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1 nt cytoplasmic RNA that is conserved across Caenorhabditis and possesses three essential secondary s
5 ht C. elegans (Orsay virus) and its relative Caenorhabditis briggsae (Santeuil virus, Le Blanc virus,
6 and Santeuil, which both specifically infect Caenorhabditis briggsae, were also found to form fibrous
10 we report that mitochondrial dysfunction in Caenorhabditis elegans activates RNAi-directed silencing
14 s involved in glial size regulation, we used Caenorhabditis elegans amphid sheath (AMsh) glia as a mo
16 of the dosage compensation complex (DCC) in Caenorhabditis elegans and demonstrate that loop anchors
17 e mechanisms underlying UPR(mt) signaling in Caenorhabditis elegans and discuss emerging connections
18 as leveraged simple model organisms, such as Caenorhabditis elegans and Drosophila melanogaster larva
19 d aging, including stem cell dysfunction, in Caenorhabditis elegans and Drosophila melanogaster model
21 ulfide reduction exclusively in endosomes in Caenorhabditis elegans and identified that exchange is m
22 e paralysis, we conducted genetic screens in Caenorhabditis elegans and isolated mutants that became
25 fication of Abeta oligomers in vitro, and in Caenorhabditis elegans and mouse hippocampal tissues.
26 mporal regulation of LAG-1/CSL expression in Caenorhabditis elegans and observed that an increase in
28 ptide F (NPF)-related neuropeptide system in Caenorhabditis elegans and show that this FLP-34/NPR-11
29 vensis is a natural pathogen of the nematode Caenorhabditis elegans and that parental exposure of ani
30 t method relying on the free-living nematode Caenorhabditis elegans and the infection of those nemato
31 mental inhibitor of the free-living nematode Caenorhabditis elegans and the plant-parasitic nematode
32 on of a long-term swim exercise protocol for Caenorhabditis elegans and we demonstrate its benefits t
34 couple these key events of the cell cycle in Caenorhabditis elegans and zebrafish through live-cell i
36 rpose, we have designed a pipeline that uses Caenorhabditis elegans as a genetic model to screen for
39 ral, biochemical, and functional analyses in Caenorhabditis elegans As capsaicin elicits heat and pai
43 ogical mechanisms that delay neuron aging in Caenorhabditis elegans by asking how neuron morphologica
44 We developed a glutamate spillover model in Caenorhabditis elegans by inactivating the conserved gli
46 ibed a physiological program in the nematode Caenorhabditis elegans called the intracellular pathogen
47 how that bloating of the intestinal lumen of Caenorhabditis elegans caused by microbial colonization
48 fied a gain-of-function (gf) mutation in the Caenorhabditis elegans CaV2 channel alpha1 subunit, UNC-
49 oteins and an unexpected direct link between Caenorhabditis elegans CFP-1 and an Rpd3/Sin3 small (SIN
54 This subunit, dSol-1, is the homolog of the Caenorhabditis elegans CUB (Complement C1r/C1s, Uegf, Bm
55 e atomic cryo-EM structures of a full-length Caenorhabditis elegans cyclic GMP-activated channel TAX-
56 actors implicated in translation fidelity in Caenorhabditis elegans decreases lifespan, and eEF2K is
64 nvestigated cytokinesis during the invariant Caenorhabditis elegans embryonic divisions and found sev
65 ression levels of APA isoforms in individual Caenorhabditis elegans embryos at different stages throu
66 robe and control the cell-division timing in Caenorhabditis elegans embryos using a combination of lo
69 report that the noncanonical sHsp HSP-17 of Caenorhabditis elegans facilitates aggregation of model
71 ng, we performed a forward genetic screen in Caenorhabditis elegans for genes involved in body size r
72 on developmental gene expression dataset for Caenorhabditis elegans from Bao, Murray, Waterston et al
73 lar range cysteamine bitartrate treatment in Caenorhabditis elegans gas-1(fc21) RC complex I (NDUFS2-
75 H Oligopaint library that targets the entire Caenorhabditis elegans genome at chromosome, three megab
76 onserved homeodomain proteins encoded by the Caenorhabditis elegans genome(2), that the complete set
78 ins FBF-1 and FBF-2 have opposite effects on Caenorhabditis elegans germline stem cell dynamics: FBF-
80 e-like glia, which extended the life span in Caenorhabditis elegans Glial XBP-1s initiated a robust c
83 rain to induce CA production directly in the Caenorhabditis elegans gut, we reveal the local effect o
87 sed by all neurons in model systems, such as Caenorhabditis elegans have left still unresolved how ne
88 Hawaiian isolates of the nematode species Caenorhabditis elegans have long been known to harbor ge
89 netic studies on the excretory canal cell of Caenorhabditis elegans have revealed many proteins that
91 TFEB and TFE3, as well as their ortholog in Caenorhabditis elegans HLH-30, play an important role in
92 nd genetic analysis, we found that catp-6, a Caenorhabditis elegans homolog of ATP13A2, was responsib
94 ere, we study the effect of different crh-1 [Caenorhabditis elegans homolog of mammalian cAMP respons
97 mechanical properties associated with ageing Caenorhabditis elegans in addition to capturing high-res
98 successfully reduced the fungal burdens in a Caenorhabditis elegans infection model by up to 96%.
