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1 leukemia cells, yeast, Escherichia coli and Enterococcus faecalis.
2 ion of IL-10 deficient (Il10(-/-)) mice with Enterococcus faecalis.
3 itogenic, protease-secreting enteric microbe Enterococcus faecalis.
4 ainst an intestinal opportunistic bacterium, Enterococcus faecalis.
5 estinal bacterial growth, mainly E. coli and Enterococcus faecalis.
6 Escherichia coli, Klebsiella pneumoniae, and Enterococcus faecalis.
7 tivity of LmPC as well as PC from pathogenic Enterococcus faecalis.
8 survival signals modulated by the bacterium Enterococcus faecalis.
9 sion in the opportunistic bacterial pathogen Enterococcus faecalis.
10 ginosa but not to the Gram-positive pathogen Enterococcus faecalis.
11 crobial treatment were notable for growth of Enterococcus faecalis.
12 positive results with vancomycin-susceptible Enterococcus faecalis.
13 ) pilus is an important virulence factor for Enterococcus faecalis.
14 ement commonly found in clinical isolates of Enterococcus faecalis.
15 Hymeglusin blocks growth of Enterococcus faecalis.
16 aphylococcus aureus and vancomycin-resistant Enterococcus faecalis.
17 ion system is a major virulence regulator in Enterococcus faecalis.
18 role of sigma(54) in the nosocomial pathogen Enterococcus faecalis.
19 vents involving nosocomial pathogens such as Enterococcus faecalis.
20 aphylococcus aureus and vancomycin-resistant Enterococcus faecalis.
21 tococcus mutans, Actinomyces naeslundii, and Enterococcus faecalis.
22 nsporter), for subspecies differentiation of Enterococcus faecalis.
23 istic pathogens Clostridioides difficile and Enterococcus faecalis.
24 important gram-positive nosocomial pathogen Enterococcus faecalis.
25 etween secretion and cell wall attachment in Enterococcus faecalis.
26 translocation for the Gram-positive bacteria Enterococcus faecalis.
27 ance of the plasmid pAD1 in its native host, Enterococcus faecalis.
28 niae, Salmonella typhimurium, S. aureus, and Enterococcus faecalis.
29 Escherichia coli, Pseudomonas aeruginosa and Enterococcus faecalis.
30 r times at room temperature in comparison to Enterococcus faecalis.
31 89), Klebsiella pneumoniae (TOP52 1721), and Enterococcus faecalis (0852) were studied, and diabetic
32 . pyogenes, 226 Streptococcus pneumoniae, 93 Enterococcus faecalis, 1,356 Enterobacteriaceae, and 227
33 (predominant microorganism in institution A: Enterococcus faecalis, 18 cultures [51.4%]; institution
34 n this study, we investigated the ability of Enterococcus faecalis 2/28, isolated from artisan cheese
35 ecium (VRE FCM) (16), vancomycin-susceptible Enterococcus faecalis (3), Aerococcus viridans (2), Baci
37 n-a two-subunit exotoxin that is secreted by Enterococcus faecalis(5,6)-as a cause of hepatocyte deat
38 d 5 methods for testing daptomycin versus 48 Enterococcus faecalis, 51 Enterococcus faecium, and 50 S
39 Herein, we describe mechanisms that link Enterococcus faecalis, a bacterium known to produce extr
40 in the recognition of muramyl dipeptide and Enterococcus faecalis, a commensal bacterium that is a c
44 ve characterization of collateral effects in Enterococcus faecalis, a gram-positive opportunistic pat
45 al instability and colon cancer triggered by Enterococcus faecalis, a human intestinal commensal bact
46 associated inflammation impacts infection by Enterococcus faecalis, a leading cause of catheter-assoc
47 ence attribute of the opportunistic pathogen Enterococcus faecalis, a leading cause of hospital-acqui
51 accounted for 34.5% of fecal coliforms, and Enterococcus faecalis accounted for 32% of enterococci.
