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1 Multiplex analysis of 191-bp amplicons from Escherichia coli O157 and 256-bp amplicons from E. coli
3 hree commercial latex reagents for detecting Escherichia coli O157 antigen (Oxoid Diagnostic Reagents
5 jor reservoir host for the zoonotic pathogen Escherichia coli O157, are known to exhibit a high degre
8 la, Campylobacter, Giardia, Cryptosporidium, Escherichia coli O157, enterotoxigenic E. coli (ETEC), a
9 ity into an agent-based simulation model for Escherichia coli O157 environmental transmission in catt
16 or for the detection of the food contaminant Escherichia coli O157:H7 (E. coli O157:H7) in complex fo
17 antibody-free, and aptamer-free detection of Escherichia coli O157:H7 (E. coli O157:H7) in water samp
18 as used for the detection of food pathogenic Escherichia coli O157:H7 (E.coli O157:H7), a dangerous s
20 Surviving predation is a fitness trait of Escherichia coli O157:H7 (EcO157) that provides ample ti
24 Here, we demonstrate that enterohemorrhagic Escherichia coli O157:H7 (EHEC) regulates virulence via
25 roduct of the eaeA gene in enterohemorrhagic Escherichia coli O157:H7 (EHEC), is required for intimat
26 h predicted a duplicated type I locus within Escherichia coli O157:H7 (EHEC), which we have named the
29 The locus of enterocyte effacement (LEE) of Escherichia coli O157:H7 (O157) encodes a type III secre
30 l models, we directly identified immunogenic Escherichia coli O157:H7 (O157) proteins expressed eithe
33 orphic amplified typing sequences (PATS) for Escherichia coli O157:H7 (O157) was previously based on
36 In the 1980s, Shiga toxin (Stx)-producing Escherichia coli O157:H7 (STEC) was identified as a caus
38 lerae O1 (the causative agent of cholera) or Escherichia coli O157:H7 (the leading cause of food-born
39 etect Shiga-like toxin gene II (SLT-II) from Escherichia coli O157:H7 after asymmetric, capillary, PC
40 nvestigation of four cases of infection with Escherichia coli O157:H7 among laboratorians from differ
41 ic region between the mutS and rpoS genes of Escherichia coli O157:H7 and closely related strains rep
42 ods with samples from children infected with Escherichia coli O157:H7 and correlated the antigen dete
45 Six protein biomarkers from two strains of Escherichia coli O157:H7 and one non-O157:H7, nonpathoge
46 The mechanisms underlying the adherence of Escherichia coli O157:H7 and other enterohemorrhagic E.
47 rial strain identification for the typing of Escherichia coli O157:H7 and other gram-negative organis
48 ins (Stx) are critical virulence factors for Escherichia coli O157:H7 and other serotypes of enterohe
51 en implicated as a major virulence factor of Escherichia coli O157:H7 and other Shiga toxin-producing
52 e important contributors to the virulence of Escherichia coli O157:H7 and other Stx-producing E. coli
53 e of commensal flora reduced colonization of Escherichia coli O157:H7 and production of Shiga toxin (
54 AMP magainin I as a recognition element for Escherichia coli O157:H7 and Salmonella typhimurium dete
56 fection of humans with Shiga toxin-producing Escherichia coli O157:H7 and Shigella dysenteriae 1 is s
57 o examine the impact of pathogenic bacteria, Escherichia coli O157:H7 and three different Salmonella
58 as fabricated by covalently grafting an anti-Escherichia coli O157:H7 antibody onto SAM-modified gold
59 n with gastrointestinal infections caused by Escherichia coli O157:H7 are at risk for the hemolytic-u
61 mmunofilter captured the targeted pathogens, Escherichia coli O157:H7 as an example for bacteria and
62 he few adhesive factors of enterohemorrhagic Escherichia coli O157:H7 associated with colonization of
64 eine residues in the passenger domain of the Escherichia coli O157:H7 autotransporter EspP at differe
65 mall linker into the passenger domain of the Escherichia coli O157:H7 autotransporter EspP effectivel
66 he C terminus of the passenger domain of the Escherichia coli O157:H7 autotransporter EspP have been
67 ient to promote the complete assembly of the Escherichia coli O157:H7 autotransporter EspP in vitro.
