ライフサイエンスコーパス: DTH

1 DTH lesions were initiated in adult rats by intracerebra

2 DTH reactions were elicited within 48 h in 16 of 29 untr

3 DTH response increased rapidly, to peak at 31-90 days af

4 DTH responses and CNS infiltration were reduced in prote

5 DTH responses in XX and XY FCG females showed no role fo

6 DTH responses to intradermal injections of HPV E7 antige

7 DTH responses to unmodified autologous melanoma were ind

8 DTH was assessed at 24, 48, 72, and 96 hours post bite i

9 A DTH response to bites was observed in 75% of individuals

10 rovision of exogenous TGF-beta or IL-10 at a DTH challenge site of allograft rejector mice caused a s

11 nhanced the ability of the mice to develop a DTH response after immunization with CneF-CFA, while ani

12 DnaK and GroEL2 for the capacity to induce a DTH response in the guinea pig model.

13 rst, rescue with either a wild-type DTH or a DTH mutant lacking neural expression leads to weak circa

14 om L. longipalpis saliva, does not produce a DTH response in these mice, suggesting that structural o

15 ype was defined as the ability to suppress a DTH response to a recall antigen in the presence of dono

16 Our findings demonstrate that a DTH reaction to a non-CNS antigen within a CNS white mat

17 dence, 19.8%) and are all caused by allergic DTH reactions.

18 ntibody significantly inhibited alloreactive DTH but did not prevent T cell priming or interferon-gam

19 (-) hosts failed to demonstrate allospecific DTH (P < 0.001).

20 ceiving B10.D2 grafts developed allospecific DTH.

21 T mice, as was the induction of allospecific DTH.

22 uvant in that myosin-specific antibodies and DTH responses both develop by 21 days postinfection or p

23 h necessary and sufficient for gastritis and DTH due to H. pylori in mice; 2) high expression of CD45

24 Gastritis and DTH were present in recipients of unfractionated splenoc

25 repertoires between individuals with IH and DTH reactions to Tri r 2.

26 pregulated in multiple sclerosis plaques and DTH lesions.

27 ing the relationships between protection and DTH reactions.

28 aive animals showed graft survival rates and DTH responses that were indistinguishable from those of

29 sured by de novo target-cell recognition and DTH priming, indicating that IN may be the preferred ROA

30 in cutaneous inflammation and angiogenesis, DTH reactions were investigated in the ear skin of wild-

31 sly that the presence of an anticryptococcal DTH response in mice is not always indicative of protect

32 occal cells (HKC), enhanced anticryptococcal DTH reactivity.

33 rotective and nonprotective anticryptococcal DTH responses are induced by different immunogens and ha

34 Even when the anticryptococcal DTH response was augmented by blocking the down-regulato

35 urately predicts time-related traits such as DTH in unobserved environments.

36 nt protein antigen tetanus toxoid as well as DTH responses were preserved in rituximab-treated RA pat

37 ts and showed the largest difference between DTH and IH responders in proliferation (mean standardize

38 induced within 2 weeks of immunization both DTH and IgG antibodies to guinea pig xenoantigens.

39 ossible to stimulate antigen-specific bovine DTH responses by using ESAT-6 in combination with a synt

40 Results from functional analyses by DTH, enzyme-linked immunospot, and immunohistofluorescen

41 inguished based on Th2 cytokine induction by DTH-associated major epitopes localizing to the amino-te

42 lografts and in donor alloantigen-challenged DTH sites in mice that have either accepted or rejected

43 microglial activation surrounding a chronic DTH EAE lesion.

44 The chronic DTH EAE lesion led to increased ligand binding in the ip

45 icroglial activation surrounding the chronic DTH EAE lesion.

46 the protective immunogen undergo a classical DTH response characterized by mononuclear cell and neutr

47 This treatment reduced T. cruzi DTH, although there was no effect on parasite-specific A

48 ferative responses in subjects with delayed (DTH), but not immediate (IH) hypersensitivity skin tests

49 The ability to depress DTH was transferred by naive spleen cells from transgeni

50 uPA(-/-) mice failed to develop DTH to SEA; did not polarize Ig production to IgE; did n

51 with 200 to 300 microg of peptide, developed DTH and EAU with scores comparable to those induced by 1

52 An alternative assay uses dithionite (DTH) to provide reduced Fd.

53 elated with the level of systemic anti-donor DTH, which varied over a 6-year interval.

