ライフサイエンスコーパス: DC

1 DC z-scan generates an intensity profile for each detect

2 DCs -containing vesicles can uniquely activate T cells,

3 DCs also have the greatest disease-associated changes in

4 DCs can also present native Ag to B cells through intera

5 DCs from Apc(Min/+)Ffar2(-/-) mice had an altered state

6 DCs of mesenteric lymph nodes (MLN) and joint regional l

7 s developed second cancers (control, n = 32; DC, n = 21; leukemia: 2 v 1).

8 BDCA-2-expressing population, Axl+Siglec-6+ DCs (ASDC), was also found to infiltrate human skin duri

9 ally increases the difficulty of designing a DC library and leads to the creation of many undesired v

10 sphaltene-rich organic phase is exposed to a DC uniform electric field.

11 Clinical translation of AC/DC and MRSI is demonstrated in a patient with mutant-IDH

12 n in DCs in vitro; they recruit and activate DCs with Th1-dominant cytokine profiles at the injection

13 ells responded to TA-loaded mCD40L-activated DC with increased proliferation and cytotoxic response (

14 show that these cells are largely activated DCs acquiring features compatible with lymph node homing

15 gen species (ROS) production in PM-activated DCs, impairing their capacity to generate functional Tre

16 Although DCs have specific characteristics that greatly enhance H

17 es inside DCs and are then transferred among DC subsets.

18 DWF and DC-GF in kidney transplant recipients represent differin

19 simultaneous use of pressure-driven flow and DC electric field, migration of particles inside microfl

20 C-SIGN (simultaneous binding to one GNP) and DC-SIGNR (intercross-linking with multiple GNPs), via a

21 Both LC-like and DC offspring from this progenitor carried the BRAF mutat

22 d electrochemical impedance spectroscopy and DC- based chronoamperometry.

23 cells, including monocytes, macrophages, and DCs isolated from specific tissues.

24 hors conclude that circulating monocytes and DCs migrate from the blood into the inflamed lungs.

25 ntaining gap junctions between monocytes and DCs.

26 Finally, we applied DC tsMSQ to study the assembly of the linker of the nucl

27 on of cDCs and CD123(+)AXL(+)CD327(+) pre/AS-DCs.

28 ents who received checkpoint blockade before DC vaccination had higher baseline MA-specific CD8 T cel

29 regulators in HIV-1 trans-infection between DC and CD4(+) T cells has the potential for the developm

30 ues down to 95 pM), but only partially block DC-SIGNR-mediated virus infection.

31 ee text] preferred-oriented Au thin films by DC plating at 5 mA/cm(2).

32 isms of Ag transfer and B cell activation by DCs remain incompletely understood.

33 racellular mechanism of B cell activation by DCs.

34 lta2(+) cells upregulates IL-12 secretion by DCs in a positive feedback loop.

35 ngomyelins; Lyso.PC.a.C18.0, PC.ae.C34.2, C3.DC..C4.OH, glutamine and SM.C16.1, being the most signif

36 n human blood stem cells engineered to carry DC-causing PARN mutations were xenotransplanted into imm

37 The rapid Defence Cascade (DC) response (startle, freeze) to sudden unexpected stim

38 the conditional ablation of Irf4 in CD11c(+) DCs results in more efficient immune control of Helicoba

39 55 deficiency, migratory (MHC(high)CD11c(+)) DCs increased significantly in RLN.

40 ithin these RLN, resident (MHC(int)CD11c(+)) DCs increased on days 14 and 21.

41 , we describe a CD1a-bearing BDCA-2+CD123int DC subset that rapidly infiltrates human skin wounds and

42 Finally, Axl(+) CD169(+) DCs were present in cancer patients and efficiently capt

43 te a nanovaccine platform targeting CD169(+) DCs to drive antitumor T cell responses.

