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1 yomavirus (MCPyV) is a human double-stranded DNA tumor virus.
2 h structure of a model helicase encoded by a DNA tumor virus.
3 tures of its genome suggest that it is a new DNA tumor virus.
4  steroid receptor superfamily and the HPV-16 DNA tumor virus.
5 licases are essential for the replication of DNA tumor viruses.
6 equired to mediate transformation induced by DNA tumor viruses.
7 ered in many cancers and is also targeted by DNA tumor viruses.
8 r similar to that of T antigens of the small DNA tumor viruses.
9 or suppressor pathway in a manner similar to DNA tumor viruses.
10 s also targeted by oncoproteins expressed by DNA tumor viruses.
11 effects associated with infections with many DNA tumor viruses.
12 the mechanisms of transformation employed by DNA tumor viruses.
13 nd Kaposi's sarcoma herpesvirus (KSHV) human DNA tumor viruses.
14 apillomaviruses are a family of nonenveloped DNA tumor viruses.
15 ession of transforming proteins derived from DNA tumor viruses.
16 ement in latent replication of these related DNA tumor viruses.
17 c single-stranded DNA elements and mammalian DNA tumor viruses.
18 plication of mammalian cells and their small DNA tumor viruses.
19  by the transforming oncoproteins of several DNA tumor viruses.
20  to have onco-suppressive properties against DNA tumor viruses.
21 made through the use of GEM models for human DNA tumor viruses.
22 om studies using immediate-early proteins of DNA tumor viruses.
23                      The oncoproteins of the DNA tumor viruses, adenovirus E1A, simian virus 40 T ant
24          While oncoproteins encoded by small DNA tumor viruses and Epstein-Barr virus (EBV) latent an
25 ression is critical for persistence of these DNA tumor viruses and most likely involved in mediating
26 n factors, bind to origins of replication in DNA tumor viruses and stimulate viral DNA replication in
27 teract with the transforming oncoproteins of DNA tumor viruses and this led to rapid advances in our
28 be effective in malignant diseases for which DNA tumor viruses are etiologic agents and that antitumo
29                                    All known DNA tumor viruses are known to target and inactivate two
30                            Oncoproteins from DNA tumor viruses associate with critical cellular prote
31 is a key target of oncoproteins expressed by DNA tumor viruses, but RNA viruses are not known to regu
32  into the host genome, is a critical step in DNA tumor virus carcinogenesis.
33       Human papillomaviruses (HPV) are small DNA tumor viruses causally associated with cervical canc
34                       The study of the small DNA tumor viruses continues to provide valuable new insi
35 xtensively to our understanding of how these DNA tumor viruses directly contribute to human cancers.
36                                         Most DNA tumor viruses disturb the cell cycle pathways by ess
37 ically interact with oncoproteins encoded by DNA tumor viruses (E7, T-Ag, E1A).
38                                        Small DNA tumor viruses encode gene products which can functio
39                            Many of the small DNA tumor viruses encode transforming proteins that func
40                                    The human DNA tumor viruses Epstein-Barr virus (EBV), Kaposi's sar
41                               Viruses of the DNA tumor virus family share the ability to transform ve
42                                    SV40 is a DNA tumor virus found in some studies to be associated a
43                         Evolution of minimal DNA tumor virus' genomes has selected for small viral on
44                                 The study of DNA tumor viruses has been invaluable in uncovering the
45                                 The study of DNA tumor viruses has revolutionized cancer biology, par
46     Human papillomavirus type 16 (HPV-16), a DNA tumor virus, has a causal role in cervical cancer, a
47                The origins of replication of DNA tumor viruses have a highly conserved feature, namel
48                                   Studies of DNA tumor viruses have provided important insights into
49                                              DNA tumor viruses have provided major insights into how
50 sive properties of AAV against adenovirus, a DNA tumor virus, have been well documented.
51 in required for T-cell transformation by the DNA tumor virus herpesvirus saimiri (HVS) and designated
52 in required for T-cell transformation by the DNA tumor virus herpesvirus saimiri (HVS) strain 484, de
53            IMPORTANCE EBV is the first human DNA tumor virus identified, discovered over 50 years ago
54                         Intervention by this DNA tumor virus in cellular translational controls is li
55 dy of the transforming proteins derived from DNA tumor viruses in experimental models of transformati
56  Polyoma virus (Py) differs from other small DNA tumor viruses in not encoding a protein that inactiv
57       Here, we identify the oncogenes of the DNA tumor viruses, including E7 from human papillomaviru
58                              Infections with DNA tumor viruses, including members of the polyomavirus
59  factors, and associate with oncoproteins of DNA tumor viruses, including simian virus 40 (SV40) larg
60 te with the transforming proteins of several DNA tumor viruses, including the large T antigen encoded
61 ctions of transforming proteins from several DNA tumor viruses, including two papillomaviruses and tw
62 The papillomavirus (PV) is a double-stranded DNA tumor virus infecting cervix, mouth, and throat tiss
63 NCE Papillomavirus (PV) is a double-stranded DNA tumor virus infecting the cutaneous and mucosal epit
64 f the best-studied oncoproteins encoded by a DNA tumor virus is adenovirus E1A, which modifies the fu
65 neral theme that has emerged from studies of DNA tumor viruses is that otherwise unrelated oncoprotei
66 i's sarcoma-associated herpesvirus (KSHV), a DNA tumor virus, is an etiological agent linked to sever
67                                          The DNA tumor virus Kaposi's sarcoma associated herpesvirus
68              Infection of cells with SV40, a DNA tumor virus known to abrogate formation of p130- and
69                  Simian virus 40 (SV40) is a DNA tumor virus known to induce cancers in laboratory an
70 ex, a hallmark of cellular transformation by DNA tumor viruses, leads to cell proliferation.
