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1  leukemia cells, yeast, Escherichia coli and Enterococcus faecalis.
2 ion of IL-10 deficient (Il10(-/-)) mice with Enterococcus faecalis.
3 itogenic, protease-secreting enteric microbe Enterococcus faecalis.
4 ainst an intestinal opportunistic bacterium, Enterococcus faecalis.
5 estinal bacterial growth, mainly E. coli and Enterococcus faecalis.
6 Escherichia coli, Klebsiella pneumoniae, and Enterococcus faecalis.
7 tivity of LmPC as well as PC from pathogenic Enterococcus faecalis.
8  survival signals modulated by the bacterium Enterococcus faecalis.
9 sion in the opportunistic bacterial pathogen Enterococcus faecalis.
10 ginosa but not to the Gram-positive pathogen Enterococcus faecalis.
11 crobial treatment were notable for growth of Enterococcus faecalis.
12 positive results with vancomycin-susceptible Enterococcus faecalis.
13 ) pilus is an important virulence factor for Enterococcus faecalis.
14 ement commonly found in clinical isolates of Enterococcus faecalis.
15                  Hymeglusin blocks growth of Enterococcus faecalis.
16 aphylococcus aureus and vancomycin-resistant Enterococcus faecalis.
17 ion system is a major virulence regulator in Enterococcus faecalis.
18 role of sigma(54) in the nosocomial pathogen Enterococcus faecalis.
19 vents involving nosocomial pathogens such as Enterococcus faecalis.
20 aphylococcus aureus and vancomycin-resistant Enterococcus faecalis.
21 tococcus mutans, Actinomyces naeslundii, and Enterococcus faecalis.
22 nsporter), for subspecies differentiation of Enterococcus faecalis.
23 istic pathogens Clostridioides difficile and Enterococcus faecalis.
24  important gram-positive nosocomial pathogen Enterococcus faecalis.
25 etween secretion and cell wall attachment in Enterococcus faecalis.
26 translocation for the Gram-positive bacteria Enterococcus faecalis.
27 ance of the plasmid pAD1 in its native host, Enterococcus faecalis.
28 niae, Salmonella typhimurium, S. aureus, and Enterococcus faecalis.
29 Escherichia coli, Pseudomonas aeruginosa and Enterococcus faecalis.
30 r times at room temperature in comparison to Enterococcus faecalis.
31 89), Klebsiella pneumoniae (TOP52 1721), and Enterococcus faecalis (0852) were studied, and diabetic
32 . pyogenes, 226 Streptococcus pneumoniae, 93 Enterococcus faecalis, 1,356 Enterobacteriaceae, and 227
33 (predominant microorganism in institution A: Enterococcus faecalis, 18 cultures [51.4%]; institution
34 n this study, we investigated the ability of Enterococcus faecalis 2/28, isolated from artisan cheese
35 ecium (VRE FCM) (16), vancomycin-susceptible Enterococcus faecalis (3), Aerococcus viridans (2), Baci
36                                              Enterococcus faecalis (48%), Enterococcus faecium (14%),
37 n-a two-subunit exotoxin that is secreted by Enterococcus faecalis(5,6)-as a cause of hepatocyte deat
38 d 5 methods for testing daptomycin versus 48 Enterococcus faecalis, 51 Enterococcus faecium, and 50 S
39     Herein, we describe mechanisms that link Enterococcus faecalis, a bacterium known to produce extr
40  in the recognition of muramyl dipeptide and Enterococcus faecalis, a commensal bacterium that is a c
41      Here, we examine the phages harbored by Enterococcus faecalis, a commensal of the human intestin
42                                              Enterococcus faecalis, a Gram-positive bacterium, and Ca
43                                              Enterococcus faecalis, a Gram-positive human commensal o
44 ve characterization of collateral effects in Enterococcus faecalis, a gram-positive opportunistic pat
45 al instability and colon cancer triggered by Enterococcus faecalis, a human intestinal commensal bact
46 associated inflammation impacts infection by Enterococcus faecalis, a leading cause of catheter-assoc
47 ence attribute of the opportunistic pathogen Enterococcus faecalis, a leading cause of hospital-acqui
48                                              Enterococcus faecalis, a leading cause of hospital-acqui
49                                  EF1143 from Enterococcus faecalis, a life-threatening pathogen that
50                                The genome of Enterococcus faecalis, a low-GC Gram-positive opportunis
51  accounted for 34.5% of fecal coliforms, and Enterococcus faecalis accounted for 32% of enterococci.
