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1 rder structure and endosomal location of the CpG oligonucleotide.
2 IL-12, and IL-6 was secreted in response to CpG oligonucleotide.
3 gins produced IFN-alpha when stimulated with CpG oligonucleotides.
4 respond to structurally distinct classes of CpG oligonucleotides.
5 he presence of agonistic Abs against CD40 or CpG oligonucleotides.
6 pG motifs abolished the protective effect of CpG oligonucleotides.
7 to explore the importance of the spacing of CpG oligonucleotides.
8 in response to cytosine-phosphate-guanosine (CpG) oligonucleotides.
9 receptor 9, and we found that the synthetic CpG oligonucleotide 2006 (CpG) reduced the frequency of
11 targeted co-delivery of antigen peptides and CpG oligonucleotides (a Toll-like receptor-9 agonist).
14 We show that siRNA synthetically linked to a CpG oligonucleotide agonist of toll-like receptor (TLR)9
15 c precursor cells isolated from human blood, CpG oligonucleotides alone were superior to GMCSF in pro
17 g conventional Toll-like receptor 9 ligands (CpG oligonucleotides), although it could be demonstrated
18 lls enhances cell proliferation triggered by CpG oligonucleotides and decreases sensitivity to dexame
20 SF) for concentrating dendritic cells (DCs), CpG oligonucleotides, and a doxorubicin-iRGD conjugate e
23 MNs coated with Ova as a model allergen and CpG oligonucleotide as an adjuvant (MNs-CIT) into the sk
24 intracerebral deposition of TLR9-activating CpG oligonucleotides, but not following non-CpG oligonuc
25 h peptide and cytosine-guanine dinucleotide (CpG) oligonucleotide, but not control oligonucleotide, s
26 e have recently shown that the TLR9 agonists CpG oligonucleotides can be used for targeted siRNA deli
27 could also produce IFN-alpha in response to CpG oligonucleotides, consistent with the observations o
29 zation and by adjuvants such as unmethylated CpG oligonucleotide (CpG) and LPS that induce T helper t
30 muramyl dipeptide (MDP), LPS, and a B-class CpG oligonucleotide (CpG-B), can substitute for gut flor
33 d stimulation of B cells with CXCL13-coupled CpG oligonucleotides (CpG-ODN) can block cancer metastas
34 es adjuvanted by a robust formulation of the CpG oligonucleotide delivered in emulsion were superior
35 coproteins adjuvanted with MF59 containing a CpG oligonucleotide elicited strong CD4(+) T helper resp
36 ted that oropharyngeal delivery of synthetic CpG-oligonucleotides elicited minimal lung inflammation
37 e contention that repeated administration of CpG-oligonucleotides enhances the effect of peptide and
40 pairs the response of subcutaneous tumors to CpG-oligonucleotide immunotherapy and platinum chemother
41 s a role for liver NK1.1(+) cells, IL-22 and CpG oligonucleotides in the induction of tolerance to is
42 ncodes Hv1, impaired mouse pDC activation by CpG oligonucleotides in vitro and in vivo, reducing IFN-
44 CpG oligonucleotides, but not following non-CpG oligonucleotide injection or after aseptic cryoinjur
47 pathway were functional, since class B and C CpG oligonucleotide ligands stimulated production of RAN
53 of functional nanodiamonds (fNDs) to deliver CpG oligonucleotides (ODNs) for sustained immunostimulat
57 on-CpG oligonucleotides, i.v. treatment with CpG oligonucleotides resulted in higher systemic concent
58 ced IL-10-secreting Treg with TLR9 agonists, CpG oligonucleotides, resulted in decreased IL-10 and IF
59 he efficiency of EBV B-CLL transformation of CpG oligonucleotide-stimulated cells by incubating patie
61 polysacharride (TLR4), zymosan (TLR2/6), and CpG oligonucleotide (TLR9) caused, in a complement-depen
62 lpha secretion from pDCs and that inhibitory CpG oligonucleotide treatment diminished HSV-induced IFN
63 ated increase in memory development, as both CpG oligonucleotide treatment or cotransfer of wild-type
64 cytokine production and tumor necrosis after CpG-oligonucleotide treatment and deficient production o
66 d uninfected individuals to the TLR9 agonist CpG oligonucleotide type B (CpG-B) in the presence and a