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1                                              EOG light peak was 714 mV OD (range: 316-1379) and 746 m
2                                              EOG recordings showed a typically decreased Arden ratio
3                                              EOGs of two subjects were obtained under the same condit
4        After the start of the medication, an EOG was performed in 19 eyes of 11 patients; 16 of these
5 ed high voltage and sub-low voltage ECoG and EOG activities, as well as decreased nuchal EMG activity
6 ce and electrophysiological testing (ERG and EOG) may indicate initial stages or more widespread inju
7                                         EEG, EOG, ECG and NIRS signals have been measured during a si
8 sly recorded odor-induced OB LFPs and either EOG or ORN neural activity showed that oscillations occu
9 troencephalogram (EEG) and electrooculogram (EOG) electrodes to evaluate sleep architecture, and vide
10  Electromyogram (EMG), and Electrooculogram (EOG), respectively.
11 lectroencephalogram (EEG), Electrooculogram (EOG), Electromyogram (EMG), Electrocardiogram (ECG) and
12  (OCT), Arden ratio, and electrooculography (EOG) light peak.
13 roretinography (ERG) and electrooculography (EOG) testing were normal and genetic testing was negativ
14 ectrocardiography (ECG), electrooculography (EOG), galvanic skin response (GSR), and head acceleratio
15 gated the application of Electrooculography (EOG) eye tracking using 14 electrodes positioned around
16 aces were assessed using electrooculography (EOG) in a measure of attentional bias.
17 s of walking tasks using electrooculography (EOG) synchronised with 3D motion analysis (VICON).
18           Interestingly, electroolfactogram (EOG) responses stimulated by either cAMP- or inositol 1,
19 ory field potential, the electroolfactogram (EOG).
20 e explore the electro-oxidation of glycerol (EOG) on platinum (Pt) in LiOH, NaOH and KOH using in sit
21 an olfactory cue but showed no difference in EOG amplitude or duration compared to controls.
22 ing finding is that the profile of intrinsic EOG response measured in surgically opened nose without
23     Octanal and other aldehydes induce large EOG responses in the rodent olfactory epithelium, sugges
24 mon Cl(-)/HCO(3)(-) exchanger AE2 had normal EOGs.
25  [electrocortical (EcoG), electo-oculargram (EOG), nuchal muscle electromyography (EMG) and breathing
26                           Electro-oculogram (EOG) and full-field electroretinogram testing results we
27 l produces changes in the electro-oculogram (EOG) similar to those caused by light, but indirect evid
28 ic examination, including electro-oculogram (EOG), short-wavelength FAF, near-infrared FAF, spectral-
29 roencephalograms (EEGs), electro-oculograms (EOGs), submental electromyogram (EMG), GG EMG (intramusc
30 D patients having normal electro-oculograms (EOGs), to examine the hypothesis that the severity of di
31 uals exhibit a reduced electro-oculographic (EOG) response to changes in light exposure and have sign
32 oretinography (ERG) and electro-oculography (EOG), and color vision testing were performed.
33 ctroretinography (ERG), electro-oculography (EOG), and Goldmann perimetry were performed.
34 ents were measured with electro-oculography (EOG).
35 retinography (ERG), and electro-oculography (EOG).
36 isual field testing and electro-oculography (EOG).
37                      Wireless acquisition of EOG further demonstrates accurate tracking of horizontal
38 he timing of the slow oscillatory changes of EOG voltage.
39             Light caused small increments of EOG voltage (termed light rises), again related to the f
40 s tested there was significant inhibition of EOG function; however, over time there was at least a pa
41                                The result of EOG testing was inconsistent with bestrophinopathy (Arde
42                         Electro-olfactogram (EOG) data demonstrated that both control and oil-exposed
43 om the olfactory organ [electro-olfactogram (EOG) or integrated neural activity], local field potenti
44 actory epithelium using electro-olfactogram (EOG) recordings from wild-type (WT) and TRPA1/V1-knock o
45            We performed electro-olfactogram (EOG) recordings on the double-mutant mice, NCKX4(-/-);CN
46 he effect of airflow on electro-olfactogram (EOG) responses and found that the MOE of mice can sense
47 ino acid cues using the electro-olfactogram (EOG) technique and found that WAF-exposed bicolor damsel
48  activities measured by electro-olfactogram (EOG) under same flow conditions.
49 behavior and recording electro-olfactograms (EOGs).
50 siological technique, electro-olfactography (EOG), was employed to determine the level of olfactory d
51 s for the measured retronasal and orthonasal EOG response at all flow rates and positions.
52 amplitude of the olfactory evoked potential (EOG) recorded inside the nose; (2) the EOG amplitude is
53 , we present electro-olfactogram recordings (EOG) demonstrating that NKCC1-deficient mice exhibit sig
54                          A bilateral reduced EOG response was detected in 1 patient.
55 B1(DeltaCaM) mice displayed markedly reduced EOG amplitude accompanied by alterations in other respon
56                        The airflow-sensitive EOG response in the MOE was attenuated when cAMP was inc
57 movements, benchmarked against gold-standard EOG and camera-based eye tracking.
58                 In comparison, gold-standard EOG attained mean absolute angular error of [Formula: se
59 tage deflections of earEOG and gold-standard EOG for saccades from [Formula: see text] to [Formula: s
60 ntal saccades using earEOG and gold-standard EOG.
61                                     Standard EOGs were recorded after oral administration of alcohol
62                                          The EOG results were abnormal in all patients.
63 tial (EOG) recorded inside the nose; (2) the EOG amplitude is correlated with the amplitude of the ol
64 cue and that oil-exposure did not affect the EOG amplitude or duration in response to oil or other cu
65 rigeminal agonists induced a decrease in the EOG response to PEA, which depended on the level of TRPA
66                   The slower decrease in the EOG voltage was evident in patients with small fields an
67 ory neurons was not affected and neither the EOG nor olfactory behavior was impaired.
68 gly suggest that the light peak phase of the EOG is temporally related to a decreased OSEL in normal
69 rt the suggestion that the light peak of the EOG may not be generated solely by hBest1.
70       However, because the light peak of the EOG of some patients with the DeltaI295, D312N, E119Q, a
71 nge in OS length corresponded to that of the EOG waveform.
72                                       On the EOG measure of attentional bias, MDMA, but not MA, incre
73 combination with an absent light rise on the EOG that outweighs the full-field ERG abnormalities, whi
74  and Cl(-)/HCO(3)(-) exchangers, reduced the EOG in epithelia from both wild-type and NKCC1 knockout
75 NKCC cotransport with bumetanide reduced the EOG in epithelia from wild-type mice but had no effect i
76 ubjects, and to compare these changes to the EOG.
77 d for androstenone and, as they do so, their EOG and OERP increase.
78 ensitivity, as determined by their unchanged EOG response amplitude.