ライフサイエンスコーパス: FBS

1 FBS (1%-8%) increased cell numbers in a dose-dependent m

2 FBS also influenced the mode of degradation by limiting

3 FBS also significantly decreased the lipid accumulation

4 FBS and IL-5 failed to inhibit or suppress the CD30 agon

5 FBS and RD were rich in Lys, Glu, Gly, Pro, Asp, Leu, Hi

6 FBS had little impact on mass loss of pure Mg during imm

7 FBS hydrolysates showed higher antioxidant activity base

8 FBS or IUPT resulted in a relatively high complication r

9 FBS reduced the mass loss of Mg-Yttrium (MgY) alloy with

10 FBS was hydrolysed to a greater extent than RD regardles

11 FBS-associated RNA is co-isolated with cell-culture deri

12 FBS-cultured cells also showed higher MsgA and NanA acti

13 s were incubated for various periods in 0.1% FBS (a concentration that maintains cell health, but doe

14 In cells kept in 0.1% FBS, there is DIO2 inhibition via FOXO1 binding to the D

15 r (2) 10% FBS with FGF and heparin or (3) 1% FBS or (4) 1% FBS with TGF-beta.

16 with FGF and heparin or (3) 1% FBS or (4) 1% FBS with TGF-beta.

17 ize keratocan when reseeded on plastic in 1% FBS or on AM.

18 In 1% FBS, AM-expanded keratocytes rapidly became alpha-SMA-ex

19 absence of FBS but not in the presence of 1% FBS.

20 s generated by shifting HAECs from 10% to 1% FBS.

21 and 1000 pg/mL TGF-beta1 or in DMEM with 1% FBS and 10 ng/mL TGF-beta1.

22 ls were grown in DMEM/F12 containing (1) 10% FBS or (2) 10% FBS with FGF and heparin or (3) 1% FBS or

23 n DMEM/F12 containing (1) 10% FBS or (2) 10% FBS with FGF and heparin or (3) 1% FBS or (4) 1% FBS wit

24 ChIP studies indicate that 4 h after 10% FBS-containing medium, FOXO1 binding markedly decreases,

25 n in response to endothelin-1, PDGF, and 10% FBS by 62%, 51%, and 29%, respectively (P<0.01), without

26 Both 10% FBS and PDGF stimulated significant keratocyte migration

27 rough the uncompressed outer matrix, but 10% FBS produced more cell-induced collagen matrix reorganiz

28 ists (by 30% to 60%, P<0.01) but also by 10% FBS (by 35%, P<0.05).

29 e cultured in DMEM/F12 medium containing 10% FBS and 5 microg/mL insulin at 37 degrees C in 5% CO(2).

30 te medium and DMEM/F12 medium containing 10% FBS in a 37 degrees C incubator supplied with 5% CO(2),

31 e cultured in DMEM/F12 medium containing 10% FBS in a 37 degrees C incubator supplied with 5% CO(2).

32 In the media containing 10% FBS, forskolin also stimulated proliferation of Schwann

33 MEM/ITS) or 10% fetal bovine serum (DMEM/10% FBS), or in a defined keratinocyte serum-free medium (KS

34 downregulated in KSFM compared with DMEM/10% FBS.

35 d by switching KSFM to DMEM/ITS and DMEM/10% FBS.

36 ays using 0.1% fetal bovine serum (FBS), 10% FBS +/- 10 microM SB, or 20 ng/ml PDGF +/- 10 microM SB.

37 cells appeared dendritic on AM, even in 10% FBS but fibroblastic on plastic.

38 In contrast, cells in 10% FBS developed a bipolar fibroblastic morphology.

39 ucleotides followed by postincubation in 10% FBS lowers endogenous p27(kip1) protein levels and promo

40 (20 ng/ml) plus heparin (5 microg/ml) in 10% FBS medium had decreased expression of alpha-SM actin pr

41 Keratocytes cultured in 10% FBS proliferated, appeared fibroblastic, and synthesized

42 ued at least to passage 6 on AM, even in 10% FBS, but was rapidly lost each time when cells on AM wer

43 hen comparing levels of cellular fill in 10% FBS, GF displayed greater wound fill than the PDL.

