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1 1 and MprF2) found in the bacterial pathogen Clostridium perfringens.
2 oss firmicutes, including the human pathogen Clostridium perfringens.
3  Escherichia coli, Bacteroides fragilis, and Clostridium perfringens.
4 ccus aureus, Clostridium acetobutylicum, and Clostridium perfringens.
5 tory activity for a bacterial NEU, NanI from Clostridium perfringens.
6 typical cholesterol-dependent cytolysin from Clostridium perfringens.
7 e(s) have been validated for this purpose in Clostridium perfringens.
8  of Clostridium botulinum, and iota toxin of Clostridium perfringens.
9 of Clostridium septicum and epsilon-toxin of Clostridium perfringens.
10  AlgR of Pseudomonas aeruginosa, and VirR of Clostridium perfringens.
11 on toxin secreted by the anaerobic bacterium Clostridium perfringens.
12 nization of specific-pathogen-free chicks by Clostridium perfringens.
13 cherichia coli, 50% GC; calf thymus, 42% GC; Clostridium perfringens, 27% GC) have been employed as t
14 ism isolated from dogs, cats, and horses was Clostridium perfringens (75, 13, and101 isolates, respec
15                The presence and abundance of Clostridium perfringens (8.4%) and Bacteroides dorei (0.
16                                              Clostridium perfringens, a human pathogen, is one of the
17  cross-react with a homologous sequence of a Clostridium perfringens adenosine triphosphate-binding c
18 idues with 90% homology to a sequence within Clostridium perfringens adenosine triphosphate-binding c
19 be immediate and direct, as in the action of Clostridium perfringens alpha toxin on red cells or plat
20 inescence assay for identifying and assaying Clostridium perfringens alpha toxin.
21     Based on comparisons with two orthologs, Clostridium perfringens alpha-toxin and Bacillus cereus
22 oximately 2 nM) of epsilon toxin produced by Clostridium perfringens and a prominent food toxin.
23 caused by the ubiquitous, anaerobic bacteria Clostridium perfringens and Clostridium septicum.
24 kholderia mallei, Burkholderia pseudomallei, Clostridium perfringens and Entamoeba histolytica.
25 caused by gas- forming bacteria - most often Clostridium perfringens and Escherichia coli.
26     NetB is a pore-forming toxin produced by Clostridium perfringens and has been reported to play a
27 obacterium hafniense, Clostridium novyi, and Clostridium perfringens and increase their activity up t
28 ization and replication of bacteria, such as Clostridium perfringens and Salmonella enterica serovar
29 for all corresponding genes of 26 strains of Clostridium perfringens and Streptococcus pneumoniae.
30 erences between the germination of spores of Clostridium perfringens and that of spores of a number o
31 H gene, encoding a hyaluronidase secreted by Clostridium perfringens, and a C. perfringens hyaluronid
32 lfitobacterium hafniense, Clostridium novyi, Clostridium perfringens, and Eggerthella lenta.
33  with enterobacteria, desulfovibrios, type E Clostridium perfringens, and Enterococcus faecalis, wher
34 ric AIPs from Lactiplantibacillus plantarum, Clostridium perfringens, and Listeria monocytogenes that
35 odborne outbreaks caused by Bacillus cereus, Clostridium perfringens, and Staphylococcus aureus were
36  bacterial IMPDHs from Campylobacter jejuni, Clostridium perfringens, and Vibrio cholerae.
37 e anaerobic sulfatase-maturating enzyme from Clostridium perfringens (anSMEcpe) catalyzes the two-ele
38                  These anSME structures from Clostridium perfringens are also the first of an AdoMet
39 s of the common human and livestock pathogen Clostridium perfringens are attributable to a formidable
40 gglutinin upon digestion with sialidase from Clostridium perfringens, Arthrobacter neurofaciens, or S
41 olysin perfringolysin O (PFO) is secreted by Clostridium perfringens as a bacterial virulence factor
42 o-beta-galactosidase designated E-ABase from Clostridium perfringens ATCC 10543 capable of liberating
43 mercially available sialidases prepared from Clostridium perfringens ATCC10543 were contaminated with
44                                    show that Clostridium perfringens beta-toxin (CPB) binds platelet
45                                              Clostridium perfringens biotype A strains are the causat
46                                              Clostridium perfringens BSH/T rapidly performed acyl tra
47 h potent activity against the human pathogen Clostridium perfringens By combining in vivo and in vitr
48                                              Clostridium perfringens can obtain sialic acid from host
49                                              Clostridium perfringens can produce up to three differen
50 ubnetworks associated with responses against Clostridium perfringens, Candida albicans, and Bacteroid
51 -beta-galactosidase (Endo-beta-Gal(GnGa)) in Clostridium perfringens capable of releasing GlcNAcalpha
52                         By producing toxins, Clostridium perfringens causes devastating diseases of b
53                                              Clostridium perfringens causes gas gangrene and gastroin
54                                The bacterium Clostridium perfringens causes severe, sometimes lethal
55 rtex by cortex-lytic enzymes (CLEs), and two Clostridium perfringens CLEs, SleC and SleM, degrade cor
56 tridia in Cluster I, including the pathogens Clostridium perfringens, Clostridium botulinum and Clost
57 y Bacillus anthracis, Staphylococcus aureus, Clostridium perfringens, Clostridium botulinum, and Clos
58 tigated a putative cell-surface adhesin from Clostridium perfringens comprising an N-terminal adhesin
59 sed method developed to specifically measure Clostridium perfringens concentrations.
