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1 a common DNA virus that is related to human Epstein-Barr virus.
2 virus, norovirus, rotavirus, parvovirus, and Epstein-Barr virus.
3 LTL attrition, with no association found for Epstein-Barr virus.
4 ovirus, adenovirus, human herpesvirus 6, and Epstein-Barr virus.
5 lla zoster virus, human cytomegalovirus, and Epstein-Barr virus.
7 s 1 (HSV-1) were 9% in saliva and 5% in GCF; Epstein-Barr virus 36% in saliva and 39% in GCF; human c
8 peptides from MS-associated foreign agents (Epstein-Barr virus and Akkermansia muciniphila), and aut
10 deceased newborn tested positive by qPCR for Epstein-Barr virus and human herpesvirus 6, including th
11 that infection with some viruses, including Epstein-Barr virus and influenza virus can elicit T cell
12 papillomaviruses and the gammaherpesviruses Epstein-Barr virus and Kaposi's sarcoma-associated herpe
13 el suitable for study of the human pathogens Epstein-Barr virus and Kaposi's sarcoma-associated herpe
16 simplex, varicella zoster, cytomegalovirus, Epstein-Barr virus and Toxoplasma gondii in patients wit
17 ommon viral targets include cytomegalovirus, Epstein-Barr virus, and adenovirus, though recent publis
20 several bacterial and viral taxa, including Epstein-Barr virus, as well as animal and plant DNA, whi
21 To this end, we report a self-replicating Epstein-Barr virus-based episomal vector for the long-te
23 , 1 CSF specimen was positive for EV-D68 and Epstein-Barr virus by real-time polymerase chain reactio
25 Many double-stranded DNA viruses, such as Epstein-Barr virus, can establish persistent infection,
26 persons positive for IgG antibodies against Epstein-Barr virus capsid antigen, 215 positive for thyr
28 at least 1 positive viremia during follow-up.Epstein-Barr virus D+/R- patients (P = 0.046) as well as
30 d at constructing a composite score based on Epstein-Barr virus DNAemia (EBVd) and simple clinical an
32 , TSPyV, HPyV9, HPyV10) and 5 herpesviruses (Epstein Barr virus (EBV), cytomegalovirus (CMV), herpes
35 2%), human herpesvirus 7 (HHV-7) (20.5%) and Epstein-Barr virus (EBV) (16.4%) were highly prevalent i
37 uman herpes virus (HHV)-6, HHV-7, chlamydia, Epstein-Barr virus (EBV) and bacterial 16S ribosomal DNA
38 to CD8 T cells specific for control viruses, Epstein-Barr virus (EBV) and cytomegalovirus (CMV), and
39 ither method, whereas other viruses, such as Epstein-Barr virus (EBV) and cytomegalovirus (CMV), were
42 circular RNAs (circRNAs) expressed from the Epstein-Barr virus (EBV) and Kaposi's sarcoma herpesviru
44 IMPORTANCE The two human gammaherpesviruses, Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated
47 ong all of the known biological carcinogens, Epstein-Barr virus (EBV) and Kaposi's sarcoma-associated
48 Clinically available drugs active against Epstein-Barr virus (EBV) and other human herpesviruses a
49 ic Burkitt lymphoma (eBL) is associated with Epstein-Barr virus (EBV) and Plasmodium falciparum malar
50 monella enterica, Aspergillus fumigatus, and Epstein-Barr virus (EBV) and samples from tuberculosis p
51 y with decitabine can upregulate immunogenic Epstein-Barr virus (EBV) antigens on Burkitt lymphoma (B
52 between polyomavirus JC (JC virus [JCV]) and Epstein-Barr virus (EBV) at sequences of JCV found infec
54 e NEC, namely the 1.56 angstrom structure of Epstein-Barr virus (EBV) BFRF1-BFLF2, as well as an incr
