain reg

256 Global resting CBF and regional CBF of right superior frontal cortex co

257 ng at rest and that the reduction in resting CBF reflected reduction in synchronized spontaneous neur

258 e results reveal an important role for RUNX3/CBF during B cell transformation and EBV latency that wa

259 rpus callosum was increased but sensorimotor CBF was decreased, particularly in the ipsilesional side

260 nterior hippocampus, while responders showed CBF increases in right middle and left posterior hippoca

261 themia and hypoxemia, and lower and sluggish CBF compared to CMS patients without cerebral edema; but

262 validation identified FA in the SN(FA-SNAv), CBF in the PFC(CBF-PFCAv) and FA in the parietal white m

263 h SN in combination with changes in FA-SNAv, CBF-PFCAv and FA-PWMAv values might serve as potential m

264 ced baseline (~6-12%) and agonist-stimulated CBF.

265 Stronger CBF-tau and sPDGFRbeta-tau correlations were observed in

266 when stratifying by amyloid status, stronger CBF-tau relationships in individuals with lower MoCA sco

267 present study provides in vivo evidence that CBFs act as essential transcriptional regulators of both

268 Collectively, our study reveals that CBFs-PIF3-phyB serves as an important regulatory module

269 at with acute exposure to high altitude, the CBF in acute mountain sickness (AMS) subjects was higher

270 s) play a role through nitric oxide, and the CBF increase produced by endothelial factors.

271 How the CBF genes themselves are activated after cold acclimatio

272 cute exposure to high altitude; however, the CBF of the brain parenchyma has not been studied to date

273 trol, thirteen subjects were included in the CBF analysis.

274 expression at low temperature, including the CBF pathway in Arabidopsis.

275 ld acclimation response likely involving the CBF regulon.

276 on-flowing capillaries or a worsening of the CBF deficit in APP/PS1 mice fed a Hfd as compared to con

277 our data suggest a major involvement of the CBF genes located in the proximal cluster, with no appar

278 1 completely reversed the attenuation of the CBF increase evoked by whisker stimulation but did not a

279 Moreover, the dynamic changes of the CBF networks elicited by acute cocaine such as heterogen

280 mechanism that explains the majority of the CBF reduction seen in two mouse models of Alzheimer's di

281 te MYB15, a transcriptional repressor of the CBF-dependent cold signaling pathway, leading to MYB15 d

282 osterior fossa tumors, additional use of the CBF-to-contrast enhancement ratio yielded sensitivity an

283 A comprehensive phylogenetic analysis of the CBF/DREB1 family members in 20 plant species from the As

284 ke Arabidopsis thaliana whose members of the CBF/DREB1 family respond only to a particular stress, le

285 ed an important role in the expansion of the CBF/DREB1 family.

286 ere we performed a genome-wide search of the CBF/DREB1 gene family in lettuce (Lactuca sativa L.) and

287 the molecular evolutionary properties of the CBF/DREB1 gene family in lettuce and a reference for gen

288 d after two exposures to smoke show that the CBF decreases gradually during both exposures, recoverin

289 ot changed by cocaine (P=0.244), whereas the CBF to the stimulation was reduced 49.9+/-2.6% (P=0.028)

290 S) subjects was higher (P < 0.05), while the CBF of non-AMS subjects was lower (P > 0.05) compared wi

291 pressure as a covariate did not alter these CBF findings (all P > 0.05).

292 caused by adhered neutrophils, contribute to CBF reduction in mouse models of AD.

293 Using CBF loci as seed regions, BOLD-fMRI data from healthy co

294 plex containing the substrate receptor (Vif, CBF-beta, Elongin-B, Elongin-C (VCBC)) and Cullin5 (CUL5

295 -protein docking was used to delineate a Vif/CBF-beta/PPP2R5 complex in which Vif is predicted to bin

296 particularly for stimulation conditions when CBF might be insufficient to cover for the energetic dem

297 ampus (Hip) and the NMSS-Mood score, whereas CBF in the Hip and the prefrontal cortex(PFC) correlated

298 We tested whether CBF/sPDGFRbeta-tau relationships differed based on Montr

299 e full spectrum of mutations coexisting with CBF translocations has not been elucidated.

300 h-throughput sequencing in 215 patients with CBF-AML enrolled in the Phase 3 Trial of Systematic Vers