ライフサイエンスコーパス: CHO cell

1 lular assay overexpressing human arginase I (CHO cells).

2 M) using flow cytometry of P2Y14R-expressing CHO cells.

3 rms diffusing in the plasma membrane of live CHO cells.

4 ed mutagenesis, and expressed the mutants in CHO cells.

5 maintained their activity on CMG2-expressing CHO cells.

6 II, and sLRP1-IV) recombinantly expressed in CHO cells.

7 ysiological properties of Kv2.1 expressed in CHO cells.

8 embrane patches from transiently transfected CHO cells.

9 of cytoplasmic CRT and R-CRT in NIH 3T3 and CHO cells.

10 h effector functions superior to PG9 made in CHO cells.

11 s and inhibits Kv7.4 currents in transfected CHO cells.

12 ere measured in human receptors expressed in CHO cells.

13 interacts with LYVE-1 when overexpressed in CHO cells.

14 e biosynthetic enzymes to produce heparin in CHO cells.

15 anced by the expression of H type II HBGA in CHO cells.

16 the lack of a unifying genomic resource for CHO cells.

17 expression of complement receptor 3 (CR3) by CHO cells.

18 activation of SM synthesis in OSBP-deficient CHO cells.

19 rs and fewer IgG aggregates and fragments in CHO cells.

20 rotein, as well as Thy-1(+)-activated EC and CHO cells.

21 an observed in mtDNA isolated from wild-type CHO cells.

22 YFP-coupled LH receptors stably expressed on CHO cells.

23 the G(1196)|G(1197) dipeptide in transfected CHO cells.

24 o the nsp2 product identified in transfected CHO cells.

25 IC2 fragments were detected in oocytes or in CHO cells.

26 diate the synthesis of unnatural proteins in CHO cells.

27 ever, neither inhibited MGAT1 in transfected CHO cells.

28 ndering the application of this technique to CHO cells.

29 y folded, soluble, and functional FXIII-A in CHO cells.

30 ed at high levels as a secreted protein from CHO cells.

31 und to the SOAT1 protein prepared from SOAT1-CHO cells.

32 in hagfish plasma and in stably transfected CHO cells.

33 ntact ER fractions prepared from SOAT1/SOAT2-CHO cells.

34 AT1 in a cell-based assay using SOAT1-/SOAT2-CHO cells.

35 ed for CyaA(1-710)/HlyA(411-1024) binding to CHO cells.

36 ant human erythropoietin (rhEPO) produced in CHO cells.

37 e generated rhC7 from Chinese hamster ovary (CHO) cells.

38 (FR) alpha-expressing Chinese hamster ovary (CHO) cells.

39 d in tyrosine-limited Chinese hamster ovary (CHO) cells.

40 (rHuEPO) expressed in Chinese hamster ovary (CHO) cells.

41 ovary cells that express alphavbeta3 (beta3-CHO) cells.

42 antibody expressed in Chinese hamster ovary (CHO) cells.

43 ae were isolated from Chinese hamster ovary (CHO) cells.

44 growth inhibition in Chinese hamster ovary (CHO) cells.

45 w cytotoxicity toward Chinese hamster ovary (CHO) cells.

46 cancer cell lines and Chinese hamster ovary (CHO) cells.

47 ic GFP (mGFP-hDAT) in Chinese hamster ovary (CHO) cells.

48 . mori and vertebrate Chinese hamster ovary (CHO) cells.

49 he plasma membrane of Chinese hamster ovary (CHO) cells.

50 kidney (HEK 293) and Chinese hamster ovary (CHO) cells.

51 ioreactors, including Chinese Hamster Ovary (CHO) cells.

52 almost identical effect on alpha2-expressing CHO cell adhesion to collagen I, but only BTT-3033 block

53 at least 80% knockdown of a GFP reporter in CHO cells after 72h.