54 ells into Bim-/- mice prevented an effective DTH response, thereby suggesting a causal link between a

55 ased type 1 cytokine production, an enhanced DTH response, and elevated production of CII-specific Ig

56 T cells at 5 days after infection, enhanced DTH, and increased mRNA levels for IFN-gamma in cornea a

57 cytokines thus leads to a state of enhanced DTH and depressed immediate-type hypersensitivity, which

58 A in inflammation, we evaluated experimental DTH reactions induced in the ear skin of transgenic mice

59 FN-gamma (IFN-gamma:IL-5 > or = 4:1) in five DTH subjects, even in the presence of Th2-dominated resp

60 that may contribute to differential footpad DTH responses using wildtype and four core genotypes (FC

61 splantation by challenging the recipient for DTH responses to donor alloantigen and evaluating the cy

62 guinea pigs that were indistinguishable from DTH responses driven by a PPD injection.

63 osure clearly distinguish this reaction from DTH.

64 By contrast, no 48-h DTH reactions occurred among 25 high risk and 32 low ris

65 Days to heading (DTH) is a crucial development stage in rice for regional

66 hile nadroparin treatment exhibited a higher DTH risk than dalteparin (hazard ratio [HR], 26.7; 95% C

67 UAS-mediated rescue of tyrosine hydroxylase (DTH) mutant (ple) flies to study the roles of dopamine i

68 , RT-PCR, and delayed-type hypersensitivity (DTH) analyses were used to examine the effects on bacter

69 osin-specific delayed-type hypersensitivity (DTH) and antibody production at 21 days postinfection.

70 osin-specific delayed-type hypersensitivity (DTH) and autoantibodies in the absence of detectable car

71 s in allergic delayed-type hypersensitivity (DTH) and bacterial chancroid skin lesions express both C

72 ermined using delayed-type hypersensitivity (DTH) and collagen-induced arthritis (CIA) models in mice

73 uppression of delayed type hypersensitivity (DTH) and in vivo lymphoproliferation correlated with MMc

74 had depressed delayed-type hypersensitivity (DTH) and splenocyte proliferative responses relative to

75 14 patients, delayed-type hypersensitivity (DTH) and/or CD4 proliferative responses developed after

76 Delayed-type hypersensitivity (DTH) assays demonstrate that CJ83193 can elicit durable

77 of measuring delayed-type hypersensitivity (DTH) following intradermal injection of recombinant solu

78 rum IgG2a, or delayed-type hypersensitivity (DTH) footpad reactions were detected.

79 gastritis and delayed-type hypersensitivity (DTH) in mice.

80 ry edema, and delayed-type hypersensitivity (DTH) in mice.

81 nor-specific, delayed-type hypersensitivity (DTH) in tolerant organ transplant recipients.

82 n Ag-specific delayed-type hypersensitivity (DTH) model in the cynomolgus macaque (Macaca fasciculari

83 revaccination delayed-type hypersensitivity (DTH) on the time course of the DTH response over 1-36 mo

84 nt model of a delayed-type hypersensitivity (DTH) reaction directed against the mycobacterium bacille

85 ve (anergic), delayed-type hypersensitivity (DTH) reaction to intradermal injection of purified prote

86 ty, toxicity, delayed-type hypersensitivity (DTH) reaction, and induction of serological and cellular

87 n a classical delayed-type hypersensitivity (DTH) reaction.

88 d by allergic delayed-type hypersensitivity (DTH) reactions and not by HIT or other rare conditions.

89 Delayed-type hypersensitivity (DTH) reactions elicited by the immunization were signifi

90 licitation of delayed-type hypersensitivity (DTH) reactions in vivo.

91 redominate in delayed-type hypersensitivity (DTH) reactions of the skin.

92 PAF inhibited delayed-type hypersensitivity (DTH) reactions to the chemical dinitrofluorobenzene only

93 ng psoriasis, delayed-type hypersensitivity (DTH) reactions, and rheumatoid arthritis.

94 and cutaneous delayed-type hypersensitivity (DTH) reactions.

95 e humoral and delayed-type hypersensitivity (DTH) reactions.

96 that induces delayed-type hypersensitivity (DTH) reactions.

97 d inhibit the delayed-type hypersensitivity (DTH) response caused by skin-homing effector memory T ce

98 duces a T(H)1 delayed-type hypersensitivity (DTH) response conferring protection against leishmaniasi

99 nduction of a delayed type hypersensitivity (DTH) response following exposure to L. longipalpis saliv

100 the effect of delayed-type hypersensitivity (DTH) response on clinical outcome.

