ライフサイエンスコーパス: CFU-E

1 e majority of cattle excrete less than 10(2) CFU E. coli O157/g feces, most studies, including those

2 No rescue of BFU-E and CFU-E growth was observed when NmU peptide was exogenous

3 The binding of BFU-E and CFU-E to the RGDS and CS-1 sites was blocked by beta1 in

4 f committed erythroid progenitors (BFU-E and CFU-E) and erythroblast differentiation in the subsequen

5 and erythroid colony-forming unit (BFU-E and CFU-E) colonies, the clonogenic assays that quantify ear

6 tion to promoting adhesion of both BFU-E and CFU-E, supported the highest levels of CFU-E migration (

7 ing erythroid burst-forming unit (BFU-E) and CFU-E progenitors purified by a new technique, we demons

8 ematopoietic progenitors (CFU-GM, BFU-E, and CFU-E colonies).

9 Bone marrow CFU-GM, BFU-E, and CFU-E colony formation was significantly reduced while p

10 ed while peripheral blood CFU-GM, BFU-E, and CFU-E was increased in the trauma patients compared with

11 t-forming unit-erythroid were increased, and CFU-E displayed increased sensitivity to suboptimal EPO

12 In contrast, CFU-E colony formation in vitro by normal fetal liver pr

13 e increased the number of one marrow-derived CFU-E from Wv/+ mice.

14 nly at a stage of erythroid differentiation (CFU-E) preceding the activation of beta-globin genes, co

15 n 1A expression also increases 3-fold during CFU-E maturation may be attributable to the action of NF

16 -forming unit and colony-forming unit (BFU-E/CFU-E) activities were significantly reduced in the homo

17 g unit-erythroid (BFU-E), and CFU-erythroid (CFU-E).

18 lopment of later stage definitive erythroid (CFU-E), mast cell and bipotential granulocyte/macrophage

19 n of human and rabbit bone marrow erythroid (CFU-E, BFU-E) and myeloid (CFU-GM) colony growth.

20 and inhibited colony-forming unit erythroid (CFU-E) and CFU granulocyte-monocyte formation in BM cult

21 (SCF) to form colony-forming unit erythroid (CFU-E) colonies.

22 pulation with colony-forming unit-erythroid (CFU-E) activity (stress CFU-E [sCFU-E]) is expanded mark

23 While colony-forming unit-erythroid (CFU-E) and burst-forming unit-erythroid (BFU-E) colonies

24 nts decreased colony-forming unit-erythroid (CFU-E) and colony-forming unit-granulocyte macrophage (C

25 d (BFU-E) and colony-forming unit-erythroid (CFU-E) formation compared with control.

26 5med immature colony-forming unit-erythroid (CFU-E) population.

27 al numbers of colony-forming unit-erythroid (CFU-E) progenitors and erythroid cells that are generate

28 ntiation from colony-forming unit-erythroid (CFU-E) progenitors in fetal liver cells.

29 cellularity, colony forming unit-erythroid (CFU-E) progenitors, and a total absence of germ cells.

30 le numbers of colony-forming unit-erythroid (CFU-E), colony-forming unit-granulocyte (CFU-G), and col

31 des inhibited colony-forming unit-erythroid (CFU-E)-derived colony growth in a concentration-dependen

32 lly increased colony-forming unit-erythroid (CFU-E).

33 re BFU-E, and colony-forming unit-erythroid (CFU-E).

34 se in marrow colony-forming units-erythroid (CFU-E).

35 enitor cells (colony-forming unit-erythroid [CFU-E], burst-forming unit [BFU]-E, and CFU-granulocyte-

36 progenitors (colony-forming units-erythroid, CFU-E) and severe anemia in cats.

37 ntities of the ACK2 antibody reduced femoral CFU-E, BFU-E, and CFU-GM content to less than half that

38 and inhibited the expansion of the immature CFU-E subset.