100 utative FAM173B orthologue from the nematode Caenorhabditis elegans Interestingly, lack of Lys-43 met
111 can modulate the developmental trajectory of Caenorhabditis elegans larvae by promoting entry into da
112 We generate CRISPR knockout strains for Caenorhabditis elegans lincRNAs and evaluate their pheno
113 -shared neurons in the nervous system of the Caenorhabditis elegans male is controlled by the tempora
117 specific isoform of CREB1/CRH-1, CRH-1e, in Caenorhabditis elegans memory formation and chemosensati
118 -helix transcription factor 30 (hlh-30), the Caenorhabditis elegans MiT/TFE ortholog, to starvation f
119 beta-induced paralysis in a transgenic Abeta Caenorhabditis elegans model and specifically target and
120 To this end, we have generated a transgenic Caenorhabditis elegans model expressing both human Abeta
122 ogical tau protein, we employed a transgenic Caenorhabditis elegans model of human tauopathy exhibiti
124 re, we develop an experimental handle in the Caenorhabditis elegans model system, in which we uncover
125 MP resistance show enhanced virulence in the Caenorhabditis elegans model, an effect that is abolishe
129 pmental expression of three conserved genes, Caenorhabditis elegans mpk-1, lag-1, and lag-3/sel-8, wh
130 e diseases, and we show that the lack of the Caenorhabditis elegans Msp1 homologue triggers an import
131 hondria are arrayed in a periodic pattern in Caenorhabditis elegans muscle, but this pattern is disru
136 to visualize alternative splicing in single Caenorhabditis elegans neurons, identifying complex spli
139 we discovered a novel coordinated action of Caenorhabditis elegans neuropeptide Y/neuropeptide F and
141 Here we report our investigations on three Caenorhabditis elegans orthologous proteins involved in
143 subunits partially rescue sensory defects in Caenorhabditis elegans osm-9 and tax-4 knock-out strains
144 of Alzheimer's disease in the model organism Caenorhabditis elegans Our results pose and answer the q
145 e we describe the emerging model system of a Caenorhabditis elegans polymodal neuron named PVD, whose
148 of integrin-mediated collagen recruitment to Caenorhabditis elegans postembryonic gonadal and pharyng
152 re, we report the identification of PLP-1, a Caenorhabditis elegans protein related to the human sing
155 dbt-1 gene underlies natural differences in Caenorhabditis elegans responses to the toxin arsenic.
156 The expression of chimeric hnRNPA2 D290V in Caenorhabditis elegans results in stress-induced glutama
157 R-generated, fluorescently tagged endogenous Caenorhabditis elegans RHEB-1 and DAF-15/Raptor are expr
159 colleagues provide evidence that exposure of Caenorhabditis elegans roundworms to 2 simple nutrients,
161 repeated induction of the flight response in Caenorhabditis elegans shortens lifespan and inhibits co
162 gene expression analyses in P. univalens and Caenorhabditis elegans show that the intestine is the ma
166 to a brief 2 degrees C temperature increase, Caenorhabditis elegans spermatocytes exhibit up to a 25-
167 bservations were recapitulated in vivo, in a Caenorhabditis elegans strain deficient in the ATP13A2 o
169 e for the whole-life culture of the nematode Caenorhabditis elegans that allows the scoring of animal
170 identify a neuronal circuit in the nematode Caenorhabditis elegans that processes information percei
171 he model agrees with experiments in the worm Caenorhabditis elegans that show the following: Life spa
172 which male-specific neurons are generated in Caenorhabditis elegans through the direct transdifferent
177 come well established that the nematode worm Caenorhabditis elegans triggers innate immune responses
178 s controlled by Munc18 (mammalian homolog of Caenorhabditis elegans uncoordinated gene 18) proteins.