101 a significant delayed-type hypersensitivity (DTH) response to adjuvant vaccine therapy (P =.0001), an

102 M) attenuated delayed-type hypersensitivity (DTH) response to methylated BSA and generation of Th17 c

103 op a vigorous delayed-type hypersensitivity (DTH) response upon intradermal virus antigen challenge.

104 Delayed-type hypersensitivity (DTH) response was detected in 7 of 10 patients for KLH a

105 se as being a delayed-type hypersensitivity (DTH) response with increased lung macrophage and Th1 cel

106 in vitro, the delayed-type hypersensitivity (DTH) response, and serum levels of CII-specific antibodi

107 s an impaired delayed-type hypersensitivity (DTH) response, manifested by a loss of skin testing to r

108 t resembles a delayed-type hypersensitivity (DTH) response.

109 onor-reactive delayed-type hypersensitivity (DTH) responses and donor-reactive alloantibody productio

110 to stimulate delayed-type hypersensitivity (DTH) responses in cattle experimentally infected with M.

111 ity to induce delayed-type hypersensitivity (DTH) responses in H37Rv-infected or BCG-vaccinated guine

112 dLAN dampened delayed type hypersensitivity (DTH) responses in male offspring.

113 ced increased delayed-type hypersensitivity (DTH) responses to autologous tumor cells in three patien

114 ine sites and delayed-type hypersensitivity (DTH) responses to autologous tumor cells indicative of T

115 stvaccination delayed-type hypersensitivity (DTH) responses to autologous tumor in 3 out of 14 treate

116 al effects on delayed type hypersensitivity (DTH) responses to donor antigens and type V collagen, an

117 The delayed-type hypersensitivity (DTH) responses to intradermal tuberculin were significan

118 titers and no delayed-type hypersensitivity (DTH) responses to Leishmania antigens.

119 We have used delayed-type hypersensitivity (DTH) responses to probe the mechanisms of drug-induced c

120 ecreased skin delayed-type hypersensitivity (DTH) responses to recall Ags in affected individuals wou

121 hat cutaneous delayed type hypersensitivity (DTH) responses to recall antigens are significantly decr

122 cific CTL and delayed-type hypersensitivity (DTH) responses were tested at the time of graft rejectio

123 allospecific delayed-type hypersensitivity (DTH) responses, and leukocytic infiltration of grafts we

124 and suppress delayed-type hypersensitivity (DTH) responses.

125 icited during delayed-type hypersensitivity (DTH) responses.

126 measure human delayed type hypersensitivity (DTH) responses.

127 n assays, and delayed-type hypersensitivity (DTH) responses.

128 igen-specific delayed-type hypersensitivity (DTH) responses.

129 Delayed-type hypersensitivity (DTH) responsiveness was assessed using the ear-swelling

130 at a distant delayed-type hypersensitivity (DTH) site would force the immune system to reveal its cu

131 PB83 elicited delayed-type hypersensitivity (DTH) skin test responses in 78% of naturally infected tu

132 by PV, and by delayed-type hypersensitivity (DTH) skin testing with PV (PV-DTH).

133 Delayed-type hypersensitivity (DTH) testing in vivo and Elispot analysis in vitro sugge

134 n of systemic delayed-type hypersensitivity (DTH) that is induced when Ags are introduced into the an

135 osin-specific delayed-type hypersensitivity (DTH) that normally develops in infected mice, although i

136 re tested for delayed-type hypersensitivity (DTH) to autologous melanoma cells, both DNP-modified and

137 ntibodies and delayed type hypersensitivity (DTH) to cardiac myosin.

138 magnitude of delayed-type hypersensitivity (DTH) to PPD in the skin.

139 ice developed delayed-type hypersensitivity (DTH) to SEA; high levels of serum immunoglobulin E (IgE)

140 ouse model of delayed-type hypersensitivity (DTH) to sMRBC.

141 Allospecific delayed-type hypersensitivity (DTH) was evaluated after transplantation in high-risk gr

142 ity as myosin delayed-type hypersensitivity (DTH) was reduced, while anti-myosin Ab production was no

143 ked cutaneous delayed-type hypersensitivity (DTH) was significantly enhanced in VPAC(2)R-null mice co

144 allospecific delayed-type hypersensitivity (DTH) were used as measures of alloreactivity.