71 y that the oncoproteins encoded by the small-DNA tumor viruses may use this mechanism to induce c-Myc
72 his summer marks the 51st anniversary of the DNA tumor virus meetings.
73                                         Many DNA tumor virus oncogenes are capable of activating and
74 ymphoblastoid cell lines (LCLs) models human DNA tumor virus oncogenesis.
75 hat shares a subset of the properties of the DNA tumor virus oncoproteins but maintains important dif
76                                              DNA tumor virus oncoproteins reduce Rb function by eithe
77 t can physically interact with two mammalian DNA tumor virus oncoproteins, simian virus 40 large-T an
78                                 Unlike other DNA tumor virus oncoproteins, which possess immortalizin
79 critical for growth regulation interact with DNA tumor virus oncoproteins.
80 1 HR1 and HR3 and short regions of two other DNA tumor virus origin-binding proteins, SV40 T antigen
81                       In cells infected with DNA tumor viruses, p53 is bound to the viral tumor antig
82 TP-binding proteins in transformation by the DNA tumor virus polyomavirus, the GTP-binding activities
83                  Next, we found that SV40, a DNA tumor virus present in approximately 50% of mesothel
84                    The oncoproteins of small DNA tumor viruses promote tumorigenesis by complexing wi
85  is the first report of STAT activation by a DNA tumor virus protein.
86                               The ability of DNA tumor virus proteins to trigger apoptosis in mammali
87                    However, unlike the small DNA tumor virus proteins, individual HCMV IE proteins ta
88 ry cells in which RB has been inactivated by DNA tumor virus proteins.
89                            It is unclear how DNA tumor viruses regulate these enzymes and how these i
90  T Ag, which suggests yet another reason why DNA tumor viruses require actively cycling host cells.
91 therefore postulated that immortalization by DNA tumor viruses results in the induction of PTKs funda
92 is increasing evidence that the transforming DNA tumor virus simian virus 40 (SV40) is associated wit
93        Infection of quiescent cells with the DNA tumor virus simian virus 40 induces expression of th
94 ies with oncoproteins encoded by other small DNA tumor viruses such as adenovirus E1A and SV40 large
95 action can be disrupted by oncoproteins from DNA tumor viruses such as adenovirus E1a that bind p107.
96                            Oncoproteins from DNA tumor viruses such as adenovirus E1a, simian virus 4
97 ed as the binding site for oncoproteins from DNA tumor viruses such as adenovirus E1a.
98                                              DNA tumor viruses such as Kaposi's sarcoma-associated he
99                                        Small DNA tumor viruses such as simian virus 40 (SV40) and pol
100                                              DNA tumor viruses such as simian virus 40 (SV40) express
101             It is widely accepted that small DNA tumor viruses, such as adenovirus, simian virus 40 a
102 s (HCMV) and IE equivalents encoded by small DNA tumor viruses, such as adenovirus.
103                           Although the small DNA tumor virus SV40 (simian virus 40) fails to replicat
104 oma-associated herpesvirus (KSHV) is a human DNA tumor virus that causes Kaposi sarcoma and several o
105 and KS.IMPORTANCE Here we show that HHV-8, a DNA tumor virus that causes Kaposi's sarcoma, infects th
106 ociated herpesvirus (KSHV) is a lymphotropic DNA tumor virus that induces Kaposi's sarcoma and AIDS-r
107                        Adenovirus is a small DNA tumor virus that is a global human pathogen and key
108           Simian virus 40 (SV40) is a potent DNA tumor virus that is known to induce primary brain ca
109      Human papillomaviruses (HPVs) are small DNA tumor viruses that are the causative agent of warts
110                       Gammaherpesviruses are DNA tumor viruses that establish lifelong latent infecti
111                Gammaherpesviruses (GHVs) are DNA tumor viruses that establish lifelong, chronic infec
112 KSHV) and Epstein-Barr virus (EBV) are human DNA tumor viruses that express nuclear antigens [latency
113  also the binding site for oncoproteins from DNA tumor viruses that inactivate Rb.
114  The human papillomaviruses (HPVS) are small DNA tumor viruses that infect epithelial cells and induc
115            Human papillomaviruses (HPVs) are DNA tumor viruses that infect mucosal and cutaneous epit
116                 Thus, like many of the small DNA tumor viruses, the first protein expressed upon HCMV
117                    The mechanism employed by DNA tumor viruses to inhibit p53-dependent transcription
118 terpretation of the p53-Tag complexes and of DNA tumor virus transformation in general.
119 s is also the first demonstration of a human DNA tumor virus upregulating TLR3, a TLR that thus far h
120       In benign lesions induced by the small DNA tumor viruses, viral genomes are typically maintaine
121       The results indicate that unlike other DNA tumor viruses which block apoptosis by inactivation

 
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