ライフサイエンスコーパス: EDS

1 EDS is a common symptom of both narcolepsy and obstructi

2 EDS mapping of a biochar produced from corn stover pyrol

3 EDS mapping showed that magnetosomes are enmeshed in a m

4 EDS results of the irradiated groups showed an increase

5 EDS results showed that the GA and EDTA solutions did no

6 EDS was also associated with presynaptic dopaminergic dy

7 EDS was measured with the Epworth Sleepiness Scale (ESS)

8 EDS-FLU produces clinically and statistically significan

9 EDS-FLU was superior on both coprimary end points (P < .

10 e products generated from 15, 27, 35, and 40 EDS-CF-PCR amplification cycles were collected and analy

11 -62 (FAVQSSSDSS(P)EENGNGDS(P)S(P)EE), 80-91 (EDS(P)DENEDEES(P)E), and 135-145 (EDES(P)DEEEEEE).

12 miting pathogen growth, that many additional EDS genes remain to be discovered, and that direct scree

13 acility using monochromatic radiation and an EDS setup.

14 , energy dispersive X-ray spectral analysis (EDS), scanning electron microscopy (SEM) and reflective

15 zed by FT-IR, TGA/DTA, SEM, BET analysis and EDS.

16 Respondents with DED and EDS >=60 (highest discomfort) fared worse on OCI, VFQ-28

17 -28R, and WPAI than respondents with DED and EDS <40 (lowest discomfort).

18 sample (conductive materials) as in DRS and EDS, or through the thin graphite foil (non-conductive m

19 FTIR and EDS analysis suggest these 2D platelets to be Boron Nitr

20 Both HRTEM and EDS results confirmed the formation of bimagnetic core-s

21 10-20 nm range and characterized by STEM and EDS for structural and elemental composition.

22 XRD and EDS confirmed the presence of two NP compositions in thi

23 TEM to study morphology, as well as XRD and EDS to study composition.

24 (EDS), and this type of EDS is classified as EDS type VI, which can also be caused by a deficiency in

25 uracy of results produced with the available EDS quantification routines in the low energy range, sim

26 an independent positive association between EDS and the consumption of some anti-pathogen drugs.

27 pharmacological therapies for treating both EDS and cataplexy, discusses concerns specific to childr

28 ion, to the same allele in two large British EDS type II families (LOD scores 4.1 and 4.3).

29 nm) that contained most of the P detected by EDS.

30 oved symptoms of eye dryness, as measured by EDS, versus placebo in participants with DED.

31 of EBOV/Mak dried on prototypic surfaces by EDS or formulations of NaOCl (>= 0.5%), PCMX (>= 0.12%),

32 Antiepileptic drugs may also cause EDS or influence sleep.

33 g pN-collagen are thinner and weaker causing EDS-like laxity of large and small joints and paraspinal

34 e locus for at least some cases of classical EDS in which the alpha1(V) and alpha2(V) genes have been

35 tely one-third of individuals with classical EDS have mutations of COL5A1 that result in haploinsuffi

36 Eight of 28 probands with classical EDS who were heterozygous for expressed polymorphisms in

37 stable dopaminergic therapy with coexistent EDS, as assessed by an Epworth Sleepiness Scale score of

38 stion and NPs were randomized to twice-daily EDS-FLU (93, 186, or 372 mug) or exhalation delivery sys

39 scular form of Ehlers-Danlos syndrome (EDS), EDS type IV, if they alter the sequence in the triple-he

40 y a combination of TEM, single-particle EDS, EDS line scans, XRD analysis, Debye Function simulations

41 has not previously been implicated in either EDS or periodontal disease and contains no known collage

42 ening questionnaires can be used to estimate EDS, but further evaluation is necessary.

