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1                                              Crigler Najjar type 1 is a rare autosomal recessive cond
2                                              Crigler-Najjar syndrome (CN) is a very rare genetic diso
3                                              Crigler-Najjar syndrome is a recessively inherited disor
4                                              Crigler-Najjar syndrome type 1 (CN-1) is a recessively i
5                                              Crigler-Najjar syndrome type I is characterized by uncon
6 utation in the UGT1A1 (HUG-Br1) isozyme of a Crigler-Najjar (CN) Type I patient.
7                              The Gunn rat, a Crigler-Najjar syndrome model animal lacking UDP-glucuro
8 h hepatic bilirubin glucuronidation, causing Crigler-Najjar syndrome type 1 or 2, respectively.
9                                 In contrast, Crigler-Najjar syndrome types I and II are rare genetic
10 easing the risk of bilirubin encephalopathy (Crigler-Najjar syndrome).
11                                          For Crigler-Najjar syndrome, an inherited disorder that resu
12 ly difficult, is now accepted management for Crigler-Najjar type I disease but remains controversial
13 yperbilirubinemia in humans that suffer from Crigler-Najjar type I disease results from lesions in th
14                                           In Crigler-Najjar type II patients and, recently, in Crigle
15 er-Najjar type II patients and, recently, in Crigler-Najjar type I patients treated with human hepato
16 o liver transplantation in the management of Crigler-Najjar syndrome, type 1.
17  hyperbilirubinemia in the Gunn rat model of Crigler-Najjar syndrome can be achieved with one injecti
18 cient hyperbilirubinemic Gunn rats (model of Crigler-Najjar syndrome type 1) resulted in hUGT1A1 expr
19 The Gunn rat is an excellent animal model of Crigler-Najjar syndrome, type 1.
20 e pathophysiology, treatment, and outcome of Crigler-Najjar type 1 syndrome (CN1) in 28 UGT1A1 c.222C
21 patients with urea cycle disorders (UCDs) or Crigler-Najjar (CN) syndrome 6 months posttransplantatio
22                            Patients with the Crigler-Najjar syndrome lack the enzyme uridine diphosph
23  vector encoding UGT1A1 in patients with the Crigler-Najjar syndrome that was being treated with phot
24 Gunn rats (n = 3), and patients (n = 4) with Crigler-Najjar type I (CN-I) syndrome.
25 his case report describes a young adult with Crigler-Najjar syndrome type II in whom kernicterus deve
26 egion of both alleles, whereas patients with Crigler-Najjar syndrome are homozygous for a defect that
27 to the induced levels noted in patients with Crigler-Najjar type 1 disease, is fatal in neonatal Ugt1