54 of RNA copy number in both HT-1080 cells and CHO cells also suggests that RBMBs can be used to image

55 orter activity to 3.6%+/-0.3% of baseline in CHO cells and 16%+/-3% in myocytes (both P<0.05), and mu

56 added exogenously is rapidly internalized by CHO cells and accumulates in nuclei in an NLS-dependent

57 ovides valuable insights into translation in CHO cells and can guide efforts to enhance protein produ

58 genous IRE1alpha repressed UPR signalling in CHO cells and deletions in the IRE1alpha locus that de-r

59 ted HS3st1 is broadly distributed throughout CHO cells and forms no detectable AT-binding sites, wher

60 Using transfected CHO cells and gene-targeted mice, we observed that CL40

61 In study 2, gD2 was produced in CHO cells and given intramuscularly with monophosphoryl

62 between P11L and G170R in stably transformed CHO cells and have studied for the first time whether a

63 exhibited prolonged transgene expression in CHO cells and HSCs up to 10 days and 14 days, in the res

64 1, CysLT2 and P2Y12 overexpressed in HEK293, CHO cells and human platelets were used and responsivene

65 cyclic AMP production in beta2AR-transfected CHO cells and induced potent dilation of isolated rat cr

66 e" effects over native hERG channels in both CHO cells and mouse atrial-derived HL-1 cells.

67 e rank order was similar for genotoxicity in CHO cells and mutagenicity in S. typhimurium, the Salmon

68 vity to 62%+/-4% and 57%+/-2% of baseline in CHO cells and myocytes, respectively (both P<0.05).

69 einizing hormone (LH) receptors expressed on CHO cells and native LH receptors on both KGN human gran

70 ly labeled lipoproteins in ILDR1-transfected CHO cells and release of CCK from isolated intestinal ce

71 of nonintegrin, non-HS receptors for FMDV on CHO cells and revealed a novel, non-RGD-dependent use of

72 nts were defective in Yop translocation into CHO cells and splenocyte-derived neutrophils and macroph

73 rified from wild-type Chinese hamster ovary (CHO) cells and HepG2 cells converted dUMP to dTMP in the

74 age endogenous H2S in Chinese hamster ovary (CHO) cells and use the developed constructs to report on

75 ed THIK-1 channels in a mammalian cell line (CHO cells) and used the phosphodiesterase inhibitor 3-is

76 ells, as this is the industrial practice for CHO cells, and developed an in silico method to identify

77 CHO Eogt reduced binding of CTD110.6 to Lec1 CHO cells, and expression of a human EOGT cDNA increased

78 Thy-1(+)-activated human dermal EC, Thy-1(+) CHO cells, and immobilized Thy-1 protein.

79 increased upon metaphase arrest in COS-1 and CHO cells, and in a pancreatic beta cell line that expre

80 ated complement activation on the surface of CHO cells, and it protected complement-sensitive intrace

81 recipitated with PP2A upon transfection into CHO cells, and PP2A/Aalpha knockdown recapitulated the i

82 us cell expression systems (Xenopus oocytes, CHO cells, and rat atrial cardiomyocytes).

83 ins were expressed in Chinese hamster ovary (CHO) cells, and their expression level on the cell surfa

84 CHO cell- and E. coli-derived rhBMP-2 in an ACS carrier

85 CHO cell- and E. coli-derived rhBMP-2 supported comparab

86 use retina, 2. Labeling of TRPM1-transfected CHO cells; and 3. Attenuation of the ERG b-wave followin

87 ore virologically relevant T lymphocytes, in CHO cells; and purifying Env with different chromatograp

88 rn analysis, HAT-L4 expressed in transfected CHO cells appeared as a 48-kDa protein.

89 mammalian cells, but Chinese hamster ovary (CHO) cells are nonpermissive for vaccinia virus (VACV).

90 While parental Chinese hamster ovary (CHO) cells are permissive for both strains, neither stra

91 Chinese hamster ovary (CHO) cells are the predominant production vehicle for bi

92 Chinese hamster ovary (CHO) cells are widely used for the production of biother

93 d the ability to infect CHO and HS-deficient CHO cells as a result of a single glutamine (Q)-to-lysin

94 as quantified using a Chinese hamster ovary (CHO) cell assay, and the descending rank order for cytot

95 fering RNA (siRNA) in Chinese hamster ovary (CHO) cells augments M3-MR signaling.

