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1 ogistic regression, adjusting for total anti-EBV nuclear antigen 1 (EBNA-1) antibodies, which have co
2 n healthy EBV carriers, transcription of the EBV nuclear antigen 1 (EBNA-1) gene is mediated by the p
5 n permissive proliferating cell lines due to EBV nuclear antigen 1 (EBNA-1) protein-mediated replicat
6 no associations were found between maternal EBV nuclear antigen 1 (EBNA-1), diffuse early antigen, o
7 dependent on genome maintenance functions of EBV nuclear antigen 1 (EBNA-1), one of six EBNAs express
8 ry cells in the tonsil express the genes for EBV nuclear antigen 1 (EBNA1) (from the Qp promoter), la
9 of 9 tumors from American patients expressed EBV nuclear antigen 1 (EBNA1) and contained standard epi
11 o DCs, but also efficiently present targeted EBV nuclear antigen 1 (EBNA1) and EBV-latent membrane pr
12 through proteasome-dependent degradation of EBV nuclear antigen 1 (EBNA1) and subsequent loss of vir
13 mimicry between the EBV transcription factor EBV nuclear antigen 1 (EBNA1) and the central nervous sy
14 mimicry between the EBV transcription factor EBV nuclear antigen 1 (EBNA1) and three human CNS protei
15 mid origin of replication, oriP, and express EBV nuclear antigen 1 (EBNA1) are stably maintained extr
16 riP) in the Epstein-Barr virus (EBV) genome, EBV nuclear antigen 1 (EBNA1) enables persistence and en
17 s episome includes two elements from EBV: an EBV nuclear antigen 1 (EBNA1) expression cassette and an
21 Ls) carry a wild-type EBV genome and express EBV nuclear antigen 1 (EBNA1) selectively from the BamHI
22 ere, IgG antibody reactivity toward ANO2 and EBV nuclear antigen 1 (EBNA1) was measured using bead-ba
23 elective increase of T cell responses to the EBV nuclear antigen 1 (EBNA1), the most consistently rec
24 se processes require a single viral protein, EBV nuclear antigen 1 (EBNA1), which binds two clusters
26 n from an integrase mutant reporter virus in EBV nuclear antigen 1-expressing cells, simian virus 40
27 ch EBV might promote autoimmunity, including EBV nuclear antigen 1-mediated molecular mimicry of huma
31 tified binding sites for Epstein-Barr virus (EBV) nuclear antigen 1 (EBNA1) in the human genome using
36 , we show that targeting Epstein-Barr virus (EBV) nuclear antigen 1 (EBNA1) to one of them, the human
37 - and promoter-regulated Epstein-Barr virus (EBV) nuclear antigen 1 (EBNA1) was evaluated in three tr
40 ly only express the less immunogenic antigen EBV nuclear antigen-1 (EBNA-1), rendering them sensitive
41 AdE1-LMPpoly has been generated that encodes EBV nuclear antigen-1 (EBNA1) fused to multiple CD8(+) T
42 gen 1 (LANA1) is functionally similar to the EBV nuclear antigen-1 (EBNA1) protein expressed during v
43 clonal antibody (2B4-1) reactive against the EBV nuclear antigen-1, we noted strong staining of tumor
44 ansgenic mice expressing Epstein-Barr virus (EBV) nuclear antigen-1 (EBNA-1) in B-cells which show a
48 V transformation of primary B cells requires EBV nuclear antigen 2 (EBNA-2) to interact with RBP-Jk t
50 y 2-fold enrichment in host regions bound by EBV nuclear antigen 2 (EBNA2) and EBNA3 transcription fa
52 nd RNA polymerase II occupancy data revealed EBV nuclear antigen 2 (EBNA2) binding at the transcripti
57 e strongly enriched in binding sites for the EBV nuclear antigen 2 (EBNA2) viral transcriptional regu
59 membrane protein 1 and, to a lesser extent, EBV nuclear antigen 2 mediated the increase in p53 level
60 EBV-mediated B cell reprogramming, including EBV nuclear antigen 2-mediated dysregulation of autoimmu
61 al for mitochondrial cristae biogenesis, via EBV nuclear antigen 2/MYC-induced CL enzyme transactivat
67 Here, we reveal a mechanism by which the EBV nuclear antigen 3A (EBNA3A) may inhibit IFNbeta indu
69 To evaluate the role of Epstein-Barr Virus (EBV) nuclear antigen 3A (EBNA3A) in the continuous proli
71 in vitro and in vivo interaction between the EBV nuclear antigen 3C (EBNA3C) and the metastatic suppr
74 The product of one of these viral genes, the EBV nuclear antigen 3C (EBNA3C), is essential for the gr
75 V small RNAs, latent membrane protein 2, and EBV nuclear antigen 3C expression in peripheral blood su
80 interaction between the Epstein-Barr virus (EBV) nuclear antigen 3C (EBNA3C) and the metastatic supp
88 -seronegative recipients was associated with EBV nuclear antigen antibody deficiency, polymorphic dis
89 programme, consisting of highly immunogenic EBV nuclear antigen (EBNA) and latent membrane proteins
91 mphoblastoid Cell Lines (LCLs) requires four EBV nuclear antigen (EBNA) oncoproteins: EBNA2, EBNALP,
92 ohistochemistry, two cases were positive for EBV nuclear antigen (EBNA)-1 (5%), one was positive for
94 s positive for latent membrane protein-1 and EBV nuclear antigen (EBNA)-4 DNAs by polymerase chain re
95 cells, and only a single viral protein, the EBV nuclear antigen (EBNA)1, is expressed via the altern
96 gamma secretion assays, was specific for the EBV nuclear antigen (EBNA)3A and latent membrane protein
99 0), kappa and lambda light chains as well as EBV nuclear antigens (EBNA2) and latent membrane protein
100 nk between the essential Epstein-Barr virus (EBV) nuclear antigen EBNA3C and the SCFSkp2 complex, pro
104 p and Cp, resulting in the expression of six EBV nuclear antigens (EBNAs) and the viral Bcl2 homologu
105 ent infection of B lymphocytes in vitro, six EBV nuclear antigens (EBNAs) are expressed from one of t
106 his may ensure against overexpression of the EBV nuclear antigens (EBNAs) prior to the transcriptiona
107 s of EBV latent membrane proteins (LMPs) and EBV nuclear antigens (EBNAs), as well as nontranslated v
108 l C promoter regulates the expression of all EBV nuclear antigens (EBNAs), some of which are very far
111 ody to both the EBV viral capsid antigen and EBV nuclear antigen, followed by a more rapid rise in an
112 /E7), EBV latent membrane protein-1 and -2A, EBV nuclear antigen, HBV-encoded X antigen, and nonstruc
113 e EBV reactivation was assessed by detecting EBV nuclear antigen IgG, viral capsid antigen IgM, and e
114 trated that the expression of this essential EBV nuclear antigen is capable of releasing the G2/M che