ライフサイエンスコーパス: Enterobacteriaceae

1 ial pathogens and intestinal anaerobes (e.g. Enterobacteriaceae).

2 udomonas aeruginosa and carbapenem-resistant Enterobacteriaceae.

3 tion regarding the ecological success of the Enterobacteriaceae.

4 ra; and a major reorganization of the family Enterobacteriaceae.

5 m, and Proteobacteria decrease accounted for Enterobacteriaceae.

6 g-resistant (including carbapenem-resistant) Enterobacteriaceae.

7 mainly focus on inducible AmpC resistance in Enterobacteriaceae.

8 in LMICs with a bloodstream infection due to Enterobacteriaceae.

9 ished sources of outbreaks of drug-resistant Enterobacteriaceae.

10 occi, vancomycin-resistant Enterococcus, and Enterobacteriaceae.

11 ta-lactamase (ESBL) and carbapenem-resistant Enterobacteriaceae.

12 es in K. pneumoniae and other members of the Enterobacteriaceae.

13 e European Survey of Carbapenemase-Producing Enterobacteriaceae.

14 tion with the mCIM to identify MBL-producing Enterobacteriaceae.

15 or of virulence and antibiotic resistance in Enterobacteriaceae.

16 olved in LCN-2-mediated host defense against Enterobacteriaceae.

17 with the dissemination of the mcr-1 genes in Enterobacteriaceae.

18 fficient to limit colonization of pathogenic Enterobacteriaceae.

19 actamase-producing, and carbapenem-resistant Enterobacteriaceae.

20 e, mediated by analogous systems, across the Enterobacteriaceae.

21 ted to the emergence of carbapenem-resistant Enterobacteriaceae.

22 and third-generation cephalosporin-resistant Enterobacteriaceae.

23 bsiella spp and from 30.4% to 71.9% in other Enterobacteriaceae.

24 ebsiella spp and from 5.9% to 68.8% in other Enterobacteriaceae.

25 side developed to target multidrug-resistant Enterobacteriaceae.

26 difficult to contain carbapenemase-producing Enterobacteriaceae.

27 fecal microbiomes, with higher abundance of Enterobacteriaceae.

28 tory flexibility in prominent members of the Enterobacteriaceae.

29 terium tuberculosis and carbapenem-resistant Enterobacteriaceae.

30 ded-spectrum beta-lactamase (ESBL) producing Enterobacteriaceae.

31 mmaproteobacteria-particularly opportunistic Enterobacteriaceae.

32 of the UK's reported carbapenemase-producing Enterobacteriaceae.

33 worldwide emergence of carbapenem-resistant Enterobacteriaceae(1-4), which are resistant to the anti

34 predominant microorganisms were the group of Enterobacteriaceae (43.0%, 52/121), followed by Enteroco

35 throughout the year, with a predominance of Enterobacteriaceae (54%) as urinary pathogens in heart,

36 ients (68%); causative pathogens were mainly Enterobacteriaceae (543/604 [90%]).

37 ruginosa (77%), Klebsiella spp. (16%), other Enterobacteriaceae (6%).

38 R sinks compared to HCP sinks (p < 0.05) for Enterobacteriaceae (76.4 vs. 32.9%) and P. aeruginosa (2

39 ent with this idea, we identified endogenous Enterobacteriaceae, a low-abundance taxon, as a keystone

40 Against Carbapenems in Klebsiella and Other Enterobacteriaceae, a prospective, multicenter, observat

41 The study tested 100 well-characterized Enterobacteriaceae, Acinetobacter baumannii, and Pseudom

42 identified 21 621 non-duplicate isolates of Enterobacteriaceae, Acinetobacter spp, and Pseudomonas a

43 Eighty Gram-negative bacilli (54 Enterobacteriaceae and 26 nonfermenting Gram-negative ba

44 nt expansion of mucosal-associated commensal Enterobacteriaceae and a decline of obligate anaerobes.