52 high activity against the oral key pathogens Enterococcus faecalis, Actinomyces naeslundii, Streptoco
53 lococcus aureus, Pseudomonas aeruginosa, and Enterococcus faecalis although PSO had an antimicrobial
55 eant dye, YOYO-1, were first developed using Enterococcus faecalis, an organism that has previously b
56 three most common species, Escherichia coli, Enterococcus faecalis and Bacteroides vulgatus, did not
58 he BC-GP correctly identified 14/15 cases of Enterococcus faecalis and Enterococcus faecium bacteremi
59 m for identification of vancomycin-resistant Enterococcus faecalis and Enterococcus faecium, VRESelec
61 coated quartz sand (IOCS) to remove two FIB (Enterococcus faecalis and Escherichia coli) suspended in
65 greater potency against vancomycin resistant Enterococcus faecalis and methicillin-resistant Staphylo
67 , involved in polysaccharide biosynthesis of Enterococcus faecalis and showed that disruption of epaB
68 kinetics of the d-Ala:d-Ser ligase VanG from Enterococcus faecalis and solved its crystal structure i
70 -positive bacteria Staphylococcus aureus and Enterococcus faecalis and two Gram-negative bacteria Esc
72 of MetAPs from Mycobacterium tuberculosis , Enterococcus faecalis , and human, three hotspots have b
73 g a microbe with host-protective properties (Enterococcus faecalis) and a pathogen (Staphylococcus au
74 both Gram positive ( Staphylococcus aureus, Enterococcus faecalis) and Gram negative bacteria (e.g.,
75 A protocol was developed for Gram-positive (Enterococcus faecalis) and Gram-negative (Escherichia co
76 genic bacteria (Photorhabdus luminescens and Enterococcus faecalis) and two nonpathogenic bacteria (E
77 roscan, 87% of Staphylococcus aureus, 90% of Enterococcus faecalis, and 88% of Enterococcus faecium i
78 us aureus (MRSA), Listeria monocytogenes and Enterococcus faecalis, and against the Gram-negative bac
79 57, Listeria innocua, Staphylococcus aureus, Enterococcus faecalis, and Bacillus anthracis, on sample
80 Bacillus anthracis, Propionibacterium acnes, Enterococcus faecalis, and both Methicillin-sensitive an
81 ve bacteria, including Bacillus subtilis and Enterococcus faecalis, and drug-sensitive and drug-resis
82 survival of non-pathogenic Escherichia coli, Enterococcus faecalis, and E. coli O157:H7 were compared
83 occus epidermidis, Streptococcus pneumoniae, Enterococcus faecalis, and Enterococcus faecium) and thr
85 tion by a pure facultative anaerobic strain, Enterococcus faecalis, and fresh mixed anaerobic sludge,
88 9.65, and 100.00% for Staphylococcus aureus, Enterococcus faecalis, and streptococci, respectively.
89 te genome plasticity in antibiotic resistant Enterococcus faecalis, and their involvement has been im
90 tive transfer and virulence functions of the Enterococcus faecalis antibiotic resistance plasmid pCF1
91 -negative Escherichia coli and Gram-positive Enterococcus faecalis applied during the first 7 months
92 resistant "superbugs." Bacteria of the genus Enterococcus faecalis are highly antibiotic-resistant no
93 combinations was assessed by application on Enterococcus faecalis as a model organism for Gram-posit
95 d-scan EPR can detect superoxide produced by Enterococcus faecalis at rates that are too low for dete
96 s aureus ATCC 29213 (0.004 to 0.015 mug/ml), Enterococcus faecalis ATCC 29212 (0.015 to 0.06 mug/ml),
97 cus aureus ATCC 29213 (0.03 to 0.12 mug/ml), Enterococcus faecalis ATCC 29212 (0.03 to 0.12 mug/ml),
98 sk only), S. aureus ATCC 29213 (broth only), Enterococcus faecalis ATCC 29212 (broth only), Streptoco
99 eudomonas aeruginosa ATCC 27853, 9 to 13 mm, Enterococcus faecalis ATCC 29212, 0.03 to 0.12 microg/ml
100 ccus aureus ATCC 29213, 0.25 to 2 mug/ml for Enterococcus faecalis ATCC 29212, 1 to 4 mug/ml for Esch
102 4028, Staphylococcus epidermidis ATCC 12228, Enterococcus faecalis ATCC 29212, and Escherichia coli D
103 pecies, including Pseudomonas putida KT2440, Enterococcus faecalis ATCC 29212, Salmonella Typhimurium