96 We report on a mammalian CHO cell-based CRISPR-Cas9 mutagenesis screen for genes

97 e Rasa3 in integrin alphaIIbbeta3-expressing CHO cells blocked Rap1 activity and integrin alphaIIbbet

98 detergent micelles, which were isolated from CHO cells, bound to bNAbs, including UCA and intermediat

99 82) results in efficient infection of mutant CHO cells but a decrease in heparin binding, whereas Arg

100 mutation (RRR to AAA) was able to penetrate CHO cells but did not translocate to the nucleus.

101 ficantly reduced activity on TEM8-expressing CHO cells but maintained their activity on CMG2-expressi

102 when transfected into Chinese hamster ovary (CHO) cells but, surprisingly, exerted "chaperone-like" e

103 ion of 14-3-3beta increased ENaC activity in CHO cells, but concomitant expression of beta1Pix attenu

104 Wildtype ribosomes isolated from CHO cells, but not those with P-stalk lesions, stimulate

105 es and the charge crossing Kv4.3 channels in CHO cells by slowing Kv4.3 inactivation kinetics.

106 so resulted in an acquired ability to infect CHO cells by type O and Asia-1 FMDV.

107 t to create a "clean" Chinese hamster ovary (CHO) cell by disrupting multiple genes to eliminate HCPs

108 solution images of lipid-binding proteins on CHO cells can be acquired with NanoSIMS imaging.

109 he therapeutic efficacy of rhEPO produced in CHO cells can be improved by shunting intracellular CMP-

110 e hamster genome as the reference upon which CHO cells can be studied and engineered for protein prod

111 CHO cell chronic cytotoxicity tests indicate that chlori

112 In Chinese hamster ovary (CHO cells), coexpression of rat Oatp1a1 and human ABCG2

113 tibodies from typical contaminants including CHO cell conditioned media, ascites fluid, DNA, and othe

114 somes, and inhibited the proliferation of WT CHO cells, confirming that it is an essential protein fo

115 ered efficiently into Chinese hamster ovary (CHO) cells constitutively expressing HSV-1 human recepto

116 nificant shifts in fluorescence intensity in CHO cell culture fluid spiked with human IgG, and detect

117 was implemented for monitoring variations in CHO cell culture media upon exposure to high temperature

118 conditions (mAb in a Chinese Hamster Ovary (CHO) cell culture harvest).

119 focus here is on the extracellular milieu of CHO cell cultures, this methodology is generally applica

120 xture extracted from HTL-WW expressed potent CHO cell cytotoxic activity, with a LC(50) at 7.5% of HT

121 -cell recordings of KCNN3 channel-expressing CHO cells demonstrated that disease-associated mutations

122 en represent those most commonly reported on CHO cell derived therapeutic antibodies.

123 ived rhBMP-2 displays comparable efficacy to CHO cell-derived rhBMP-2 in vitro and in small-animal mo

124 li-derived rhBMP-2 compared to the benchmark CHO cell-derived rhBMP-2 using an established large-anim

125 Chinese hamster ovary (CHO) cell-derived recombinant human bone morphogenetic p

126 rain efficiently bound to or infected mutant CHO cells devoid of GAG expression.

127 CHO cells do not express CAR or HBGAs and are resistant

128 demand for antibody expression increased in CHO cells during the production phase, or in plasma B ce

129 pling to cAMP production in HEK293 cells and CHO cells ectopically expressing the receptor and in Mad

130 hly optimized growth conditions, recombinant CHO cells engineered to produce a model human IgG clone

131 content cell-based assay in the presence of CHO cells engineered with GFP-PDK1.

132 zed in this pathway may be novel targets for CHO cell engineering.