45 dance of Bacteroidaceae, Clostridiaceae, and Enterobacteriaceae and a lower relative abundance of Bif

46 ance genes, tet(X3) and tet(X4), in numerous Enterobacteriaceae and Acinetobacter that were isolated

47 al porins in small-molecule translocation in Enterobacteriaceae and consider the crucial contribution

48 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae and CRE carriage among solid organ tr

49 eened IncX4 plasmids among 2,470 isolates of Enterobacteriaceae and determined the mcr-1 positive iso

50 e, prolonged carriage of multidrug-resistant Enterobacteriaceae and distinct antibiotic-driven patter

51 ses in mesophile, psychrophile, Pseudomonas, Enterobacteriaceae and H(2)S producing bacterial counts

52 nts of mesophile, psychrophile, Pseudomonas, Enterobacteriaceae and H(2)S-producing bacteria were obt

53 ranscription factor EutR is conserved in the Enterobacteriaceae and is required for ethanolamine sens

54 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae and MDR Acinetobacter baumannii.

55 coding Ssp6-like effectors are widespread in Enterobacteriaceae and often linked with T6SS genes.

56 iscordant development of bacterial genera of Enterobacteriaceae and Parabacteroides species in the fi

57 susceptibility testing breakpoints for both Enterobacteriaceae and Pseudomonas aeruginosa This break

58 ting potent antibacterial activities against Enterobacteriaceae and Pseudomonas aeruginosa were ident

59 loxacin and levofloxacin breakpoints for the Enterobacteriaceae and Pseudomonas aeruginosa, daptomyci

60 of infections caused by carbapenem-resistant Enterobacteriaceae and Pseudomonas aeruginosa, which are

61 esistance to beta-lactam antibiotics in many Enterobacteriaceae and Pseudomonas aeruginosa.

62 chromosomal and extrachromosomal MGEs within Enterobacteriaceae and Pseudomonas, and an additional Ac

63 We show that increased Enterobacteriaceae and reduced Clostridia abundance dist

64 rrent with limited total viable count (TVC), Enterobacteriaceae and reduced conversion of pro-polyphe

65 in intrinsic antibacterial activity against Enterobacteriaceae and restoration of beta-lactam activi

66 A combination of Enterobacteriaceae and spore-forming bacteria, but not c

67 common errors included misinterpretation of Enterobacteriaceae and Staphylococcus genus results.

68 infant gut are predominantly associated with Enterobacteriaceae and Staphylococcus, siderophore-like

69 The facultative anaerobic taxa Enterobacteriaceae and Streptococcaceae were increased i

70 ts with cirrhosis showed higher abundance of Enterobacteriaceae and Streptococcus and a reduction in

71 the healthy status by increased abundance of Enterobacteriaceae and Streptococcus spp. and, functiona

72 se are the class B (metallo) enzyme NDM-1 of Enterobacteriaceae and the class D (OXA) enzymes of Acin

73 RMANOVA, p = 0.01), with higher abundance of Enterobacteriaceae and Veillonella, in those born to H.

74 oplane, shifts were detected in the families Enterobacteriaceae and Xanthomonadaceae following the fi

75 ition (e.g., Lactobacillus, Bacteroides, and Enterobacteriaceae) and approximately 9% higher beta-glu

76 g resistant strains and carbapenem-resistant Enterobacteriaceae, and most anaerobic pathogens.

77 cia alcalifaciens, all members of the family Enterobacteriaceae, appeared to be transcriptionally act

78 Enterobacteriaceae are among the first colonizers of neo

79 Escherichia coli and other Enterobacteriaceae are among the most common pathogens o

80 Carbapenemase-producing Enterobacteriaceae are an emerging threat to hospitals w

81 ociated infections, demonstrating that these Enterobacteriaceae are emerging nosocomial pathogens.

82 The biofilms of Enterobacteriaceae are fortified by assembly of curli am

83 Enterobacteriaceae are intestinal residents, but general

84 nd the likelihood of ampC induction by other Enterobacteriaceae are less clear.

85 Carbapenem-resistant Enterobacteriaceae are resistant to most beta-lactam ant

86 Enterobacteriaceae as the index isolate was found to be

87 d at understanding the host response against Enterobacteriaceae as well as for the development of ant

88 Enterobacteriaceae, as the index isolate, was found to b

89 he number of intrinsic resistance genes from Enterobacteriaceae, as well as with a reduction in the r

90 recipient clinical and laboratory strains of Enterobacteriaceae bacteria.