43 -to-use disinfectant spray with 58% ethanol (EDS), were tested at contact times of 0, or 0.5 to 10 mi

44 e prefrontal cortical neurons and facilitate EDS is consistent with findings implicating LC-norepinep

45 rious analytical techniques including FESEM, EDS, TEM, AFM, FTIR, Raman, Fluorescence and Absorption

46 exhalation delivery system with fluticasone (EDS-FLU) capable of high/deep drug deposition improves o

47 A predictive index for EDS was generated, which included seven baseline charact

48 agility of OI and indirectly responsible for EDS symptoms, by interference with N-propeptide removal.

49 he previously identified EDS5 gene, and four EDS genes that have not been previously described.

50 13.3% and 4.3% reported regular and frequent EDS, respectively.

51 The current study suggests that frequent EDS is independently associated with future vascular eve

52 Moreover, the association with frequent EDS was statistically significant for stroke (HR, 2.10;

53 roke was confined to the group with frequent EDS, and was 1.73x as much as in the group that reported

54 so observed in the cultured fibroblasts from EDS probands.

55 -GQDs are characterized by XRD, HRTEM, FTIR, EDS and PL.

56 h as in the group that reported never having EDS (HR, 1.73; 95% confidence interval [CI], 1.15-2.60),

57 The HRSEM/EDS/XPS analysis confirmed successful immobilization of

58 iodontal management of a case of hypermobile EDS (type III) associated with atypical gingivitis.

59 If EDS persists despite adequate treatment for either disor

60 learning and the lowest dose (2 ng) improved EDS.

61 re change from baseline to days 42 and 14 in EDS.

62 point was change from baseline to day 84 in EDS.

63 Mean changes from baseline in EDS also significantly favored lifitegrast on days 42 (T

64 icantly greater improvement from baseline in EDS versus those receiving placebo (treatment effect [TE

65 cle control levels which was not the case in EDS + Sham animals.

66 Furthermore, in EDS cells at the 7+4d stage, the reduced beta-adrenergic

67 Improvement in EDS was observed as early as day 14.

68 t LT resulted in significant improvements in EDS, as assessed by the Epworth Sleepiness Scale score (

69 ith controls consistent with joint laxity in EDS patients.

70 very similar to that previously observed in EDS VIA human and Plod1(-/-) mouse tissues, suggesting t

71 mal connective tissue biogenesis observed in EDS, we analyzed mice heterozygous for a targeted inacti

72 euro-ophthalmic symptoms that might occur in EDS patients after mild traumatic brain injury.

73 V collagen appears to have a causal role in EDS types I and II, which show phenotypic overlap and ma

74 nective tissues is consistent with a role in EDS, and our patient's skin fibroblasts do not synthesiz

75 the M4 module were consistent with roles in EDS and a newly identified hub gene of the M4 module (Na

76 investigate baseline predictors of incident EDS.

77 parameters appear to worsen with increasing EDS.

78 ications shaped the evolution of HAE-induced EDS in Nicotiana.

79 JHS/EDS-HT patients reported dry eye symptoms more commonly

80 ent in 77 small fibre neuropathy and 167 JHS/EDS-HT patients without itch.

81 ia with abnormal vitreous was found in 7 JHS/EDS-HT eyes (15.9%) and 0 controls (P = .01).

82 s included comparing ocular anomalies in JHS/EDS-HT and control eyes.

83 irmer I test were significantly lower in JHS/EDS-HT eyes (P < .0001).

84 sphere index was significantly higher in JHS/EDS-HT group (P < .01).

85 atients with an established diagnosis of JHS/EDS-HT and 44 eyes of 22 age- and gender-matched control

86 es were significantly more common in the JHS/EDS-HT group (13.6%; P < .05).

87 tent association of eye anomalies in the JHS/EDS-HT group included xerophthalmia, steeper corneas, pa

88 By confocal microscopy, the JHS/EDS-HT group showed lower density of cells in the superf

89 lers-Danlos syndrome hypermobility type (JHS/EDS-HT), characterized by idiopathic chronic itch with p

90 sessment and management of patients with JHS/EDS-HT.

