ライフサイエンスコーパス: Desulfovibrio

1 may include sulfate-reducing species (e.g., Desulfovibrio).

2 increase of Ruminococcaceae and reduction of Desulfovibrio.

3 ing to Acetobacterium, Sulfurospirillum, and Desulfovibrio.

4 revealed a community shift toward the genus Desulfovibrio.

5 may include sulfate reducing species such as Desulfovibrio.

6 occocus_2 (-0.628; 95% CI: -0.91, -0.36) and Desulfovibrio (-2.05; 95% CI: -2.31, -1.78).

7 m (a sulfur oxidizer and NO3(-) reducer) and Desulfovibrio (a SO4(2-) reducer) become dominant in Sta

8 Desulfovibrio africanus strain Walvis Bay is a Hg-methyl

9 Desulfovibrio africanus strain Walvis Bay is an anaerobi

10 single historical precedent, the enzyme from Desulfovibrio africanus.

11 Desulfovibrio alaskensis G20 (formerly Desulfovibrio des

12 Consequently, we investigated the ability of Desulfovibrio alaskensis G20 to evolve perchlorate resis

13 nd in Desulfovibrio desulfuricans DSM642 and Desulfovibrio alaskensis G20, which use isethionate but

14 ative transcriptional and mutant analyses of Desulfovibrio alaskensis strain G20 and Desulfovibrio vu

15 uating methanogenic environments, we studied Desulfovibrio alaskensis strain G20 grown in chemostats

16 eptor availability was evaluated by studying Desulfovibrio alaskensis strain G20 under varying respir

17 n the sulphate-reducing Deltaproteobacterium Desulfovibrio alaskensis.

18 s in the genera Prevotella, Bifidobacterium, Desulfovibrio and Bacteroides and correlates with brain

19 rial populations, such as those of the genus Desulfovibrio and Bilophila.

20 ed dominance of Sulfurospirillum, Rhizobium, Desulfovibrio and four members of the Clostridiales fami

21 Expansion of Desulfovibrio and loss of Clostridia were key features a

22 hydrogenotrophic methanogens and syntrophic Desulfovibrio and the decrease of aceticlastic methanoge

23 ding Prevotella, Mitsuokella, Fusobacterium, Desulfovibrio, and bacteria belonging to the families Ru

24 sed abundance of Clostridium, Lactobacillus, Desulfovibrio, and Methylobacterium and an increased ten

25 ulin A targeting of Clostridia and increased Desulfovibrio antagonized the colonization of beneficial

26 criptomes showed that nitrogenase genes from Desulfovibrio bacteria were expressed in six samples sug

27 ell genomic amplicons from Desulfobulbus and Desulfovibrio (class Deltaproteobacteria) to better unde

28 ydrate intake was positively associated with Desulfovibrio (coefficient = 13.16; FDR-adjusted P = 0.0

29 ochromes c3 (Norway 4 and 9974) than for the Desulfovibrio (D.) gigas, D. vulgaris, and D. desulfuric

30 om the anaerobic sulfate-reducing bacterium, Desulfovibrio (D.) vulgaris (Hildenborough).

31 The steady state kinetics of a Desulfovibrio (D.) vulgaris superoxide reductase (SOR) t

32 In this context, the methylating SRB Desulfovibrio dechloracetivorans (strain BerOc1) was inc

33 dimers bind strongly to apo-flavodoxin from Desulfovibrio desulfuricans (30 degrees C, 20 mM Hepes,

34 of homodimeric desulfoferrodoxin (dfx) from Desulfovibrio desulfuricans (ATCC 27774).

35 Fe clusters in an Fe-S protein isolated from Desulfovibrio desulfuricans (ATCC 27774).

36 ic growth of the sulphate-reducing bacterium Desulfovibrio desulfuricans (strain G11) with hydrogen a

37 io vulgaris Hildenborough, but two copies in Desulfovibrio desulfuricans 27774, which can use nitrate

38 Importantly, electroactive and corrosive Desulfovibrio desulfuricans and Desulfovibrio vulgaris w

39 he inoculum, including Jonquetella anthropi, Desulfovibrio desulfuricans and Dialister invisus.