133 By knocking out these genes, the engineered CHO cells exhibited activation of cellular immune respon

134 CHO cells express two RGD-binding integrins (alpha5beta1

135 In addition, when CHO cells expressed gamma318, gamma323, or gammaFL with

136 and did not increase further in MV-infected CHO cells expressing >/=4,620 CD46 copies/cell, there wa

137 However, CHO cells expressing both hCAR and the type B HBGA were

138 CHO cells expressing CD36 with mutated Lys-164 had impai

139 in DAT-positive immortalized DA neurons and CHO cells expressing DAT decreased the magnitude and rat

140 ing [(35)S]GTPgammaS binding was assessed in CHO cells expressing either human D2 or D3 receptors.

141 interact with itself in trans by binding to CHO cells expressing full-length LINGO-1.

142 T6, and JAK3 phosphorylation was observed in CHO cells expressing gamma323 and gammaFL but not in gam

143 occurred for K(+) currents representing IKr (CHO cells expressing hERG; IC50=219+/-21 mumol/L) and IK

144 ac and McK(gKDelta31-68) viruses entered all CHO cells expressing HSV-1 receptors via a pH-independen

145 000 synthetic small molecules was done using CHO cells expressing human AQP4 and a human NMO recombin

146 c binding to beta2AR in H9c2 cardiomyocytes, CHO cells expressing human beta2AR, and rat aorta.

147 CHO cells expressing human CD36 were SSO-treated, and th

148 capsaicin and acid-induced calcium influx in CHO cells expressing human TRPV1.

149 r into CHO-PILRalpha cells, while it entered CHO cells expressing HVEM and nectin-1 more efficiently

150 In CHO cells expressing integrin alphaIIbbeta3, co-expressi

151 essing hERG; IC50=219+/-21 mumol/L) and IKs (CHO cells expressing KCNQ1+KCNE1; IC50=184+/-12 mumol/L)

152 X-1, whereas little increase was observed in CHO cells expressing only LOX-1.

153 CHO cells expressing rbMate1 truncated at residue Gly-54

154 nhibitor of icilin-induced calcium influx in CHO cells expressing recombinant rat TRPM8.

155 Furthermore, when GalNAc-T3 was expressed in CHO cells expressing the MUC2 C terminus, the second thr

156 ranslocation assay in Chinese hamster ovary (CHO) cells expressing an NFkappaB-EGFP reporter.

157 e cardiac myocytes or Chinese hamster ovary (CHO) cells expressing the mouse or human subunits.

158 om both E. coli and Chinese hamster ovaries (CHO) cell expression platforms; however, isotopic labeli

159 ection with certain ssRNA and dsRNA viruses, CHO cells fail to generate a significant interferon (IFN

160 gp120-alpha4beta7 interactions similarly to CHO cell fibronectin.

161 CHO cell fibronectins were able to mediate the binding o

162 Chinese hamster ovary (CHO) cells, first isolated in 1957, are the preferred pr

163 membrane of isolated Chinese Hamster Ovary (CHO) cells following exposure to nsPEF.

164 oxP site of the alphoid(tetO)-HAC in hamster CHO cells from where the HAC may be MMCT-transferred to

165 profiles observed offer potential to direct CHO cell function during culture through medium design a

166 parental CHO cells over the course of three CHO cell generations.

167 Chlorination of wastewaters produced CHO cell genotoxicity comparable to chloramination, 3.9

168 The estimated demand of NSs towards CHO cell glycosylation can be used to rationally design

169 tric coefficients for NS consumption towards CHO cell glycosylation.

170 lipids to estimate the demand of NSs towards CHO cell glycosylation.

171 eptors in the membrane of stably transfected CHO cells has been observed.

172 cosylation in multiple cell lines, including CHO cells, HeLa cells, normal and patient fibroblasts, i

173 o cell lines and to primary cells, including CHO cells, hiPSC-CMs, and human astrocytes derived in 3D

174 er gene expression in Chinese Hamster Ovary (CHO) cells, Human Immortalized Myelogenous Leukemia (K56

175 hen Env trimers are truncated or produced in CHO cells.IMPORTANCE A protective HIV-1 vaccine will lik

176 lating cAMP accumulation in receptor-bearing CHO cells in a concentration-dependent manner.