91 We used this system for the two known EDS mutations (Gly-to-Val) in the middle at Gly-910 and

92 Longitudinally, EDS in PD was associated with non-tremor dominant phenot

93 There were no differences in mean EDS scores between the 2 groups at the first and second

94 The mean EDS scores of the 126 case women in the first month afte

95 r discriminating between 13 subjects with no EDS/snoring and 21 patients with EDS and snoring were id

96 he remaining 103 subjects (14 nonsnoring non-EDS, 21 snoring non-EDS, 68 snoring with EDS).

97 jects (14 nonsnoring non-EDS, 21 snoring non-EDS, 68 snoring with EDS).

98 tance of type V collagen in the causation of EDS type II, and the novel collagen mutation indicates t

99 nclear, and data on biological correlates of EDS in PD are limited.

100 ical presentation confirmed the diagnosis of EDS type VIII.

101 However, isolating the primary etiology of EDS can be challenging and may be multifactorial.

102 ls with a rare recessively inherited form of EDS (characterized by joint hypermobility, skin hyperext

103 ome (EDS) type IV is the most severe form of EDS.

104 1 overlap with those of the vascular form of EDS.

105 -year-old Caucasian female with a history of EDS type III presented with erythematous mucogingival le

106 ilies with autosomal dominant inheritance of EDS, there appears to be linkage to loci that contain th

107 e precise role of TNX in the pathogenesis of EDS is uncertain, this patient's findings suggest a uniq

108 erials in use for testing the performance of EDS in the low energy range is included.

109 Predictors of EDS are unclear, and data on biological correlates of ED

110 omatic range, from the usual presentation of EDS type IV and thus have been excluded from analysis.

111 lers-Danlos syndrome (EDS), and this type of EDS is classified as EDS type VI, which can also be caus

112 east some cases of the hypermobility type of EDS, a condition marked by gross joint laxity and chroni

113 For the hypermobile type of EDS, the molecular underpinnings remain unknown.

114 etic therapies are available for any type of EDS.

115 dy, manifestations of the different types of EDS are present, to varying degrees, in virtually every

116 Fourteen different types of EDS are recognized, of which the molecular cause is know

117 ndings have been reported with some types of EDS.

118 tinct alpha1(I)-osteogenesis imperfecta (OI)/EDS phenotype.

119 Thus, these OI/EDS collagen mutations are directly responsible for the

120 Patients with OI/EDS form a distinct subset of osteogenesis imperfecta (O

121 In each child with OI/EDS, we identified a mutation in the first 90 residues o

122 The dose-response relationship for CRF on EDS performance resembled an inverted U-shaped curve wit

123 This review is focused on EDS due to OSA and narcolepsy and addresses some of the

124 The influence of dopaminergic therapy on EDS is dose dependent.

125 aOCl (0.5% or 1%), PCMX (0.12% to 0.48%), or EDS for >= 5 min.

126 Energy dispersive X-ray spectrometry (EDX or EDS) is a technique often implemented on scanning electr

127 ct times >= 2.5 min, by 0.5% and 1% NaOCl or EDS (> 4 log(10)) at contact times >= 5 min, and by 0.12

128 zed by a combination of TEM, single-particle EDS, EDS line scans, XRD analysis, Debye Function simula

129 Erysiphe orontii, suggesting that particular EDS genes have pathogen-specific roles in conferring res

130 lly driven synchronized continuous flow PCR (EDS-CF-PCR) configuration carried out in a microfabricat

131 In early PD, EDS increases significantly over time and is associated

132 At the end of the double-blind period, EDS-FLU (all doses) significantly improved all 4 definin

133 of CHD, stroke, or dementia, and self-rated EDS as never, rare, regular, or frequent in response to

134 ng high sensitivity energy dispersive X-ray (EDS) mapping and electron energy loss spectroscopy (EELS

135 ificantly versus those in patients receiving EDS-placebo (-21.1 to -21.4 vs -11.7 at week 16, P < .05

136 In most patients (68%), those receiving EDS-FLU reported "much" or "very much" improvement.

137 high-resolution TEM and SEM, nanometer-scale EDS mapping, and DTA.

