戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 he protein stability of Ctk1 (the major Ser2 CTD kinase).
2 AK components and thus may represent a novel CTD kinase.
3 s distinct from its ability to function as a CTD kinase.
4 ciency similar to Tax, also failed to bind a CTD kinase.
5 DK13 has been demonstrated to be a bona fide CTD kinase.
6 implicating it as the cyclin subunit of this CTD kinase.
7 recruits poised RNAPII PIC lacking the Kin28 CTD kinase.
8  a complex regulatory network governs Pol II CTD kinases.
9 ation by a relay of transcriptionally active CTD kinases.
10 se genes encode an Srb/mediator component, a CTD kinase, a CTD phosphatase, and a protein involved in
11           The Tat-, E1A- and VP16-associated CTD kinase activities detected in our assay also appear
12 rential requirement of Rad3 DNA helicase and CTD kinase activities in damage-specific incision versus
13                                          The CTD kinase activities that interact with E1A and VP16 ar
14  the contributions of TFIIH DNA helicase and CTD kinase activities to efficient promoter escape by RN
15                An inhibitor of CDK9 and CDK7 CTD kinase activities, TAF7 also binds to BRD4 and inhib
16  resulting in the inhibition of their Pol II CTD kinase activities.
17 iption through its well-described RNA Pol II CTD kinase activity and also through the Cdc2-activating
18         We show that CDK8 is associated with CTD kinase activity and that CDK8 co-fractionates with E
19 are sufficient for Mediator to enhance Kin28 CTD kinase activity and that Mediator enhances phosphory
20 rotein, Rsp5, is capable of stimulating this CTD kinase activity as well.
21                           Regulation of CDK7-CTD kinase activity by p16(INK4A) thus may represent an
22 d suggest a mechanism for the enhancement of CTD kinase activity by the Mediator complex.
23 cell lysate demonstrates that Tat-associated CTD kinase activity elutes in two peaks.
24  indicates that the E1A- and VP16-associated CTD kinase activity has a molecular size of approximatel
25 DK13 purified from nuclear extracts displays CTD kinase activity in vitro, as anticipated.
26 d VP16 specifically interact with a cellular CTD kinase activity in vitro.
27 K12 purified from nuclear extracts manifests CTD kinase activity in vitro.
28                                          The CTD kinase activity is induced upon mitogenic stimulatio
29      This association greatly stimulates the CTD kinase activity of general transcription factor TFII
30 and that this phosphorylation stimulates the CTD kinase activity of Kin28p.
31 g kinase in TFIIH was unable to activate the CTD kinase activity of P-TEFb.
32  stimulated PBLs results in induction of the CTD kinase activity of the cyclin T1/CDK9 complex, which
33 ated to MO15, the catalytic component of the CTD kinase activity of the general transcription factor
34 ion in vivo, and recombinant Cak1 stimulates CTD kinase activity of the purified Bur1-Bur2 complex in
35 o-fractionates with E1A- and VP16-associated CTD kinase activity over several chromatography columns.
36 V irradiation or actinomycin D, which induce CTD kinase activity, and that UV inhibition can be rescu
37 abundance, which represses or activates CDK9 CTD kinase activity, respectively.
38 e levels of cyclin T1 protein and associated CTD kinase activity, suggests that the cyclin T1/CDK9 pa
39 cterized that Tat associated with a cellular CTD kinase activity, whereas Tax did not.
40 th of these cyclin C complexes retain potent CTD kinase activity.
41 responsible for the E1A- and VP16-associated CTD kinase activity.
42 sely, CDK9 phosphorylates BRD4 enhancing its CTD kinase activity.
43  of a high molecular weight complex that has CTD kinase activity.
44 y of TFIIH to activate transcription and its CTD kinase activity.
45  kinase activity but also by modulating CDK7-CTD kinase activity.
46 k1p), is not a component of TFIIH, and lacks CTD kinase activity.