60 P = .1 and P = .01 for consecutive samples); Clostridium perfringens continued to be more prevalent i
61 against pathogenic bacterial sialidases from Clostridium perfringens (CpNanI) and Vibrio cholerae.
62 c properties against human pathogens such as Clostridium perfringens, define two hairpin domains givi
63 te pretreatment with sialidase purified from Clostridium perfringens did (P < 0.05).
64                   The genome of the pathogen Clostridium perfringens encodes two proteins, GerO and G
65 y cpb2, is implicated in the pathogenesis of Clostridium perfringens enteritis.
66                                  Domain I of Clostridium perfringens enterotoxin (cCPE) binds to the
67        A noncytotoxic C-terminal fragment of Clostridium perfringens enterotoxin (cCPE) is a natural
68                                              Clostridium perfringens enterotoxin (CPE) action starts
69 ious epidemiological studies have implicated Clostridium perfringens enterotoxin (CPE) as a virulence
70                                              Clostridium perfringens enterotoxin (CPE) causes food po
71                                              Clostridium perfringens enterotoxin (CPE) causes the gas
72                                              Clostridium perfringens enterotoxin (CPE) causes the sym
73                                              Clostridium perfringens enterotoxin (CPE) has a unique m
74                                              Clostridium perfringens enterotoxin (CPE) has recently b
75                                              Clostridium perfringens enterotoxin (CPE) induces cytoly
76                               After binding, Clostridium perfringens enterotoxin (CPE) initially loca
77                                              Clostridium perfringens enterotoxin (CPE) is a major cau
78                                              Clostridium perfringens enterotoxin (CPE) is a pore-form
79                                              Clostridium perfringens enterotoxin (CPE) is a pore-form
80                                              Clostridium perfringens enterotoxin (CPE) is responsible
81                                              Clostridium perfringens enterotoxin (CPE) is responsible
82                                              Clostridium perfringens enterotoxin (CPE) is the etiolog
83 d the potential superantigenic properties of Clostridium perfringens enterotoxin (CPE) on human perip
84         The previous model for the action of Clostridium perfringens enterotoxin (CPE) proposed that
85 ost in vitro studies exploring the action of Clostridium perfringens enterotoxin (CPE) utilize either
86 ies performed to investigate the topology of Clostridium perfringens enterotoxin (CPE) when this toxi
87                                              Clostridium perfringens enterotoxin (CPE), a 35-kDa poly
88  relationship and mechanism of action of the Clostridium perfringens enterotoxin (CPE), a series of r
89                                              Clostridium perfringens enterotoxin (CPE), a single poly
90 -affinity intestinal epithelial receptor for Clostridium perfringens enterotoxin (CPE), and is suffic
91 y receptors, respectively, for the cytotoxic Clostridium perfringens enterotoxin (CPE), in this study
92                                              Clostridium perfringens enterotoxin (CPE), the virulence
93 , claudin-4 and -3 serve as the receptor for Clostridium perfringens enterotoxin (Cpe).
94  claudins (e.g., Claudin-4) as receptors for Clostridium perfringens enterotoxin (CPE).
95                                              Clostridium perfringens enterotoxin causes the gastroint
96                                              Clostridium perfringens enterotoxin is a common cause of
97 the localization of the genes for the CPE-R (Clostridium perfringens enterotoxin receptor, CPETR1) an
98                                         CPE (Clostridium perfringens enterotoxin) is the major virule
99  for the sporulation-associated synthesis of Clostridium perfringens enterotoxin, a common cause of f
100 xins, such as botulinum neurotoxins (BoNTs), Clostridium perfringens epsilon toxin (ETX), staphylococ
101                                              Clostridium perfringens epsilon toxin belongs to the aer
102 the motion of single membrane receptors, the Clostridium perfringens epsilon-toxin (CPepsilonT) recep
103                                          The Clostridium perfringens epsilon-toxin causes a severe, o
104                                          The Clostridium perfringens epsilon-toxin is responsible for
105 d (Staphylococcus aureus, Bacillus subtilis, Clostridium perfringens, Escherichia coli), except Enter
106 ntestinal inhabitants: Bacteroides fragilis, Clostridium perfringens, Escherichia coli, Klebsiella pn
107 ia (FIBs; Escherichia coli, enterococci, and Clostridium perfringens) exhibited biphasic decay patter
108              To examine the early effects of Clostridium perfringens exotoxins on tissue perfusion, a
109                                              Clostridium perfringens food poisoning is caused by type
110 me that the anaerobic gram-positive pathogen Clostridium perfringens forms biofilms.