68 ere we show that the passenger domain of the Escherichia coli O157:H7 autotransporter EspP is release
69 ent secretion of the passenger domain of the Escherichia coli O157:H7 autotransporter EspP requires t
70 se toxin CyaA fused to the C terminus of the Escherichia coli O157:H7 autotransporter EspP to test th
73 complex interact with the beta domain of the Escherichia coli O157:H7 autotransporter extracellular s
79 sensor chips were developed for detection of Escherichia coli O157:H7 based on the surface immobiliza
80 d toxin homologous to a predicted toxin from Escherichia coli O157:H7 but had apparently lost the try
81 or/bioreactor was developed for detection of Escherichia coli O157:H7 by chemically immobilizing anti
84 ated to an outbreak of Shiga toxin-producing Escherichia coli O157:H7 caused by the consumption of ra
90 udied interactions of antibodies with single Escherichia coli O157:H7 cells and demonstrated a capabi
91 ed by lateral flow strip detection of viable Escherichia coli O157:H7 cells by detecting the RNA of t
93 nce of the sensor was evaluated by capturing Escherichia coli O157:H7 cells on the antibody-stamped l
94 differentiate 10(3) and 10(6) CFU/uL initial Escherichia coli O157:H7 concentrations compared to NTC
95 0, and 20 muM) on Listeria monocytogenes and Escherichia coli O157:H7 cultivated in tryptic soy broth
97 question, we separated the production of an Escherichia coli O157:H7 exoprotein (OtpA) and its trans
98 effector protein Tir from enterohemorrhagic Escherichia coli O157:H7 expressed in epithelial cells i
100 irms on-chip detection and identification of Escherichia coli O157:H7 from colony isolates in a color
101 ivity ( approximately 95%) in isolating live Escherichia coli O157:H7 from cultures containing 1000 E
106 r of protocols for the cultural detection of Escherichia coli O157:H7 in clinical fecal specimens hav
107 aditional fecal culture for the detection of Escherichia coli O157:H7 in experimentally infected and
108 ay for rapid detection and quantification of Escherichia coli O157:H7 in meat and water samples based
109 n of the O-antigen biosynthesis cluster from Escherichia coli O157:H7 in strain 1244 resulted in the
110 ous detection of Salmonella, Legionella, and Escherichia coli O157:H7 in tap water and wastewater.
111 ources that support the growth of pathogenic Escherichia coli O157:H7 in the mammalian intestine have
112 The largest reported outbreak of waterborne Escherichia coli O157:H7 in the United States occurred i
115 The use of antibiotics for treatment of Escherichia coli O157:H7 infection has become controvers
116 94, 21 cases (19 primary and 2 secondary) of Escherichia coli O157:H7 infection were identified in th
120 ts were identified with laboratory-confirmed Escherichia coli O157:H7 infection; 52 residents had blo
128 ultilocus-genotyping methods have shown that Escherichia coli O157:H7 is a geographically disseminate
141 magnetoelastic immunosensor for detection of Escherichia coli O157:H7 is described, based on immobili
142 A hemolytic determinant of enterohemorrhagic Escherichia coli O157:H7 is encoded on a 90-kbp plasmid
143 a toxin 2 (Stx2) from the foodborne pathogen Escherichia coli O157:H7 is encoded on a temperate bacte
145 Rapid detection of the foodborne pathogen Escherichia coli O157:H7 is of vital importance for publ
152 recommended that all stools be cultured for Escherichia coli O157:H7 on selective medium as well as
153 netic nanoparticles functionalized with anti-Escherichia coli O157:H7 or anti-Salmonella typhimurium
154 estimate the frequency of enterohemorrhagic Escherichia coli O157:H7 or O157:nonmotile (EHEC O157) i
155 ic uremic syndrome after oral infection with Escherichia coli O157:H7 or other Shiga toxin--producing
159 ss outbreaks resulting from contamination of Escherichia coli O157:H7 remain a serious concern in foo
160 R from Mu-like prophage sequences present in Escherichia coli O157:H7 Sakai, Haemophilus influenzae R
162 ment (LEE) from EDL933, an enterohemorrhagic Escherichia coli O157:H7 serovar originally isolated fro
165 ve constructed NheI and XhoI optical maps of Escherichia coli O157:H7 solely from genomic DNA molecul
171 Promoter alterations in the csgD gene of Escherichia coli O157:H7 strains ATCC 43894 and ATCC 438
173 The rapid and accurate identification of Escherichia coli O157:H7 strains is central to reducing
176 onocytogenes, but was less effective against Escherichia coli O157:H7 than free LAE, which was correl
177 entified StcE, a metalloprotease secreted by Escherichia coli O157:H7 that cleaves the serpin C1 este
178 quired genomic regions of enterohaemorrhagic Escherichia coli O157:H7 that regulate expression of the
179 and duration of fecal shedding of wild-type Escherichia coli O157:H7 to that of an isogenic intimin
181 Here we identify the enterohaemorrhagic Escherichia coli O157:H7 type III effector EspG as a reg
182 microbalance for the detection of pathogenic Escherichia coli O157:H7 using TCEP-reduced antibody wit
183 eal-time PCR assay to detect the presence of Escherichia coli O157:H7 using these fluorogenic reporte
184 Efficient photocatalytic disinfection of Escherichia coli O157:H7 was achieved by using a C70 mod
185 demonstrated in a model experiment in which Escherichia coli O157:H7 was detected at concentrations
187 mpedance immunosensor for rapid detection of Escherichia coli O157:H7 was developed by immobilizing a
188 a enrichment and detection system for viable Escherichia coli O157:H7 was developed using a piezoelec
190 polar fimbriae to intestinal colonization by Escherichia coli O157:H7 was evaluated in sheep, convent
196 a toxin (Stx)-producing, food-borne pathogen Escherichia coli O157:H7 will develop a life-threatening
197 l RNA samples, the assay truly discriminated Escherichia coli O157:H7's 16s rRNA from closely related
199 genes) than from the gram-negative organism (Escherichia coli O157:H7), preventing unbiased detection
205 erization of an effector protein (NleL) from Escherichia coli O157:H7, a widespread pathogen causing
207 tensively to investigate the epidemiology of Escherichia coli O157:H7, although it has not been evalu
208 cterium in the order: Enterococcus faecalis, Escherichia coli O157:H7, and Escherichia coli D21f2.