47 XPD subunit, and a carboxyl-terminal domain (CTD) kinase activity catalyzed by its CDK7 subunit.
48  its RNA polymerase II COOH-terminal domain (CTD)-kinase activity, resulted in preferential inhibitio
49  yeast Cak1 kinase, in order to uncouple the CTD kinase and CAK activities of Mcs6, revealed an unant
50           Cyclin K is associated with potent CTD kinase and Cdk kinase (CAK) activity in vitro and co
51 ults identify Bur1 as a fourth S. cerevisiae CTD kinase and provide striking functional similarities
52 laries between activators with(out) a linked CTD kinase and regulated transcription by RNA polymerase
53 between viral transactivators and a cellular CTD kinase and suggests that at least two CTD kinases ma
54  purified calf thymus RNAP IIA with specific CTD kinases and used as substrates for FCP1.
55 with both CDK7 and CDK9 (putative RNA pol II CTD kinases) and that CDKN1C blocks their ability to pho
56 role for metazoan CDK7 as a broadly required CTD kinase, and as a CAK essential for cell cycle progre
57 ymerase II transcription machinery by MAPKs, CTD kinases, and phosphatases constitutes an essential m
58 emonstrate that metazoan CDK12 and CDK13 are CTD kinases, and that CDK12 is orthologous to yeast Ctk1
59  of the CTD; affect the Kin28, Bur1, or Ctk1 CTD kinases; and affect the CTD phosphatase Fcp1.
60 tro and in vivo under conditions where other CTD kinases are inactive.
61 olymerase (Pol) II carboxyl-terminal domain (CTD) kinase associated with transcription factor (TF) II
62  activity results in Kin28 being the primary CTD kinase at initiation.
63 al orthologs of the Saccharomyces cerevisiae CTD kinase Bur1/Bur2, a putative yeast P-TEFb.
64          We conclude that the nuclear RNAPII-CTD kinase CDK12 cooperates with mTORC1, and controls a
65 d Set2, but in a manner stimulated by Pol II CTD kinase Cdk7/Kin28.
66                         We report that three CTD kinases, CDK7, CDK9, and BRD4, engage in cross-talk,
67  Ser(2), Thr(4) phosphorylation requires the CTD kinase CDK9 and is evolutionarily conserved from yea
68  by a dominant-negative mutant of the pol II CTD kinase, CDK9, and by low concentrations of the CDK9
69 n T1, a regulatory subunit of the TAK/P-TEFb CTD kinase complex.
70 lymerase II carboxyl terminal domain (RNAPII CTD) kinase complex (CTK complex) is known as a positive
71 at the ability of p16(INK4A) to inhibit CDK7-CTD kinase contributes to the capacity to induce cell cy
72                  About 60% of Tat-associated CTD kinase copurifies with CDK2 kinase activity and cont
73  among Nrd1p, the pol II CTD, Nab3p, and the CTD kinase CTDK-I.
74 eletion of the RNA pol II C-terminal domain (CTD) kinase Ctk1, or partial deletion of the CTD, result
75 sidual Ser2P in cells lacking the major Ser2 CTD kinase, CTK1.
76 s but does not require the RNA polymerase II CTD kinase Ctk1p.
77 NA polymerase II (Pol II) C-terminal domain (CTD) kinases cyclin-dependent kinase 7 (CDK7) and CDK9 a
78 e studied the properties of a Tat-associated CTD kinase derived from mitogenically stimulated human p
79 w that Ctk1, the serine 2 C-terminal domain (CTD) kinase for RNA polymerase II (RNAP II), regulates H
80                           In contrast, TFIIH CTD kinase has a pronounced preference for RNAPII incorp
81 ition, we show that O-GlcNAc transferase and CTD kinase have different CTD repeat requirements for en
82                      Two primary S2 position CTD kinases have been identified in higher eukaryotes: P
83 ion, we have analyzed the ability of a known CTD kinase, human Cdk8, to modulate HIV-1 LTR-driven gen
84 etic screen for suppressors of loss of yeast CTD kinase I (CTDK-I) function (by deletion of the catal
85                                              CTD kinase I (CTDK-I) of Saccharomyces cerevisiae is req
86 red the relationship between Srb10-Srb11 and CTD kinase I (CTDK-I), another member of the cdk-cyclin
87 ain (CTD) affinity column created with yeast CTD kinase I and the CTD of RNA polymerase II was used t
88               These results demonstrate that CTD kinase I modulates the elongation efficiency of RNA
89  templates, we examined the effects of yeast CTD kinase I or CTD kinase inhibitors on transcription a
90 itors, whereas both are greatly increased by CTD kinase I; in contrast, transcription initiation is m
91                       The C-terminal domain (CTD) kinase I (CTDK-1) complex is the primary RNA Polyme
92 onal specificity of phosphorylation by yeast CTD kinase-I (CTDK-I), an enzyme implicated in various n
93                   More broadly, our atlas of CTD kinases implicates Pol II as a direct regulatory end
94 ons demonstrate that c-Abl can function as a CTD kinase in vitro as well as in vivo.
