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1                                              DMVECs expressed a mixed profile of factors that can con
2                                              DMVECs were isolated using magnetic-activated cell sorti
3  p38 MAPK pathway in wounds, db/db MSCs, and DMVECs.
4 uced dermal microvascular endothelial cells (DMVEC) as detected by indirect immunofluorescence assay
5 uman dermal microvascular endothelial cells (DMVEC) in culture results in the conversion of cobblesto
6 uman dermal microvascular endothelial cells (DMVEC) to form colonies of proliferating latently infect
7 n of dermal microvascular endothelial cells (DMVEC) transforms the cells from a cobblestone-like mono
8 n of dermal microvascular endothelial cells (DMVEC) with KSHV.
9 cted dermal microvascular endothelial cells (DMVEC).
10      Dermal microvascular endothelial cells (DMVECs) are an understudied cell type in HTS.
11 n of dermal microvascular endothelial cells (DMVECs) with KSHV recapitulates many of the features of
12 uman dermal microvascular endothelial cells (DMVECs) with KSHV than supernatants from BC-3 or BCP-1 P
13 b/db dermal microvascular endothelial cells (DMVECs), as well as remedied paracrine angiogenic functi
14  and dermal microvascular endothelial cells (DMVECs).
15 ted, immortalized dermal microvascular EC (E-DMVEC).
16  was strongly induced in latently infected E-DMVEC, whereas the expression levels of the IGF-IR remai
17 stic focus formation seen in KSHV-infected E-DMVEC.
18                                          HTS DMVECs have both a discordant response to cellular insul
19                                          HTS DMVECs were 17.59% less permeable than normal DMVECs (p
20   After injury, NS DMVECs were 28.4% and HTS DMVECs were 18.8% more permeable than uninjured controls
21 ranscript and protein-level responses in HTS DMVECs differ from those in normal skin (NS).
22  inserts were used to form monolayers of HTS DMVECs and NS DMVECs.
23 e a functional and expression profile of HTS DMVECs.
24 n lymphoma cells and telomerase-immortalized DMVEC infected directly with cell-free virus.
25 tion of spindle cell colonies and plaques in DMVEC cultures provides for the first time a quantitativ
26                            KSHV infection in DMVECs was associated with a change from a cobblestone t
27 th increased c-Kit expression, KHSV-infected DMVEC displayed enhanced proliferation in response to th
28  the early lytic cycle both in KSHV-infected DMVEC and in the body cavity-based lymphoma BCBL1 PEL ce
29    All spindle-shaped cells in KSHV-infected DMVEC cultures express the latency-associated nuclear pr
30  showed that transformation of KSHV-infected DMVEC was inhibited by small interfering RNA directed at
31 ulins 3 and 5 was decreased in KSHV-infected DMVEC, fibulins 1C/1D were increased, and fibulins 4, 6,
32  profiles of KSHV-infected and mock-infected DMVEC.
33 ls and of KSHV (JSC-1) persistently infected DMVEC cells displayed cytoplasmic vGCR protein expressio
34 itro enhanced proliferation of KSHV-infected DMVECs in the presence of free heme.
35 MVECs were 17.59% less permeable than normal DMVECs (p < 0.05).
36 rentially expressed genes between HTS and NS DMVECs, of which 10 were upregulated and 21 were downreg
37 used to form monolayers of HTS DMVECs and NS DMVECs.
38                             After injury, NS DMVECs were 28.4% and HTS DMVECs were 18.8% more permeab
39      Gene expression in HTS DMVECS versus NS DMVECs was measured.
40 tisense oligomers, we observed inhibition of DMVEC proliferation and foci formation using antisense m
41 ncluding VEGF secretion and the promotion of DMVEC migration and vasculature formation.
42 KSHV-induced morphological transformation of DMVEC.
43 onent of the KSHV-mediated transformation of DMVEC.
44                     Further investigation of DMVECs is warranted to elucidate their contribution to H
45 spindle cell conversion phenotype in primary DMVEC cultures that is directly associated with latent s
46 estone-like phenotype of adjacent uninfected DMVEC.
47 man genes in infected compared to uninfected DMVEC cultures in both the presence and absence of TPA.
48 ion profiles of KSHV-infected and uninfected DMVECs.