ライフサイエンスコーパス: E. faecalis

1 E. faecalis activates EGFR through production of H(2)O(2

2 E. faecalis also colonizes the female reproductive tract

3 E. faecalis co-exists with Escherichia coli and other pa

4 E. faecalis infections are associated with a high mortal

5 E. faecalis is intrinsically resistant to several antibi

6 E. faecalis MN1 inhibited interleukin-8 production from

7 E. faecalis must be able to adapt its physiology based o

8 E. faecalis tolerates high concentrations of GIT antimic

9 E. faecalis translocation and subsequent sepsis may be c

10 The virulence of 28 E. faecalis isolates representing 24 multilocus sequence

11 This revealed that 6 E. faecalis isolates behaved in a commensal manner with

12 Further analysis revealed that active E. faecalis physiology was important for inhibition of h

13 We adapted E. faecalis OG1RF (a laboratory strain) and S613TM (a cl

14 ameloriated with antibiotics active against E. faecalis.

15 showed complete bacteriocin activity against E. faecalis, but neither BacL1 nor BacA protein alone sh

16 ely express the bacteriocin activity against E. faecalis.

17 esent study, we show that antibodies against E. faecalis LTA also bind to type 1 LTA from other gram-

18 immunization with rabbit antibodies against E. faecalis LTA promoted the clearance of bacteremia by

19 single very major error was obtained against E. faecalis, while vancomycin-intermediate S. aureus (VI

20 eover, the sera conferred protection against E. faecalis and E. faecium strains in a mouse infection

21 herapeutic and preventive strategies against E. faecalis and beyond.

22 ironmental lytic phages with tropism against E. faecalis, we found that these phages require the ente

23 All E. faecalis isolates were judged to be S by the referenc

24 prgU gene pairs are widely distributed among E. faecalis isolates and other enterococcal and staphylo

25 eemed to be a strain-independent trait among E. faecalis isolates.

26 crobial peptide activity developed either an E. faecalis or Pseudomonas aeruginosa urinary tract infe

27 Finally, a screen of an E. faecalis transposon mutant library identified other g

28 Finally, we show that an E. faecalis epa mutant strain is deficient in intestinal

29 Colonization with an E. faecalis strain carrying a conjugation-defective pPD1

30 (LOD) determined for HAdV 41, Phi X 174, and E. faecalis was 35 GU/muL, 1 GU/muL, and 5 x 10(3) GU/mu

31 Furthermore, S. aureus and E. faecalis form biofilms normally without polyamines, a

32 require polyamines for growth, S. aureus and E. faecalis grow normally over multiple subcultures in t

33 yamine N-acetyltransferases in S. aureus and E. faecalis represent a new paradigm for bacterial polya

34 stem of primary colonic epithelial cells and E. faecalis-infected macrophages (in vitro), with an int

35 )) and two AR bacterial strains (E. coli and E. faecalis, at 10(5) CFU mL(-1)) were spiked in real WW

36 major role in control of ace expression and E. faecalis virulence.

37 tion of Staphylococcus spp., E. faecium, and E. faecalis and its ability to ascertain mecA, vanA, and

38 rial carbohydrate metabolism in general, and E. faecalis PTS-gluconate in particular, during inflamma

39 antimicrobial activity for P. gingivalis and E. faecalis.

40 mmon NICU organisms including K. oxytoca and E. faecalis and increases in common adult organisms incl

41 2) For L. plantarum, S. pyogenes, and E. faecalis, the effects of Pi are distinguishable from

42 ) homologues encoded by S. aureus USA300 and E. faecalis acetylate spermidine, spermine and norspermi

43 tioned media (CCM) from E. faecalis V583 and E. faecalis lacking the gelatinase gene (gelE).

44 n of solar exposure for AR E. coli, while AR E. faecalis was more resistant to the disinfection proce

45 splacement assays using PGTs from S. aureus, E. faecalis, and E. coli.

46 l activity against VanA resistant bacteria ( E. faecalis , VanA VRE) at a level accurately reflecting

47 dy aimed to evaluate the association between E. faecalis and oral cancer and to determine the underly

48 sues suggests a possible association between E. faecalis infection and oral carcinogenesis.

49 A link between E. faecalis and EGFR signaling in oral cancer has not be

50 ) and EGFR (gefitinib) significantly blocked E. faecalis-induced EGFR activation and cell proliferati

51 phonuclear neutrophils to readily clear both E. faecalis and multidrug-resistant E. faecium.