111                                              Clostridium perfringens gas gangrene is a fulminant infe
112                                              Clostridium perfringens gas gangrene is characterized by
113                                              Clostridium perfringens gas gangrene is characterized by
114 t the site of infection are two hallmarks of Clostridium perfringens gas gangrene.
115                                      Several Clostridium perfringens genotype E isolates, all associa
116                       A published complex of Clostridium perfringens GH125 enzyme with a nonhydrolyza
117      PFO, a virulence factor of the organism Clostridium perfringens, has almost the same molecular m
118 ly, the genome sequences of three strains of Clostridium perfringens have been completed and we ident
119 acillus anthracis, Campylobacter jejuni, and Clostridium perfringens IMPDHs.
120 robiology methods are not suitable to detect Clostridium perfringens in formalin-fixed, paraffin-embe
121  as a competitive exclusion agent to control Clostridium perfringens in poultry.
122 characterized by Anaerobiospirillum loss and Clostridium perfringens increases.
123            Necrotic enteritis (NE) caused by Clostridium perfringens infection has reemerged as a pre
124                                              Clostridium perfringens iota-toxin consists of two separ
125                                              Clostridium perfringens iota-toxin is a binary toxin con
126                                              Clostridium perfringens is a common cause of food-borne
127                                              Clostridium perfringens is a Gram-positive anaerobic pat
128                                              Clostridium perfringens is a Gram-positive, anaerobic sp
129                                              Clostridium perfringens is a leading cause of food-poiso
130                                              Clostridium perfringens is a ubiquitous and versatile pa
131                                              Clostridium perfringens is an anaerobic Gram-positive pa
132                                              Clostridium perfringens is an anaerobic toxin-producing
133                                              Clostridium perfringens is an anaerobic, Gram-positive b
134                                              Clostridium perfringens is an extracellular pathogen whi
135                                              Clostridium perfringens is an important foodborne pathog
136                                              Clostridium perfringens is an important human and animal
137                                              Clostridium perfringens is capable of producing up to 15
138 turnoff assay for phospholipase C (PLC) from Clostridium perfringens is developed based on the revers
139                  A neuraminidase enzyme from Clostridium perfringens is non-covalently immobilized in
140            Necrotic enteritis (NE) caused by Clostridium perfringens is one of the most detrimental i
141                                     Although Clostridium perfringens is recognized as an important ca
142                                              Clostridium perfringens is the cause of several human di
143                                              Clostridium perfringens is the most common cause of clos
144                                              Clostridium perfringens is the second leading cause of b
145                                              Clostridium perfringens is the third most frequent cause
146  potent pore forming toxin (PFT) produced by Clostridium perfringens, is responsible for the pathogen
147 logical studies suggested that cpb2-positive Clostridium perfringens isolates are associated with gas
148 s such as norovirus and toxigenic strains of Clostridium perfringens, Klebsiella oxytoca, Staphylococ
149 his model is challenged by the glycosylating Clostridium perfringens large cytotoxin (TpeL toxin) tha
150 and in vitro activity on human substrates of Clostridium perfringens NagJ, a close homologue of human
151       A polyclonal antibody prepared against Clostridium perfringens neuraminidase 1) detected surfac
152 ysis, we were able to identify inhibitors of Clostridium perfringens neuraminidase present in a root
153             C. difficile was codetected with Clostridium perfringens, norovirus, sapovirus, parechovi
154 omic sequences, but only in three bacterial (Clostridium perfringens, Oenococcus oeni, and Leuconosto
155 e monomeric cytolysin secreted by pathogenic Clostridium perfringens, oligomerizes and forms large po
156 ontrast, perfringolysin O (PFO), secreted by Clostridium perfringens, only binds to membranes contain
157           Whether expressed in C. difficile, Clostridium perfringens, or Escherichia coli, TxeR stimu
158                                              Clostridium perfringens perfringolysin O (PFO or theta-t
159  S. pyogenes gene that closely resembles the Clostridium perfringens pfoR gene, exerts a negative eff
160                                              Clostridium perfringens phospholipase C (PLC) and perfri
161 ws 60% amino acid sequence identity with the Clostridium perfringens phospholipase C, or alpha-toxin
162 s (G-) and to the transfer protein TcpC from Clostridium perfringens plasmid pCW3 (G+).