52 high activity against the oral key pathogens Enterococcus faecalis, Actinomyces naeslundii, Streptoco
53 lococcus aureus, Pseudomonas aeruginosa, and Enterococcus faecalis although PSO had an antimicrobial
54                          Multidrug-resistant Enterococcus faecalis, an opportunistic human pathogen,
55 eant dye, YOYO-1, were first developed using Enterococcus faecalis, an organism that has previously b
56 three most common species, Escherichia coli, Enterococcus faecalis and Bacteroides vulgatus, did not
57 acterial biofilm and faecal samples included Enterococcus faecalis and Enterobacter hormaechei.
58 he BC-GP correctly identified 14/15 cases of Enterococcus faecalis and Enterococcus faecium bacteremi
59 m for identification of vancomycin-resistant Enterococcus faecalis and Enterococcus faecium, VRESelec
60 f clinical strains of Staphylococcus aureus, Enterococcus faecalis and Enterococcus faecium.
61 coated quartz sand (IOCS) to remove two FIB (Enterococcus faecalis and Escherichia coli) suspended in
62 the nonpathogenic commensal bacteria strains Enterococcus faecalis and Escherichia coli.
63 n to be involved in bile salts resistance of Enterococcus faecalis and in virulence.
64 rain except those from two related bacteria, Enterococcus faecalis and Lactococcus lactis.
65 greater potency against vancomycin resistant Enterococcus faecalis and methicillin-resistant Staphylo
66                                          For Enterococcus faecalis and other enterococcal species, re
67 , involved in polysaccharide biosynthesis of Enterococcus faecalis and showed that disruption of epaB
68 kinetics of the d-Ala:d-Ser ligase VanG from Enterococcus faecalis and solved its crystal structure i
69                  The Gram-positive bacterium Enterococcus faecalis and the fungus Candida albicans ar
70 -positive bacteria Staphylococcus aureus and Enterococcus faecalis and two Gram-negative bacteria Esc
71        Risk factors included isolation of VR Enterococcus faecalis and use of linezolid or clindamyci
72  of MetAPs from Mycobacterium tuberculosis , Enterococcus faecalis , and human, three hotspots have b
73 g a microbe with host-protective properties (Enterococcus faecalis) and a pathogen (Staphylococcus au
74  both Gram positive ( Staphylococcus aureus, Enterococcus faecalis) and Gram negative bacteria (e.g.,
75  A protocol was developed for Gram-positive (Enterococcus faecalis) and Gram-negative (Escherichia co
76 genic bacteria (Photorhabdus luminescens and Enterococcus faecalis) and two nonpathogenic bacteria (E
77 roscan, 87% of Staphylococcus aureus, 90% of Enterococcus faecalis, and 88% of Enterococcus faecium i
78 us aureus (MRSA), Listeria monocytogenes and Enterococcus faecalis, and against the Gram-negative bac
79 57, Listeria innocua, Staphylococcus aureus, Enterococcus faecalis, and Bacillus anthracis, on sample
80 Bacillus anthracis, Propionibacterium acnes, Enterococcus faecalis, and both Methicillin-sensitive an
81 ve bacteria, including Bacillus subtilis and Enterococcus faecalis, and drug-sensitive and drug-resis
82 survival of non-pathogenic Escherichia coli, Enterococcus faecalis, and E. coli O157:H7 were compared
83 occus epidermidis, Streptococcus pneumoniae, Enterococcus faecalis, and Enterococcus faecium) and thr
84 lyzed, in particular, Staphylococcus aureus, Enterococcus faecalis, and Escherichia coli.
85 tion by a pure facultative anaerobic strain, Enterococcus faecalis, and fresh mixed anaerobic sludge,
86  with poor response to Streptococcus mutans, Enterococcus faecalis, and Lactobacillus casei.