91 onged labor with a first pregnancy, a higher Enterobacteriaceae/Bacteroidaceae ratio at 3 months was

92 was the dominant path to overweight; higher Enterobacteriaceae/Bacteroidaceae ratios and Clostridioi

93 342 health-care-associated infections, with Enterobacteriaceae being the most frequently found (113

94 eta-lactam (APBL) therapy within 48 hours of Enterobacteriaceae bloodstream infections (BSIs) on 90-d

95 <=48 hours of APBL for empirical therapy of Enterobacteriaceae BSI after adjustment for the propensi

96 hospitalized for >48 hours for treatment of Enterobacteriaceae BSI at Palmetto Health hospitals in C

97 Among 808 patients with Enterobacteriaceae BSI, 414 and 394 received >48 and <=4

98 f patients with bloodstream infection due to Enterobacteriaceae (BSI-E).

99 incidence of CDI in hospitalized adults with Enterobacteriaceae BSIs.

100 obactin is secreted by members of the family Enterobacteriaceae, but many other bacteria including Ps

101 rt for the treatment of carbapenem-resistant Enterobacteriaceae, but resistance in 5% to >40% isolate

102 pecifically inhibits the iron acquisition of Enterobacteriaceae by binding and neutralizing the bacte

103 ncept that dysbiosis-associated expansion of Enterobacteriaceae can be viewed as a microbial signatur

104 ntifying and overcoming barriers to updating Enterobacteriaceae carbapenem breakpoints in Los Angeles

105 Continued use of obsolete Enterobacteriaceae carbapenem breakpoints is common in c

106 y of California laboratories, using obsolete Enterobacteriaceae carbapenem breakpoints.

107 mase (ESBL) production, carbapenem-resistant Enterobacteriaceae, carbapenem-resistant acinetobacter s

108 notype that we observed among members of the Enterobacteriaceae Characterization of S. flexneri 2457T

109 Institute (CLSI) published revisions to the Enterobacteriaceae ciprofloxacin and levofloxacin breakp

110 In patients with ESBL-producing Enterobacteriaceae, clinical cure rates were 87.5% (14/1

111 ith a set of other representative species of Enterobacteriaceae, closely related to E. coli, indicate

112 r extended-spectrum beta-lactamase-producing Enterobacteriaceae colonization, and colonization was as

113 f any ColR isolate and the emergence of ColR Enterobacteriaceae (ColR-E).

114 Genomes of many Enterobacteriaceae contain conserved gene clusters encod

115 These results suggest that commensal Enterobacteriaceae contribute to colonization resistance

116 Protection conferred by endogenous Enterobacteriaceae could be modelled by inoculating mice

117 carbapenemase-producing carbapenem-resistant Enterobacteriaceae (CP-CRE) is an important element of t

118 our understanding of carbapenemase-producing Enterobacteriaceae (CPE) and which will be valuable for

119 Carbapenemase-producing Enterobacteriaceae (CPE) are a significant threat to pub

120 Carbapenemase-producing Enterobacteriaceae (CPE) are emerging worldwide, limitin

121 n and infection with carbapenemase-producing Enterobacteriaceae (CPE) in Chicago-area long-term acute

122 bal dissemination of carbapenemase-producing Enterobacteriaceae (CPE) is a more recent problem that,

123 ly identification of carbapenemase-producing Enterobacteriaceae (CPE) is essential to prevent their d

124 le treatment against carbapenemase-producing Enterobacteriaceae (CPE) is unknown.

125 ed with 27 different carbapenemase-producing Enterobacteriaceae (CPE) isolates and 34 different non-C

126 Rapid detection of carbapenemase-producing Enterobacteriaceae (CPE) represents a major challenge fo

127 ith activity against carbapenemase-producing Enterobacteriaceae (CPE) with metallo-beta-lactamases (M

128 and 161 (64.7%) had carbapenemase-producing Enterobacteriaceae (CPE).