104 tracts showed antimicrobial activity against Enterococcus faecalis, Bacillus subtilis, Escherichia co
105 nted here had either Enterococcus faecium or Enterococcus faecalis bacteremia caused by both vancomyc
109 ers were then shown to inhibit the growth of Enterococcus faecalis biofilms that play a role in early
110 photoinactivation of a laboratory strain of Enterococcus faecalis, but depressed photoinactivation o
111 iorated sepsis-induced death associated with Enterococcus faecalis, but not Escherichia coli, infecti
112 and vanA or vanB in Enterococcus faecium and Enterococcus faecalis) by the BC-GP assay also was asses
114 tural snapshots from the type II-A system of Enterococcus faecalis Cas1 and Cas2 during spacer integr
117 Upon sensing of the peptide pheromone cCF10, Enterococcus faecalis cells carrying pCF10 produce three
119 alf-life of the metK transcript in vivo when Enterococcus faecalis cells were depleted for SAM, but n
122 ructure of the ligand binding segment of the Enterococcus faecalis collagen binding MSCRAMM ACE (micr
124 The ethanolamine utilization (eut) locus of Enterococcus faecalis, containing at least 19 genes dist
125 an SDMH is involved in biofilm formation in Enterococcus faecalis, contributing to oxidant productio
126 on from the antagonistic prgX/prgQ operon in Enterococcus faecalis controlling conjugative transfer o
127 nt bioinformatic studies have suggested that Enterococcus faecalis could serve as a model system to b
128 present in other pathogenic bacteria such as Enterococcus faecalis, Coxiella burnetii, and Clostridiu
130 In other gram-positive bacteria, such as Enterococcus faecalis, disulfide bonds are formed in sec
131 ngly difficult to treat infections caused by Enterococcus faecalis due to its high levels of intrinsi
132 port and retention behavior of two model FIB Enterococcus faecalis (E. faecalis) and Escherichia coli
135 hat Streptococcus gordonii (S. gordonii) and Enterococcus faecalis (E. faecalis) were frequent isolat
137 ins of diverse origin, including the species Enterococcus faecalis, E. faecium, E. casseliflavus, and
138 inosa (Pa), Legionella pneumophila (Lp), and Enterococcus faecalis (Ef) by using anti-infective, anti
139 applied single-molecule FRET methods to the Enterococcus faecalis (Efa) Cas1-Cas2 system to establis
141 The pheromone-responsive plasmid pCF10 of Enterococcus faecalis encodes a putative cell wall hydro
142 y named celBA) of the opportunistic pathogen Enterococcus faecalis, encodes a 6-phospho-beta-glucosid
143 transposon Tn916 from the bacterial pathogen Enterococcus faecalis, encodes a putative ArdA (alleviat
144 Expression of ace (adhesin to collagen of Enterococcus faecalis), encoding a virulence factor in e
145 ated that the ebp operon and the ace gene of Enterococcus faecalis, encoding endocarditis- and biofil
147 re several species of enterococci, including Enterococcus faecalis, Enterococcus faecium, Enterococcu
148 s (Bacillus cereus group, Enterococcus spp., Enterococcus faecalis, Enterococcus faecium, Staphylococ
149 am-positive pathogens Staphylococcus aureus, Enterococcus faecalis, Enterococcus faecium, Streptococc
150 ides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia coli K12, E. coli O15
151 ved with the type of bacterium in the order: Enterococcus faecalis, Escherichia coli O157:H7, and Esc
152 robial CAUTI and frequently cocolonizes with Enterococcus faecalis, Escherichia coli, Providencia stu
153 sis revealed that hsdS allelic variations in Enterococcus faecalis exert significant impact on gene e
154 lococcus aureus (MRSA), vancomycin-resistant Enterococcus faecalis/faecium (VREfc/VREfm), and ciprofl
155 es to PG composition in vancomycin-resistant Enterococcus faecalis following the growth in presence o
156 al species (seven Escherichia coli and three Enterococcus faecalis ) for all ten patient samples.