163                                    Spores of Clostridium perfringens possess high heat resistance, an
164                                              Clostridium perfringens possesses at least two functiona
165                                   Strains of Clostridium perfringens produce a two-domain enterotoxin
166                   The gram-positive anaerobe Clostridium perfringens produces a large arsenal of toxi
167 ssociated O-acetyl sialic acid--but not from Clostridium perfringens resulted in an increase in RN639
168   Likewise, MazF-cd expression in E. coli or Clostridium perfringens resulted in growth arrest.
169 e crystal structure of ligand-free NanE from Clostridium perfringens reveals a modified triose-phosph
170 pathogenesis, application of the approach to Clostridium perfringens reveals heterogeneous expression
171  reduced disease-associated bacteria such as Clostridium perfringens, Ruminococcus gnavus, and Klebsi
172               The human pathogenic bacterium Clostridium perfringens secretes an enterotoxin (CpE) th
173                                  Infusion of Clostridium perfringens sialidase to the injury site mar
174            The carbonic anhydrase (Cpb) from Clostridium perfringens strain 13, the only carbonic anh
175             The complete genome sequences of Clostridium perfringens strain ATCC 13124, a gas gangren
176                                              Clostridium perfringens strains (type A) isolated from a
177                                         Many Clostridium perfringens strains produce NanI as their ma
178                                              Clostridium perfringens strains produce severe diseases,
179 ess and purify a representative chimera from Clostridium perfringens (termed CperHydR) and apply vari
180 2-aminobenzoic acid using neuraminidase from Clostridium perfringens that cleaves sialic acid monomer
181 peL is a recently identified LCT produced by Clostridium perfringens that has received relatively lim
182                            Pathogens such as Clostridium perfringens, the causative agent of necrotic
183                                              Clostridium perfringens, the most broadly distributed pa
184                                 For example, Clostridium perfringens, the species with the highest va
185  initially attributed to norovirus; however, Clostridium perfringens toxicoinfection was subsequently
186                                              Clostridium perfringens type A food poisoning is the sec
187                                  About 5% of Clostridium perfringens type A isolates carry the cpe ge
188                                              Clostridium perfringens type A isolates carrying an ente
189                                              Clostridium perfringens type A isolates causing food poi
190                                              Clostridium perfringens type A isolates producing entero
191                                              Clostridium perfringens type A strains producing enterot
192                             Alpha toxin from Clostridium perfringens type A, a phospholipase C, has b
193 clostridial glucosylating toxins produced by Clostridium perfringens type A, B, and C strains.
194            Culture confirmed the presence of Clostridium perfringens type A.
195                                              Clostridium perfringens type B and type C isolates, whic
196                                              Clostridium perfringens type B causes enteritis and ente
197            The important veterinary pathogen Clostridium perfringens type B is unique for producing t
198                                              Clostridium perfringens type C isolates cause enteritis
199                                              Clostridium perfringens type C isolates cause enterotoxe
200                                              Clostridium perfringens type C isolates, which cause ent
201                                              Clostridium perfringens type C strains are the only non-
202                               The ability of Clostridium perfringens type C to cause human enteritis
203                  Recombinant beta-toxin from Clostridium perfringens type C was found to increase the
204 usative organism of enteritis necroticans is Clostridium perfringens type C, an anaerobic gram-positi
205                 Previous studies showed that Clostridium perfringens type D animal disease strain CN3
206                                              Clostridium perfringens type D causes disease in sheep,
207                                              Clostridium perfringens type D enterotoxemias have signi
208                                              Clostridium perfringens type D isolates are important in
209                                              Clostridium perfringens type D isolates cause enterotoxe
210                                              Clostridium perfringens type D strains cause enterotoxem
211 silon-toxin, the primary virulence factor of Clostridium perfringens type D, causes mortality in live
212                                              Clostridium perfringens type E isolates produce iota-tox
213 iota toxin, a binary enterotoxin produced by Clostridium perfringens type E, were studied by fluoresc
214 ration-initiated hydrolysis catalyzed by the Clostridium perfringens unsaturated glucuronyl hydrolase
215  and in the pathogenesis of cystic fibrosis; Clostridium perfringens VirR, a regulator of virulence f
216                                              Clostridium perfringens was also isolated by microbiolog
217          An alpha/beta-type SASP, Ssp2, from Clostridium perfringens was expressed at significant lev
218 ogens using virulence factors as indicators; Clostridium perfringens was revealed as a likely pathoge
219 4 mo, counts of Clostridioides difficile and Clostridium perfringens were ~90% (P < 0.001) and ~65% (
220 ignificant increases in genus Sutterella and Clostridium perfringens when compared to healthy dogs.
221                              Here, using the Clostridium perfringens ZmpA, ZmpB, and ZmpC M60 peptida

 
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