145 e disease and delayed-type hypersensitivity (DTH), a convenient model for two-photon imaging of Tem c

146 allospecific delayed-type hypersensitivity (DTH), and cytokine expression were compared among the re

147 e in reducing delayed-type hypersensitivity (DTH), even in RAGE(-/-) mice by 50% (p < 0.001).

148 ctions, i.e., delayed-type hypersensitivity (DTH), in response to an intradermal injection of specifi

149 ling test for delayed-type hypersensitivity (DTH), in vitro proliferation assays, and cytokine assays

150 Delayed-type hypersensitivity (DTH), suggesting T-cell responses, was seen in 14 of 19

151 resulted in a delayed-type hypersensitivity (DTH)-like EAE lesion.

152 rine model of delayed-type hypersensitivity (DTH).

153 s measured by delayed-type hypersensitivity (DTH).

154 allospecific delayed-type hypersensitivity (DTH).

155 nd suppressed delayed-type hypersensitivity (DTH).

156 uppression of delayed-type hypersensitivity (DTH).

157 s, a modified delayed-type-hypersensitivity (DTH) protocol was developed to induce innate and adaptiv

158 ion (positive delayed-type hypersensitivity [DTH+]), or (iii) no evidence of infection (DTH-).

159 iferation and delayed type hypersensitivity [DTH]) and B-cell (antibody) responses specific to CM wer

160 displayed an only slight reduction of 16% in DTH, explained by compensatory reciprocal upregulation o

161 otpad, CD4(+) T cells were hyporesponsive in DTH to donor type HLA-B Ags and derivative allopeptides.

162 was accompanied by a progressive increase in DTH responsiveness.

163 lly, P5 induced IL-5 and IL-10 production in DTH, but not IH subjects (p = 0.003 (IL-5), p = 0.024 (I

164 double-knockout mice show a 27% reduction in DTH reaction.

165 h Leishmania major as well as a reduction in DTH responses to Leishmania Ag.

166 mall interfering RNA show a 35% reduction in DTH, and ALCAM(-/-) RAGE(-/-) double-knockout mice show

167 aa 41-60) induced the strongest response in DTH subjects and showed the largest difference between D

168 and DnaK/GroEL2/Rv0685, were found to induce DTH responses in H37Rv-infected or BCG-vaccinated guinea

169 BCG-induced DTH responsiveness appears to decline more rapidly in tr

170 in response to dinitrofluorobenzene-induced DTH that correlated with a reduced number of T cells in

171 leas and bed bugs, the strong saliva-induced DTH response may reflect an adaptation of the fly to man

172 D9 + CD81+ EVs that suppressed sMRBC-induced DTH in a miRNA-150-dependent manner.

173 y in the three patients with vaccine-induced DTH responses.

174 [DTH+]), or (iii) no evidence of infection (DTH-).

175 ShK-186, a specific Kv1.3 blocker, inhibited DTH and suppressed Tem cell enlargement and motility in

176 ereby providing a possible mechanism for its DTH-enhancing properties.

177 for a cell-mediated immune defect with lower DTH responses and macrophages that have a decreased abil

178 Using this new protocol, we measured DTH responses induced by killed bacteria or PHA in the p

179 In mice treated with imatinib mesylate, DTH was reduced in comparison to sham-injected controls.

180 Significantly more DTH-positive patients developed a recurrence than DTH-ne

181 WYE-151650 was efficacious in mouse DTH and CIA models.

182 ion of primed allogeneic T cells in a murine DTH model.

183 C57BL/6 mice did not develop cardiac myosin DTH upon immunization with T. cruzi extract.

184 els of myosin IgG and did not exhibit myosin DTH or myocarditis upon T. cruzi infection.

185 osin-immunized mice, the magnitude of myosin DTH in the two groups was statistically equivalent.

186 ly into the test site also suppressed myosin DTH.

187 14 of 19 volunteers receiving LT and CS6; no DTH was seen in subjects receiving CS6 alone.

188 0%] of six IV patients; P =.005) and de novo DTH (seven [87.5%] of eight IN patients v two [33.3%] of

189 y helps to improve the predictive ability of DTH in unobserved environments.

190 ervations was demonstrated by attenuation of DTH in WT and RAGE(-/-) animals pretreated with CD166/AL

191 Finally, the development of DTH was dependent on the schedule of administration of t

192 te population responsible for improvement of DTH responses were identified as being NK1.1 positive.

