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1 suggests a speciation of PV from a C-cluster coxsackie A virus (C-CAV) ancestor through mutation of t
2                                              Coxsackie A virus (CAV) serotypes 1, 11, 13, 15, 17, 18,
3 iruses, including PV type 1 (PV1), PV2, PV3, coxsackie A virus 11 (CAV11), CAV13, CAV17, CAV20, CAV21
4 ces closely related to certain cocirculating coxsackie A virus isolates.
5 p that contains enterovirus 71 and 11 of the coxsackie A viruses.
6          Pairwise comparisons suggested that coxsackie A11 and A15 viruses should be classified as st
7 d as strains of the same serotype, as should coxsackie A13 and A18 viruses.
8 cacious by oral administration in treating a Coxsackie A21 infection in CD-1 mice.
9 en 100 and 500 and occurs independent of the coxsackie Ad receptor.
10 fic adapter, sCARhMFE, composed of sCAR [the coxsackie/Ad receptor (CAR) ectodomain] and MFE-23 (a si
11 arget cells depends upon the presence of the coxsackie adenovirus cell surface receptor, CAR, which i
12                                          The Coxsackie Adenovirus Receptor (CAR) has primarily been s
13 rminal knob of its fiber coat protein to the Coxsackie adenovirus receptor (CAR) protein.
14 ell as binding to native cellular receptors (coxsackie adenovirus receptor (CAR), integrins).
15 tion with antibody for Dynamin 2 but not for Coxsackie adenovirus receptor or for integrin alpha(v).
16 s an integrin binding RGD-4C motif, allowing Coxsackie adenovirus receptor-independent infection of c
17 hange with the loss of ZO-1, Connexin-45 and Coxsackie-adenovirus (CAR) proteins were reduced in atri
18                                          The coxsackie-adenovirus receptor (CAR) is a transmembrane r
19  digestive or respiratory routes require the Coxsackie-adenovirus receptor (CAR) to infect the epithe
20 ing antibodies, widespread expression of the coxsackie-adenovirus receptor (CAR), and adenovirus sequ
21 ile some serotype D adenoviruses bind to the coxsackie-adenovirus receptor (CAR), the ability of Ad30
22  mediate cell attachment in vitro when using coxsackie-adenovirus receptor (CAR)-containing cell line
23 lian cells via interaction of fiber with the coxsackie-adenovirus receptor (CAR).
24 1, and Ad14) that use receptor(s) other than coxsackie-adenovirus receptor and CD46 were able to trig
25 bstacles is the highly variable level of the coxsackie-adenovirus receptor expression on human primar
26 only used oncolytic adenoviruses targeted to coxsackie-adenovirus receptor or CD46.
27 s is expression of the primary receptor, the coxsackie-adenovirus receptor.
28 ressed glycosylphosphatidylinositol-modified coxsackie-adenovirus receptor.
29 rimary receptor for Ad35 (CD46) but not Ad5 (coxsackie-adenovirus receptor; CAR).