129 Carbapenem-resistant Enterobacteriaceae (CRE) are a serious public health thr

130 Carbapenem-resistant Enterobacteriaceae (CRE) are among the most severe threa

131 Carbapenem-resistant Enterobacteriaceae (CRE) are an urgent threat with poten

132 Carbapenem-resistant Enterobacteriaceae (CRE) are associated with considerabl

133 Carbapenem-resistant Enterobacteriaceae (CRE) are high-priority bacterial pat

134 Carbapenem-resistant Enterobacteriaceae (CRE) are multidrug-resistant pathoge

135 g detects a fraction of carbapenem-resistant Enterobacteriaceae (CRE) carriers.

136 Here, using carbapenem-resistant Enterobacteriaceae (CRE) clinical isolates, we reveal th

137 2010, the incidence of carbapenem-resistant Enterobacteriaceae (CRE) has been increasing in Singapor

138 ears, the prevalence of carbapenem-resistant Enterobacteriaceae (CRE) has risen substantially, and th

139 easing dissemination of carbapenem-resistant Enterobacteriaceae (CRE) in both humans and animals pose

140 re a major reservoir of carbapenem-resistant Enterobacteriaceae (CRE) in healthcare facilities, contr

141 nd clinical outcomes of carbapenem-resistant Enterobacteriaceae (CRE) in sentinel US hospitals.

142 The rise in carbapenem-resistant Enterobacteriaceae (CRE) infections has created a global

143 Twenty patients with carbapenem-resistant Enterobacteriaceae (CRE) infections were treated with me

144 tality in nonbacteremia carbapenem-resistant Enterobacteriaceae (CRE) infections.

145 Carbapenem-resistant Enterobacteriaceae (CRE) is an emergent microorganism of

146 of infections caused by carbapenem-resistant Enterobacteriaceae (CRE) is crucial for proper treatment

147 Two collections of carbapenem-resistant Enterobacteriaceae (CRE) isolates were evaluated, includ

148 Carbapenem-resistant Enterobacteriaceae (CRE) spread regionally throughout he

149 g organisms (ESBL), and carbapenem-resistant Enterobacteriaceae (CRE) using nares, skin (axilla/groin

150 Carbapenem-resistant Enterobacteriaceae (CRE), Acinetobacter baumannii (CRAB)

151 d fecal coliforms (FC), carbapenem-resistant Enterobacteriaceae (CRE), blaNDM-1, and selected extende

152 asive infections due to carbapenem-resistant Enterobacteriaceae (CRE), leading to higher rates of cli

153 Thirty-seven carbapenem-resistant Enterobacteriaceae (CRE)-infected patients were treated

154 ant bacteria, including carbapenem-resistant Enterobacteriaceae (CRE).

155 ing medium (OPP-C), and carbapenem-resistant Enterobacteriaceae (CRE).

156 pathogens, particularly carbapenem-resistant Enterobacteriaceae (CREs), present a major and growing t

157 agnosed patients with carbapenem-susceptible Enterobacteriaceae (CSE) and carbapenem-resistant Entero

158 via a novel analysis of carbapenem-resistant Enterobacteriaceae data reported to the National Healthc

159 alence of the bee pathogen-containing family Enterobacteriaceae declined with temperature, suggesting

160 ophilic bacteria, yeast, mold and pathogenic Enterobacteriaceae decreased as irradiation dose increas

161 ctale and Bifidobacterium and an increase of Enterobacteriaceae, Desulfovibrio sp., and mainly Akkerm

162 for the detection of polymyxin resistance in Enterobacteriaceae, developed by P.

163 that O(2) and NO(3) respiration confer upon Enterobacteriaceae during expansion.

164 h extended-spectrum beta-lactamase-producing Enterobacteriaceae during ICU-hospitalization.

165 ments across opportunistic pathogens such as Enterobacteriaceae (e.g., Escherichia coli) and non-ferm

166 h extended spectrum beta-lactamase-producing Enterobacteriaceae (EPE) in previously-colonized individ

167 l Extended-spectrum Beta-lactamase producing Enterobacteriaceae (ESBL-E) carriage in community-dwelli

168 l extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-E) carriage in community-dwelli

169 f extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-E) in clinical stool samples.

170 o extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-E) strengthens the requirement

171 f extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-E), compared with 2.9% of non-P

172 o extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-E).