40 We show here that FMN binding to Desulfovibrio desulfuricans apo-flavodoxin is faster and

41 derstand this reaction, we analyzed a set of Desulfovibrio desulfuricans apoflavodoxin variants with

42 48-residue single-domain alpha/beta protein, Desulfovibrio desulfuricans apoflavodoxin.

43 O2 vary greatly; the [FeFe]-hydrogenase from Desulfovibrio desulfuricans ATCC 7757, an anaerobe, is i

44 One case of primary Desulfovibrio desulfuricans bacteremia in an immunocompe

45 This unique GRE is also found in Desulfovibrio desulfuricans DSM642 and Desulfovibrio ala

46 ermal unfolding of the apo and holo forms of Desulfovibrio desulfuricans flavodoxin, which noncovalen

47 In this work, the ability of Desulfovibrio desulfuricans formate dehydrogenase (Dd FD

48 Desulfovibrio desulfuricans G20 grows and reduces 20 mM

49 on insertion mutant has been identified in a Desulfovibrio desulfuricans G20 mutant library that does

50 of the type I tetraheme cytochrome c(3) from Desulfovibrio desulfuricans G20 was determined to 1.5 An

51 Desulfovibrio alaskensis G20 (formerly Desulfovibrio desulfuricans G20) is a Gram-negative meso

52 This study delves into how Desulfovibrio desulfuricans G20, a representative SRM, u

53 n mutant library containing 5,760 mutants of Desulfovibrio desulfuricans G20.

54 in two strains: Desulfovibrio sp. BerOc1 and Desulfovibrio desulfuricans G200 (which exhibit differen

55 The sulfate reducing bacterium Desulfovibrio desulfuricans inhabits both the human gut

56 The [FeFe] hydrogenase from Desulfovibrio desulfuricans is exceptionally active and

57 owever, when these particles were exposed to Desulfovibrio desulfuricans ND132 (a known Hg methylator

58 nd hgcB, required for mercury methylation by Desulfovibrio desulfuricans ND132 and Geobacter sulfurre

59 he FeRB Geobacter sulfurreducens and the SRB Desulfovibrio desulfuricans ND132 as model organisms, we

60 n of stable-isotope enriched (201)HgCl(2) by Desulfovibrio desulfuricans ND132 in short-term washed c

61 xperiments with the Hg-methylating bacterium Desulfovibrio desulfuricans ND132 to elucidate the role

62 educens PCA and a sulfate-reducing bacterium Desulfovibrio desulfuricans ND132 under nonsulfidic cond

63 Using a model Hg-methylating bacterium, Desulfovibrio desulfuricans ND132, we evaluated Hg-DOM-s

64 ic bacteria Geobacter sulfurreducens PCA and Desulfovibrio desulfuricans ND132.

65 g bacteria: Geobacter sulfurreducens PCA and Desulfovibrio desulfuricans ND132.

66 ylate dissolved elemental Hg(0) as shown for Desulfovibrio desulfuricans ND132.

67 i-inflammatory phenotype within 7 d, whereas Desulfovibrio desulfuricans resulted in a proinflammator

68 Desulfovibrio desulfuricans strain ND132 is an anaerobic

69 ducted on biofilm and planktonic cultures of Desulfovibrio desulfuricans strains M8 and ND132.

70 Desulfovibrio desulfuricans was isolated from the blood

71 ence of the sulfate-reducing bacterium (SRB) Desulfovibrio desulfuricans(T) (DSM-642).

72 mpylobacter coli, Campylobacter upsaliensis, Desulfovibrio desulfuricans, Desulfovibrio gigas, and De

73 hylators included Desulfobulbus propionicus, Desulfovibrio desulfuricans, Desulfovibrio magneticus an

74 Recombinant versions of Desulfovibrio desulfuricans, Desulfovibrio vulgaris and

75 rcina barkeri and the delta-purple bacterium Desulfovibrio desulfuricans, respectively, while the 12-

76 he homologous protein desulfoferrodoxin from Desulfovibrio desulfuricans.