87 three different organisms (Escherichia coli, Enterococcus faecalis, and Staphylococcus aureus).
88 9.65, and 100.00% for Staphylococcus aureus, Enterococcus faecalis, and streptococci, respectively.
89 te genome plasticity in antibiotic resistant Enterococcus faecalis, and their involvement has been im
90 tive transfer and virulence functions of the Enterococcus faecalis antibiotic resistance plasmid pCF1
91 -negative Escherichia coli and Gram-positive Enterococcus faecalis applied during the first 7 months
92 resistant "superbugs." Bacteria of the genus Enterococcus faecalis are highly antibiotic-resistant no
93  combinations was assessed by application on Enterococcus faecalis as a model organism for Gram-posit
94                             The emergence of Enterococcus faecalis as a significant nosocomial pathog
95 d-scan EPR can detect superoxide produced by Enterococcus faecalis at rates that are too low for dete
96 s aureus ATCC 29213 (0.004 to 0.015 mug/ml), Enterococcus faecalis ATCC 29212 (0.015 to 0.06 mug/ml),
97 cus aureus ATCC 29213 (0.03 to 0.12 mug/ml), Enterococcus faecalis ATCC 29212 (0.03 to 0.12 mug/ml),
98 sk only), S. aureus ATCC 29213 (broth only), Enterococcus faecalis ATCC 29212 (broth only), Streptoco
99 eudomonas aeruginosa ATCC 27853, 9 to 13 mm, Enterococcus faecalis ATCC 29212, 0.03 to 0.12 microg/ml
100 ccus aureus ATCC 29213, 0.25 to 2 mug/ml for Enterococcus faecalis ATCC 29212, 1 to 4 mug/ml for Esch
101                                              Enterococcus faecalis ATCC 29212, a vancomycin-sensitive
102 4028, Staphylococcus epidermidis ATCC 12228, Enterococcus faecalis ATCC 29212, and Escherichia coli D
103 pecies, including Pseudomonas putida KT2440, Enterococcus faecalis ATCC 29212, Salmonella Typhimurium
104 tracts showed antimicrobial activity against Enterococcus faecalis, Bacillus subtilis, Escherichia co
105 nted here had either Enterococcus faecium or Enterococcus faecalis bacteremia caused by both vancomyc
106  of synergistic gentamicin for uncomplicated Enterococcus faecalis bacteremia in children.
107 ective endocarditis (IE) among patients with Enterococcus faecalis bacteremia.
108 ed as a critical structural component of the Enterococcus faecalis biofilm matrix.
109 ers were then shown to inhibit the growth of Enterococcus faecalis biofilms that play a role in early
110  photoinactivation of a laboratory strain of Enterococcus faecalis, but depressed photoinactivation o
111 iorated sepsis-induced death associated with Enterococcus faecalis, but not Escherichia coli, infecti
112 and vanA or vanB in Enterococcus faecium and Enterococcus faecalis) by the BC-GP assay also was asses
113                                The bacterium Enterococcus faecalis cannot synthesize heme but can acq
114 tural snapshots from the type II-A system of Enterococcus faecalis Cas1 and Cas2 during spacer integr
115                                              Enterococcus faecalis Cas1-Cas2 selectively binds to a s
116                                          The Enterococcus faecalis cell wall-anchored protein Ace is
117 Upon sensing of the peptide pheromone cCF10, Enterococcus faecalis cells carrying pCF10 produce three
118                       The mating response of Enterococcus faecalis cells carrying the conjugative pla
119 alf-life of the metK transcript in vivo when Enterococcus faecalis cells were depleted for SAM, but n