193 The inhibition of DTH reactions was abrogated by the addition of neutraliz

194 f pertussis toxin reversed the inhibition of DTH.

195 ligation and puncture demonstrated a loss of DTH for the 7 d following cecal ligation and puncture; h

196 sion or Bim deficiency prevented the loss of DTH.

197 ents who generated the greatest magnitude of DTH response and also displayed a strong clinical respon

198 monstrate the durability and T(H)1 nature of DTH to sand fly bites in humans living in a cutaneous le

199 Here, we describe the duration and nature of DTH to sand fly saliva in humans from an endemic area of

200 the thymus did not affect down-regulation of DTH.

201 The relevance of DTH to sand fly bites in humans living in a leishmaniasi

202 e findings raise questions about the role of DTH as an important mediator of cardiac allograft injury

203 we tested the hypothesis that suppression of DTH is mediated by tolerogenic properties of the apoptot

204 er, in contrast to the linked suppression of DTH seen when a given B-LCL expressed donor-type HLA-B a

205 on) and expression (efferent suppression) of DTH.

206 f this DTH-positive group was double that of DTH-negative patients (59.3% v 29.3%; P <.001).

207 Using a local adoptive transfer of DTH assay, we found that gamma delta T cells were requir

208 Furthermore, the treatment of DTH effector cells with sMRBC-induced EVs decreased the

209 Uncoupling of DTH reactivity and protection has been demonstrated in o

210 The effects of EV's action on DTH effector cells were evaluated cytometrically.

211 cy for classification of subjects into IH or DTH groups.

212 ptide recognition after > or =20 mo in IH or DTH subjects.

213 unized mice also exhibited a decrease in OVA DTH.

214 uency of cellular responses to the parasite (DTH).

215 bility of some persons to mount a persistent DTH response probably reflects genetic background and/or

216 The ability to maintain a positive DTH response to the C albicans skin test was comparable

217 cognition pattern correlated with a positive DTH test to normal kidney cells despite no evidence of i

218 In contrast, positive DTH responses to DNP-modified autologous melanoma cells

219 ly longer in patients who developed positive DTH to unmodified tumor cells (25.2% v 12.3%; P <.001).

220 ity as measured by increased postvaccination DTH responses against autologous tumors.

221 the single best predictor of postvaccination DTH.

222 mismatched heart transplant exhibited potent DTH, T-cell proliferation and antibody responses to CM a

223 vival with vaccine-DTH responses but not PPD-DTH indicates a treatment-specific effect.

224 Prevaccination DTH was the single best predictor of postvaccination DTH

225 more, RTL201 inhibited T cell proliferation, DTH responses, and cytokine mRNA expression in the eye,

226 range, 10.7 to 93.6 months), an increased PV-DTH response seemed to be associated with improved 5-yea

227 ersensitivity (DTH) skin testing with PV (PV-DTH).

228 Donor-reactive DTH responses were not detected in reconstituted SCID mi

229 (4) Donor-reactive DTH sites of allograft rejector mice displayed a broad a

230 he ability of intestinal helminths to reduce DTH responses may have clinical implications for the use

231 ternal and paternal exposure to dLAN reduced DTH responses in female offspring.

232 All three patients manifested regulated DTH responses to HA-1H peptide.

233 ty, indicating a role for strictly regulated DTH and dopamine in robust circadian rhythmicity.

234 individuals developed induration resembling DTH, and the cellular infiltrates contained monocytes an

235 ignificantly increased spleen Th1 responses, DTH reaction, and serum IgG2a levels along with decrease

236 are important for the induction of a robust DTH response.

237 cultures established from seven IH and seven DTH subjects.

238 h swine, which failed to mount a significant DTH response to CM and displayed low and transient anti-

239 e with donor alloantigens at a distant site (DTH challenge).

240 tween both mDC allogeneic responses and skin DTH responses with clinical disease severity as measured

241 rectly corresponded to the magnitude of skin DTH reactions to recall Ags in both sarcoidosis subjects

242 The CD8+ TE cells mediated HA-1-specific DTH and produced interferon-gamma.

243 pulation that suppressed an antigen-specific DTH response.

244 h cardiac myosin developed T. cruzi-specific DTH and antibodies.

245 O mice adoptively transferred donor-specific DTH and induced apoptosis of BALB/c corneal endothelial

246 ively transfer suppression of donor-specific DTH.