77 nse of the cytochrome c nitrite reductase of Desulfovibrio desulfuricans.

78 hat resembled the H(trans) state of DdH from Desulfovibrio desulfuricans.

79 truncus, CC-115, Collinsella, Coprobacillus, Desulfovibrio, Dorea, Eubacterium, and Ruminococcus, whi

80 n of germ-free mice with Clostridia, but not Desulfovibrio, down-regulated genes that control lipid a

81 n Lactobacillus, Streptococcus, Clostridium, Desulfovibrio, Enterococcus, Fusobacterium, and several

82 ve not been identified to the species level, Desulfovibrio fairfieldensis and D. desulfuricans have b

83 cathodic electron uptake by Fe(0) -corroding Desulfovibrio ferrophilus IS5 and showed that electron u

84 ted using a lithotrophically-grown bacterium Desulfovibrio ferrophilus strain IS5, which is known to

85 Relative abundances changed for Desulfovibrio from 29.8% at PQO to 16.1% at GC.

86 mutants V74M L122A, V74M L122M, and V74M of Desulfovibrio fructosovorans [NiFe]-hydrogenase.

87 The heterodimeric [NiFe] hydrogenase from Desulfovibrio fructosovorans catalyzes the reversible ox

88 line of the -sensitive NiFe hydrogenase from Desulfovibrio fructosovorans.

89 The sulfate-reducing bacteria of the Desulfovibrio genus make three distinct modified tetrapy

90 SORs from Desulfovibrio gigas and Treponema pallidum showed simila

91 ional structures of the oxidised and reduced Desulfovibrio gigas cytochrome c(3) in solution were sol

92 parison of the [3Fe-4S]+ clusters in FdI and Desulfovibrio gigas ferredoxin II, refined at 1.7 A reso

93 nctions in the energy-yielding metabolism of Desulfovibrio gigas of the Bacteria domain.

94 d molybdoenzyme aldehyde oxidoreductase from Desulfovibrio gigas suggest that both enzymes utilize a

95 tallographically known aldehyde oxidase from Desulfovibrio gigas) and the higher pKa to substrate.

96 er upsaliensis, Desulfovibrio desulfuricans, Desulfovibrio gigas, and Desulfovibrio vulgaris, exhibit

97 s rubredoxins from Clostridium pasteurianum, Desulfovibrio gigas, Desulfovibrio vulgaris, and Pyrococ

98 ubredoxin:dioxygen oxidoreductase (ROO) from Desulfovibrio gigas.

99 e structure of a closely related enzyme from Desulfovibrio gigas.

100 QI0027(T) is the first Desulfovibrio human isolate for which nitrogen fixation

101 ic adaptive flexibility that likely sustains Desulfovibrio in naturally fluctuating methanogenic envi

102 Eleven previous cases of Desulfovibrio infection are reviewed; most arose from a

103 Antimicrobial susceptibilities of 36 Desulfovibrio isolates are presented.

104 of shoulder hemiarthroplasty infection with Desulfovibrio legallii.

105 sted strains of Campylobacter, Desulfomonas, Desulfovibrio, Leptotrichia, Mobiluncus, Peptostreptococ

106 ., and the s-OTUs Marinobacter, Alteromonas, Desulfovibrio, Limnobacter, Sphingomonas, Methyloversati

107 us propionicus, Desulfovibrio desulfuricans, Desulfovibrio magneticus and Geobacter sulfurreducens.

108 Here, we use Desulfovibrio magneticus RS-1 to uncover the mechanisms

109 he process of magnetite biomineralization in Desulfovibrio magneticus sp. RS-1, the only reported spe

110 Desulfovibrio magneticus sp. strain RS-1 forms bullet-sh

111 eviously observed in the anaerobic bacterium Desulfovibrio magneticus(6).