181 regulates its trafficking and degradation in Caenorhabditis elegans We found that the WD40-repeat pro
182 multipotent vulval precursor cells (VPCs) of Caenorhabditis elegans We have previously shown that thi
183 ned by the number of traps after exposure to Caenorhabditis elegans While some strains were highly se
190 EB) transcription factor significantly slows Caenorhabditis elegans' reproductive decline, an early h
192 on in HD models (cells, primary neurons, and Caenorhabditis elegans) increases mutant HTT exon 1 phos
193 dopsis thaliana), rice (Oryza sativa), worm (Caenorhabditis elegans), and human (Homo sapiens) cells
196 ere we develop tests for IIA in the nematode Caenorhabditis elegans, an animal with only 302 neurons,
199 bolism without affecting eating behaviors in Caenorhabditis elegans, and identified specific odors th
201 e, whereas Smn from Drosophila melanogaster, Caenorhabditis elegans, and Schizosaccharomyces pombe wa
202 of a related nonparasitic nematode species, Caenorhabditis elegans, and the parasitic nematode Haemo
203 europeptide Y/neuropeptide F in the nematode Caenorhabditis elegans, and we discovered that it is req
206 tant proteins remain enriched at synapses in Caenorhabditis elegans, but show defects in active-zone
207 nd prodigiosin antibiotics, and infection in Caenorhabditis elegans, but up-regulating flagellar moti
208 herefore, simple organisms like the nematode Caenorhabditis elegans, combining relevant advantages of
211 is regard, we summarize recent studies using Caenorhabditis elegans, Drosophila melanogaster, Danio r
212 ets across species inclusive of Arabidopsis, Caenorhabditis elegans, Drosophila, human, mouse, rat, y
213 erotonin-dependent behavior of the roundworm Caenorhabditis elegans, egg laying, to perform a behavio
214 ically infects the laboratory model nematode Caenorhabditis elegans, encodes a fibrous protein delta
215 hat govern the formation of gap junctions in Caenorhabditis elegans, five of which are already suppor
216 zing proteome-wide mass spectrometry data of Caenorhabditis elegans, however, we show that the levels
217 cells as well as Drosophila melanogaster and Caenorhabditis elegans, indicating suitability for use i
229 velopment in a subpopulation of synchronized Caenorhabditis elegans, sets processes in motion that in
237 ntrations in most studied species, including Caenorhabditis elegans, the molecular mechanisms behind
244 lls, a population of epidermal stem cells in Caenorhabditis elegans, to study the influence of temper
245 usly shown that 2 paralogous tetraspanins in Caenorhabditis elegans, TSP-12 and TSP-14, function redu
248 Using the touch receptor neurons (TRNs) of Caenorhabditis elegans, we find that mec-15(-) mutants d
249 ructions of spermatocyte meiotic spindles in Caenorhabditis elegans, we find the lagging X chromosome
250 ors of heat-shock-induced gene expression in Caenorhabditis elegans, we found a new allele of hsf-1 t
253 n coupled with deep-sequencing (TRAP-seq) in Caenorhabditis elegans, we have obtained high coverage p
254 encing and genomic and genetic approaches in Caenorhabditis elegans, we reveal an unprecedented role
256 and siRNAs in regulating gene expression in Caenorhabditis elegans, we subjected small RNAs and mRNA
258 increased in young amyloid B (AB) expressing Caenorhabditis elegans, whereas it is not in wild type s
259 odels zebrafish, Drosophila melanogaster and Caenorhabditis elegans, which command superb research re
292 imized to measure activity and quiescence in Caenorhabditis larvae and adults and in Drosophila larva
294 omosome in hemizygous (XY) males, whereas in Caenorhabditis nematodes, expression is halved for both
296 logenetic relationships of C. bovis to other Caenorhabditis species and reveal the changes in genome
298 preserving, are more highly conserved across Caenorhabditis species, and are enriched in specific cis