57 an herpesviruses, cytomegalovirus (HCMV) and Epstein-Barr virus (EBV) both impair the activity of APC
60 As the first discovered human cancer virus, Epstein-Barr virus (EBV) causes Burkitt's lymphoma and n
65 ting tumor cells (CTCs) and plasma levels of Epstein-Barr virus (EBV) DNA are sensitive prognostic to
66 n each sample, we measured levels of CMV and Epstein-Barr virus (EBV) DNA by droplet digital PCR (ddP
68 sorder (HVLPD) typically have high levels of Epstein-Barr virus (EBV) DNA in T cells and/or natural k
72 -1), HSV-2, human cytomegalovirus (HCMV) and Epstein-Barr virus (EBV) entry glycoproteins have define
75 's sarcoma-associated herpesvirus (KSHV) and Epstein-Barr Virus (EBV) establish life-long infections
81 for both T- and B-cell infection.IMPORTANCE Epstein-Barr virus (EBV) has a well-described tropism fo
83 stricted to the replication cycle.IMPORTANCE Epstein-Barr virus (EBV) has significant oncogenic poten
85 al cells in infection and persistence of the Epstein-Barr virus (EBV) have long been difficult to res
87 omprehensive EBV regulome in LCLs.IMPORTANCE Epstein-Barr virus (EBV) immortalization of resting B ly
88 om anal samples; 2.7% (95% CI: 0.7-4.7%) for Epstein-Barr virus (EBV) immunoglobulin M (IgM) positivi
90 ally permissive phenotype of the herpesvirus Epstein-Barr virus (EBV) indicate that upon exposure to
92 isorder (PTLD) are those who acquire primary Epstein-Barr virus (EBV) infection after solid organ tra
93 essive head and neck cancer characterized by Epstein-Barr virus (EBV) infection and dense lymphocyte
94 al carcinoma (NPC) is highly associated with Epstein-Barr virus (EBV) infection and exhibits remarkab
96 ene cause X-linked magnesium deficiency with Epstein-Barr virus (EBV) infection and neoplasia (XMEN),
99 and environmental risk factors, among which Epstein-Barr virus (EBV) infection is a strong suspect.
114 erial artificial chromosome (BAC).IMPORTANCE Epstein-Barr virus (EBV) infects the majority of the wor
116 s to treat EBV-associated disease.IMPORTANCE Epstein-Barr virus (EBV) is a causative agent of various
128 ation or its role in Zta function.IMPORTANCE Epstein-Barr virus (EBV) is a ubiquitous human pathogen
145 novel mechanisms of host control.IMPORTANCE Epstein-Barr virus (EBV) is transmitted orally, replicat
147 program and DNA methylation state.IMPORTANCE Epstein-Barr virus (EBV) latency and carcinogenesis invo
149 In this study, we provide evidence that Epstein-Barr virus (EBV) latent membrane protein 1 (LMP1
153 s: cytomegalovirus mismatch (18.5% [10/54]), Epstein-Barr virus (EBV) mismatch (EBV) (9.3% [5/54]), a
156 ressive lymphoid tumor which is occasionally Epstein-Barr virus (EBV) positive and is further subtype
158 n lymphomas (HIV-NHLs), which are frequently Epstein-Barr virus (EBV) positive or human herpesvirus t
164 Recent studies reported that posttransplant Epstein-Barr virus (EBV) replication is frequent and ind
165 ect of subclinical cytomegalovirus (CMV) and Epstein-Barr virus (EBV) replication on CD4(+) and CD8(+
168 This study identifies an interaction between Epstein-Barr virus (EBV) SM protein and cellular helicas
171 sequences from an enlarged set of about 200 Epstein-Barr virus (EBV) strains, including many primary
173 e remarkable task of lifelong infection, the Epstein-Barr virus (EBV) switches between four viral gen
178 fically manipulate the HIV-1 genome but also Epstein-Barr virus (EBV) which, similar to KSHV, belongs