120                                          The Enterococcus faecalis class IIa bacteriocin MC4-1 encode
121   Biofilm production is a major attribute of Enterococcus faecalis clinical isolates.
122 ructure of the ligand binding segment of the Enterococcus faecalis collagen binding MSCRAMM ACE (micr
123                                           In Enterococcus faecalis, conjugation of a Cas9-targeted pl
124  The ethanolamine utilization (eut) locus of Enterococcus faecalis, containing at least 19 genes dist
125  an SDMH is involved in biofilm formation in Enterococcus faecalis, contributing to oxidant productio
126 on from the antagonistic prgX/prgQ operon in Enterococcus faecalis controlling conjugative transfer o
127 nt bioinformatic studies have suggested that Enterococcus faecalis could serve as a model system to b
128 present in other pathogenic bacteria such as Enterococcus faecalis, Coxiella burnetii, and Clostridiu
129                     Here we characterize the Enterococcus faecalis Csn2 protein as a double-stranded
130     In other gram-positive bacteria, such as Enterococcus faecalis, disulfide bonds are formed in sec
131 ngly difficult to treat infections caused by Enterococcus faecalis due to its high levels of intrinsi
132 port and retention behavior of two model FIB Enterococcus faecalis (E. faecalis) and Escherichia coli
133                     In the present study, an Enterococcus faecalis (E. faecalis) DNA biosensor (ef-bi
134                                              Enterococcus faecalis (E. faecalis) is one of the indica
135 hat Streptococcus gordonii (S. gordonii) and Enterococcus faecalis (E. faecalis) were frequent isolat
136             This approach is demonstrated on Enterococcus faecalis (E. faecalis), which served as tar
137 ins of diverse origin, including the species Enterococcus faecalis, E. faecium, E. casseliflavus, and
138 inosa (Pa), Legionella pneumophila (Lp), and Enterococcus faecalis (Ef) by using anti-infective, anti
139  applied single-molecule FRET methods to the Enterococcus faecalis (Efa) Cas1-Cas2 system to establis
140           Upon sensing of peptide pheromone, Enterococcus faecalis efficiently transfers plasmid pCF1
141    The pheromone-responsive plasmid pCF10 of Enterococcus faecalis encodes a putative cell wall hydro
142 y named celBA) of the opportunistic pathogen Enterococcus faecalis, encodes a 6-phospho-beta-glucosid
143 transposon Tn916 from the bacterial pathogen Enterococcus faecalis, encodes a putative ArdA (alleviat
144    Expression of ace (adhesin to collagen of Enterococcus faecalis), encoding a virulence factor in e
145 ated that the ebp operon and the ace gene of Enterococcus faecalis, encoding endocarditis- and biofil
146 B-containing Enterococcus faecium (ENFM) and Enterococcus faecalis (ENFS) isolates.
147 re several species of enterococci, including Enterococcus faecalis, Enterococcus faecium, Enterococcu
148 s (Bacillus cereus group, Enterococcus spp., Enterococcus faecalis, Enterococcus faecium, Staphylococ
149 am-positive pathogens Staphylococcus aureus, Enterococcus faecalis, Enterococcus faecium, Streptococc
150 ides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia coli K12, E. coli O15
151 ved with the type of bacterium in the order: Enterococcus faecalis, Escherichia coli O157:H7, and Esc
152 robial CAUTI and frequently cocolonizes with Enterococcus faecalis, Escherichia coli, Providencia stu
153 sis revealed that hsdS allelic variations in Enterococcus faecalis exert significant impact on gene e
154 lococcus aureus (MRSA), vancomycin-resistant Enterococcus faecalis/faecium (VREfc/VREfm), and ciprofl
155 es to PG composition in vancomycin-resistant Enterococcus faecalis following the growth in presence o
156 al species (seven Escherichia coli and three Enterococcus faecalis ) for all ten patient samples.
157 TDB, Escherichia coli, Bacillus subtilis and Enterococcus faecalis, from the guts of the desert woodr
158                                          The Enterococcus faecalis Fsr quorum system has been shown t
159                                          The Enterococcus faecalis gene coaB encodes a novel monofunc
160                           CVRE distinguishes Enterococcus faecalis (green colonies) from Enterococcus
161 ia coli (group A, 38.4%; group B, 39.3%) and Enterococcus faecalis (group A, 32.7%; group B, 33.2%).