95 thermore, direct competition between OGT and CTD kinase in vivo could generate multiple functionally
96 ily phosphorylated in front of the lesion by CTD kinases in the presence of ATP.
97 sactivators for the ability to interact with CTD kinases in vitro.
98 r elongation-phase C-terminal repeat domain (CTD) kinase in Saccharomyces cerevisiae, CTDK-I.
99 sphorylate the Rpb1 carboxy-terminal domain (CTD) kinase in vitro, it has no strong specificity withi
100 1-beta-d-ribofuranosylbenzimidazole (DRB), a CTD kinase inhibitor.
101 ion initiation complex and is blocked by the CTD-kinase inhibitor H8.
102 xamined the effects of yeast CTD kinase I or CTD kinase inhibitors on transcription and CTD phosphory
103                                              CTD kinase inhibitors, however, have little effect on in
104 NA polymerase II and treatment of cells with CTD kinase inhibitors, including DRB (5,6-dichloro-1-bet
105                                     However, CTD kinase inhibitors, such as 5,6-dichloro-1-beta-D-rib
106               In cultured cells treated with CTD kinase inhibitors, the dephosphorylation of RNAPII o
107 d CTD phosphorylation are greatly reduced by CTD kinase inhibitors, whereas both are greatly increase
108                          In yeast, the major CTD kinase is a subunit of the general transcription fac
109 VP16, suggesting that the interaction with a CTD kinase is relevant for the transactivation function
110 s of the pol II C-terminal domain (CTD), the CTD kinase Kin28 and the holoenzyme subunit Srb2 all inh
111         Therefore, only the TFIIH-associated CTD kinase Kin28 appears necessary for proper capping en
112  (6-AU) and inactivation of pol II, TFIIE or CTD kinases Kin28 and Ctk1, this mark shifted to the 3'
113 ns between ESS1 and genes encoding the known CTD kinases (KIN28, CTK1, BUR1, and SRB10).
114                                          The CTD kinase, Kin28, is required for binding, and the CTD
115                 In contrast, DYRK1A, another CTD kinase known to control transcription of a subset of
116 ar CTD kinase and suggests that at least two CTD kinases may mediate responses to viral transactivato
117 sCTD extracted from cells mutated in several CTD kinases or phosphatases showed the expected changes
118 ex (SEC) with the RNA polymerase II (Pol II) CTD kinase P-TEFb.
119 s with cdk9 and cyclin T1, components of the CTD kinase P-TEFb.
120 shed by differential recruitment of the Ser2 CTD kinase, P-TEFb.
121                    We sought to identify the CTD kinase responsible for capping enzyme targeting.
122  snRNA promoters recruit factors including a CTD kinase(s) whose snRNA-specific phosphorylation patte
123  not stimulate the activity of several other CTD kinases, suggesting that the specific enhancement of
124      Previously, we reported that a cellular CTD kinase, TAK, is targeted by the human immunodeficien
125 ished by inactivation of Kin28, the serine 5 CTD kinase that promotes the transition from initiation
126 CDK12 (CG7597) is a transcription-associated CTD kinase, the ortholog of yCtk1.
127 on factor-b) (CycT1:CDK9) C-terminal domain (CTD) kinase to the HIV-1 promoter.
128  by recruitment of carboxyl-terminal domain (CTD) kinases to the HIV-1 promoter.
129                                              CTD kinases with different phosphoryl acceptor specifici
130    Furthermore, recruitment of FACT requires CTD kinases, yet FACT is dispensable for p21(CIP1) expre

 
Page Top