44 Substance P induced CD301b(+) DC migration to the dLN where they initiated T helper-2

45 TL responses via Ag transfer to splenic CD8+ DCs in a manner independent of monocyte APC activity.

46 e contributed to intrathymic dendritic cell (DC) differentiation, predominantly of plasmacytoid dendr

47 ate (Zol) treatment impaired dendritic cell (DC) functions and increased bacterial load in alveolar b

48 ost upregulated gene was the dendritic cell (DC) marker CD209a only in PVPV-Akt1KO thyroids.

49 Dendritic cell (DC) maturation is a prerequisite for the induction of ad

50 gerhans cells (LC), the only dendritic cell (DC) population of the epidermis.

51 ion of pDCs from bone marrow dendritic cell (DC) progenitors and their maintenance is driven by the t

52 -CSF has been widely used in dendritic cell (DC) research, the mechanisms, factors, and signals regul

53 attributed to a loss of the dendritic cell (DC) subset conventional DC2.

54 Recently, another dendritic cell (DC) subset, termed merocytic DCs (mcDCs), was defined.

55 Dendritic cell (DC) subsets are abundantly present in genital and intest

56 t diminishes the efficacy of dendritic cell (DC)-based cancer immunotherapy.

57 ls (DCs) in vitro and in vivo, we map T cell-DC interaction preferences, and discover regulatory T ce

58 Analysis of T cell-DC pairs reveals an interaction-specific program between

59 nses inflammation-associated Langerhans cell/DC to gain an enhanced capacity to promote Treg cells vi

60 anger signals recognized by dendritic cells (DC) facilitating the establishment of an anti-BP CD4 T c

61 (BMDC) as well as pulmonary dendritic cells (DC) from RSV-infected mice upregulated the expression of

62 Dendritic cells (DC) play an essential role in innate immunity and radiat

63 c injury to the myocardium, dendritic cells (DC) respond to cardiomyocyte necrosis, present cardiac a

64 hans cells (LC), and dermal dendritic cells (DC).

65 damages that are sensed by dendritic cells (DCs) and activates the host cytosolic DNA sensing STING/

66 ecruitment to phagosomes in dendritic cells (DCs) and downstream TLR signaling are essential to initi

67 Dendritic cells (DCs) and other innate immune cells patrol the oral mucos

68 NVP was readily taken up by dendritic cells (DCs) and promoted DC maturation and antigen presentation

69 Dendritic cells (DCs) are a diverse group of specialized antigen-presenti

70 Dendritic cells (DCs) are antigen-presenting cells controlling T cell act

71 Dendritic cells (DCs) are antigen-presenting cells subdivided in speciali

72 Dendritic cells (DCs) are critical for the differentiation of pathogen-sp

73 Dendritic cells (DCs) are highly susceptible to extrinsic signals that mo

74 Dendritic cells (DCs) are one of the earliest targets of HIV-1 infection

75 Dendritic cells (DCs) are professional APCs, which sample Ags in the peri

76 ine whereby patient-derived dendritic cells (DCs) are programmed to induce Th17 responses to the OC a

77 Airway dendritic cells (DCs) are recognized as important factors in the mechanis

78 were primed via antigen and dendritic cells (DCs) at fever temperatures, the Th2 switch was abrogated

79 Dendritic cells (DCs) develop in the bone marrow from haematopoietic prog

80 A elements in skin-resident dendritic cells (DCs) exhibit the highest enrichment of SSc-associated si

81 Conventional dendritic cells (DCs) in particular exist as two major subpopulations in

82 To examine the role of lung dendritic cells (DCs) in T follicular helper (Tfh)-cell induction, a T-ce

83 ion of tolerogenic CD103(+) dendritic cells (DCs) in the spleen.

