1 143 nonduplicate Enterobacter isolates (
102 E. cloacae complex, 41 E. aerogenes) were tested, includ
2 After E. cloacae immunization, MZ B cells were responsible for
3 All E. cloacae complex strains, 89% of K. pneumoniae, and ha
4 lasmid was present in K. oxytoca,E. coli
and E. cloacae isolates from unlinked patients within the sa
5 Enterobacterales (K. pneumoniae, E. coli
and E. cloacae) harboured multiple cephalosporin and carbape
6 cking of particles and bacteria (E. coli
and E. cloacae) with developmental age and exposure to NEC s
7 es of M. morganii, C. freundii, E. coli,
and E. cloacae).
8 In both control
and E. cloacae colonized pigs, HuNoV infection of enterocyte
9 An outbreak of S. marcescens
and E. cloacae bacteremia in a surgical intensive care unit
10 two case patients yielded S. marcescens
and E. cloacae.
11 cter spp., P. aeruginosa, K. pneumoniae,
and E. cloacae.
12 complement-mediated killing of H. pylori
and E. cloacae, indicating its potential as a cross-species
13 The bacterial isolate was identified
as E. cloacae using a polyphasic approach that included phe
14 skin and skin-structure infections caused
by E. cloacae and K. pneumoniae, and for treatment of commu
15 ebsiella pneumoniae and Enterobacter
cloacae/
E. cloacae complex, the most commonly found isolates, we
16 culation of either wound model with E.
coli,
E. cloacae, or A. baumannii, produces the typical 'S'-sh
17 This
contrasted E. cloacae where there was no temporally changing patter
18 However, in our institution the MIC(90)
for E. cloacae bloodstream isolates is 16 microg/ml.
19 cal reevaluation of cefepime breakpoints
for E. cloacae may be prudent.
20 rogenes isolates and 6.0% of the results
for E. cloacae isolates tested with the Vitek system.
21 f the membrane-bound selenate reductase
from E. cloacae SLD1a-1.
22 However,
E. cloacae carries a chromosomally encoded ampC conferri
23 ts and shows that MDR plasmid acquisition
in E. cloacae was not indicative of success in the hospital
24 pigs, reduced HuNoV shedding was observed
in E. cloacae colonized pigs, characterized by significantl
25 PmrAB is dispensable for CAMP resistance
in E. cloacae.
26 nst several Gram-negative bacteria
including E. cloacae but not against Gram-positive bacteria.
27 Serratia
marcescens,
E. cloacae, and Enterobacter kobei presented high rates
28 bla genes revealed that one-third (15/45)
of E. cloacae bloodstream isolates produced SHV-type extend
29 lution) was achieved in only 76/114 (67%)
of E. cloacae isolates (65 susceptible, 11 resistant).
30 Colonization
of E. cloacae also reduced HuNoV titers in intestinal tissu
31 To test the influences
of E. cloacae on HuNoV infectivity and to determine whether
32 In this study involving clinical isolates
of E. cloacae and E. aerogenes, susceptibility testing meth
33 The beta-lactamase
of E. cloacae GC1, an extended spectrum mutant of the P99 e
34 also effectively blocked adhesion of pili
or E. cloacae, but no effect was observed with nonspecific
35 All but two of the ESBL-
producing E. cloacae isolates, but none of the non-ESBL-producing
36 The adhesion of
radiolabeled E. cloacae to HT-29 cells was concentration and temperat
37 sal population underlies multidrug-
resistant E. cloacae infections within hospitals.
38 systematic collection of multidrug-
resistant E. cloacae isolated between 2001 and 2011 from bloodstre
39 016 and 7 previously determined PB-
resistant E. cloacae isolates from JMI Laboratories.
40 In
summary,
E. cloacae inhibited HuNoV infectivity, and B cells were
41 We demonstrate
that E. cloacae lipid A is modified with l-Ara4N to induce CA
42 of H2O2 in the leaves of plants 3h after
the E. cloacae inoculation, according to a mechanism involvi
43 Unlike wild-type (WT) forms, such as
the E. cloacae P99 and Citrobacter freundii enzymes, the ES
44 he vast majority of clinical isolates of
the E. cloacae complex, predominantly in (sub)species that f
45 clinically most prevalent subspecies of
the E. cloacae complex.
46 determinant was highly conserved within
the E. cloacae complex and mediated resistance to up to 600
47 h indicates extensive dissemination of
these E. cloacae clones across the UK and Ireland.
48 methods were generally unreliable with
these E. cloacae isolates.
49 Thus E. cloacae NR is not a good candidate for enzymatic prod
50 of bleb-related endophthalmitis secondary
to E. cloacae infection.
51 ed production of DEX-specific Ab titers
upon E. cloacae rechallenge.
52 evalent pathogens causing EOS in Bukavu
were E. cloacae complex, K. pneumoniae, and S. marcescens.
53 Catheter infection
with E. cloacae occurred in 50% of roll plates and 80% of bro
54 in vivo, we colonized gnotobiotic pigs
with E. cloacae and inoculated pigs with 2.74 x 10(4) genome
55 r HuNoV in gnotobiotic pigs, with or
without E. cloacae colonization.