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1                                              EF-1 alpha co-migrates with GST-EF-3 on glutathione-Seph
2                                              EF-1, as in recoverin, does not bind calcium because it
3                    When elongation factor 1 (EF-1) from rabbit reticulocytes was examined as substrat
4 eukaryotic elongation factor 1A-1 (eEF1A-1), EF-1-delta, eEF1gamma, 14-3-3epsilon, and 14-3-3zeta/del
5 e cooperativity between EF-4, EF-3 and EF-2, EF-1 and allostery involving the four EF-hands.
6 in as translation elongation factor-1 alpha (EF-1 alpha).
7 s calcium first, followed by EF-3, EF-2, and EF-1 and determined the four affinity constants by model
8 dentified active synthetic peptides AG73 and EF-1 from the sequence of laminin alpha1 LG4 for binding
9 coimmunoprecipitation of WNV genomic RNA and EF-1 alpha, using an anti-EF-1 alpha antibody.
10 NV genomic RNA and EF-1 alpha, using an anti-EF-1 alpha antibody.
11      Mg(2+) binds constitutively to CaBP1 at EF-1 with an apparent dissociation constant (K(d)) of 30
12 osomes involves a direct interaction between EF-1 alpha and EF-3.
13 vidence suggest a direct interaction between EF-1 alpha and EF-3.
14 ciation constant for the interaction between EF-1 alpha and WNV 3' SL RNA was calculated to be 1.1 x
15 ys demonstrated that the interaction between EF-1 alpha and WNV 3' SL RNA was specific.
16           However, binding of Ca(2+) to both EF-1 and EF-3 is necessary for ALG-2 interaction with Al
17 ined with partially purified EF-1, with both EF-1 and ribosomes contributing to stimulation of elonga
18 osed patch of hydrophobic residues formed by EF-1 and EF-2 (Leu24, Trp27, Phe31, Phe45, Phe48, Phe49,
19 ndent EF-3-ATPase activity are influenced by EF-1 alpha.
20 nterference of EF-3/anti-EF-3 interaction by EF-1 alpha but not by EF-2.
21 s of the protein synthesis elongation factor EF-1.
22 he binding activity of the WNV 3' SL RNA for EF-1 alpha by approximately 60%.
23 lyclonal antibody to EF-3 immunoprecipitates EF-1 alpha along with EF-3.
24  alanine substitutions within the AG73 (M1), EF-1 (M2, M3), and alpha-dystroglycan binding sites (M4,
25 he48 that includes the EF-hand-related motif EF-1 is essential both for activation of RetGC at low Ca
26                         Dephosphorylation of EF-1 alpha by calf intestinal alkaline phosphatase inhib
27  methods, shows that the N-terminal helix of EF-1 is close to the membrane interface.
28 o associates with the bacterial homologue of EF-1 for its replicase function, implicating a possible
29 e interaction indicated that one molecule of EF-1 alpha binds to each molecule of WNV 3' SL RNA.
30                        Specific mutations of EF-1 residues in full-length PLC delta1 reduce enzyme ac
31                           Phosphorylation of EF-1 resulted in a 2-2.6-fold stimulation of EF-1 activi
32 timulation involves coordinate regulation of EF-1 and ribosomes through phosphorylation by multipoten
33 EF-1 resulted in a 2-2.6-fold stimulation of EF-1 activity, as measured by poly(U)-directed polypheny
34 e also demonstrate that the alpha subunit of EF-1 remains tightly bound to the L protein in the inact
35 s for the beta, gamma, and delta subunits of EF-1 from rabbit were cloned, and proteins of 225, 437,
36 alyze the role of the individual subunits of EF-1 in elongation, the cDNAs for the beta, gamma, and d
37  the expression of the different subunits of EF-1 may contribute to alterations not only in protein s
38 d virions also package all three subunits of EF-1.
39 esults were obtained with partially purified EF-1, with both EF-1 and ribosomes contributing to stimu
40 s and one additional EF-hand-like structure, EF-1, that deviates form the EF-hand consensus sequence.
41 ee regions, encompassing the N-terminus, the EF-1 motif, and the EF-3 motif, were likely involved in
42 chment on LG4 through syndecans and that the EF-1 site is for cell spreading activity through integri
43 GCAP-specific amino acid residues within the EF-1 can create a contact surface for binding GCAP-2 to
44 ound that various point mutations within the EF-1 domain can specifically affect the ability of GCAP-
45                   Point mutations within the EF-1 region can interfere with both the activation of th
46 2+) binding to EF-3, while Ca(2+) binding to EF-1 has little effect on surface exposure of hydrophobi
47                              Ca(2+) binds to EF-1 with low affinity (K(d) >100 microM), and no Ca(2+)
48                             Interaction with EF-1 alpha at the high-activity binding site was sequenc