179 that recognized five common viral pathogens: Epstein-Barr virus (EBV), adenovirus (AdV), cytomegalovi
180 cellular functions, we investigated whether Epstein-Barr virus (EBV), an oncoherpesvirus, exploits i
181 s B virus (HBV), human papillomavirus (HPV), Epstein-Barr virus (EBV), and BK Virus (BKV), suggesting
182 ection of closely related DNA viruses: KSHV, Epstein-Barr virus (EBV), and herpes simplexvirus-2 (HSV
185 murine gammaherpesvirus 68 (MHV68), BGLF5 in Epstein-Barr virus (EBV), and vhs in herpes simplex viru
186 treating EBV-associated lymphomas.IMPORTANCE Epstein-Barr virus (EBV), as the first human tumor virus
187 iral response to HSV-1 and the related virus Epstein-Barr virus (EBV), as well as influenza A virus (
188 itative PCR assays of cytomegalovirus (CMV), Epstein-Barr virus (EBV), BK virus (BKV), adenovirus (AD
189 ain reaction for periodontal viruses such as Epstein-Barr virus (EBV), cytomegalovirus (CMV) and Herp
191 were infected with an oncogenic recombinant Epstein-Barr virus (EBV), encoding enhanced firefly luci
192 ce of known tumor-associated viruses such as Epstein-Barr virus (EBV), hepatitis B virus (HBV) and hu
193 viremia episodes with cytomegalovirus (CMV), Epstein-Barr virus (EBV), human herpesvirus 6 (HHV-6), h
194 ncogenic gammaherpesviruses, including human Epstein-Barr virus (EBV), human Kaposi's sarcoma-associa
197 re, we show that the large dsDNA herpesvirus Epstein-Barr virus (EBV), which is the causative agent o
198 n follicular helper T cells (TFH cells), and Epstein-Barr virus (EBV), which persists in B cells.
202 s), is an appealing therapeutic approach for Epstein-Barr virus (EBV)-associated malignancies of late
203 Several therapeutic strategies targeting Epstein-Barr virus (EBV)-associated tumors involve upreg
204 rs the formation of PDX tumors that resemble Epstein-Barr virus (EBV)-driven B-cell lymphomas rather
205 Lymphomatoid granulomatosis (LYG) is a rare Epstein-Barr virus (EBV)-driven B-cell lymphoproliferati
206 triggered by the gly-ala repeat sequence of Epstein-Barr virus (EBV)-encoded EBNA1, results in PI3Kd
208 ite advances in T-cell immunotherapy against Epstein-Barr virus (EBV)-infected lymphomas that express
209 cause a T-cell primary immunodeficiency with Epstein-Barr virus (EBV)-lymphoproliferative disorders.
212 ty-two cytomegalovirus (CMV)-positive and 41 Epstein-Barr virus (EBV)-positive plasma samples, togeth
213 sed throughout infection, can be detected in Epstein-Barr virus (EBV)-positive tumors, and manipulate
215 vely, detected in cytomegalovirus (CMV)- and Epstein-Barr virus (EBV)-responsive CD4+ T cells followi
216 ncies for influenza-specific (A2/M1(58)) and Epstein-Barr virus (EBV)-specific (A2/BMLF(1280)) (~1.38
217 reinvigorated by the implementation of novel Epstein-Barr virus (EBV)-specific IgA and IgG antibodies
218 nt of LUBAC, interacts with LMP1 and IRF7 in Epstein-Barr virus (EBV)-transformed cells and that LUBA
228 reptococcus mutans, and Human herpesvirus 4 (Epstein-Barr virus [EBV]) were more prevalent in childre
230 al infections (HIV, hepatitis C virus [HCV], Epstein-Barr virus [EBV], and cytomegalovirus [CMV]) hav
231 to control antigens (cytomegalovirus [CMV], Epstein-Barr virus [EBV], and influenza virus [FLU]) or
232 ADV], BK virus [BKV], cytomegalovirus [CMV], Epstein-Barr virus [EBV], human herpesvirus 6A [HHV-6A],
233 titis B core [HBc], hepatitis C virus [HCV], Epstein-Barr virus [EBV], or cytomegalovirus [CMV]) in K