209 ts, with an emphasis on Salmonella enterica, Escherichia coli O157:H7, and Listeria monocytogenes.
210 ve bacteria, including Campylobacter jejuni, Escherichia coli O157:H7, and multidrug resistant Klebsi
211 pXO1 from Bacillus anthracis, and pO157 from Escherichia coli O157:H7, as well as the broad host rang
212 versity and survival of the invading species Escherichia coli O157:H7, assessed by using the marked d
213 to the strong interaction between AuNPs and Escherichia coli O157:H7, AuNPs attached to the surface
214 e technique for rapid detection of low-level Escherichia coli O157:H7, Bacillus subtilis var. niger s
215 To better define the bovine reservoir of Escherichia coli O157:H7, cattle were tested monthly by
216 analyses of previously sequenced strains of Escherichia coli O157:H7, EDL933 and Sakai, localized th
217 r flagellar filaments from enterohemorrhagic Escherichia coli O157:H7, enteropathogenic E. coli O127:
219 ca serotype Enteritidis (S. enteritidis) and Escherichia coli O157:H7, has generated increasing inter
221 E) was used to compare Wisconsin isolates of Escherichia coli O157:H7, including 39 isolates from a 1
222 llowing infection by the intestinal pathogen Escherichia coli O157:H7, is due to the ability of the p
224 ht emitting diodes (LEDs) against pathogenic Escherichia coli O157:H7, Listeria monocytogenes, Pseudo
225 ates of the recently emerged clonal pathogen Escherichia coli O157:H7, making them a very useful mole
226 spersion of pathogens (Campylobacter jejuni, Escherichia coli O157:H7, non-O157 E. coli, Listeria mon
229 effacing (A/E) bacterial pathogens, such as Escherichia coli O157:H7, pose a serious threat to publi
230 inst Staphylococcus aureus, Bacillus cereus, Escherichia coli O157:H7, Pseudomonas aeruginosa and Sal
231 -MNP induced strong dose-dependent bacteria (Escherichia coli O157:H7, Salmonella enterica serovar Ty
232 against common foodborne pathogens including Escherichia coli O157:H7, Salmonella enteritidis, Salmon
233 (LOD) was <5 CFU/mL of the tested pathogens (Escherichia coli O157:H7, Salmonella typhimurium, and Li
234 osis, the 5' 350 bp of the 16S rRNA genes of Escherichia coli O157:H7, Salmonella typhimurium, Salmon
235 isolates of food-borne pathogens (currently Escherichia coli O157:H7, Salmonella, Shigella, and List
238 lly identify a variety of bacterium, such as Escherichia coli O157:H7, through antibody-antigen inter
239 olorimetric immunoassay for the detection of Escherichia coli O157:H7, using antibody-directed liposo
240 swab samples cultured in our laboratory for Escherichia coli O157:H7, we frequently isolated E. coli
242 that of a recently identified stcE gene from Escherichia coli O157:H7, which encoded a protein (StcE)
243 network is important for the pathogenesis of Escherichia coli O157:H7, which has captured many virule
244 nts, specifically pathogenic species such as Escherichia coli O157:H7, which is a highly infectious a
245 gen is an essential part of the detection of Escherichia coli O157:H7, which is recognized as a major
264 2009 outbreak of Shiga-like toxin-producing Escherichia coli O157 infection associated with a pettin
265 obacter infection, 2.7 {CI, 1.5-4.8}; OR for Escherichia coli O157 infection, 7.4 {CI, 2.1-26.1}]).
269 Detailed laboratory characterization of Escherichia coli O157 is essential to inform epidemiolog
272 erformed pulsed-field gel electrophoresis on Escherichia coli O157 isolates (n = 318) from 199 health
273 nd ribotyping, were used to characterize 207 Escherichia coli O157 isolates from food animals, foods
274 addition of the target ligand (in this case Escherichia coli O157) leads to the formation of a nanop
275 mate different species of pathogens, such as Escherichia coli O157, Listeria innocua, Staphylococcus
278 l enteropathogens: Aeromonas, Campylobacter, Escherichia coli O157, other Shiga toxin-producing E. co
280 tic thrombocytopenic purpura (TTP) during an Escherichia coli O157 outbreak occurred in 1996 in centr
281 or 0/2 Campylobacter-positive specimens, 0/4 Escherichia coli O157-positive specimens, 0/9 Salmonella
282 nal pathogens, such as Shiga toxin-producing Escherichia coli O157 (STEC O157), is key to rapidly ide
285 ks of vero cytotoxin (shiga toxin) producing Escherichia coli O157 (VTEC O157) infection have stimula
288 ransmission of a microparasite, in this case Escherichia coli O157, within a multigroup system, namel