138 We compared mean Edinburgh Depression Scale (EDS) scores from a group of women (n = 126 cases) at 32

139 scores of the Everyday Discrimination Scale (EDS).

140 the endoplasmic reticulum (ER) occurs in SCD-EDS patients.

141 splastic form of Ehlers-Danlos syndrome (SCD-EDS), a heritable connective tissue disorder.Those previ

142 We propose that SCD-EDS is the result of vesicular zinc trapping and ER zinc

143 score >/=2.0 (0-4 scale), eye dryness score (EDS) >/=40 (0-100 visual analogue scale [VAS]), and hist

144 ts with DED completed the eye dryness score (EDS) visual analogue scale, Ocular Comfort Index (OCI),

145 biosensor was characterized by XRD, FE-SEM, EDS, FT-IR and differential pulse voltammetry.

146 were characterized systematically by FE-SEM, EDS, UV/Vis., FTIR spectroscopy, powder XRD, and XPS tec

147 tion of graphene oxide according to the SEM, EDS, XRD, XPS, Raman and TGA results.

148 SEM-EDS analysis shows that the fossils are composed of alum

149 SEM-EDS measurements of SO2-exposed goethite revealed small

150 SEM-EDS was used to obtain the elemental distribution along

151 wavelength dispersive spectrometer) and SEM-EDS (scanning electron microscopy analysis using an ener

152 o measure changes in pixel intensity and SEM-EDS for compositional analysis.

153 or quantification of the morphology; and SEM-EDS was utilized to locate the impurity within the alpha

154 uorescence spectroscopy, micro-FTIR, and SEM-EDS.

155 th energy-dispersive X-ray spectrometer (SEM-EDS) analysis, which it is a analysis that shows the che

156 to energy dispersive X-ray spectrometry (SEM-EDS) and benchtop ED-XRF analyses confirmed these result

157 copy and energy-dispersive spectrometry (SEM-EDS), suggesting anthropogenic sources.

158 py-X-ray energy dispersive spectroscopy (SEM-EDS) and X-ray photoelectron spectroscopy (XPS), whereas

159 croscopy-energy dispersive spectroscopy (SEM-EDS) technique, with the significant advantage of drasti

160 croscopy-energy-dispersive spectroscopy (SEM-EDS).

161 th energy-dispersive X-ray spectroscopy (SEM-EDS).

162 th energy dispersive X-ray spectroscopy (SEM-EDS).

163 action products were characterized using SEM-EDS and synchrotron muXRD and muXRF.

164 SEM/EDS revealed characteristic surface weathering on the pl

165 SEM/EDS showed diminished content of calcium (Ca) after 15 s

166 energy dispersive X-ray spectroscopy (FE-SEM/EDS) and Brunauer-Emmett-Teller (BET) analysis.

167 copy (OM), scanning electron microscopy (SEM/EDS), micro-infrared spectroscopy (muFTIR/muSR-FTIR), an

168 croscopy/energy dispersive spectrometry (SEM/EDS) after demineralization (n = 3).

169 us energy-dispersive X-ray spectroscopy (SEM/EDS), and Fourier transform infrared (FTIR) micro-spectr

170 microscopy and energy dispersive system (SEM/EDS) analysis.

171 xcluded conditions for 6 months prior to SEM/EDS and muXANES analysis to determine V host phases and

172 rsal task and (b) an extradimensional shift (EDS) task.

173 learning, and extradimensional set shifting (EDS) at different doses.