177 CdtA subunit remains on the cell surface of CHO cells in association with cholesterol-containing and

178 s stably expressed in Chinese hamster ovary (CHO) cells increased AMPK activity and AMPK phosphorylat

179 Parallel studies in transfected CHO cells indicate that Kv1.1, Kv1.2, Kv7.2/7.3, and mBK

180 d the genotoxicity to Chinese hamster ovary (CHO) cells induced by municipal secondary wastewater eff

181 residue of most repeats, which in wild-type CHO cells is glycosylated with the typical sialylated co

182 In CHO cells, Kv1.1, Kv1.2, and Kv7.2/7.3 K(+) currents wer

183 be expressed from this construct in a stable CHO cell line and purified at an acceptable yield using

184 2A peptides to generate a stably transfected CHO cell line constitutively secreting high levels of re

185 trates, the expression of the Fc domain in a CHO cell line in the presence of an alpha-mannosidase in

186 a web-based tool for integrative analysis of CHO cell line omics data that provides an interactive vi

187 We designed a CHO cell line overexpressing the London mutated betaAPP

188 formation was evaluated for the same clonal CHO cell line producing a BisAb using fed-batch and perf

189 Then, we established a stable CHO cell line that produces rhEPO in the context of repr

190 produced in stable cell lines derived from a CHO cell line with titers over 1 g/L.

191 w of protein translation in an IgG-producing CHO cell line, measured with ribosome profiling.

192 expressed with mMOR-1 in a Tet-Off inducible CHO cell line, mMOR-1G has no effect on mMOR-1 mRNA expr

193 of an IgG1-producing Chinese hamster ovary (CHO) cell line for 18-25 days.

194 ative proteomics data were obtained from two CHO cell lines (CHO-S and CHO DG44) and compared with se

195 These detailed comparisons of CHO cell lines and hamster tissues will enhance understa

196 The developed design matrix and engineered CHO cell lines enables systematic studies towards improv

197 resequenced and analyzed the genomes of six CHO cell lines from the CHO-K1, DG44 and CHO-S lineages.

198 Although genetic heterogeneity among CHO cell lines has been well documented, a systematic, n

199 Using CHO cell lines stably expressing human ABCA1 or ABCG1, w

200 Here we used wild-type and mutant CHO cell lines with alterations in tubulin to demonstrat

201 dentified hamster genes missing in different CHO cell lines, and detected >3.7 million single-nucleot

202 d4, whose knockdown was tested in additional CHO cell lines, confirming the induced high productivity

203 ins to citrullinated collagen was studied by CHO cell lines, each overexpressing 1 of the 4 human col

204 A protein kinase catalytic subunit-deficient CHO cell lines, which are defective for the nonhomologou

205 ration sites and flanking sequences in three CHO cell lines.

206 sphatidylcholine (LPC) compared to SP2/0 and CHO cell lines.

207 lycan (GAG)-deficient Chinese hamster ovary (CHO) cell lines and soluble GAGs, we found that an argin

208 Chinese hamster ovary (CHO) cell lines are widely used in industry for biologic

209 tion assays on mutant Chinese hamster ovary (CHO) cell lines defective in various stages of glycan ch

210 expressed in a set of Chinese hamster ovary (CHO) cell lines under conditions of full glycosylation,

211 racterized a panel of Chinese hamster ovary (CHO) cell lines with inducible transgenes encoding tagge

212 genetically unstable Chinese hamster ovary (CHO) cell lines with only draft genome assemblies availa

213 For Chinese Hamster ovary (CHO) cell lines, key indicators of metabolic events link

214 In contrast to the whole CHO cell lysate which contained a variety of fatty acids

215 e attachment were significantly decreased in CHO cell mutants defective in glycosaminoglycan synthesi

216 ntibodies produced in Chinese hamster ovary (CHO) cells often exhibit a slight yellow-brown color, bu

217 integrin alphaIIbbeta3-mediated spreading of CHO cells on fibrinogen.

