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1 nsport of proteins in male and female flies (Drosophila melanogaster).
2 as invertebrates (Caenorhabditis elegans and Drosophila melanogaster).
3 mutations in ribosomal protein (Rp) genes in Drosophila melanogaster.
4 bodies from the first instar larva and adult Drosophila melanogaster.
5 incompatibilities (EGIs) in the model insect Drosophila melanogaster.
6 d, and compared with data on a population of Drosophila melanogaster.
7 to-cell heterogeneity in their expression in Drosophila melanogaster.
8 ageing in the antennal ears of the fruit fly Drosophila melanogaster.
9 mal organ size, regulates ovariole number in Drosophila melanogaster.
10 tion library for a developmental enhancer in Drosophila melanogaster.
11 hod for a low input sample, a single outbred Drosophila melanogaster.
12 we conducted a genetic suppressor screen in Drosophila melanogaster.
13 enance of male germline stem cells (GSCs) in Drosophila melanogaster.
14 the transition between hunger and satiety in Drosophila melanogaster.
15 disclosed a new in vivo function for USEs in Drosophila melanogaster.
16 resembles that of standard homing drives in Drosophila melanogaster.
17 zog (CG5830), through a multimodal screen in Drosophila melanogaster.
18 fected cells and to drive hyperlocomotion in Drosophila melanogaster.
19 anisms, including Caenorhabditis elegans and Drosophila melanogaster.
20 ring, is based on replication competition in Drosophila melanogaster.
21 dressed for small animals including the fly, Drosophila melanogaster.
22 insights into ovariole number regulation in Drosophila melanogaster.
23 ent changes in the metabolomes of long-lived Drosophila melanogaster.
24 e genes in their original genomic context in Drosophila melanogaster.
25 and female responses to dietary variation in Drosophila melanogaster.
26 Here, we address these problems using Drosophila melanogaster.
27 isease, we utilized a new, allelic series of Drosophila melanogaster.
28 the neuroprotective effects of PI3K in adult Drosophila melanogaster.
29 ressed bitter gustatory receptors (Grs) from Drosophila melanogaster.
30 largely unknown, even for the model species Drosophila melanogaster.
31 evoid of lamin and nuclear pore complexes in Drosophila melanogaster.
32 ions that give rise to the sensory organs of Drosophila melanogaster.
33 y this fundamental problem, we used the fly, Drosophila melanogaster.
34 ry that links egg laying to mating status in Drosophila melanogaster.
35 properties of the early olfactory system of Drosophila melanogaster.
36 of identified cell types in the HD system of Drosophila melanogaster.
37 ne neurons that predict behavioral choice in Drosophila melanogaster.
38 hygrosensory, and memory systems in the fly Drosophila melanogaster.
39 ents mating decisions in the brain of female Drosophila melanogaster.
40 erization of candidates were performed using Drosophila melanogaster.
41 ins, we screened for genetic interactions in Drosophila melanogaster.
42 es in behaviour compared to its close cousin Drosophila melanogaster(6,7), which are linked to its ex
44 cting PCB metabolism and toxicity, we tested Drosophila melanogaster, a well-known model system for g
45 s like the mammalian prostate and the paired Drosophila melanogaster accessory glands secrete seminal
46 sotope-resolved metabolomics to show that in Drosophila melanogaster, Acetobacter pomorum (Ap) and La
48 s (Oga(D133N) and Oga(KO) , respectively) in Drosophila melanogaster Adult Oga(D133N) and Oga(KO) fli
50 A polymerase II CTD phosphatase (Ssu72, from Drosophila melanogaster), an essential CTD phosphatase t
51 impacts of imidacloprid are investigated in Drosophila melanogaster, an experimental organism expose
52 s) genome-wide in mammalian cells as well as Drosophila melanogaster and Caenorhabditis elegans, indi
53 the traditional biomedical models zebrafish, Drosophila melanogaster and Caenorhabditis elegans, whic
55 ram and single sensillum recordings in adult Drosophila melanogaster and inhibited OR-mediated olfact
56 we discuss aspects of the circadian clock in Drosophila melanogaster and mammals, including the compo
57 rly or late in life, of both male and female Drosophila melanogaster and measure the effects on court