30 T cells, transgenic (Tg) mice expressing the coxsackie/adenovirus receptor (CAR) in their T cell comp
31 o adenovirus infection because they lack the Coxsackie/adenovirus receptor (CAR) needed for virus att
32 sCAR-MFE), which fuses the ectodomain of the coxsackie/adenovirus receptor (sCAR) with a single-chain
33 abeled Ad binding and with the expression of coxsackie/adenovirus receptor but not with the expressio
34 n the expression of fiber receptors, such as coxsackie/adenovirus receptor, and alpha(v) integrins on
35 evels because these cells have low levels of Coxsackie and adenovirus receptor (CAR) and alpha(v) int
36                              The presence of coxsackie and adenovirus receptor (CAR) and alpha(v) int
37 -FT285 infection, which is controlled by the Coxsackie and adenovirus receptor (CAR) and the type A o
38                  Recently, we identified the coxsackie and adenovirus receptor (CAR) as the entry rec
39 per, we reported a significant difference in coxsackie and adenovirus receptor (CAR) from several hum
40  the HuNoV entry receptor(s) is unknown, the coxsackie and adenovirus receptor (CAR) has recently bee
41                            Expression of the Coxsackie and Adenovirus Receptor (CAR) is frequently re
42                                          The coxsackie and adenovirus receptor (CAR) is identified as
43                                              Coxsackie and adenovirus receptor (CAR) is not only a hi
44                                              Coxsackie and adenovirus receptor (CAR) is the primary c
45  to investigate the modulatory effect of the coxsackie and adenovirus receptor (CAR) on ventricular c
46                                          The coxsackie and adenovirus receptor (CAR) plays key roles
47                Binding of JAML to its ligand Coxsackie and adenovirus receptor (CAR) provides costimu
48                         One such receptor is Coxsackie and Adenovirus Receptor (CAR) that binds to Ju
49 oteins with functional binding sites for the coxsackie and adenovirus receptor (CAR) were cleared rap
50 ed cells lost expression of the cell surface coxsackie and adenovirus receptor (CAR) within 24 h post
51 oversial, potentially including sialic acid, coxsackie and adenovirus receptor (CAR), integrins, and
52 ith domain I of its human cellular receptor, coxsackie and adenovirus receptor (CAR), is presented he
53 r expression of the primary Ad receptor, the coxsackie and adenovirus receptor (CAR), known to be dow
54 termed globin transcription factor 1 (GATA1)-coxsackie and adenovirus receptor (CAR), that expressed
55  higher GFP expression than naked Ad in both coxsackie and adenovirus receptor (CAR)-positive and -ne
56 s its attachment to a cell surface receptor, coxsackie and adenovirus receptor (CAR).
57 ion via binding interactions with epithelial coxsackie and adenovirus receptor (CAR).
58 sly suggested attachment receptors, CD46 and Coxsackie and Adenovirus Receptor (CAR).
59     beta cells strongly express cell-surface coxsackie and adenovirus receptor (CXADR) genes, which c
60 ike protein (JAML) on T cells and its ligand coxsackie and adenovirus receptor (CXADR) within tumor t
61                                          The coxsackie and adenovirus receptor (CXADR), a TJ protein
62                  Recently, a cDNA clone (the coxsackie and adenovirus receptor [CAR]) encoding a 46-k
63  increase was not observed in the absence of Coxsackie and Adenovirus Receptor cell expression, disco
64 cally inhibited by HCBP1 peptide rather than coxsackie and adenovirus receptor specific antibody.
65                                         CAR (Coxsackie and Adenovirus Receptor) is the primary dockin
66 xpression of related proteins JAM-C and CAR (Coxsackie and adenovirus receptor) was also observed in
67    However, a single-chain anti-CD40/soluble Coxsackie and adenovirus receptor-fusion protein (G28/sC
68 ne protein (CLMP) is structurally related to coxsackie and adenovirus receptor.
69 initial cell surface attachment receptor for Coxsackie and group C adenoviruses.
70                                          The coxsackie- and adenovirus receptor (CAR) functions as th
71 nce of the main receptor for adenovirus, the coxsackie- and adenovirus receptor (CAR).
72  We identified loss-of-function mutations in Coxsackie- and adenovirus receptor-like membrane protein
73                    The structures of polio-, coxsackie-, and rhinovirus polymerases have revealed a c
74                                          All coxsackie B (CB) viruses can initiate infection by attac
75 the inefficiency is due to an absence of the coxsackie B and adenovirus type 2 and 5 receptor (CAR) f
76 l-differentiated human airway epithelia, the coxsackie B and adenovirus type 2 and 5 receptor (CAR) r
77  receptors for the adenovirus fiber protein, coxsackie B and adenovirus type 2 and 5 receptor (CAR),
78 totype laboratory strains suggest that all 6 coxsackie B serotypes interact with a 46-kDa protein rec
79         Many serotypes of echovirus (EV) and Coxsackie B virus (CBV) bind human decay-accelerating fa
80                                          The coxsackie B virus and adenovirus receptor (CAR) is a mem
81 rmissive hamster cells became susceptible to coxsackie B virus attachment and infection.
82 dy interactions between low-passage clinical coxsackie B virus isolates and the two receptors.