218 However, in SLC35A3-knockout CHO cells, only limited changes were observed; GlcNAc wa

219 plying a mouse whole-genome siRNA library to CHO cells, optimized the protocol for suspension culture

220 either as research-grade material in 293 and CHO cells or as two independent lots of clinical materia

221 luorescently labeled PSA or PSA-NCAM to live CHO cells or hippocampal neurons expressing MARCKS as a

222 tic cell immunoreceptor (DCIR) constructs in CHO cells or on DCs were examined by using fluorescent m

223 mited contribution to integrin activation in CHO cells or platelets.

224 lt male Hound Labrador mongrel dogs received CHO cell- or E. coli-derived rhBMP-2 (0.2 mg/mL) in an a

225 e doxycycline-induced cells or from parental CHO cells over the course of three CHO cell generations.

226 invading and damaging endothelial cells and CHO cells overexpressing GRP78.

227 ibits glucagon-stimulated cAMP production in CHO cells overexpressing the human glucagon receptor wit

228 CHO cells overexpressing the human insulin receptor and

229 A production is enhanced more than 2-fold by CHO cell overexpression of integrin beta(3).

230 supernatant during a high-cell-concentration CHO cell perfusion culture.

231 Inside living CHO cells, PhDY-Chol mimics the behavior of cholesterol,

232 ion phase increased antibody productivity in CHO cells, possibly by redirecting antibody molecules fr

233 CHO cells preadapted to oxidative stress resist the effe

234 In semi-intact SOAT1-/SOAT2-CHO cells prepared by a treatment with digitonin (plasma

235 integrin activation, ATX generates LPA from CHO cells primed with bee venom phospholipase A(2), and

236 n underlies BisAb aggregation formation in a CHO cell process.

237 unction, and overexpression of N-cadherin in CHO cells promoted barrier function.

238 Expression of M3R in CHO cells promoted plasma membrane localization of YFP-P

239 Using a model of CHO cell protein secretion, we predict that the eliminat

240 N-linked and O-GalNAc glycosites across the CHO cell proteome, and have derived stoichiometric coeff

241 peat (CRISPR)-Cas9 knockout of chSAMD9L from CHO cells removed the restriction for VACV, while ectopi

242 gamma transactivation activities in a Tet-on CHO cell reporter system, RORalpha co-activator assays a

243 tions to promote the broader use of omics in CHO cell research.

244 However, LRP6 knockdown in wild type CHO cells resulted in a much greater decline in LDL bind

245 dase-treated or sialic acid-deficient mutant CHO cells revealed a 3-15-fold increase in relative bind

246 itu proximity ligation assay (PLA) assays in CHO cells revealed the presence of cell-surface complexe

247 lity of this approach through an analysis of CHO cell samples.

248 (50) values determined on human recombinant (CHO) cells showed very similar inhibitory activities alb

249 in as well as elevated enzymatic activity in CHO cells stably expressing CGalT.

250 Recombinant CHO cells stably expressing hCAR or the type B HBGA alon

251 CHO cells stably expressing human CD36 released severalf

252 SR48692 (5a) using a FLIPR calcium assay in CHO cells stably expressing rat NTS2.

253 CHO cells stably expressing rbSGLT1 were probed by using

254 Knockdown of SET by siRNA in CHO cells stably expressing the M3-MR did not alter agon

255 forded complement resistance, we grew NDV in CHO cells stably transfected with CD46 or HeLa cells, wh

256 Chinese hamster ovary (CHO) cells stably expressing a Tet-on RORalpha or RORgam

257 In CHO cells, stably expressing alphaIIbbeta3 integrins, vi

258 a lack of arginase activity also in several CHO cell strains (CHO-DP12, CHO-S, and DUXB11) and other

259 ty to coactivate integrin alphaIIbbeta3 in a CHO cell system when coexpressed with talin.

260 ay yield organics with lower genotoxicity to CHO cells than chlorine-based disinfection.