157 TDB, Escherichia coli, Bacillus subtilis and Enterococcus faecalis, from the guts of the desert woodr
161 ia coli (group A, 38.4%; group B, 39.3%) and Enterococcus faecalis (group A, 32.7%; group B, 33.2%).
162 -positive bacteria Staphylococcus aureus and Enterococcus faecalis have lost either all or most polya
163 idium perfringens, Escherichia coli), except Enterococcus faecalis, human milk was more antimicrobial
164 positive organisms Staphylococcus aureus and Enterococcus faecalis in comparison with known analogues
165 of the pheromone receptor protein PrgZ from Enterococcus faecalis in complex with the heptapeptide c
166 lar function in Streptococcus agalactiae and Enterococcus faecalis In conclusion, the elucidation of
167 pheromone plasmids increase the virulence of Enterococcus faecalis in experimental pathogenesis model
169 e wetland, while for the bacterial indicator Enterococcus faecalis, inactivation results were compara
170 induce high levels of (p)ppGpp production in Enterococcus faecalis, indicating that this nosocomial p
171 e, infection of catheter-implanted mice with Enterococcus faecalis induced the specific expression of
172 icantly decreased the burden of a subsequent Enterococcus faecalis infection in the nematode intestin
173 us gentamicin (AG) combinations for treating Enterococcus faecalis infective endocarditis (EFIE).
174 study, we compare outcomes in patients with Enterococcus faecalis infective endocarditis treated in
189 One of the well-studied virulence factors of Enterococcus faecalis is a secreted bacterial protease,
190 e (SAS) RelQ from the Gram-positive pathogen Enterococcus faecalis is a sequence-specific RNA-binding
196 sphate-dependent tyrosine decarboxylase from Enterococcus faecalis is followed by transformation of d
198 The Gram-positive opportunistic pathogen Enterococcus faecalis is frequently responsible for noso
202 olling pheromone-induced plasmid transfer in Enterococcus faecalis is the most thoroughly studied gen
207 e of a clinical pair of vancomycin-resistant Enterococcus faecalis isolates from the blood of a patie
209 , Clostridium species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella oxytoca, Klebsiella pn
210 ecent clinical isolates of Escherichia coli, Enterococcus faecalis, Klebsiella pneumoniae, and Pseudo
211 : Pseudomonas aeruginosa, Proteus mirabilis, Enterococcus faecalis, Klebsiella pneumoniae, Escherichi
212 Staphylococcus aureus, Enterococcus faecium, Enterococcus faecalis, Klebsiella pneumoniae, Pseudomona
213 ntestinal microbiota (Lactobacillus reuteri, Enterococcus faecalis, Lactobacillus crispatus and Clost
215 creasing sensitivity in the following order: Enterococcus faecalis LDH2 </= Lactococcus lactis LDH2 <
217 a monoclonal antibody developed against the Enterococcus faecalis major pilus protein EbpC, we ident
219 e visualization of conformational changes in Enterococcus faecalis MDD that describe sequential steps
221 lope homeostasis), from daptomycin-resistant Enterococcus faecalis not only reversed resistance to 2
223 ed molecular patterns, including heat-killed Enterococcus faecalis or CpG DNA, led to increased Ikapp
227 onoclonal antibody to the major component of Enterococcus faecalis pili, EbpC, labels polymerized pil
229 l-prolyl cis-trans isomerase, as well as the Enterococcus faecalis polysaccharide diheteroglycan, are
232 d the GIT microbiota of MAT mothers, whereas Enterococcus faecalis predominated within the MAT infant
233 EfbA is a PavA-like fibronectin adhesin of Enterococcus faecalis previously shown to be important i
236 ost & Microbe, Keogh et al. (2016) show that Enterococcus faecalis promotes Escherichia coli biofilm
237 uconostoc mesenteroides, Bacillus cereus and Enterococcus faecalis proving its antimicrobial action.