58 pressing it in the indirect flight muscle of Drosophila melanogaster and measuring alterations to mus
60 We purified the three SARAH domains from Drosophila melanogaster and performed an unbiased pulldo
61 functions have been extensively explored in Drosophila melanogaster and some other Dipteran species,
63 mulated data (calibrated from experiments on Drosophila melanogaster) and real data from five distinc
65 pathway has only been fully characterized in Drosophila melanogaster, and apoptosis-related proteins
66 tion-level data from a Zambian population of Drosophila melanogaster, and find that it has experience
68 ports have demonstrated that the fruit flies Drosophila melanogaster are capable of visual cue-driven
69 he Hippo pathway was initially discovered in Drosophila melanogaster as a key regulator of tissue gro
74 t advances in neurogenetics have highlighted Drosophila melanogaster as an exciting model to study ne
78 compounds were found to be nonmutagenic in a Drosophila melanogaster assay and exhibited a promising
79 ccessible method for non-invasive imaging of Drosophila melanogaster at high resolution using micro-c
80 res of human SERINC5 and its orthologue from Drosophila melanogaster at subnanometer and near-atomic
81 he whole adult brain (3.64 x 10(7) um(3)) of Drosophila melanogaster at the nanometer scale with high
82 an electron micrograph dataset for an entire Drosophila melanogaster brain, we reconstruct the first
84 initiation of subcellular lumen formation in Drosophila melanogaster, but not much is known on the wh
85 vision has been extensively characterised in Drosophila melanogaster, but substantially less is known
87 ition and organization of the centromeres of Drosophila melanogaster by combining long-read sequencin
88 manipulate the polyandry levels in groups of Drosophila melanogaster by deletion of the female sex pe
89 nonsynonymous mutations in humans, mice, and Drosophila melanogaster by examining patterns of polymor
90 nificantly prevent disease, whereas Smn from Drosophila melanogaster, Caenorhabditis elegans, and Sch
92 the serotonergic system in the vinegar fly, Drosophila melanogaster, can modulate walking speed in a
94 of Bruchpilot (Brp), a key component of the Drosophila melanogaster CAZ, participates in SV tetherin
95 uctures of the Schizosaccharomyces pombe and Drosophila melanogaster CENP-C cupin domains at 2.52 and
96 sexually dimorphic aIPg neurons in the adult Drosophila melanogaster central brain whose optogenetic
97 tron microscopy volume, we studied the adult Drosophila melanogaster circuitry associated with antenn
98 predictions, we generated a ClvR element in Drosophila melanogaster ClvR (tko) is located on chromos
99 e real-time dynamics of purified recombinant Drosophila melanogaster CMG unwinding DNA with single-mo
100 Moreover, neuronal overexpression of DCP1 in Drosophila melanogaster confers longevity in adults, whi
103 recent studies using Caenorhabditis elegans, Drosophila melanogaster, Danio rerio, and Mus musculus.
105 ats are transcribed in many cells throughout Drosophila melanogaster development, enriched in neurons
106 of Cell, Cassidy et al. (2019) show that, in Drosophila melanogaster, developmental abnormalities res
108 nt insights have been provided by studies of Drosophila melanogaster diversifying along the thermal g
109 s study cytokinesis regulators visualized in Drosophila melanogaster (Dm) cells were found to localiz
111 he T and Tn glycoform O-glycoproteome of the Drosophila melanogaster embryo, and determine that Miner
114 , calorimetry, and biochemical assays - that Drosophila melanogaster embryogenesis utilizes 10 mJ of
116 ping protein-protein interaction networks in Drosophila melanogaster embryos on a system-wide level.
117 ved version of Poly-Ribo-Seq and apply it to Drosophila melanogaster embryos to extend the catalog of
118 q datasets derived from Spiroplasma-infected Drosophila melanogaster embryos, to search for signals o
120 isualize the transcriptional dynamics of the Drosophila melanogaster even-skipped gene at single-cell
125 s from transgenic Caenorhabditis elegans and Drosophila melanogaster expressing fluorescent proteins.