235 rom both adenovirus-associated (VA) RNAs and Epstein-Barr virus-encoded small RNAs (EBERs) with respe
236 lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, enterovirus, cytomegalovirus, and ch
237 ctors included a high prevalence of previous Epstein-Barr virus exposure and a relatively low immunol
238 globulin plus GVHD prophylaxis group died of Epstein-Barr virus hepatitis, but no deaths were attribu
241 type) or BGLF5 (the KSHV protein homolog in Epstein-Barr virus) in 293L/Q129H cells restored the vir
242 positive test results for cytomegalovirus or Epstein-Barr virus, indicating possible cross-reactivity
244 ll migration, the G protein-coupled receptor Epstein-Barr virus-induced gene 2 (EBI2 or GPR183) direc
246 sions: In COPD lungs, we found lung EBI2(+) (Epstein-Barr virus-induced gene 2-positive) OX-40L-expre
248 se that expressed the extrafollicular marker Epstein-Barr virus-induced protein 2 (EBI2), but signifi
249 report that in human airway epithelial cells Epstein-Barr virus induces TRIM29, a member of the TRIM
251 s, such as the incidence of acute rejection, Epstein-Barr virus infection, sepsis, biliary and vascul
256 xic effects of Zta overexpression.IMPORTANCE Epstein-Barr virus infects most people across the world
257 nt for diverse functions of EBNA1.IMPORTANCE Epstein-Barr virus is a human gammaherpesvirus that is c
258 trategies in the clinical setting.IMPORTANCE Epstein-Barr virus is associated with many different can
260 r efficient viral DNA replication.IMPORTANCE Epstein-Barr virus is the causative agent of infectious
262 s in the blood of patients with myocarditis: Epstein Barr virus (n=11, 41%), human pegivirus (n=1, 4%
263 s B virus (n = 10), cytomegalovirus (n = 9), Epstein-Barr virus (n = 5), and rotavirus A (n = 3) were
264 B virus (n = 2), human pegivirus 1 (n = 2), Epstein-Barr virus (n = 9), and Orungo virus (n = 1), a
265 n vIRF1, which is identical to that found in Epstein-Barr virus nuclear antigen 1 (EBNA1) that intera
266 ting tumor cells requires the interaction of Epstein-Barr virus nuclear antigen 1 (EBNA1) with the vi
268 es to the host cell nucleolus where it binds Epstein-Barr virus nuclear antigen 1-binding protein 2 (
270 utologous VSTs specific for cytomegalovirus, Epstein-Barr virus, or adenovirus and genetically modifi
272 y cutaneous acral CD8(+) T-cell lymphoma and Epstein-Barr virus positive (EBV(+)) mucocutaneous ulcer
280 tes EBV reactivation from latency.IMPORTANCE Epstein-Barr virus represents an important human pathoge
281 element (HRE) located within the promoter of Epstein-Barr virus's (EBV's) latent-lytic switch BZLF1 g
282 bodies (aIRR = 2.03); in recipients who were Epstein-Barr virus-seronegative at the time of transplan
284 he atomic structure of the portal protein of Epstein-Barr virus, solved by cryo-electron microscopy a
285 ion marker CD57, and fewer HDV-specific than Epstein-Barr virus-specific CD8(+) T cells were 2B4(+)CD
291 HLA-A*02:01 and HLA-B*27:05 expressed on the Epstein-Barr virus-transformed B cell line Jesthom and M
294 HSV UL37 with the human cytomegalovirus and Epstein-Barr virus UL37 homologs revealed that Y480 was
297 iherpesviral and anticancer drugs.IMPORTANCE Epstein-Barr virus, which is nearly ubiquitous in humans
298 virus type 1, human herpesvirus type 6, and Epstein-Barr virus) with change in leukocyte telomere le
299 viral infections with human herpesvirus 6 or Epstein-Barr virus within 100 days after transplant incr