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1 DMPP (1-4 microM) had no effect on the rate of spontaneo
2 DMPP produced robust increases with minimal release obse
3 DMPP-induced [3H]-5-HT release was neither sensitive to
6 the use of nitrification inhibitors such as DMPP (3,4-dimethylpyrazole phosphate) has been proposed.
7 erved, since the N(2)O mitigation induced by DMPP was lower than in control soil, demonstrating that
10 receptor agonist dimethylphenylpiperazinium (DMPP) on the evoked release of ACh from motor terminals
13 s (unfertilized, fertilized and fertilized + DMPP) were assayed under two contrasting soil water cont
14 gonists studied here were similar except for DMPP, which seemed to be a partial agonist compared with
16 pivotal role of the aromatic signal, for HA-DMPP association, and both alkyl methyl groups, for FA-D
17 of the IAS smooth muscle by EFS (0.25-5 Hz), DMPP, and VIP caused a significant increase in the HO ac
18 st evidence that the nitrification inhibitor DMPP can inhibit the N2 O production from nitrifier-indu
20 C), 1,1-dimethyl-4-phenylpiperzinium iodide (DMPP), cytisine (CYT) and epibatidine (EPI) were investi
21 y 1,1-dimethyl-4-phenyl-piperazinium iodide (DMPP) approximately nicotine > acetylcholine > carbachol
22 t 1,1-dimethyl-4-phenyl-piperazinium iodide (DMPP) depolarized PPN neurons early in development, then
23 ulant dimethyl diphenyl piperazinium iodide (DMPP), and neuropeptide vasoactive intestinal polypeptid
25 1,1-Dimethyl-4-phenyl-piperazinium iodide (DMPP, 20 microM) or alpha,beta-methylene ATP (50-100 mic
26 t 1,1-dimethyl-4-phenyl-piperazinium iodide (DMPP; in vivo and in vitro) and by high K+ (in vitro); a
29 n of evoked ACh release at 50 Hz by 2 microM DMPP was abolished by 10 microM of the calmodulin antago
34 hat PROG co-applied with DMPP inhibited peak DMPP-induced current up to 68% and with 3 min pre-incuba
35 , whereas 1,1-dimethyl-4-phenylpiperazinium (DMPP) was less potent than ACh in some cells (as in the
36 (10 muM), 1,1-dimethyl-4-phenylpiperazinium (DMPP; 100 muM) or luminal nutrients (Ensure(R)), in the
37 agonist, 1,1-dimethyl-4-phenylpiperizinium (DMPP, 10(-10)-10(-7) M), for 24 h significantly increase
38 e focused on 3,4-dimethylpyrazole phosphate (DMPP) by placing it in contact with increasing concentra
39 on inhibitor 3,4-dimethylpyrazole phosphate (DMPP) significantly reduced the N2 O production from the
42 sing an electrophysiological technique, that DMPP can produce changes in the evoked ACh release from
47 echniques revealed that PROG co-applied with DMPP inhibited peak DMPP-induced current up to 68% and w
49 d that both humic substances interacted with DMPP, with HA exhibiting a significantly greater affinit