162 -positive bacteria Staphylococcus aureus and Enterococcus faecalis have lost either all or most polya
163 idium perfringens, Escherichia coli), except Enterococcus faecalis, human milk was more antimicrobial
164 positive organisms Staphylococcus aureus and Enterococcus faecalis in comparison with known analogues
165  of the pheromone receptor protein PrgZ from Enterococcus faecalis in complex with the heptapeptide c
166 lar function in Streptococcus agalactiae and Enterococcus faecalis In conclusion, the elucidation of
167 pheromone plasmids increase the virulence of Enterococcus faecalis in experimental pathogenesis model
168                           PCR did not detect Enterococcus faecalis in five BC-confirmed cases.
169 e wetland, while for the bacterial indicator Enterococcus faecalis, inactivation results were compara
170 induce high levels of (p)ppGpp production in Enterococcus faecalis, indicating that this nosocomial p
171 e, infection of catheter-implanted mice with Enterococcus faecalis induced the specific expression of
172 icantly decreased the burden of a subsequent Enterococcus faecalis infection in the nematode intestin
173 us gentamicin (AG) combinations for treating Enterococcus faecalis infective endocarditis (EFIE).
174  study, we compare outcomes in patients with Enterococcus faecalis infective endocarditis treated in
175                                              Enterococcus faecalis is a commensal and pathogen of hum
176                                              Enterococcus faecalis is a commensal bacterium found in
177                                              Enterococcus faecalis is a commensal bacterium of the hu
178                      The EF1143 protein from Enterococcus faecalis is a distant homolog of deoxynucle
179                                              Enterococcus faecalis is a Gram-positive commensal bacte
180                                              Enterococcus faecalis is a gram-positive commensal bacte
181                                              Enterococcus faecalis is a gram-positive organism respon
182                                              Enterococcus faecalis is a human intestinal commensal th
183                                              Enterococcus faecalis is a human intestinal pathobiont w
184                   The Gram-positive pathogen Enterococcus faecalis is a leading agent of nosocomial i
185                                              Enterococcus faecalis is a leading cause of nosocomial i
186                                              Enterococcus faecalis is a member of the intestinal and
187                                              Enterococcus faecalis is a member of the mammalian gastr
188                                        Since Enterococcus faecalis is a natural heme auxotroph and ca
189 One of the well-studied virulence factors of Enterococcus faecalis is a secreted bacterial protease,
190 e (SAS) RelQ from the Gram-positive pathogen Enterococcus faecalis is a sequence-specific RNA-binding
191                                              Enterococcus faecalis is an established nosocomial patho
192                                              Enterococcus faecalis is an opportunistic pathogen respo
193                  The Gram-positive bacterium Enterococcus faecalis is both a colonizer of the gastroi
194                                              Enterococcus faecalis is both a common commensal of the
195                                              Enterococcus faecalis is considered to be the most impor
196 sphate-dependent tyrosine decarboxylase from Enterococcus faecalis is followed by transformation of d
197                                              Enterococcus faecalis is frequently associated with poly
198     The Gram-positive opportunistic pathogen Enterococcus faecalis is frequently responsible for noso
199                                              Enterococcus faecalis is one of the most frequently isol
200                                              Enterococcus faecalis is part of the human intestinal mi
201                      Expansion of intestinal Enterococcus faecalis is sufficient to exacerbate ethano
202 olling pheromone-induced plasmid transfer in Enterococcus faecalis is the most thoroughly studied gen
203                                              Enterococcus faecalis is the third most frequent cause o
204                                              Enterococcus faecalis is unusual in that it encodes two
205                                              Enterococcus faecalis isolates (n = 1,112) were highly s
206        Most previous studies have found that Enterococcus faecalis isolates do not show significant a
207 e of a clinical pair of vancomycin-resistant Enterococcus faecalis isolates from the blood of a patie
208              Eighty-one endocarditis-derived Enterococcus faecalis isolates that were collected from
209 , Clostridium species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella oxytoca, Klebsiella pn
210 ecent clinical isolates of Escherichia coli, Enterococcus faecalis, Klebsiella pneumoniae, and Pseudo
211 : Pseudomonas aeruginosa, Proteus mirabilis, Enterococcus faecalis, Klebsiella pneumoniae, Escherichi
212 Staphylococcus aureus, Enterococcus faecium, Enterococcus faecalis, Klebsiella pneumoniae, Pseudomona
213 ntestinal microbiota (Lactobacillus reuteri, Enterococcus faecalis, Lactobacillus crispatus and Clost
214                                           In Enterococcus faecalis, lateral transfer of conjugative p
215 creasing sensitivity in the following order: Enterococcus faecalis LDH2 </= Lactococcus lactis LDH2 <
216                                              Enterococcus faecalis, long implicated in serious system
217  a monoclonal antibody developed against the Enterococcus faecalis major pilus protein EbpC, we ident
218                                              Enterococcus faecalis may contribute to periodontal brea
219 e visualization of conformational changes in Enterococcus faecalis MDD that describe sequential steps
220 occus (n = 5), and daptomycin-nonsusceptible Enterococcus faecalis (n = 6).