238 species such as Staphylococcus epidermidis, Enterococcus faecalis, Pseudomonas aeruginosa, and Klebs
239 the AMP-modifications with Escherichia coli, Enterococcus faecalis, Pseudomonas aeruginosa, Staphyloc
240 induced stresses (Brochothrix thermosphacta, Enterococcus faecalis, Pseudomonas fluorescens, Salmonel
241 atural enterococci diversity, 11 isolates of Enterococcus faecalis recovered from freshwater watershe
246 t side of the heart caused by streptococcus, Enterococcus faecalis, Staphylococcus aureus, or coagula
247 polysaccharide antigen (epa) gene cluster of Enterococcus faecalis strain OG1RF was shown to be atten
250 ng a collagenolytic rodent-derived strain of Enterococcus faecalis (strain E2), and on the second day
253 of five E. faecium strains but none of five Enterococcus faecalis strains consistently developed res
257 fect of the sealer to multispecies bacteria (Enterococcus faecalis, Streptococcus gordonii, Actinomyc
258 including E. coli, P. aeruginosa, S. aureus, Enterococcus faecalis, Streptococcus pyogenes, Streptoco
259 aive or inactivated Staphylococcus aureus or Enterococcus faecalis, suggesting that in vivo, Eater di
261 ve Escherichia coli Symbio and Gram-positive Enterococcus faecalis Symbio or placebo from week 5 unti
262 ) from the opportunistic nosocomial pathogen Enterococcus faecalis synthesizes a specific lysoform li
263 endocarditis- and biofilm-associated pili of Enterococcus faecalis that are also important in experim
264 rk, we identify LiaR-independent pathways in Enterococcus faecalis that regulate cell membrane adapta
266 y for growth of the human bacterial pathogen Enterococcus faecalis The final enzyme in this pathway,
268 st infection with the opportunistic pathogen Enterococcus faecalis through promotion of host-microbio
269 ic activity of SrtA is key to the ability of Enterococcus faecalis to bind mucin (a major component o
272 on on the antibacterial host defense against Enterococcus faecalis translocation was investigated.
275 e setup was tested with Escherichia coli and Enterococcus faecalis, two bacterial strains that are co
276 usly, our research demonstrated that dietary Enterococcus Faecalis UC-100 substituting antibiotics en
277 ons were tested against Escherichia coli and Enterococcus faecalis urinary tract infection isolates.
278 e required for DAF-16-mediated resistance to Enterococcus faecalis using a C. elegans killing assay.
279 served that a female was mono-colonized with Enterococcus faecalis vaginally as tested in aerobic cul
280 cus anginosus group (</=0.06 microg/mL), and Enterococcus faecalis (vancomycin susceptible, </=0.25 m
281 tient with recalcitrant vancomycin-resistant Enterococcus faecalis (VRE) and 2 patients with infectio
282 occus aureus (MRSA) and vancomycin-resistant Enterococcus faecalis (VRE), and another undergoes spont
288 l and pathogenic host-microbe interaction of Enterococcus faecalis was explored using a Caenorhabditi
292 nic Escherichia coli (UPEC) or Gram-positive Enterococcus faecalis, we used a mouse transurethral ins
293 omonas gingivalis and the endodontic species Enterococcus faecalis were grown to early log phase and
295 vibrios, type E Clostridium perfringens, and Enterococcus faecalis, whereas the reverse was true for
296 adenovirus 41, Phi X 174) and the bacterium Enterococcus faecalis, which are relevant for water hygi
297 n Tn1546, acquired from vancomycin-resistant Enterococcus faecalis, which is known to alter cell wall
298 n pCF10, an antibiotic resistance plasmid of Enterococcus faecalis, which negatively regulates conjug
299 ms are absent from multidrug-resistant (MDR) Enterococcus faecalis, which only possess an orphan CRIS
300 g of one organism containing an orphan SelD, Enterococcus faecalis, with 75Se revealed a protein cont