126 functions demonstrated in isolated brains of Drosophila melanogaster expressing hDAT DeltaN336 and wi
128 sequestering Polo to the microtubules during Drosophila melanogaster female meiosis and suggest that
131 red an intracellular mechanism that prepares Drosophila melanogaster FGF Branchless (Bnl) for cytonem
133 base editing on the native Atpalpha gene in Drosophila melanogaster flies and retraced the mutationa
134 and courtship behavior were examined of wild Drosophila melanogaster flies isolated from two contrast
135 ion of recombinant Hb-ugt-1 (rHb-ugt-1) into Drosophila melanogaster flies resulted in diminished upr
137 ne the position of 27 sarcomeric proteins in Drosophila melanogaster flight muscles with a quasimolec
138 ess data from 202 fully sequenced hemiclonal Drosophila melanogaster fly lines to perform a genome-wi
139 eep and neural injury responses, we examined Drosophila melanogaster following the removal of antenna
140 lenge the male germline stem cells (GSCs) of Drosophila melanogaster for the production of specialize
142 vo genome assemblies for two wild strains of Drosophila melanogaster from the Drosophila Genetic Refe
144 high-quality, manually curated TE libraries: Drosophila melanogaster (fruit fly), Danio rerio (zebraf
145 PLD blocks ethanol-mediated hyperactivity in Drosophila melanogaster (fruit fly), demonstrating that
150 ments for the sequenced, inbred lines of the Drosophila melanogaster Genetic Reference Panel (DGRP) a
151 of recently collected isofemale lines in the Drosophila melanogaster Genetic Reference Panel (DGRP) c
152 (Entomophthoromycota), in 20 lines from the Drosophila melanogaster Genetic Reference Panel (DGRP).
154 se RNA-sequencing of BCBM development with a Drosophila melanogaster genetic screen, and identify Rab
155 ied more than 20,000 euchromatic SVs from 14 Drosophila melanogaster genome assemblies, of which ~40%
157 pressor alleles after P-elements invaded the Drosophila melanogaster genome in the mid-twentieth cent
164 ent methods of measuring these parameters in Drosophila melanogaster have low temporal resolution and
165 across the animal kingdom, where studies on Drosophila melanogaster have revealed that sleep phenoty
166 icase family is evolutionarily conserved, as Drosophila melanogaster have three family members: DmBlm
167 t cases of recent selection in the genome of Drosophila melanogaster Here we use a naive population a
169 infection model in the genetically amenable Drosophila melanogaster host, in which parasite spores o
170 et al. (2018) demonstrated in the fruit fly Drosophila melanogaster how an enzyme from specific gut
171 ity modulates the courtship behavior of male Drosophila melanogaster in an age-dependent manner.
172 of injured axons, dendrites, and synapses in Drosophila melanogaster In both male and female raw hypo
174 is decline have been explored intensively in Drosophila melanogaster in recent years and are now star
175 d mutation parameters that are realistic for Drosophila melanogaster In the presence of crossing over
176 d knockdowns in Saccharomyces cerevisiae and Drosophila melanogaster, including carriers, membrane ho
178 t cause different reproductive phenotypes in Drosophila melanogaster influence the mRNA transcriptome
180 mitochondrial protein trafficking pathway in Drosophila melanogaster involving the mitochondria-assoc
186 se patterns, we exposed larvae and adults of Drosophila melanogaster isogenic lines derived from a na
187 l-basal polarity and epithelial integrity in Drosophila melanogaster It is now clear that Scribble ac
188 dy, we used a recently isolated DNA virus of Drosophila melanogaster, Kallithea virus (KV; family Nud
190 transcription factor (bHLH)/PAS proteins in Drosophila melanogaster known as germ cell-expressed (Gc
191 us studies from our laboratory revealed that Drosophila melanogaster lacking RhoGAP18B and Ras Suppre
192 ays to obtain precise optogenetic control in Drosophila melanogaster larvae expressing the light-gate
193 us stimuli, such as parasitoid wasp attacks, Drosophila melanogaster larvae generate a curling and ro
194 rganisms, such as Caenorhabditis elegans and Drosophila melanogaster larvae, to investigate the genet
197 um for cells, Caenorhabditis elegans gonads, Drosophila melanogaster larval brain, mouse retina and b
199 on in wild populations of the model organism Drosophila melanogaster Like for most viruses discovered
200 n the alcohol dehydrogenase (ADH) protein of Drosophila melanogaster, like genetic variation in many
202 the dosage of fission and fusion genes in a Drosophila melanogaster loss-of-function model and found
204 de in vivo map of miRNA-mRNA interactions in Drosophila melanogaster, making use of single nucleotide
206 3, histone H4 is inherited asymmetrically in Drosophila melanogaster male germline stem cells undergo
207 ith fluorescent sperm labeling, we show that Drosophila melanogaster males exercise independent contr
211 ic alterations in the brains of an inducible Drosophila melanogaster model of AD expressing the Arcti
214 nteric diseases, we report on the use of the Drosophila melanogaster model to identify a novel functi
225 At the embryonic neuromuscular junction of Drosophila melanogaster, mutation or knockdown of many k
229 es but has also reportedly inserted into the Drosophila melanogaster nuclear export factor gene nxf2.