173 f extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-PE) who receive cephalosporin-b

174 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae (ESBL-PE), with undefined clinical co

175 r extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-PE).

176 cs approach, we found that E. coli and other Enterobacteriaceae expressing the Yersinia HPI also secr

177 ia (e.g., species of the Lachnospiraceae and Enterobacteriaceae families).

178 , Escherichia coli, and other members of the Enterobacteriaceae family are common human pathogens tha

179 molecular identification of species from the Enterobacteriaceae family in a variety of clinical speci

180 However, only the Enterobacteriaceae family of human gut-associated Proteo

181 dominance of pathogenic bacteria within the Enterobacteriaceae family, and a paucity of strictly ana

182 umoniae and Escherichia coli are part of the Enterobacteriaceae family, being common sources of commu

183 al Klebsiella michiganensis, a member of the Enterobacteriaceae family, was sufficient for colonizati

184 ant components of the biofilms formed by the Enterobacteriaceae family.

185 and only observed with other members of the Enterobacteriaceae family.

186 ted P = 0.25] but decreased pro-inflammatory Enterobacteriaceae (FDR-corrected P = 0.25).

187 bactin is a secondary metabolite produced by Enterobacteriaceae for acquiring iron, an essential meta

188 Enteritidis competes with commensal Enterobacteriaceae for oxygen.

189 cular characterization of colistin-resistant Enterobacteriaceae For the MicroScan colistin well, cate

190 Many Enterobacteriaceae fortify their outer membrane with cat

191 llance program collected 103,960 isolates of Enterobacteriaceae from 2008 to 2014.

192 sign retains high discriminatory power among Enterobacteriaceae genera and species, with only particu

193 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae generally cannot be treated with peni

194 , a histidine kinase encoded within the core Enterobacteriaceae genome that promotes the activation o

195 o reveal functionally unique features of the Enterobacteriaceae GusRs.

196 Transmissible carbapenem-resistance in Enterobacteriaceae has been recognized for the last 2 de

197 actamase- (ESBL) and carbapenemase-producing Enterobacteriaceae has significantly increased in recent

198 rbapenems in Klebsiella pneumoniae and Other Enterobacteriaceae) has contributed seminal multicenter

199 a self-transmissible MGE functioning in the Enterobacteriaceae, has evolved a second target cell att

200 Although carbapenemase-producing Enterobacteriaceae have received the most attention beca

201 Carbapenem-resistant Enterobacteriaceae have recently become an important cau

202 Proteins of the Hha-family, conserved among enterobacteriaceae, have been implicated in dynamically

203 ant bacteria, including carbapenem-resistant Enterobacteriaceae, have increased in frequency, resulti

204 l early colonisers such as Streptococcus and Enterobacteriaceae, iii) domination of Bifidobacteriacea

205 hothrix thermosphacta, Pseudomonas spp., and Enterobacteriaceae in AP meat compared to NAP meat.

206 XA-48 is the most prevalent carbapenemase in Enterobacteriaceae in Europe and the Middle East, but it

207 cks the striking irinotecan-induced bloom of Enterobacteriaceae in immune-deficient mice.

208 h staphylococci in 14 patients, fermentative Enterobacteriaceae in nine patients and streptococci in

209 h extended-spectrum beta-lactamase-producing Enterobacteriaceae in previously noncolonized and noninf

210 y implicates urinary tract Lactobacillus and Enterobacteriaceae in protective and pathogenic roles fo

211 ition, the CREST study (Carbapenem-Resistant Enterobacteriaceae in Solid Organ Transplant Patients) h

212 was associated with increasing domination by Enterobacteriaceae in the IgA-unbound fraction of the mi

213 ability of respiratory electron acceptors to Enterobacteriaceae in the lumen of the colon.

