1 easing the risk of bilirubin encephalopathy (Crigler-Najjar syndrome).

2                                          For Crigler-Najjar syndrome, an inherited disorder that resu

3 egion of both alleles, whereas patients with Crigler-Najjar syndrome are homozygous for a defect that

4  hyperbilirubinemia in the Gunn rat model of Crigler-Najjar syndrome can be achieved with one injecti

5                                              Crigler-Najjar syndrome (CN) is a very rare genetic diso

6                                              Crigler-Najjar syndrome is a recessively inherited disor

7                            Patients with the Crigler-Najjar syndrome lack the enzyme uridine diphosph

8                              The Gunn rat, a Crigler-Najjar syndrome model animal lacking UDP-glucuro

9  vector encoding UGT1A1 in patients with the Crigler-Najjar syndrome that was being treated with phot

10                                              Crigler-Najjar syndrome type 1 (CN-1) is a recessively i

11 h hepatic bilirubin glucuronidation, causing Crigler-Najjar syndrome type 1 or 2, respectively.

12 cient hyperbilirubinemic Gunn rats (model of Crigler-Najjar syndrome type 1) resulted in hUGT1A1 expr

13                                              Crigler-Najjar syndrome type I is characterized by uncon

14 his case report describes a young adult with Crigler-Najjar syndrome type II in whom kernicterus deve

15 o liver transplantation in the management of Crigler-Najjar syndrome, type 1.

16 The Gunn rat is an excellent animal model of Crigler-Najjar syndrome, type 1.

17                                 In contrast, Crigler-Najjar syndrome types I and II are rare genetic