84 Migratory dendritic cells (DCs) in tumors transport antigens and share them with ly

85 actions between T cells and dendritic cells (DCs) in vitro and in vivo, we map T cell-DC interaction

86 oclasts (OCs) and much less dendritic cells (DCs) induce significant expansion and functional activat

87 T cell activation by dendritic cells (DCs) involves forces exerted by the T cell actin cytoske

88 The formation of mammalian dendritic cells (DCs) is controlled by multiple hematopoietic transcripti

89 Dendritic cells (DCs) of the cDC2 lineage initiate allergic immunity and

90 Dendritic cells (DCs) participate in the pathogenesis of several diseases

91 Dendritic cells (DCs) play a central role in the early immune response ag

92 Dendritic cells (DCs) play a critical role in shaping adaptive immunity.

93 ent tissue macrophages, and dendritic cells (DCs) present in every tissue in the body.

94 were studied; in addition, dendritic cells (DCs) purified from the draining lymph nodes of tape-stri

95 s, we identify a cluster of dendritic cells (DCs) that we name 'mature DCs enriched in immunoregulato

96 to be populated by vascular dendritic cells (DCs) that, during hypercholesterolemia, initiate atheros

97 s on human monocyte-derived dendritic cells (DCs) to investigate how they polarize naive CD4(+) T(H)

98 -p65 and IL-6 expression in dendritic cells (DCs), as well as aryl hydrocarbon receptor (AhR) express

99 and CD103+ cross-presenting dendritic cells (DCs), as well as up-regulation of MHC class I and down-r

100 whereas Tlr9 deficiency in dendritic cells (DCs), plasmacytoid DCs, and neutrophils had no discernab

101 Tconvs) and proinflammatory dendritic cells (DCs), which are both implicated in GVHD pathogenesis.

102 e the functions of PD-L1 on dendritic cells (DCs), which highly express PD-L1.

103 R1(+) macrophages, DCIR2(+) dendritic cells (DCs), which play a key role in priming Tfh responses, we

104 n mouse bone marrow-derived dendritic cells (DCs).

105 promoting the maturation of dendritic cells (DCs).

106 onses via interactions with dendritic cells (DCs).

107 The hotspots for MSMs were central DC and non-MSM in south DC.

108 CIA-DCs differentially expressed genes associated with immun

109 subset of intestinal cDCs, which we term CIA-DCs.

110 Cells identified as classical DC subsets, cDC1s and cDC2s, and lacking Fcgr1 (encoding

111 Classical DCs include type 1 and type 2 subsets, which can detect

112 Type 1 classical DCs (cDC1), which depend on the transcription factor Bat

113 Here, we describe a dual-color (DC) extension of the fluorescence z-scan technique, whic

114 , we describe the development of dual-color (DC) tsMSQ to analyze NE heteroprotein complexes built fr

115 This chemo-immunotherapy that concentrates DCs to present endogenous tumor antigens generated in si

116 NOP10, or NHP2 cause dyskeratosis congenita (DC), a disorder characterized by telomere attrition.

117 y disorders, such as dyskeratosis congenita (DC), for which there are no effective treatments.

118 of OX40L and define a mechanism controlling DC expression of OX40L in vivo.

119 overcome several limitations of conventional DC-driven electroluminescent devices, including the requ

120 Type 1 conventional DCs are essential for PD-L1 blockade and they upregulate

121 cross-presenting pDCs required conventional DCs (cDCs) to achieve cross-priming in vivo by transferr

122 , and ability to synergize with conventional DCs (cDCs) in generating potent tumor antigen-specific C

123 Decompressive craniectomy (DC) is often required to manage rising intracranial pres

124 on alternating as opposed to direct current (DC) schemes.

125 r followed by application of direct current (DC), thus inducing electrolysis and creating localized p

126 The classic theory of direct-current (DC) insulator-based dielectrophoresis (iDEP) considers t

127 hanisms in recurrent tumor cells that dampen DC antigen presentation and recruit innate-like CD8(+) T

128 IFTA was similarly associated with decreased DC-GS.

129 somal TLR responses in PI4KIIalpha-deficient DCs are restored by reexpression of wild-type PI4KIIalph

130 entiation and maturation of GM-CSF-dependent DCs are insufficiently known.