52 differentiation of DCs were not affected by E. faecalis, expression of the autophagy-related protein

53 LTA promoted the clearance of bacteremia by E. faecalis and S. epidermidis in mice.

54 h culture-positive endophthalmitis caused by E. faecalis between January 1, 2002, and December 31, 20

55 quired for pathogenesis, GIT colonization by E. faecalis is poorly understood.

56 in contributes to intestinal colonization by E. faecalis.

57 anii actively prevents urease enhancement by E. faecalis, P. stuartii, and E. coli Importantly, these

58 g the bioactivities of peptides generated by E. faecalis.

59 M1M were not induced by E. faecalis Ag in cultures of I-MLNM, whereas normal mou

60 he mechanism of lectin pathway inhibition by E. faecalis, we purified and characterized cell wall car

61 etramic acid compounds naturally produced by E. faecalis MN1, may be useful in prevention of diseases

62 acteriocin 41 (Bac41) is produced by certain E. faecalis clinical isolates, and it is active against

63 To identify and characterize E. faecalis genes that are key to intestinal colonizatio

64 porcine gut in response to diets containing E. faecalis were similar to the response to which contai

65 Our findings link cytolytic E. faecalis with more severe clinical outcomes and incre

66 eriophages can specifically target cytolytic E. faecalis, which provides a method for precisely editi

67 ects of bacteriophages that target cytolytic E. faecalis.

68 e presence of cytolysin-positive (cytolytic) E. faecalis correlated with the severity of liver diseas

69 or the 2 sera was observed against different E. faecalis and E. faecium strains.

70 value: 37 nM) and successfully discriminated E. faecalis from 20 different Enterococcus and non-Enter

71 lineages were predominant among endocarditis E. faecalis isolates recovered during this time period.

72 on prior to urinary catheterization enhanced E. faecalis colonization, suggesting that implant-mediat

73 bacterial composition in the colon, enhanced E. faecalis survival within macrophages, and increased p

74 cally and phylogenetically the environmental E. faecalis were indistinguishable from their enteric co

75 of this phage and show that it can eradicate E. faecalis biofilms formed in vitro on a standard polys

76 rainwater tank samples followed by AS, esp (E. faecalis variant), and cylA genes which were detected

77 We also evaluated E. faecalis interactions with human platelets and found

78 mouse vaginal colonization model to explore E. faecalis vaginal carriage and demonstrate that both v

79 PTS permits growth in gluconate, facilitates E. faecalis intestinal colonization, and exacerbates col

80 vior of two model FIB Enterococcus faecalis (E. faecalis) and Escherichia coli (E. coli) was examined

81 the present study, an Enterococcus faecalis (E. faecalis) DNA biosensor (ef-biosensor) was fabricated

82 Enterococcus faecalis (E. faecalis) is one of the indicator bacteria that have

83 nii (S. gordonii) and Enterococcus faecalis (E. faecalis) were frequent isolates after treatment with

84 ch is demonstrated on Enterococcus faecalis (E. faecalis), which served as target in eleven rounds of

85 of microbiota in the distal gut of pigs fed E. faecalis UC-100 substituting antibiotics, this study

86 re generated in MLNs of these mice following E. faecalis stimulation.

87 For E. faecalis strains, only GS exhibits a rapid bactericid

88 For E. faecalis, results were similar in original water samp

89 he ef-biosensor was successfully applied for E. faecalis detection in human samples.

90 t identification of genetic determinants for E. faecalis commensal and pathogenic interactions with M

91 ence of the catheter itself is essential for E. faecalis persistence in the bladder.

92 PCR/ESI-MS again found genetic evidence for E. faecalis at levels comparable to the pretreatment lev

93 ibrary to identify novel genes important for E. faecalis colonization and persistence in the vaginal

94 inhibitory concentration was 0.57 mg/ml for E. faecalis and 1.15 mg/ml for all the other strains.

95 aureus ATCC 29213, 0.016 to 0.12 mug/ml for E. faecalis ATCC 29212, 0.008 to 0.03 mug/ml for S. pneu

96 0.1 mM), the toxicity of 10 mg L(-1) PCP for E. faecalis and fresh anaerobic sludge was detected in 1

97 nce of OG1RF_12399-12402 is not required for E. faecalis colonization of the mouse intestine but is a

98 m, 87%, 83%, 98%, and 80%, respectively, for E. faecalis, and all 100% for S. aureus.