214 develop NEC first experience an increase in Enterobacteriaceae in the portion of the microbiota not

215 typic detection of carbapenemase activity in Enterobacteriaceae In this issue of the Journal of Clini

216 ated UTIs and acute pyelonephritis caused by Enterobacteriaceae, including multidrug-resistant strain

217 ice S100 at birth prevented the expansion of Enterobacteriaceae, increased numbers of T-regulatory ce

218 ological pH, Escherichia coli and many other Enterobacteriaceae infect human gastrointestinal and uri

219 n extended-spectrum beta-lactamase-producing Enterobacteriaceae infection (risk ratio, 49.62 [95% CI,

220 The incidence of carbapenem-resistant Enterobacteriaceae infection did not change significantl

221 g the 90 ESBL-PE carriers, manifestations of Enterobacteriaceae infection included travelers' diarrhe

222 t extended-spectrum beta-lactamase-producing Enterobacteriaceae infection were 95.1% (95% CI, 54.7-99

223 Carbapenems resistant Enterobacteriaceae infections are increasing worldwide r

224 Symptoms suggesting Enterobacteriaceae infections were recorded prospectivel

225 Twenty patients with carbapenem-resistant Enterobacteriaceae infections were treated with meropene

226 y innate lymphoid cells type 3 (ILC3) during Enterobacteriaceae infections.

227 infections caused by carbapenemase-producing Enterobacteriaceae is a global health concern.

228 f the OXA-48-type class D beta-lactamases in Enterobacteriaceae is challenging.

229 Fifteen clinical Enterobacteriaceae isolates (harboring NDM [n = 7], VIM

230 ESBL and bla(OXA-48)-type Enterobacteriaceae isolates included E. coli, Kluyvera,

231 Clinical Enterobacteriaceae isolates which lack smvA and smvR als

232 Fifty-eight Enterobacteriaceae isolates with ciprofloxacin MICs of 0

233 nt with beta-lactam antibiotics, susceptible Enterobacteriaceae isolates would become sufficiently pe

234 e developed a validation panel comprising 10 Enterobacteriaceae isolates, 5 Gram-positive cocci, 5 Gr

235 8 K-SeT) were compared by using a set of 166 Enterobacteriaceae isolates, including isolates producin

236 mcr-1-positive Enterobacteriaceae (MCRPE) have attracted substantial me

237 rd-generation cephalosporin resistance among Enterobacteriaceae, mediated by the spread of extended-s

238 tibiotic screening against E. coli and other Enterobacteriaceae members.

239 ibiotic, fosfomycin, to treat ESBL-producing Enterobacteriaceae (one that has completed enrollment an

240 nonfermenting gram-negative bacilli, but not Enterobacteriaceae or other pathogens.

241 th no difference between clusters except for Enterobacteriaceae organisms (the level was higher in cl

242 g extended-spectrum beta-lactamase-producing Enterobacteriaceae outbreaks or data on pediatric popula

243 HIV-infected participants and enrichment of Enterobacteriaceae (P = .02) in participants with low CD

244 articularly due to an increase in the family Enterobacteriaceae (p = 0.002) and Escherichia coli (p =

245 -AG) are protected from enteric infection by Enterobacteriaceae pathogens.

246 sented in this study suggest that pathogenic Enterobacteriaceae persist much longer than their more b

247 r extended-spectrum beta-lactamase-producing Enterobacteriaceae phenotypic confirmation.

248 Family Enterobacteriaceae (phylum Proteobacteria), family Lacto

249 udomonas aeruginosa, Acinetobacter spp., and Enterobacteriaceae pose urgent public health threats.

250 precedented outbreak of carbapenem-resistant Enterobacteriaceae, primarily involving KPC-producing Kl

251 iofilm formation, Escherichia coli and other Enterobacteriaceae produce an extracellular matrix consi

252 m-negative pathogens (Staphylococcus aureus, Enterobacteriaceae, Pseudomonas aeruginosa, and Acinetob

253 f extended-spectrum beta-lactamase-producing Enterobacteriaceae ranged from 5% to 10%.

254 is drives expansion of facultative anaerobic Enterobacteriaceae, regardless of their pathogenic poten

255 velopment of antimicrobial therapies against Enterobacteriaceae-related infections.

256 Enterobacteriaceae represent a diverse and medically imp

257 rveillance definition for CPE to include all Enterobacteriaceae resistant to any carbapenem tested.