131 enterocolitica-infected bone marrow-derived DCs and purified MLN DCs, which may explain the mechanis

132 turation, C/EBPbeta(-/-) bone marrow-derived DCs matured much faster enabling them to activate and po

133 In SPPL2a(-/-) bone marrow-derived DCs, Dectin-1 is redistributed to endosomal compartments

134 Aspergillus-infected monocyte-derived DCs and neutrophils recruit pDCs, which promote neutroph

135 Activation of FPR3 in human monocyte-derived DCs leads to inhibition of IL-12 production.

136 iyamotoi is phagocytosed by monocyte-derived DCs, causing upregulation of activation markers and prod

137 , respectively, in LC and Langerin(+) dermal DC and in all CD11c(+) cells.

138 CD301b(+) dermal DCs respond to allergens encountered in vivo, but not in

139 nce induction, while talin1-deficient dermal DCs unexpectedly accumulated in the dermis despite their

140 TF HIV-1 phenotype and the role of different DC subsets in establishing infection.

141 on nanocalorimetry (ITC), molecular docking (DC), and molecular dynamics (MD) were used to characteri

142 required for TLR signalosome assembly during DC activation.

143 tworks control differential aspects of early DC progenitor versus late-stage DC cell fate decisions.

144 ologic condition that often occurs following DC as a result of the unrepaired skull.

145 ere, we report that primary fibroblasts from DC patients and late generation telomerase knockout mice

146 te differentiation toward TAMs and away from DCs via suppression of DC-promoting transcription factor

147 However, to what extent innate cues from DCs dictate transcriptional changes in T cells remains e

148 ogated via increased production of IL12 from DCs at fever temperatures.

149 Furthermore, DCs from IL-9R(-/-) mice induced more GM-CSF production

150 ene carbonate-graft-dodecanol) (mPEG-b-PCC-g-DC) polymeric nanoparticles (NPs) by solvent evaporation

151 : (i) the g-gap dipeptide composition (g-gap DC), pseudo-amino acid composition (PseAAC), auto-correl

152 dues at the tip of the LBL for binding of HC/DC to lipid membranes.

153 d amplitude to radially confine ions, a high DC field, and a wide exit ring electrode.

154 ependent on SignR3 (murine ortholog of human DC-SIGN).

155 Our results indicate that human DC subsets, such as CD141+CD11c+ and CD1c+CD11c+ myeloid

156 zed mouse model that mounts functional human DCs enables preclinical assessment of the immunogenicity

157 ogy, we reveal that IRF5 deficiency in human DCs, or macrophages, corresponded with reduced virus-ind

158 d immune pathways were also studied in human DCs.

159 ow these findings can be translated to human DCs and their progenitors.

160 ad increased IL1B+ T-regulatory cells, IL1B+ DCs and IL1B+ plasmacytoid DCs, IL1B+ monocytes, and few

161 ir tumors had even higher numbers of IL27(+) DCs.

162 cterized the local phylodynamics of HIV-1 in DC using next-generation sequencing (NGS) data.

163 We found that changes in DC-induced deposition of bacteria depended on the relati

164 Clec9a(-/-) mice deficient in DC cross-priming are protected from persistent immune-me

165 hese results suggest that KDM6 expression in DC enhances proinflammatory innate cytokine production t

166 7 HIV positives non-MSMs (n=12078) living in DC, end of 2018.

167 ts are central to control particle motion in DC-iEK devices.

168 Thus, C/EBPbeta plays a unique role in DC maturation and immunostimulatory functionality and em

169 ammaRIIa and spleen tyrosine kinase (Syk) in DC activation and showed that they were both strongly im

170 In DCs, blockade of ERK signaling similarly inhibited IL-10

171 e formation of preassembled TLR complexes in DCs at steady state via direct interaction with MyD88 an

172 of these barriers, phagolysosomal escape in DCs.