99 mosomal genetic determinants responsible for E. faecalis biofilm-mediated infection, we used a rabbit

100 ding these observations, colon biopsies from E. faecalis-colonized IL-10(-/-) mice exhibited crypt hy

101 to concentrated conditioned media (CCM) from E. faecalis V583 and E. faecalis lacking the gelatinase

102 he mevalonate diphosphate decarboxylase from E. faecalis (MDDEF).

103 ur investigations demonstrate that GelE from E. faecalis can regulate enteric epithelial permeability

104 Both the homologous gene from E. faecalis V583 (EF1861) and E. coli panE functionally

105 Gelatinase (GelE) purified from E. faecalis V583 was used to confirm the ability of this

106 We generated E. faecalis strains containing deletions/point mutations

107 that can be exploited to alter heterogeneous E. faecalis populations.

108 s study is the first to report heterogeneous E. faecalis bacteremia.

109 lowing disruption of intestinal homeostasis, E. faecalis can overgrow, cross the intestinal barrier,

110 The present study examined how E. faecalis influences the differentiation of murine bon

111 ors required for commensalism, we identified E. faecalis genes that are upregulated in the gut of M.

112 the absence of the silicone tubing implant, E. faecalis induced only minimal inflammation and was ra

113 ry response induced by urinary implantation, E. faecalis produced biofilm and high bladder titers in

114 This antiapoptotic activity in E. faecalis-infected cells was dependent on the activati

115 ying overproduction of PrgB-like adhesins in E. faecalis and other clinically-important Gram-positive

116 cci: (i) the accretion of mobile elements in E. faecalis V583 renders it incompatible with commensal

117 e virulence map that explains enhancement in E. faecalis virulence and contributes to a deeper compre

118 EF2638 represents an authentic Rex factor in E. faecalis that influences the production or detoxifica

119 racytoplasmic function (ECF) sigma factor in E. faecalis, and that the deletion of sigV increases the

120 ratio of occurrence of ace and gelE genes in E. faecalis was much higher at 7.96 and 6.40 times, resp

121 rrence of ace, gelE, efaA, and asa1 genes in E. faecalis were found to be much higher compared to the

122 We have in E. faecalis monitored production of the enzyme polypepti

123 oly(ADP-ribose) polymerase were inhibited in E. faecalis-infected cells, indicating that E. faecalis

124 bpB, and EbpC) have not been investigated in E. faecalis.

125 egulating expression of proteins involved in E. faecalis carbohydrate uptake and utilization.

126 that EfbA is an important factor involved in E. faecalis endocarditis and that rEfbA immunization is

127 of tetracycline-resistance plasmid pCF10 in E. faecalis.

128 protection against antimicrobial peptides in E. faecalis at a significant cost in bacterial fitness.

129 duction of conjugation by a sex pheromone in E. faecalis.

130 hway is a phosphotransferase system (PTS) in E. faecalis strain OG1RF that phosphorylates gluconate a

131 to investigate the role of this receptor in E. faecalis-induced permeability.

132 al mechanisms of cephalosporin resistance in E. faecalis is lacking.

133 rocally regulate cephalosporin resistance in E. faecalis, dependent on the kinase activity of IreK.

134 inants of cholate and lysozyme resistance in E. faecalis, IreK was the only one found to be required

135 ant regulator of antimicrobial resistance in E. faecalis.

136 ive regulator of cephalosporin resistance in E. faecalis.

137 dence of in vivo-expressed antisense RNAs in E. faecalis.

138 factor beta1 [TGF-beta1]) were suppressed in E. faecalis-induced DCs, while IL-1beta, tumor necrosis

139 l teichoic acid (WTA) synthesis gene tagB in E. faecalis V583 that exhibited an increased susceptibil

140 Our study indicates that in E. faecalis, modification of peptidoglycan by secondary

141 anding of how (p)ppGpp promotes virulence in E. faecalis and other bacterial pathogens is still lacki

142 ng system, a major regulator of virulence in E. faecalis.

143 oxidative stress tolerance and virulence in E. faecalis.

144 imicrobial resistance for bacteria including E. faecalis, E. coli, K. pneumoniae and S. aureus.

145 3) Addition of Pi inhibits E. faecalis LDH2, whereas in the absence of FBP, Pi is a

146 To investigate E. faecalis factors required for commensalism, we identi

147 FBP, Pi is an activator of S. pyogenes LDH, E. faecalis LDH1, and L. lactis LDH1 and LDH2 at pH 6.

148 etermine the underlying mechanisms that link E. faecalis to EGFR signaling.