258 reduction the acquisition of ESBL-producing Enterobacteriaceae (RR, 0.28 [95% CI, .11-.69] for STD+A

259 In Enterobacteriaceae, several membrane transporters are in

260 We included 219 MGEs described in 7 Enterobacteriaceae species isolated from human, animal a

261 bacter cloacae (n = 62, 14.4%) were the main Enterobacteriaceae species.

262 pressed by enterotoxigenic E. coli and other Enterobacteriaceae species.

263 ctive analysis of an additional 720 clinical Enterobacteriaceae spectra found an approximately 11,109

264 against multidrug-resistant bacteria such as Enterobacteriaceae spp, Pseudomonas aeruginosa, Stenotro

265 h tests were challenged with 294 isolates of Enterobacteriaceae spp., Pseudomonas aeruginosa, and Aci

266 lood cultures experimentally inoculated with Enterobacteriaceae, Staphylococcus epidermidis or Staphy

267 f extended-spectrum beta-lactamase-producing Enterobacteriaceae subsequent infection and increased mo

268 nic amine moiety addition to lipid A in many Enterobacteriaceae such as E. coli and Salmonella.

269 mmation reprograms the metabolic pathways of Enterobacteriaceae, such as Escherichia coli LF82, in th

270 xtended-spectrum cephalosporin resistance in Enterobacteriaceae suggestive of extended-spectrum beta-

271 IM was conducted as previously described for Enterobacteriaceae Test performance was compared to the

272 that these genes may be disseminated by non-Enterobacteriaceae that are not detected as part of stan

273 ted between CP-CRE and members of the family Enterobacteriaceae that do not produce carbapenemases.

274 with pre-IBD exhibited notable expansions of Enterobacteriaceae that exacerbated low-grade mucosal in

275 ble to aminoglycosides (78.8% vs. 68.6%) and Enterobacteriaceae that produce extended-spectrum beta-l

276 e practice of screening clinical isolates of Enterobacteriaceae that test as susceptible to carbapene

277 ologic eradication, including eradication of Enterobacteriaceae that were not susceptible to aminogly

278 ations of susceptibility among MBL-harboring Enterobacteriaceae The addition of EDTA at concentration

279 for 1.4% (1,493/103,960) of all isolates of Enterobacteriaceae The most frequently identified carbap

280 ramework for colonization resistance against Enterobacteriaceae, these mechanistic insights point to

281 Carbapenem-resistant Enterobacteriaceae threaten human health, since carbapen

282 diated destruction of the microbiota enables Enterobacteriaceae to expand to high densities in the co

283 otics reduce colonization resistance against Enterobacteriaceae to pinpoint possible control points f

284 cimens enabled unambiguous classification of Enterobacteriaceae to the species level in 22 of 27 (81.

285 In the outer membrane of Enterobacteriaceae, trimeric porins control the cellular

286 ative transfer of F plasmids residing in the Enterobacteriaceae was first reported in the 1940s, yet

287 atients with monomicrobial bacteremia due to Enterobacteriaceae was not associated with a detrimental

288 ty of metallo-beta-lactamase (MBL)-harboring Enterobacteriaceae We also evaluated the addition of EDT

289 o carbapenemase PCR, for a collection of 125 Enterobacteriaceae We also investigated the impact of an

290 ty to aztreonam, ceftazidime, and meropenem; Enterobacteriaceae were also tested against ertapenem an

291 Multiresistant bacteria like Enterobacteriaceae were isolated from one quarter of all

292 m each medium identified as P. aeruginosa or Enterobacteriaceae were tested for susceptibility to azt

293 roved assay for dnaJ-based identification of Enterobacteriaceae which boasts increased broad-range sp

294 ically important Gram-negative member of the Enterobacteriaceae, which has increasingly been recogniz

295 interpretation associated with the mCIM for Enterobacteriaceae will likely lead to its adoption by c

296 ine increases bacterial fitness and provides Enterobacteriaceae with a growth advantage against compe

297 The proportions of non-Salmonella Enterobacteriaceae with extended spectrum beta-lactamase

298 ted resistance mechanism in human and animal Enterobacteriaceae, with a wide geographical distributio

299 e mCIM was easy to perform and interpret for Enterobacteriaceae, with results in less than 24 h and e

300 ource driving a post-antibiotic expansion of Enterobacteriaceae within the large bowel.