112 Here, we identify a single-subunit OST from Desulfovibrio marinus with relaxed substrate specificity

113 A-phylotype related to the sulphate-reducing Desulfovibrio oxamicus DSM1925, whereas the ArrA sequenc

114 fide production by the prevalent gut species Desulfovibrio piger increases the minimum inhibitory con

115 e set to better understand how the growth of Desulfovibrio piger is affected by, and affects the grow

116 spiration in E. coli only in the presence of Desulfovibrio piger, a sulfide-producing representative

117 vironmental factors that impact the niche of Desulfovibrio piger, the most common SRB in a surveyed c

118 M. smithii or the sulfate-reducing bacterium Desulfovibrio piger.

119 trite inhibited the dominating H2-scavenging Desulfovibrio population, and sustained the formation of

120 examined how anaerobes such as Bacteroides, Desulfovibrio, Pyrococcus and Clostridium spp. struggle

121 transformation of Hg species in two strains: Desulfovibrio sp. BerOc1 and Desulfovibrio desulfuricans

122 erium and an increase of Enterobacteriaceae, Desulfovibrio sp., and mainly Akkermansia muciniphila in

123 iniphilia and the hydrogen sulfide producing Desulfovibrio sp10575755 that was reduced with butyrate

124 ple tests were useful for characterizing the Desulfovibrio species and differentiating them from othe

125 The four Desulfovibrio species could be distinguished from each o

126 2S and were desulfoviridin positive, and all Desulfovibrio species except D. piger were motile.

127 to the 10-microg colistin disk separated the Desulfovibrio species from most of the other genera, whi

128 All Desulfovibrio species produced H2S and were desulfovirid

129 es implicated in syntrophic metabolism among Desulfovibrio species suggest considerable variation in

130 Genomic data from oral Desulfobulbus and Desulfovibrio species were compared to other available d

131 E. coli and Desulfovibrio spp levels (each associated with different

132 ntral role in the bioenergetic metabolism of Desulfovibrio spp.

133 that relative abundance of Azospira oryzae, Desulfovibrio, Stenotrophomonas, and Rhodocyclaceae was

134 ates the absence of a conserved gene core in Desulfovibrio that would determine the ability for a syn

135 um by increasing Lactobacillus and decreased Desulfovibrio The net effect of these changes was improv

136 uate the metabolic flexibility of syntrophic Desulfovibrio to adapt to naturally fluctuating methanog

137 The adaptation capability of Desulfovibrio to natural fluctuations in electron accept

138 r part and near the ectosymbiont 'Candidatus Desulfovibrio trichonymphae' in the anterior part of the

139 es (SS) were associated with enterobacteria, desulfovibrios, type E Clostridium perfringens, and Ente

140 obiota associated with inflammation, such as Desulfovibrio, Tyzzerella, and Lachnospiraceae_ge, and i

141 roquinones for one-electron reduction in the Desulfovibrio vulgaris ( D. vulgaris) flavodoxin ( E sq/

142 ssed from a gene-fusion with flavodoxin from Desulfovibrio vulgaris (2C9/FLD).

143 bane cluster in HdrB from a non-methanogenic Desulfovibrio vulgaris (Dv) Hildenborough organism.

144 (1.50-2.48 angstrom resolution) of CODH from Desulfovibrio vulgaris (DvCODH) heterologously expressed

145 e trinuclear center, bacterial ferritin from Desulfovibrio vulgaris (DvFtn) and its E130A variant was

146 used at the genetic level to flavodoxin from Desulfovibrio vulgaris (FLD) to create the chimeric CYP2

147 om the anaerobic sulfate-reducing bacterium, Desulfovibrio vulgaris (Hildenborough), has a hemerythri

148 on protein of unknown function isolated from Desulfovibrio vulgaris (Hildenborough).

149 The periplasmic hydrogenase of Desulfovibrio vulgaris (Hildenbourough) is an all Fe-con

150 -catalase oxidative stress defense system in Desulfovibrio vulgaris (strain Hildenborough).

151 in this work by examination of an engineered Desulfovibrio vulgaris 2Fe-SOR variant, C13S, in which o

152 ant versions of Desulfovibrio desulfuricans, Desulfovibrio vulgaris and Methanosarcina barkeri AhbA/B

153 cerevisiae to environmental microbes such as Desulfovibrio vulgaris and Shewanella oneidensis.