149 faecalis LDH2 </= Lactococcus lactis LDH2 < E. faecalis LDH1 < L. lactis LDH1 </= Streptococcus pyog

150 ated with enhanced survival from M. luteus + E. faecalis infection.

151 ssociate CcpA with the production of a major E. faecalis virulence factor, providing new insights int

152 The pathogenesis of many E. faecalis infections, including endocarditis and cathe

153 owever, little is known about the mechanisms E. faecalis uses to colonize and compete for stable gast

154 In unsaturated porous media, E. faecalis cells seemed to prefer to attachment at air/

155 sses, we hypothesized that (p)ppGpp mediates E. faecalis virulence through regulation of metal homeos

156 merous animal models have been used to mimic E. faecalis infections, but none of them is considered i

157 thogenic E. coli isolates and (10(0) CFU/mL) E. faecalis and E. faecium strains were detected within

158 Monomicrobial E. faecalis bacteremia (hazard ratio [HR], 3.60; 95% con

159 Monomicrobial E. faecalis bacteremia, community acquisition, prostheti

160 ys were orally infected with 10(6) CFU/mouse E. faecalis.

161 wing exposure to CCM from parental or mutant E. faecalis strains indicated paracellular permeability.

162 a heptapeptide pheromone produced by native E. faecalis present in the fecal consortium.

163 Following vaginal colonization, we observed E. faecalis in vaginal, cervical, and uterine tissue.

164 bp expression was restricted to 30 to 72% of E. faecalis cells, consistent with a bistability mode of

165 Analyses of E. faecalis mutants exhibiting defects in antimicrobial

166 comprehensive functional genomic analysis of E. faecalis.

167 ntified signatures suggest that ancestors of E. faecalis resided in extra-enteric habitats, challengi

168 and characterized cell wall carbohydrates of E. faecalis wild type and V583DeltatagB.

169 ollection is not a random sample of cases of E. faecalis endocarditis, these results indicate that no

170 s study, we investigated the contribution of E. faecalis to mixed-species infection when iron availab

171 ns were selected from a wild-type culture of E. faecalis OG1RF.

172 , each with less than 28 SNP differences, of E. faecalis and E. hormaechei.

173 Here, we show that the dynamics of E. faecalis communities exposed to antibiotics can be su

174 ults provide the first evidence that EfbA of E. faecalis plays a role in UTIs, probably contributing

175 Much of the negative effect of E. faecalis on C. albicans was due to the inhibition of

176 PCR/ESI-MS detected genetic evidence of E. faecalis in all CSF samples, but the level of detecti

177 ollected from the peritoneal lavage fluid of E. faecalis-infected mice showed reduced levels of apopt

178 g activity against the cell wall fraction of E. faecalis in the absence of BacA.

179 We report the high frequency of E. faecalis infection in oral tumors and the clinical as

180 ith human platelets and found that growth of E. faecalis in BHI plus serum significantly enhanced adh

181 We have also shown that growth of E. faecalis in brain heart infusion (BHI) serum enhances

182 One of the hallmarks of E. faecalis pathogenesis is its unusual ability to toler

183 calis isolates underscores the importance of E. faecalis as a reservoir of VGs in the fresh water aqu

184 Incubation of E. faecalis with heme increased growth and restored cata

185 Interaction of E. faecalis with host cells and production of H(2)O(2) i

186 were observed for daptomycin in isolates of E. faecalis and 2 ME, 1 for high-level gentamicin resist

187 c hepatitis have increased faecal numbers of E. faecalis.

188 The pathogenesis of E. faecalis infection relies in part on its capacity to

189 and is implicated in the pathophysiology of E. faecalis.

190 so report the GIT colonization phenotypes of E. faecalis mutants lacking selected sortase-dependent p

191 heromone-responsive, conjugative plasmids of E. faecalis have retained Prg-like surface functions ove

192 In addition, the composition ratio of E. faecalis could be decreased in multispecies microecol

193 roviding new insights into the regulation of E. faecalis pathogenesis.

194 The resistance of E. faecalis to several classes of antibiotics and its ca

195 To study the role of E. faecalis Rex, we purified EF2638 and evaluated its DN

196 probe was selected from 16S rRNA sequence of E. faecalis and immobilized on a gold electrode surface

197 We undertook whole genome sequencing of E. faecalis associated with bloodstream infection in the

198 gh-level aminoglycoside resistance status of E. faecalis.