221 lope homeostasis), from daptomycin-resistant Enterococcus faecalis not only reversed resistance to 2
222 Deletion mutants of the two sortase genes of Enterococcus faecalis OG1RF were constructed.
223 ed molecular patterns, including heat-killed Enterococcus faecalis or CpG DNA, led to increased Ikapp
224                                          The Enterococcus faecalis pathogenicity island (PAI) encodes
225                                              Enterococcus faecalis pCF10 transfers at high frequencie
226                                              Enterococcus faecalis pheromone-responsive plasmids prod
227 onoclonal antibody to the major component of Enterococcus faecalis pili, EbpC, labels polymerized pil
228             The par stability determinant of Enterococcus faecalis plasmid pAD1 is the only antisense
229 l-prolyl cis-trans isomerase, as well as the Enterococcus faecalis polysaccharide diheteroglycan, are
230 reduction in Lactobacillus spp. and promoted Enterococcus faecalis populations.
231                          Multidrug-resistant Enterococcus faecalis possess numerous mobile elements t
232 d the GIT microbiota of MAT mothers, whereas Enterococcus faecalis predominated within the MAT infant
233   EfbA is a PavA-like fibronectin adhesin of Enterococcus faecalis previously shown to be important i
234                                          The Enterococcus faecalis prg and pcf genes of plasmid pCF10
235                                              Enterococcus faecalis PrgA, encoded by the conjugative p
236 ost & Microbe, Keogh et al. (2016) show that Enterococcus faecalis promotes Escherichia coli biofilm
237 uconostoc mesenteroides, Bacillus cereus and Enterococcus faecalis proving its antimicrobial action.
238  species such as Staphylococcus epidermidis, Enterococcus faecalis, Pseudomonas aeruginosa, and Klebs
239 the AMP-modifications with Escherichia coli, Enterococcus faecalis, Pseudomonas aeruginosa, Staphyloc
240 induced stresses (Brochothrix thermosphacta, Enterococcus faecalis, Pseudomonas fluorescens, Salmonel
241 atural enterococci diversity, 11 isolates of Enterococcus faecalis recovered from freshwater watershe
242                                              Enterococcus faecalis resistance to aminopenicillins, va
243 occi, as homologs from Bacillus subtilis and Enterococcus faecalis retain this ability.
244 R (D191N) first identified from the pathogen Enterococcus faecalis S613.
245                   We studied the survival of Enterococcus faecalis, Salmonella spp., E. coli O157 and
246 t side of the heart caused by streptococcus, Enterococcus faecalis, Staphylococcus aureus, or coagula
247 polysaccharide antigen (epa) gene cluster of Enterococcus faecalis strain OG1RF was shown to be atten
248           Overexpression of the Fst toxin in Enterococcus faecalis strain OG1X leads to defects in ch
249                                          The Enterococcus faecalis strain, which was not inhibited, p
250 ng a collagenolytic rodent-derived strain of Enterococcus faecalis (strain E2), and on the second day
251                                              Enterococcus faecalis strains are commensal bacteria in
252                                              Enterococcus faecalis strains are resident intestinal ba
253  of five E. faecium strains but none of five Enterococcus faecalis strains consistently developed res
254                                              Enterococcus faecalis strains secrete multiple peptides
255  have been shown to opsonize nonencapsulated Enterococcus faecalis strains.
256 hogenicity island (PAI) found among virulent Enterococcus faecalis strains.