83 g increases risk of chronic pancreatitis (4) Coxsackie B virus may increase severity of alcoholic chr
84 viruses, including parainfluenza virus 5 and Coxsackie B virus, that enter at the plasma membrane.
85                                              Coxsackie B viruses (CVB) are enteroviruses that have be
86 ole of recombination in the evolution of the coxsackie B viruses (CVB), we determined the partial seq
87 amily (IgSF) and functions as a receptor for coxsackie B viruses (CVBs).
88  human enterovirus B species (HEV-Bs) (e.g., coxsackie B viruses [CVBs] and echoviruses) have been im
89 r (CAR) mediates attachment and infection by coxsackie B viruses and many adenoviruses.
90 ion for heart transplantation worldwide, and coxsackie B viruses are detected in about one-third of i
91                                Although many coxsackie B viruses interact with decay accelerating fac
92                                              Coxsackie B viruses interact with two putative cell surf
93 In this report, we describe experiments with coxsackie B viruses with a cell type-specific propagatio
94 s species B, which contains the echoviruses, coxsackie B viruses, coxsackievirus A9, and enterovirus
95                                              Coxsackie-B-viruses (CVB) are frequent causes of acute m
96 ybridization with a target ssDNA specific of Coxsackie B3 virus was monitored using electrochemical i
97 HIP1 and HIP1R led to decreased infection by Coxsackie B3 virus.
98 at iNOS is induced in mice infected with the Coxsackie B3 virus.
99 iated with a moderate rate of diabetes after Coxsackie B4 and vaccinia virus infection.
100                                              Coxsackie B4 virus is strongly associated with the devel
101   We infected different strains of mice with Coxsackie B4 virus to discriminate between the two possi
102 s shown to display moderate activity against coxsackie B4 virus.
103 of LEW*1WR1 rats, whereas H-1, vaccinia, and Coxsackie B4 viruses did not.
104 n monocytes respond to infection with either Coxsackie (CV), encephalomyocarditis (EMCV), influenza A
105  2 (HSV-2) virus, and enteroviruses (polio-, coxsackie-, echo-, and enteroviruses 68 and 71) from per
106 active opsonophagocytic Ab responses against coxsackie, picorna, and influenza viruses and demonstrat
107  derived from glutamic acid decarboxylase or coxsackie virus (each of which has been associated with
108 tivator, m-3M3FBS, protects mice from severe Coxsackie virus A 16 (CVA16) infection.
109  mouth disease is caused by a new lineage of Coxsackie virus A6 and is characterised by high fever an
110 ool samples taken on day 6 of illness showed Coxsackie virus A6.
111 they express low levels of the high-affinity Coxsackie virus and adenovirus receptor (CAR).
112 ncer risk through differential regulation of Coxsackie virus and adenovirus receptor gene expression
113  cardiomyocytes were not more susceptible to Coxsackie virus B3 (CVB3) infection than control cells.
114 -altering mutations previously identified in coxsackie virus B3 (CVB3) were mapped onto the nsp12-RdR
115                                  Enterovirus Coxsackie virus B3 (CVB3)-the most common cause of viral
116  et al. show that infection of NOD mice with Coxsackie virus B3 or lymphocytic choriomeningitis virus
117       Infection of prediabetic NOD mice with Coxsackie virus B3 or lymphocytic choriomeningitis virus
118  These results show that diabetes induced by Coxsackie virus infection is a direct result of local in
119 pport of this, infection of the cells with a Coxsackie virus isolated originally from the pancreas of
120 d-19 differs from both influenza and typical coxsackie virus myocarditis.
121  complexes did not require expression of the coxsackie virus-Ad receptor (CAR) since similar data wer
122 ovel finding that human erythrocytes present Coxsackie virus-adenovirus receptor (CAR) providing an A
123 ion of the ligand MIP1alpha protects against Coxsackie virus-associated myocarditis.
124 r) is the primary docking receptor for typeB coxsackie viruses and subgroup C adenoviruses.
125 ), Severe acute respiratory syndrome (SARS), coxsackie viruses, and rhinoviruses.

 
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