199 Thus, we hypothesized that this strain of E. faecalis may make anti-inflammatory factors which blo

200 f nematodes exposed to pathogenic strains of E. faecalis and Staphylococcus aureus, including multidr

201 The 70S ribosome structure of E. faecalis now extends our knowledge of bacterial ribos

202 ependent cleavage of Ace from the surface of E. faecalis, confirming that GelE specifically reduces A

203 es the in vitro antibiotic susceptibility of E. faecalis isolated from periodontitis patients in the

204 cultures containing Enterococcus faecium or E. faecalis was 100% sensitive and specific.

205 in prostates challenged with either UPEC or E. faecalis was equal or greater than primary infection

206 cal isolates, and it is active against other E. faecalis strains.

207 ABC mutants, suggesting involvement of other E. faecalis factors in urinary tract colonization or inf

208 the intestine, pPD1 is transferred to other E. faecalis strains by conjugation, enhancing their surv

209 s investigation indicated that, unlike other E. faecalis virulence traits, phage03-like elements were

210 faecalis V583 during competition with other E. faecalis strains in vitro and in vivo.

211 laces indigenous enterococci and outcompetes E. faecalis lacking pPD1.

212 n and epithelial translocation of pathogenic E. faecalis during severe microbial dysbiosis and was am

213 Periodontal E. faecalis exhibited substantial in vitro resistance to

214 itro inhibitory activity against periodontal E. faecalis, and may be clinically useful in treatment o

215 and teicoplanin-susceptible (VanB phenotype) E. faecalis were responsible for major and minor errors.

216 The false-positive E. faecalis strains were typed by Diversilab Rep-PCR (bi

217 tential targets for interventions to prevent E. faecalis infections.

218 is known about specific factors that promote E. faecalis vaginal colonization and subsequent infectio

219 (-/-) mice attenuated infection and promoted E. faecalis colonization resistance by restoring the div

220 Although factors promoting E. faecalis colonization of intestines are not fully kno

221 Upon hymeglusin inactivation of purified E. faecalis mvaS, the thioester adduct is more stable th

222 sistance by electrostatic repulsion, renders E. faecalis more resistant to killing by defensins and l

223 treat a broad range of antibiotic-resistant E. faecalis infections.

224 eventing colonization by multidrug-resistant E. faecalis could therefore be a valuable approach towar

225 As a result, E. faecalis showed the least affinity to sand in freshwa

226 Secreted E. faecalis proteins induced permeability in epithelial

227 Secreted E. faecalis proteins induced permeability in the colonic

228 o bind putative promoter segments of several E. faecalis genes in an NADH-responsive manner, indicati

229 In Manduca sexta, E. faecalis is an infrequent member of the commensal gut

230 The mobility of the gram-positive species E. faecalis was much more sensitive to solution chemistr

231 A specific E. faecalis DNA probe was selected from 16S rRNA sequenc

232 in gelatinase-and-cytolysin-negative strain E. faecalis JH2-2.

233 fish is a novel, powerful model for studying E. faecalis pathogenesis, enabling us to dissect the mec

234 Pure cultures of 47 subgingival E. faecalis clinical isolates were each inoculated onto

235 in vitro activity against human subgingival E. faecalis clinical isolates, and would likely be ineff

236 ure of vancomycin-resistant and -susceptible E. faecalis (VSEfs), which has important implications fo

237 lation of several bacteriophages that target E. faecalis strains isolated from the oral cavity of pat

238 ents was more effective against E. coli than E. faecalis.

239 ne is the more preferred substrate, and that E. faecalis SSAT is almost as efficient as human SSAT wi

240 ically in the presence of gluconate and that E. faecalis strains lacking, or harboring a single point

241 It is concluded that E. faecalis promotes the differentiation of bone marrow

242 t the clinical findings and demonstrate that E. faecalis can induce EGFR activation and cell prolifer

243 polymicrobial infection, we discovered that E. faecalis and C. albicans negatively impact each other

244 E. faecalis-infected cells, indicating that E. faecalis protects macrophages from apoptosis by inhib

245 Interestingly, we observe that E. faecalis transiently maintains CRISPR targets despite

246 Thus, we propose that E. faecalis V583 uses phage particles to establish and m

247 Here we show that E. faecalis harbouring pPD1 replaces indigenous enteroco

248 We show that E. faecalis significantly augments E. coli biofilm growt

249 We show that E. faecalis strain V583 produces a composite phage (PhiV

250 We show that E. faecalis upregulates OG1RF_12399 transcription specif

251 Interaction energy calculations suggest that E. faecalis retention was largely governed by the combin

252 The E. faecalis mvaS-hymeglusin cocrystal structure (1.95 A)

253 The E. faecalis strains were from two sequence types (ST191

254 h defined bacterial consortia containing the E. faecalis strains and measured inflammation and bacter

255 ietary treatments: the basal diet group, the E. faecalis group, and the antibiotic group on d 0, 14,

256 ilus subcellular compartmentalization in the E. faecalis Ebp pilus system.