173 signals also induce tolerogenic functions in DCs.

174 lence in mice by boosting early infection in DCs, resulting in the greater activation of antiviral re

175 es MyD88-dependent TLR signaling pathways in DCs through a novel mechanism with implications for anti

176 activate, and induce epitope presentation in DCs in vitro; they recruit and activate DCs with Th1-dom

177 g one receptor, the farnesoid X receptor, in DCs enhanced the generation of T(reg) cells and imposed

178 se metabolites elicited similar responses in DCs as compared to respective RWEs.

179 ytotoxicity were not different from those in DCs.

180 edge, on how protein aggregates could induce DC and T cell activation via the FcgammaRIIa-Syk signali

181 ith ovalbumin (OVA), the E. faecalis-induced DCs showed reduction in CD4(+) OVA-specific OT-II T cell

182 We hypothesized that Chlamydia-infected DCs and epithelial cells present overlapping sets of Chl

183 nded type 2/type 22 T cells and inflammatory DCs, and by a unique inflammatory fibroblast that may in

184 e characterized by expansion of inflammatory DCs (CD1A(+)FCER1A(+)) and tissue-resident memory T cell

185 -glycans can potently and completely inhibit DC-SIGN-mediated augmentation of Ebola virus glycoprotei

186 Exposure of NK cells to innate DC-derived costimulatory factors triggered their helper

187 ed to the LN within discrete vesicles inside DCs and are then transferred among DC subsets.

188 The major intestinal DC subsets (CD103(+)CD11b(-), CD103(-)CD11b(+), and CD10

189 ynamic regime beyond the widely-investigated DC limit which enables the coupling between the Weyl fer

190 We investigated DCs and the connection between mucosa and joints in a mu

191 n wild type and Clec9a-depleted mice lacking DC cross-priming function.

192 Furthermore, the LAMP3 + DC subgroup may be able to recruit regulatory T cells, p

193 n(-) migratory DCs revealed that Langerin(+) DCs became activated in the MC903 model, whereas these c

194 3, Bcl3, and Id2, which render pDCs and late DC progenitors refractory to physiological stimuli contr

195 ween two closely related tetrameric lectins, DC-SIGN (simultaneous binding to one GNP) and DC-SIGNR (

196 t promoted the precursors of M2 macrophages, DCs and regulatory T cells.

197 ates human skin wounds and comprises a major DC population.

198 A late-stage bone marrow DC progenitor expressed low amounts of LIFR and develope

199 microscopy, we find that during maturation, DC cortical stiffness increases via a process that invol

200 f dendritic cells (DCs) that we name 'mature DCs enriched in immunoregulatory molecules' (mregDCs), o

201 dendritic cell (DC) subset, termed merocytic DCs (mcDCs), was defined.

202 sis of Langerin(+) and Langerin(-) migratory DCs revealed that Langerin(+) DCs became activated in th

203 rogram between pathogen-presenting migratory DCs and T cells.

204 ted bone marrow-derived DCs and purified MLN DCs, which may explain the mechanism underlying the impa

205 Aspergillus-infected murine Mo-DCs and neutrophils recruited pDCs to the lung by releas

206 respond to IL-18 and to effectively modulate DC function may contribute to disturbances in proper imm

207 ere is currently much interest in modulating DC function to improve cancer immunotherapy.

208 ansmission, it is becoming evident that most DC subsets also have virus restriction mechanisms that e

209 Here, we discuss genetic models of mouse DC development and function that have aided in correlati

210 itiating phagosomal TLR4 responses in murine DCs by generating a phosphatidylinositol-4-phosphate (Pt

211 such as CD141+CD11c+ and CD1c+CD11c+ myeloid DCs, distribute throughout several organs in HIS mice in

212 plasmacytoid dendritic cells; CD1c + myeloid DCs; neutrophils; macrophages; natural killer cells (NK)

213 ity of 7 fT Hz(-1/2) at a low frequency near DC is achieved using only one laser beam with 15 uW of p

214 and reversible covalent PROTACs, with <10 nM DC(50)'s and >85% degradation.