174 e often mediated by early defense signaling (EDS) rapidly activated by the perception of herbivore as

175 ues, such as electrical discharge sintering (EDS) or resistive sintering (RS), have been intensively

176 Excessive daytime sleepiness (EDS) affects 10-20% of the population and is associated

177 links between excessive daytime sleepiness (EDS) and vulnerability to infectious diseases in a large

178 orders such as excessive daytime sleepiness (EDS) as well as other sleep or cognitive disorders.

179 icacy of LT on excessive daytime sleepiness (EDS) associated with PD.

180 sessed whether excessive daytime sleepiness (EDS) at baseline was associated with subsequent coronary

181 Excessive daytime sleepiness (EDS) can be caused by insufficient sleep but is also a m

182 Excessive daytime sleepiness (EDS) is common and disabling in Parkinson's disease (PD)

183 en obesity and excessive daytime sleepiness (EDS), although for the most part the genetic variance in

184 aracterized by excessive daytime sleepiness (EDS), cataplexy, nighttime sleep disturbances, and REM-s

185 to symptoms of excessive daytime somnolence (EDS).

186 ure and cause excessive day-time somnolence (EDS).

187 ce of energy dispersive X-ray spectrometers (EDS) in the low energy range below 1 keV.

188 Energy Dispersive X-Ray Spectrometry (EDS) and Raman spectroscopy analysis indicate the presen

189 (XRD), energy dispersive X-ray spectrometry (EDS), UV-vis absorption, and extended X-ray absorption f

190 cope(SEM) and energy dispersive spectrometry(EDS).

191 Energy dispersed spectroscopy (EDS) was employed to reveal that the LAH treatment depos

192 py (SEM) and energy dispersive spectroscopy (EDS) analysis of production debris indicate a complex, m

193 confirmed by energy-dispersive spectroscopy (EDS) and by the fluorescence properties of the granules.

194 on (BSE) and energy-dispersive spectroscopy (EDS) mapping of the same particle, the Pb measured in th

195 Using the electron dispersive spectroscopy (EDS) technique, the particles are confirmed to come from

196 imaging and energy dispersive spectroscopy (EDS) were proved the right synthesis of the GNRs and cor

197 rticle X-ray energy dispersive spectroscopy (EDS) were used to characterize the bimetallic DENs befor

198 scopy (TEM), energy dispersive spectroscopy (EDS), and infrared spectroscopy of adsorbed CO showed th

199 scopy (TEM), energy dispersive spectroscopy (EDS), UV-visible spectroscopy and X-ray diffraction (XRD

200 coupled with energy dispersive spectroscopy (EDS), were used to characterize crystallite morphology a

201 s like energy dispersive x-ray spectroscopy (EDS) and electron energy loss spectroscopy (EELS), the d

202 d with energy-dispersive X-ray spectroscopy (EDS) and UV-visible spectrometry.

203 using energy-dispersive X-ray spectroscopy (EDS) and X-ray diffraction (XRD), and microindentation w

204 plying energy-dispersive X-ray spectroscopy (EDS) mapping to the study of two intermetallic phases of

205 es and energy-dispersive X-ray spectroscopy (EDS) showed that Pt-black was growing along GrO(x).

206 ked by energy-dispersive X-ray spectroscopy (EDS) study.

207 M) and energy dispersive X-ray spectroscopy (EDS) to determine their corresponding major element comp

208 M) and energy dispersive X-ray spectroscopy (EDS) to investigate the correlation of the permittivity

209 ts and energy dispersive X-ray spectroscopy (EDS) was used for mineral content distribution.

210 Energy dispersive X-ray spectroscopy (EDS) was used to confirm the elemental composition of th

211 ), and energy dispersive X-ray spectroscopy (EDS) were used to characterize galvanostatically deposit

212 (XRD), energy-dispersive X-ray spectroscopy (EDS), and Raman spectroscopy studies, the continuous lig

213 scopy, energy-dispersive X-ray spectroscopy (EDS), and surface-sensitive cycling voltammetry.