257 of Fibrobacteres phylum and 12 genera in the E. faecalis group and antibiotics group were lower than

258 as 12 genera were uniquely identified in the E. faecalis group on d 14 and 28.

259 Bacterial abundance and diversity in the E. faecalis group, bacterial diversity in the antibiotic

260 Here, we report the cryo-EM structure of the E. faecalis 70S ribosome to a global resolution of 2.8 a

261 We report here the identification of the E. faecalis bacteriocin, EntV, produced from the entV (e

262 ontaneous mutants that allowed growth of the E. faecalis DeltafabI strain on fatty acid-free medium.

263 ed in the eutT-eutG intergenic region of the E. faecalis eut cluster.

264 The presence of multiple VGs in most of the E. faecalis isolates underscores the importance of E. fa

265 A comparison of the E. faecalis LiaR, E. faecium LiaR, and the LiaR homolog

266 hnology (RIVET) to identify promoters on the E. faecalis OG1RF chromosome that were specifically acti

267 When pulsed with ovalbumin (OVA), the E. faecalis-induced DCs showed reduction in CD4(+) OVA-s

268 Here, we expressed and purified the E. faecalis OG1RF EfbA and confirmed that this protein b

269 Compared to the wild type, the E. faecalis ccpA mutant had an impaired ability to adher

270 nd intestinal epithelial cell lines with the E. faecalis strains and measured intracellular bacterial

271 Therefore, E. faecalis infection of oral tumor tissues suggests a p

272 Thus, E. faecalis modulates its local environment by contribut

273 Here, we studied for the first time E. faecalis pathogenesis in zebrafish larvae.

274 PhiV1/7 confers an advantage to E. faecalis V583 during competition with other E. faecal

275 mer EF508 exhibited high binding affinity to E. faecalis cells (K(D)-value: 37 nM) and successfully d

276 ity differences among seven chlorophenols to E. faecalis and fresh mixed anaerobic sludge were elucid

277 suggest that multiple factors contribute to E. faecalis persistence in the reproductive tract.

278 Several factors contributing to E. faecalis virulence have been identified.

279 Higher sorption of DOM to E. faecalis was roughly correlated with higher photoinac

280 nsport chains in SCV S. aureus and wild-type E. faecalis results in reduced growth rate but provides

281 s significantly more H(2)O(2) than wild-type E. faecalis.

282 Ultrastructurally, E. faecalis was identified in single-membrane vacuoles,

283 uggests that for children with uncomplicated E. faecalis bacteremia, the addition of low-dose gentami

284 A is present, results in the lysis of viable E. faecalis cells.

285 for mobile genetic elements shapes in vitro E. faecalis populations.

286 irradiated mice 1-4 d postinfection, whereas E. faecalis was not isolated from MLNs of normal mice.

287 The mechanisms by which E. faecalis may contribute to the initiation and progres

288 comparing clinical outcomes of children with E. faecalis bacteremia without endocarditis receiving am

289 er that involves long-term colonization with E. faecalis (in vivo).

290 y testing results for 45 of 46 cultures with E. faecalis or E. faecium.

291 model of colonization of the mouse gut with E. faecalis, without disrupting the microbiota, to evalu

292 cationic human beta-defensins interact with E. faecalis at discrete septal foci, and this exposure d

293 A total of 344 patients with E. faecalis bacteremia were included, all examined using

294 evalence of 26% definite IE in patients with E. faecalis bacteremia, suggesting that echocardiography

295 3, we included 647 consecutive patients with E. faecalis bacteremia.

296 chocardiography in consecutive patients with E. faecalis bacteremia.

297 hy should be considered in all patients with E. faecalis bacteremia.

298 amined the prevalence of IE in patients with E. faecalis bacteremia.

299 ermine the prevalence of IE in patients with E. faecalis bacteremia.

300 4 (17%), 2 (9%), and 1 (4%) samples yielded E. faecalis, E. mundtii, E. casseliflavus, E. faecium, E