247 l grafts and exhibited strong donor-specific DTH.

248 ients tested with positive envelope-specific DTH.

249 In this case, myosin-specific DTH and Ab production were significantly reduced.

250 eatment also reduced cardiac myosin-specific DTH and antibody production.

251 status and induced mild guinea pig-specific DTH only after 5 weeks.

252 interleukin-10, which could restore a strong DTH response to donor B-LCL.

253 the protein cocktails tested induced strong DTH responses in H37Rv-infected guinea pigs.

254 expression of CD8, and capacity to suppress DTH, which is mediated by previously immunized T cells.

255 COL-3 suppressed DTH responses to donor antigens and type V collagen, abr

256 s in ICAM-1-/- mice, along with a suppressed DTH response to donor alloantigens after transplantation

257 In addition, Th2 clones suppressed DTH responses mediated by Th1 clones in vivo and blocked

258 P-1alpha but not KC significantly suppressed DTH and sharply reduced neutrophil accumulation in the e

259 ced a CD8+ regulatory T cell that suppressed DTH by TRAIL production.

260 ositive patients developed a recurrence than DTH-negative patients.

261 We conclude that DTH analysis can readily detect donor antigen-linked sup

262 These results indicate that DTH response against saliva provides most or all of the

263 The DTH response to C albicans was measured 2-3 days followi

264 iltration and gene expression profile at the DTH biopsy site corresponds to immunosuppression of an A

265 Although TNF-alpha is increased at the DTH reaction site in mice immunized with the nonprotecti

266 cells, and amount of gamma interferon at the DTH reaction site.

267 mor necrosis factor alpha (TNF-alpha) at the DTH reaction site.

268 Ex vivo phenotyping of T cells at the DTH site indicated that this response is mediated by act

269 ating the cytokine profiles displayed at the DTH site.

270 us retinal beta-gal expression depressed the DTH response and proliferation assays after beta-gal imm

271 In contrast, FICZ exacerbated the DTH response and promoted Th17 cells.

272 In the DTH model, assessment of T cell infiltration and gene ex

273 injury, there was a further decrease in the DTH response of aged injured mice, compared with aged sh

274 d what role, if any, neutrophils play in the DTH to a viral antigen.

275 e main leukocyte population infiltrating the DTH reaction site is the neutrophil.

276 l allow a more thorough understanding of the DTH response and may provide a platform for more rapid a

277 rsensitivity (DTH) on the time course of the DTH response over 1-36 months after vaccination were stu

278 neutrophils are a critical component of the DTH response to viral antigen.

279 , but not interleukin-10, at the site of the DTH response.

280 cytokine mRNA expression at the site of the DTH response.

281 Second, the DTH rescue strain deficient in neural dopamine selective

282 They are recruited to the DTH test site by MIP-2 and MIP-1alpha, where they can be

283 a broad array of cytokine mRNAs, whereas the DTH sites of allograft acceptor mice displayed only IL-4

284 2-week-old thymocytes did not improve their DTH response after immunization.

285 had significant apoptosis but retained their DTH responses.

286 This DTH was also antigen specific, since immunization with s

287 tokines establishes the T(H)1 nature of this DTH response.

288 The OS of this DTH-positive group was double that of DTH-negative patie

289 y than expected from heterozygous parents to DTH+ offspring (P = 0.0006), and haplotypes containing T

290 Four or eight weeks after transfer, DTH to H. pylori Ags was determined by footpad injection

291 First, rescue with either a wild-type DTH or a DTH mutant lacking neural expression leads to w

292 GFbeta) or interleukin-10 (IL10) can uncover DTH responses to donor alloantigens in cardiac allograft

293 The correlation of survival with vaccine-DTH responses but not PPD-DTH indicates a treatment-spec

294 MPB83, and MPB64 failed to stimulate in vivo DTH in cattle that had been experimentally infected with

295 Using the trans vivo DTH test, we identified four regulators, and four nonreg

296 The mechanism by which DTH responses were induced was elucidated by histologic

297 1), and OS was significantly correlated with DTH to vaccine (P =.0001) but not with DTH to purified p

298 profile in draining lymph nodes of mice with DTH, treatment with I3C and DIM decreased the expression

299 with DTH to vaccine (P =.0001) but not with DTH to purified protein derivative (PPD), a control anti

300 d between individuals with VL and those with DTH+ phenotypes.