154 ndent pairings (cocultures) of the bacterium Desulfovibrio vulgaris and the archaeon Methanococcus ma

155 Using Desulfovibrio vulgaris as a model SRB organism, we compa

156 on and protein abundance data collected from Desulfovibrio vulgaris by DNA microarray and liquid chro

157 operties of the soluble C-terminal domain of Desulfovibrio vulgaris CcmE, dvCcmE'.

158 Cytochrome c-553 from Desulfovibrio vulgaris exhibits a highly exposed heme an

159 Two mutants of the Desulfovibrio vulgaris flavodoxin, T12H and N14H, were g

160 In the Desulfovibrio vulgaris flavodoxin, the elimination of th

161 nse regulators (RR) were identified from the Desulfovibrio vulgaris genome.

162 microarray and proteomic data collected from Desulfovibrio vulgaris grown under three different condi

163 lavodoxin from the sulfate-reducing bacteria Desulfovibrio vulgaris has been proposed, based on elect

164 s for two syntrophic cocultures, Dhc195 with Desulfovibrio vulgaris Hildenborough (-13.0 +/- 2.0 per

165 lms of sulfate-reducing bacterium (SRB) like Desulfovibrio vulgaris Hildenborough (DvH) can facilitat

166 le, most abundant multi-protein complexes in Desulfovibrio vulgaris Hildenborough (DvH) that are larg

167 pecific transcripts, causing a population of Desulfovibrio vulgaris Hildenborough (DvH) to collapse a

168 , we couple formate dehydrogenase (FDH) from Desulfovibrio vulgaris Hildenborough (DvH) to metal oxid

169 ogical, proteomic and biochemical studies of Desulfovibrio vulgaris Hildenborough and mutants affecte

170 hic co-culture containing lactate-fermenting Desulfovibrio vulgaris Hildenborough and solvent-dechlor

171 Desulfovibrio vulgaris Hildenborough belongs to a class

172 ytochrome c from Rhodobacter sphaeroides and Desulfovibrio vulgaris Hildenborough cytochrome c(3)).

173 We therefore tested a deposited strain of Desulfovibrio vulgaris Hildenborough for its capacity to

174 desulfothioredoxin (Dtrx) from the anaerobe Desulfovibrio vulgaris Hildenborough has been identified

175 Desulfovibrio vulgaris Hildenborough is a model organism

176 of the anaerobic, sulfate-reducing organism Desulfovibrio vulgaris Hildenborough to low-oxygen expos

177 The response of exponentially growing Desulfovibrio vulgaris Hildenborough to pH 10 stress was

178 The ability of Desulfovibrio vulgaris Hildenborough to reduce, and ther

179 ctivation of modA and modBC genes by TunR in Desulfovibrio vulgaris Hildenborough was confirmed in vi

180 istribution in central metabolic pathways of Desulfovibrio vulgaris Hildenborough was examined using

181 For the sulfate-reducing bacterium Desulfovibrio vulgaris Hildenborough, 347 of the 3634 ge

182 In Desulfovibrio vulgaris Hildenborough, a model bacterium

183 5' RNA sequencing to identify transcripts in Desulfovibrio vulgaris Hildenborough, a model sulfate-re

184 Desulfovibrio vulgaris Hildenborough, a sulfate-reducing

185 gene in the model sulfate-reducing bacterium Desulfovibrio vulgaris Hildenborough, but two copies in

186 o plays a role during fermentative growth of Desulfovibrio vulgaris Hildenborough, by studying two st

187 Sulfate-reducing microbes, such as Desulfovibrio vulgaris Hildenborough, cause "souring" of

188 reductase, formate dehydrogenase (FDH) from Desulfovibrio vulgaris Hildenborough, is interfaced with

189 nalysis of a model sulfate-reducing microbe, Desulfovibrio vulgaris Hildenborough, suggested the use

190 In the model sulfate-reducing microbe Desulfovibrio vulgaris Hildenborough, the gene DVU_0916

191 ion of cationic metabolites in the bacterium Desulfovibrio vulgaris Hildenborough.