215 A unique feature of DC-iEK devices is that nonlinear EK effects are enhanced

216 This study underlines the importance of DC-mediated priming in identifying novel regulators of T

217 o received radiotherapy, and the presence of DC correlates with gene expression of LGP2 in the tumor

218 ance with E2F1 being a negative regulator of DC maturation, C/EBPbeta(-/-) bone marrow-derived DCs ma

219 owth retardation, and cellular senescence of DC fibroblasts.

220 glycoprotein modifications, the stability of DC-SIGN is better correlated with the number of glycosyl

221 rd TAMs and away from DCs via suppression of DC-promoting transcription factor Irf4.

222 The upregulation of DC-STAMP and matrix metalloproteases was observed on the

223 ovement in outcome or safety with the use of DC compared with anthracycline-based chemotherapy.

224 ulting in enhanced cross-priming capacity of DCs and tumor-specific CD8(+) T cell activation.

225 n the mechanism underlying the impairment of DCs in RLN induced by FTY720.

226 Maturation of DCs induced by diverse proinflammatory conditions promot

227 by CD4(+) T cells through the modulation of DCs.

228 burgdorferi We investigated the response of DCs to two different strains of B. miyamotoi using in vi

229 that chitosan facilitates transmigration of DCs across the vaginal epithelium in the mouse female re

230 MC-derived IL-13 acted on DCs from draining lymph nodes of OVA-sensitized skin to

231 anism supporting the effect of aggregates on DCs and consequently on the T cell response.

232 ally with bone marrow-derived macrophages or DCs from Tpl2(-/-) mice.

233 ustion of CD8(+) T cells, and overactivating DCs, leading to their death.

234 in 1,2-dioleoyl-sn-glycero-3-phosphocholine (DC(18:1)PC) lipid bilayer.

235 k 1,2-dierucoyl-sn-glycero-3-phosphocholine (DC(22:1)PC) but not in thin 1,2-dioleoyl-sn-glycero-3-ph

236 offers a practical approach to measuring pig DC responses, and potentially pig affect and welfare, un

237 Plasmacytoid DCs (pDCs), the major producers of type I interferon, ar

238 iency in dendritic cells (DCs), plasmacytoid DCs, and neutrophils had no discernable effect on diseas

239 tory cells, IL1B+ DCs and IL1B+ plasmacytoid DCs, IL1B+ monocytes, and fewer group 1 innate lymphoid

240 -IFTA = 1 and t >= 2, ti >= 2 had postbiopsy DC-GS similar to CA TCMR.

241 criteria for CA TCMR had similar postbiopsy DC-GS as those with CA TCMR.

242 ompartment showing that AXL(+)SIGLEC6(+) pre-DCs mapped exclusively to the DC2 pathway.

243 -TENGs and over 10-fold compared to previous DC-TENGs.

244 of cytotoxic CD8(+) T cells by cross-priming DC contributes to exacerbation of postischemic inflammat

245 Cross-priming DC have the ability to activate both CD4(+) helper and C

246 drive the maturation of high IL-12-producing DCs.

247 ken up by dendritic cells (DCs) and promoted DC maturation and antigen presentation.

248 The immune scavenger protein DC-SIGN interacts with glycosylated proteins and has a p

249 py-based cell counting were used to quantify DC field effects on the deposition of bacterial strains

250 nOVAmax induced a regulatory DC phenotype evidenced by a decrease of the activation m

251 able T cell responses, and efficacy required DCs and host expression of CD40.

252 milar changes in both migratory and resident DCs were also detected on day 14 in MLN of TNFRp55(-/-)

253 icles is a dominant pathway to load resident DCs for presentation to T cells.

254 gens and share them with lymph node resident DCs through cross-presentation.

