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1 ous risk factors for PB (which also includes Cheyne-Stokes respiration).
2 tive and central sleep apnea associated with Cheyne-Stokes respiration.
3 normal left ventricular function, and severe Cheyne-Stokes respiration.
4 d and effective therapy for life-threatening Cheyne-Stokes respiration.
5 per hour (serum level, 5.6 microg/mL), both Cheyne-Stokes respiration and oxygen desaturation were m
7 pnea and central sleep apnea associated with Cheyne-Stokes respiration as potentially modifiable risk
9 y rises and falls in ventilation with apnea (Cheyne-Stokes respiration [CSR]) or without apnea (perio
14 ography revealed that 12 of 12 patients with Cheyne-Stokes respiration experienced increases in end-e
16 index, >/=5 vs. <5), central sleep apnea or Cheyne-Stokes respiration, obstructive apnea-hypopnea in
21 rkably irregular versus classic instability (Cheyne-Stokes respiration), suggesting our mechanistic u
22 entral apnea (OR, 9.97; 95% CI, 2.72-36.50), Cheyne-Stokes respiration with central apnea (OR, 6.31;
23 95% confidence interval [CI], 1.18-5.66) and Cheyne-Stokes respiration with central sleep apnea (OR,