230 dy the neuronal circuitry that allows larval Drosophila melanogaster of either sex to negotiate this
231 documented the presence of the fruit fly or Drosophila melanogaster on alcohol-containing food sourc
232 tsetse fly antenna and that of the fruit fly Drosophila melanogaster One morphological type of sensil
233 ) resolution X-ray structural information on Drosophila melanogaster Orai in an open conformation (Ho
235 ene sets associated with development rate in Drosophila melanogaster Our transcriptional data also su
237 se deep genomic population sequencing of two Drosophila melanogaster populations to measure selection
238 uppression tactics have been well studied in Drosophila melanogaster, primarily during RNA, but not D
239 study the hydrophobic membrane motifs of two Drosophila melanogaster proteins, GPAT4 and ALG14, that
241 re, we make the unexpected discovery that in Drosophila melanogaster PTEN reduces PtdIns(4,5)P(2) lev
245 n during early nervous system development in Drosophila melanogaster requires precise regulation of g
247 SCAF4 ortholog CG4266 in the model organism Drosophila melanogaster resulted in impaired locomotor f
248 e-based RNA interference screen using stable Drosophila melanogaster S2 cells expressing the enhanced
252 Syntenic analysis of Glossina relative to Drosophila melanogaster shows reduced structural conserv
254 um falciparum microgametocytes and human and Drosophila melanogaster sperm), very little is known abo
256 ed in a population of fully sequenced inbred Drosophila melanogaster strains when flies developed in
258 e alphaPS3 integrin, scab) in the fruit fly (Drosophila melanogaster) suggests that gastrulation in t
259 ically manipulated the social environment of Drosophila melanogaster, testing individual flies and dy
260 more likely to encode essential functions in Drosophila melanogaster than ancient, conserved ZAD-ZNF
263 wo genetically modified invertebrate models (Drosophila melanogaster) that develop invasive or non-in
264 of the two evolutionarily conserved genes in Drosophila melanogaster, the authors found that their kn
268 In this study, a transcriptomic analysis in Drosophila melanogaster third instar larvae was carried
269 xpression of human EZHIP reduces H3K27me3 in Drosophila melanogaster through a conserved mechanism.
270 ine neuron loss and locomotor dysfunction in Drosophila melanogaster through an aberrant increase in
272 lbut virus, is common in wild populations of Drosophila melanogaster To begin to understand the proce
273 ) neurons in the optic lobe of the fruit fly Drosophila melanogaster to characterize divergent proper
275 used the pregrastrula-patterning network of Drosophila melanogaster to demonstrate that loss in accu
281 e developed a novel experimental paradigm in Drosophila melanogaster to study the mechanisms underlyi
284 rolled by SIN3 and SAM synthetase (SAM-S) in Drosophila melanogaster Using several approaches, includ
285 ection conferred by Spiroplasma to its host, Drosophila melanogaster varies with strain of attacking
286 that Chlamydomonas VIG1, an ortholog of the Drosophila melanogaster Vasa intronic gene (VIG), is req
287 ells and for mapping of neuronal activity in Drosophila melanogaster via genetically encoded Ca(2+) i
289 xamining the diversity of enteric neurons in Drosophila melanogaster, we identify a key role for gut-
290 of P-element derived transgene insertions in Drosophila melanogaster, we show here that raising flies
292 Here, using genetic techniques possible in Drosophila melanogaster, we tested the hypothesis that v
293 Or22a, and Or71a from the common fruit fly, Drosophila melanogaster, were recombinantly expressed, p
294 onal immediate early gene; and 3) in vivo in Drosophila melanogaster, where developmental exposures t
295 se body size is larger than the model insect Drosophila melanogaster, which enabled us to more easily
296 in vivo, we studied the unique Rh50 gene of Drosophila melanogaster, which encodes two isoforms, Rh5
297 recently published structure of spastin from Drosophila melanogaster, which forms a six-subunit spira
298 rformed targeted assays on selected genes in Drosophila melanogaster, which we suspected of being inv
300 of Drosophila simulans, a close relative of Drosophila melanogaster with which it co-occurs on both