39 , different H-ras proteins were expressed in CFU-E progenitors and early erythroblasts with the use o

40 he majority of microRNAs (miRNAs) present in CFU-E erythroid progenitors are down-regulated during te

41 er GM-CSF or IL-3 also leads to reduction in CFU-E numbers and hematocrits but does not significantly

42 o induce strong expression of a transgene in CFU-E stage cells.

43 mechanism by which glucocorticoids increase CFU-E formation.

44 mately 50% to 70% of BFU-E and 60% to 80% of CFU-E bound to the carboxy-terminal HBD and to the CS-1

45 e RGDS sequence in contrast to 75% to 85% of CFU-E.

46 BD alone, although they promoted adhesion of CFU-E, failed to support significant levels of migration

47 d self-renewal, which increases formation of CFU-E cells > 20-fold.

48 ured cells, is essential for the function of CFU-E progenitors.

49 .25 to 1 mmol/L, SNP inhibited the growth of CFU-E by 30% to 75%.

50 25 to 1 mmol/L, SNAP inhibited the growth of CFU-E by 33% to 100%.

51 E and CFU-E, supported the highest levels of CFU-E migration (11-fold above background).

52 We measured migration of CFU-E on fragments of FN containing each cell binding re

53 escued BFU-E and yielded a greater number of CFU-E than observed with control.

54 Maximal numbers of CFU-E and burst-forming unit-erythroid were increased, a

55 The numbers of CFU-E increased modestly in the femur, and approximately

56 nic H-ras promotes abnormal proliferation of CFU-E progenitors and early erythroblasts and supports t

57 onic retroviral system, and their effects on CFU-E colony formation and erythroid differentiation wer

58 FLVCR is upregulated on CFU-E, indicating that heme export is important in prima

59 T-HSC, MPP, premegakaryocytic/erythroid, Pre CFU-E, Pre GM, MkP, and granulocyte-macrophage compartme

60 e erythropoiesis beyond the late progenitor (CFU-E) stage was drastically inhibited by the EpoR mutat

61 H-ras, not dominant-negative H-ras, reduced CFU-E colony formation.

62 and enhances the formation of Epo-sensitive CFU-E progenitors.

63 ming unit-erythroid (CFU-E) activity (stress CFU-E [sCFU-E]) is expanded markedly to restore the eryt

64 must have occurred in vivo before or at the CFU-E progenitor stage.

65 iesis failure occurs in these animals at the CFU-E/proerythroblast stage, a point at which the transf

66 rovide evidence that the transition from the CFU-E to erythroblasts is critically dependent on c-Myb

67 From the CFU-E/proerythroblast (CD71(+) Ter119(-) cells) stage on

68 se expression is confined exclusively to the CFU-E erythroid precursor cells, but not in mature eryth

69 )CD105(+)CD36(+) cells as LEP giving rise to CFU-E, in a hierarchical fashion.

70 ent growth of erythroid colony-forming unit (CFU-E) and erythropoietin hypersensitivity, and Southern

71 conventional erythroid colony-forming unit (CFU-E) assays have been obtained concerning the role of

72 t (BFU-E) and erythroid colony-forming unit (CFU-E) numbers only in the adult bone marrow of the trip

73 that enhance erythroid colony-forming unit (CFU-E) production, we studied the mechanism by which glu

74 re, EpoR(+/-) erythroid colony-forming unit (CFU-E) progenitors are reduced both in frequency and in

75 myb knockdown erythroid colony-forming unit (CFU-E) stage progenitors displayed an immature phenotype

76 unit (BFU-E), erythroid colony-forming unit (CFU-E), and colony-forming unit-granulocyte macrophage-e

77 development (erythroid colony-forming unit [CFU-E] stage).

78 progenitors (erythroid colony-forming unit [CFU-E]) are reduced in p85alpha-/- fetal livers compared

79 progenitors (erythroid colony-forming unit [CFU-E]) were reduced in K-Ras(-/-) fetal livers compared