192 a biomass hydrolysate of the soil bacterium Desulfovibrio vulgaris Hildenborough.

193 In this work, we studied FlxABCD of Desulfovibrio vulgaris Hildenborough.

194 2+) complex with the [NiFe]-hydrogenase from Desulfovibrio vulgaris immobilized on a functionalized e

195 owth of the model sulfate-reducing bacterium Desulfovibrio vulgaris in the absence of sulfate or a sy

196 Flavodoxin from Desulfovibrio vulgaris is a low molecular weight (15 000

197 Cytochrome c(553) (cyt c(553)) from Desulfovibrio vulgaris is a small helical heme protein t

198 e (57)Fe-labelled fully reduced Ni-R form of Desulfovibrio vulgaris Miyazaki F [NiFe]-hydrogenase.

199 Antibodies raised against Desulfovibrio vulgaris Rbr reacted with both native and

200 Phascolopsis gouldii hemerythrin (Pg-Hr) and Desulfovibrio vulgaris rubrerythrin (Dv-Rr), have been e

201 mixed-valent (Fe(2+),Fe(3+)) diiron site of Desulfovibrio vulgaris rubrerythrin (Rbr(mv)) were deter

202 The X-ray crystal structure of recombinant Desulfovibrio vulgaris rubrerythrin (Rbr) that was subje

203 asurement on a similar protein isolated from Desulfovibrio vulgaris showed that the protein contains

204 s of Desulfovibrio alaskensis strain G20 and Desulfovibrio vulgaris strain Hildenborough growing synt

205 The capacity of Desulfovibrio vulgaris to reduce U(VI) was studied previ

206 Stopped-flow mixing of the Desulfovibrio vulgaris two-iron superoxide reductase (2F

207 angstrom resolution cryo-EM structure of the Desulfovibrio vulgaris type I-C Cascade, revealing the m

208 tion, the properties of a beta-class CA from Desulfovibrio vulgaris were dramatically enhanced.

209 nd corrosive Desulfovibrio desulfuricans and Desulfovibrio vulgaris were identified within the biofil

210 re present in the sulfate-reducing bacterium Desulfovibrio vulgaris, although it grows only poorly on

211 Most of these operons were also conserved in Desulfovibrio vulgaris, an additional metal reducing org

212 ostridium pasteurianum, Desulfovibrio gigas, Desulfovibrio vulgaris, and Pyrococcus furiosus.

213 br(red) from the sulfate reducing bacterium, Desulfovibrio vulgaris, as well as its azide adduct (Rbr

214 revious work revealed how a sulfate reducer (Desulfovibrio vulgaris, Dv) and a methanogen (Methanococ

215 brio desulfuricans, Desulfovibrio gigas, and Desulfovibrio vulgaris, exhibited significantly relaxed

216 rmations using two genomes from each domain: Desulfovibrio vulgaris, Pseudomonas aeruginosa, Archaeog

217 scriptomic and proteomic data collected from Desulfovibrio vulgaris.

218 nctional urea transporter from the bacterium Desulfovibrio vulgaris.

219 rom the anaerobic sulfate-reducing bacterium Desulfovibrio vulgaris.

220 el for flavin binding to the flavodoxin from Desulfovibrio vulgaris.

221 MN) and riboflavin to the apoflavodoxin from Desulfovibrio vulgaris.

222 s on recombinant two-iron SOR (2Fe-SOR) from Desulfovibrio vulgaris.

223 lavodoxins from Clostridium beijerinckii and Desulfovibrio vulgaris.

224 corresponding with baseline adenomas, while Desulfovibrio was enriched both in stool and in mucosal

225 inical isolates representing four species of Desulfovibrio were characterized using 16S rRNA gene seq

226 s, formate dehydrogenase, and cytochromes of Desulfovibrio were found in high abundance near the elec

227 e relative abundances of Christensenella and Desulfovibrio were higher in the constipation group.

228 Sulfate-reducing bacteria from the genus Desulfovibrio, which have been implicated in microbially