214 ESEM), energy dispersive X-ray spectroscopy (EDS), atomic force microscopy (AFM) and grazing incidenc

215 (TEM), energy dispersive X-ray spectroscopy (EDS), fluorescence spectroscopy, and mass spectrometry,

216 d with energy-dispersive X-ray spectroscopy (EDS), quasi in situ X-ray photoelectron spectroscopy (XP

217 RTEM), energy-dispersive X-ray spectroscopy (EDS), selected area electron diffraction (SAED), Raman s

218 PXRD), energy-dispersive X-ray spectroscopy (EDS), X-ray photoelectron spectroscopy (XPS), and (10)B

219 RTEM), energy-dispersive X-ray spectroscopy (EDS), X-ray photoelectron spectroscopy (XPS), Raman spec

220 using energy dispersive X-ray spectroscopy (EDS).

221 D) and energy dispersive X-ray spectroscopy (EDS).

222 In early developmental stage (EDS) cells (from 7+3d to 7+5d), the stimulatory response

223 STEM-EDS mapping shows very close agreement with the ASV-dete

224 In the case of PtCl(4)(2-), STEM-EDS mapping shows AuPt alloy NPs with 3.9 +/- 1.3% and 4

225 h Energy-Dispersive X-ray Spectroscopy (STEM-EDS) mapping provide quantitation of the extent of Ag an

226 h energy dispersive X-ray spectroscopy (STEM-EDS).

227 y/energy-dispersive X-ray spectroscopy (STEM/EDS) and inductively coupled plasma atomic emission spec

228 roscopy/energy dispersive spectroscopy (STEM/EDS), inductively coupled plasma-atomic emission spectro

229 Patients with subclinical EDS and brain injury may experience a slower, less compl

230 al course of seven patients with subclinical EDS and mild traumatic brain injury are presented.

231 ith incident "elevated depressive symptoms" (EDS: CES-D(total) >= 16) among AA (HR = 1.28, 95% CI: 1.

232 e of the subtypes of Ehlers-Danlos syndrome (EDS) and cutis laxa.

233 ive rise to forms of Ehlers-Danlos syndrome (EDS) because of partial or complete skipping of exon 6,

234 ed mutations causing Ehlers-Danlos syndrome (EDS) classic type result in haploinsufficiency of proalp

235 Ehlers-Danlos syndrome (EDS) designates a heterogeneous group of connective tiss

236 use model of classic Ehlers Danlos syndrome (EDS) had decreased biomechanical stiffness compared with

237 Ehlers-Danlos syndrome (EDS) is a group of collagen disorders primarily affectin

238 Ehlers-Danlos syndrome (EDS) is a heterogeneous group of connective-tissue disor

239 Vascular Ehlers-Danlos syndrome (EDS) is a rare connective tissue disorder with serious h

240 Vascular Ehlers-Danlos syndrome (EDS) results from mutations in the formation of type III

241 Ehlers-Danlos syndrome (EDS) type I (the classical variety) is a dominantly inhe

242 Vascular Ehlers-Danlos syndrome (EDS) type IV is the most severe form of EDS.

243 Ehlers-Danlos syndrome (EDS) type IV results from mutations in the COL3A1 gene,

244 Ehlers-Danlos syndrome (EDS) types I and II, which comprise the classical variet

245 As in Ehlers-Danlos syndrome (EDS) VIIA/B, fibrils containing pN-collagen are thinner

246 lassical form of the Ehlers-Danlos syndrome (EDS), a heritable connective-tissue disorder that severe

247 phoscoliotic type of Ehlers-Danlos syndrome (EDS), and this type of EDS is classified as EDS type VI,

248 the vascular form of Ehlers-Danlos syndrome (EDS), EDS type IV, if they alter the sequence in the tri

249 e cases of classical Ehlers-Danlos syndrome (EDS), in which aberrant collagen fibrils are associated

250 e characteristics of Ehlers-Danlos syndrome (EDS).

251 with the features of Ehlers-Danlos syndrome (EDS).