257 fect of the sealer to multispecies bacteria (Enterococcus faecalis, Streptococcus gordonii, Actinomyc
258 including E. coli, P. aeruginosa, S. aureus, Enterococcus faecalis, Streptococcus pyogenes, Streptoco
259 aive or inactivated Staphylococcus aureus or Enterococcus faecalis, suggesting that in vivo, Eater di
260 ve Escherichia coli Symbio and gram-positive Enterococcus faecalis Symbio or its placebo.
261 ve Escherichia coli Symbio and Gram-positive Enterococcus faecalis Symbio or placebo from week 5 unti
262 ) from the opportunistic nosocomial pathogen Enterococcus faecalis synthesizes a specific lysoform li
263 endocarditis- and biofilm-associated pili of Enterococcus faecalis that are also important in experim
264 rk, we identify LiaR-independent pathways in Enterococcus faecalis that regulate cell membrane adapta
265                    In this study, we show in Enterococcus faecalis that SecA and Sortase A, required
266 y for growth of the human bacterial pathogen Enterococcus faecalis The final enzyme in this pathway,
267                                           In Enterococcus faecalis, the regulatory nucleotides pppGpp
268 st infection with the opportunistic pathogen Enterococcus faecalis through promotion of host-microbio
269 ic activity of SrtA is key to the ability of Enterococcus faecalis to bind mucin (a major component o
270                       The strategies used by Enterococcus faecalis to evade recognition by human comp
271                               The ability of Enterococcus faecalis to form robust biofilms on host ti
272 on on the antibacterial host defense against Enterococcus faecalis translocation was investigated.
273    Thermally injured mice are susceptible to Enterococcus faecalis translocation.
274                                              Enterococcus faecalis transposon insertion mutants were
275 e setup was tested with Escherichia coli and Enterococcus faecalis, two bacterial strains that are co
276 usly, our research demonstrated that dietary Enterococcus Faecalis UC-100 substituting antibiotics en
277 ons were tested against Escherichia coli and Enterococcus faecalis urinary tract infection isolates.
278 e required for DAF-16-mediated resistance to Enterococcus faecalis using a C. elegans killing assay.
279 served that a female was mono-colonized with Enterococcus faecalis vaginally as tested in aerobic cul
280 cus anginosus group (</=0.06 microg/mL), and Enterococcus faecalis (vancomycin susceptible, </=0.25 m
281 tient with recalcitrant vancomycin-resistant Enterococcus faecalis (VRE) and 2 patients with infectio
282 occus aureus (MRSA) and vancomycin-resistant Enterococcus faecalis (VRE), and another undergoes spont
283 ptococcus pyogenes, and vancomycin-resistant Enterococcus faecalis (VRE).
284 ancomycin-resistant Enterococcus faecium and Enterococcus faecalis (VRE).
285 TRSA, respectively) and vancomycin-resistant Enterococcus faecalis (VRE).
286                         Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial
287                         Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial
288 l and pathogenic host-microbe interaction of Enterococcus faecalis was explored using a Caenorhabditi
289              Under sterile conditions, 1 muL Enterococcus faecalis was inoculated inside the implants
290                   The gram-positive pathogen Enterococcus faecalis was previously reported to produce
291           Here, using a laboratory strain of Enterococcus faecalis, we developed a novel Caenorhabdit
292 nic Escherichia coli (UPEC) or Gram-positive Enterococcus faecalis, we used a mouse transurethral ins
293 omonas gingivalis and the endodontic species Enterococcus faecalis were grown to early log phase and
294 However, the AcpAs of Lactococcus lactis and Enterococcus faecalis were inactive.
295 vibrios, type E Clostridium perfringens, and Enterococcus faecalis, whereas the reverse was true for
296  adenovirus 41, Phi X 174) and the bacterium Enterococcus faecalis, which are relevant for water hygi
297 n Tn1546, acquired from vancomycin-resistant Enterococcus faecalis, which is known to alter cell wall
298 n pCF10, an antibiotic resistance plasmid of Enterococcus faecalis, which negatively regulates conjug
299 ms are absent from multidrug-resistant (MDR) Enterococcus faecalis, which only possess an orphan CRIS
300 g of one organism containing an orphan SelD, Enterococcus faecalis, with 75Se revealed a protein cont

 
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