261 romoters for biopharmaceutical production in CHO cells that exhibited precisely designed activity dyn

262 iminished in mitochondria isolated from glyA CHO cells that lack SHMT2 activity, as well as mitochond

263 AV2.5T binds but fails to be internalized by CHO cells that lack surface expression of sialic acid.

264 CHO cells that overexpress exogenous GULP (FL) demonstra

265 In mutant Chinese hamster ovary (CHO) cells that do not add galactose (Gal) to the GlcNAc

266 lex N-glycans of Lec8 Chinese hamster ovary (CHO) cells that lack UDP-Gal transporter activity and ex

267 In CHO cells, the AMPK activator 5-aminoimidazole-4-carboxa

268 In trigeminal neurons and CHO cells, the manipulation of cellular PIP(2) led to si

269 hat integrates the reported glycoproteome of CHO cells, the number of N-linked and O-GalNAc glycosyla

270 In CHO cells, this coincident signaling mechanism is involv

271 regulated kinases 1 and 2 (ERK1/2) in intact CHO cells to identify potential agonistic effects as wel

272 As part of an effort to bioengineer CHO cells to produce heparin, we previously showed that

273 explored the basis for the susceptibility of CHO cells to RNA virus infection.

274 acute genotoxicity in Chinese hamster ovary (CHO) cells to compare the toxicity of analogous N-nitros

275 CHO cells transiently transfected with 24p3R or distal t

276 well as mitochondria isolated from wild-type CHO cells treated with methotrexate, a DHFR inhibitor.

277 When added to CHO cells, U-X crosslinked to NPC1.

278 We show that Chinese hamster ovary (CHO) cells used to express recombinant gp120 produced fi

279 of-function screen in Chinese hamster ovary (CHO) cells using insertional mutagenesis to identify gen

280 as uracil levels in mtDNA isolated from glyA CHO cells was 40% higher than observed in mtDNA isolated

281 rTNF-alpha3 produced in CHO cells was bioactive in different cell lines and prim

282 The oligomeric nature of Gi1 in live CHO cells was demonstrated by means of Forster resonance

283 fluorescent labeling of microtubules in live CHO cells was demonstrated with a long-wavelength photoa

284 we show that DLL1-induced NOTCH signaling in CHO cells was enhanced by LFNG, but this did not occur i

285 data for specific growth and productivity of CHO cells, we observe that the demand of NSs towards gly

286 nsfer with functional receptors expressed in CHO cells, we show that the cleft of the amino-terminal

287 d 9-13 toward FRalpha- and FRbeta-expressing CHO cells were only partly reflected in binding affiniti

288 R profiles might affect macrophage function, CHO cells were transfected with SR-AI/II, and phagocytos

289 e cell suspensions of Chinese hamster ovary (CHO) cells were plated on flasks and irradiated with 3,

290 blood cells (RBC), or Chinese hamster ovary (CHO) cells, were immobilized on the inside of the flow c

291 e reporter fusions in Chinese hamster ovary (CHO) cells, where the putative cis element required for

292 ocedure to vesiculate Chinese hamster ovary (CHO) cells, widely used for the expression of recombinan

293 Initially, we found that treating CHO cells with a potent inhibitor of GSL biosynthesis in

294 s, since it occurs even in erbB3-transfected CHO cells with disproportionally small amounts of erbB2.

295 FULL LENGTH (FL)) co-expressed in L-cells or CHO cells with wild-type (WT) IL-4Ralpha.

296 ent of fibroblasts or Chinese hamster ovary (CHO) cells with 25OH caused a 50-70% reduction in Golgi-

297 G protein by 350% in Chinese hamster ovary (CHO) cells with genetically induced expression of AT1 an

298 rvey of bioengineered Chinese Hamster Ovary (CHO) cells with knock-in/out enzymes involved in protein

299 Treating Chinese hamster ovary (CHO) cells with monoHANs followed by the release from th

300 y stably transfecting Chinese Hamster Ovary (CHO) cells with plasmids encoding the rat angiotensin ty