255 During an immune response, DCs undergo a maturation process that optimizes their ab

256 However, selecting DCs for sets of sequences that covary at multiple positi

257 g exposure to activation of allergic-skewing DCs and the initiation of an allergic immune response.

258 ed the capacity of LPS-stimulated human skin DCs to express IL-12 and promote IFN-gamma secretion by

259 SOCS3(-/-) DCs slightly attenuated BMDC-induced immunogenic toleran

260 er, also under steady-state conditions, some DCs are characterized by inherent tolerogenic properties

261 or MSMs were central DC and non-MSM in south DC.

262 cts of early DC progenitor versus late-stage DC cell fate decisions.

263 ally, we showed that B. miyamotoi-stimulated DCs induced proliferation of naive CD4(+) and CD8(+) T c

264 parable results were obtained by stimulating DCs directly with RWEs.

265 Strikingly, DC expression of OX40L is redundant in a chronic intesti

266 Strikingly, DCs from BALB/c mice cocultured with CD83-deficient CD4(

267 performed on model systems demonstrate that DC tsMSQ properly identifies heteroprotein complexes and

268 The DC power creates acidic (pH < 2) and alkaline (pH > 12.9

269 es facilitates the T cells activation by the DC vaccine.

270 We characterized the DC population in the heart and mediastinal lymph nodes a

271 inverse model to estimate emissions from the DC-Baltimore region.

272 odels exceeded 0.85 in all age groups in the DC and 0.84 in the VC, indicating well-validated diagnos

273 included in the HLH group and 34 each in the DC and HC groups.

274 ammation, the lack of IRF4 expression in the DC compartment alleviates inflammation and goblet cell m

275 In the DC, sensitivity values ranged between 0.75 and 0.97 and

276 o studies and demonstrate the utility of the DC tsMSQ technique for characterizing NE heteroprotein c

277 Modelling, restricted to the DC component, showed good agreement with measured result

278 tion in Weyl semimetals is restricted to the DC regime.

279 Thus, the DCs epigenome links inherited susceptibility and clinica

280 vitro, LPS-stimulated migratory TNFRp55(-/-) DCs of MLN increased IL-12/23p40 compared with wild-type

281 tes displayed antitumor activity superior to DC vaccines in several cancer models, including aggressi

282 high- and low-risk LCH patients gave rise to DCs and LC-like cells in vitro, but the driver mutation

283 These "induced tolerogenic DCs" help to moderate immune responses such as those to

284 Here, we report that tumor DCs are linked to the clinical outcome of patients with

285 AE upon adoptive transfer than did wild-type DCs.

286 Here, we used DCs stimulated with specific pathogens to prime CD4 T ce

287 es its great potential in electric vehicles, DC micro-grids etc.

288 ient Databases from 19 states and Washington DC, we analyzed 1157 patients from early-adopter states

289 Washington, DC continues to experience a generalized HIV-1 epidemic.

290 ong individuals in the Baltimore-Washington, DC area by self-reported race/ethnicity.

291 th higher incomes, who reside in Washington, DC, USA, have congressional experience and who engage in

292 icles can uniquely activate T cells, whereas DCs lacking them do not.

293 al load in alveolar bone in vivo and whether DC inhibition alone predisposed the animals to osteonecr

294 This may explain why DCs respond faster and more vigorously to TLR ligand bin

295 hat were previously infected by feeding with DC-microinjected nymphal ticks.

296 ted increased levels in ALS as compared with DCs and HCs.

297 expression and IL-12 secretion compared with DCs cocultured with CD4(+) conventional T cells from CD8

298 s as a major T cell subtype interacting with DCs in mouse draining lymph nodes.

299 ntify key genes involved in a pathway within DCs that is exploited by HIV-1 to traffic to the VS.IMPO

300 = 1 (vs 0) was associated with worse 3-year DC-GS: (i-IFTA = 0, 81.7%, [95% CI 77.7 to 85.9%]); i-IF