252 s are typical of the Ehlers-Danlos syndrome (EDS).

253 The Ehlers-Danlos syndromes (EDS) are a heterogeneous group of hereditary disorders o

254 , or 372 mug) or exhalation delivery system (EDS)-placebo for 24 weeks (16 double-blind plus 8 open-l

255 Products were characterized by SEM, (S)TEM, EDS, XPS, and SQUID.

256 ution process and characterized by XRD, TEM, EDS, and XPS.

257 d with magnetic skyrmions, so that XRD, TEM, EDS, SAED and HREM investigations were carried out for s

258 have been characterized by powder XRD, TEM, EDS, selected area electron diffraction, and nanobeam el

259 TEM-EDS analysis at 336 h showed secondary precipitates enri

260 croscopy-energy dispersive spectroscopy (TEM-EDS) and field flow fractionation (FFF-ICP-MS).

261 at % Ti, consistently determined by XRD, TEM-EDS, and ICP-OES, was distributed uniformly across the a

262 y process, as confirmed by DLS, SLS, and TEM/EDS analysis.

263 relations, are consistent with the idea that EDS as a proxy of altered sleep quality/quantity may aff

264 h linkage could be excluded, indicating that EDS-VIII is a genetically heterogeneous disorder.

265 d impair learning of the 5 reversals and the EDS task.

266 th similar characteristics who completed the EDS at 32 weeks' gestation during the same summer period

267 ntrations (KCl) to reduce the current in the EDS-CF-PCR device during cycling.

268 The results showed that the EDS-CF-PCR format produced results similar to that of a

269 best quantitative physiological correlate to EDS.

270 one-half of the mutations that give rise to EDS types I and II are likely to lie in the COL5A1 gene,

271 or SC65 mutations may cause as yet undefined EDS variants.

272 the first system capable of being used under EDS or RS conditions independently combining current con

273 s in a patient with a previously undiagnosed EDS type VIII.

274 Vascular EDS is a serious disorder with high mortality, which doe

275 Vascular EDS is associated with a shortened overall survival due

276 old decrease in arterial rupture in vascular EDS patients.

277 nt of complications associated with vascular EDS.

278 Ehlers-Danlos VIII (EDS-VIII) is an autosomal dominant disorder characterize

279 or on both coprimary end points (P < .001 vs EDS-placebo, all doses).

280 25% of patients at week 24 versus 8.7% with EDS-placebo (P <= .014, all comparisons).

281 Dopaminergic therapy was associated with EDS at years 2 and 3, as dose increased.

282 Poor healing associated with EDS excluded surgical excision and necessitated the use

283 onnective tissue dysfunction associated with EDS.

284 ariables were assessed for associations with EDS for up to 3 years.

285 ar 1 showed no significant associations with EDS.

286 first report on the folding of collagen with EDS mutations, which demonstrates local delays in the tr

287 Comparable with EDS patients, they had decreased aortic stiffness and te

288 -fos profiles was positively correlated with EDS performance.

289 in 33 unrelated individuals or families with EDS type IV, mutations that affect splicing, of which 30

290 t should be useful in analyzing linkage with EDS and other disease phenotypes.

291 coding region of COL5A1, in one patient with EDS type I.

292 cts with no EDS/snoring and 21 patients with EDS and snoring were identified by receiver operator cur

293 olding of type III collagen in patients with EDS.

294 urgical management options for patients with EDS.

295 kage search in a large Swedish pedigree with EDS-VIII and established linkage to a 7-cM interval on c

296 Analysis of four further pedigrees with EDS-VIII revealed two consistent with linkage to 12p13 a

297 non-EDS, 21 snoring non-EDS, 68 snoring with EDS).

298 0% specificity for identifying subjects with EDS.

299 FESEM, XRD, XPS, EDS and ATR analysis were employed to characterize the s

300 The sorbent was characterized by SEM, XRD, EDS, and FT-IR.