44 levels of wound fill for PDLs and GFs in 10% FBS.

45 active when cultured in the presence of 10% FBS (fetal bovine serum), with a replication time of 1-3

46 as a negative control, and a culture of 10% FBS served as a positive control.

47 ease of [Ca2+] to 1.8 mM and addition of 10% FBS synergistically downregulated the keratocan promoter

48 In the presence of 10% FBS, rBPI(21) and XMP.Z increased BRP growth by 91% +/-

49 response of either cell type compared to 10% FBS alone.

50 The addition of SB to 10% FBS did not significantly affect the wound fill response

51 Relative to 10% FBS, PDLs stimulated with PDGF showed significantly (P <

52 CGF and in positive controls exposed to 10% FBS.

53 n a cell model transitioning from 0.1 to 10% FBS.

54 l was conferred by incubating cells with 10% FBS and IL-5.

55 Stimulation of Rb cells with 10% FBS resulted in an increase in CTCF expression and a dec

56 washed twice with MEM supplemented with 10% FBS, and fibroblasts in treatment and control groups wer

57 re either scrape wounded or treated with 10% FBS, PDGF, or FGF-2 for 6 hours.

58 orneal and 293T fibroblasts treated with 10% FBS, PDGF, or FGF-2.

59 er serum-starved cells were treated with 10% FBS, PDGF, or FGF-2.

60 hen incubated in media supplemented with 10% FBS, TGFbeta1, TGFbeta2, platelet-derived growth factor

61 ane matrix (AM) in DMEM, with or without 10% FBS, in serum-free DMEM containing insulin-transferrin-s

62 A culture of 0.2% FBS alone served as a negative control, and a culture of

63 by growth for 48 hours in DMEM/F12 with 0.2% FBS and subsequently were either scrape wounded or treat

64 In differentiation-promoting media (2% FBS), IUGR and control myoblasts had similar percentages

65 esponded to fluid flow in the presence of 2% FBS, confirming that P2Y2 purinoceptors are responsible

66 creases in [Ca(2+)](i) in the presence of 2% FBS.

67 DMEM) plus 2% fetal bovine serum (FBS) or 2% FBS plus EMD (100 microg/ml), with and without ascorbic

68 e effect on SVF cells, when compared with 2% FBS control.

69 owth of BRPs, even above that induced by 20% FBS.

70 ells were seeded, maintained overnight in 4% FBS to permit cell attachment, washed, and incubated for

71 itated by 0.9 mM Ca(2+) but suppressed by 5% FBS in KSFM at passage 0.

72 concentration to 0.9 mM, with or without 5% FBS.

73 that VEGF protects MM patient cells against FBS starvation-induced apoptosis.

74 In addition, both humic acid and FBS protein effectively lowered the amounts of fullerene

75 Humic acid and FBS significantly changed the characteristics of fullere

76 an mRNAs were downregulated by TGF-beta1 and FBS.

77 In contrast, both EGF and FBS significantly increased [(3)H]thymidine incorporatio

78 ne, whereas in the presence of CGF + EGF and FBS they remained elevated for up to 20 h.

79 to activate ERK1 and ERK2; however, EGF- and FBS-induced activation of ERKs was not different from th

80 ptor, integrin alpha5beta1, inhibited Fn and FBS-mediated invasion but did not specifically inhibit L

81 Proteins found in human and FBS bound exogenous HNP-2 and retrocyclin-1, and compete

82 Insulin-like growth factor I and FBS activate NADPH oxidase through transcriptional up-re

83 With poor ICHS and FBS as references, individuals with ideal ICHS and FBS s

84 was to compare the effectiveness of ICHS and FBS in predicting the presence and extent of subclinical

85 references, individuals with ideal ICHS and FBS showed lower adjusted odds of having atherosclerotic

86 criminating accuracy were found for ICHS and FBS with respect to the presence of plaques (C-statistic

87 e between 20 and 400 nm from human serum and FBS using ultracentrifugation and sucrose gradient centr

88 f the antenatal management strategy applied; FBS, IUPT, or IVIG with or without corticosteroids.

89 Latino, 31% African American, and a baseline FBS of 9.3 +/- 2.4 points.

90 -ATP channel opener) administered 1 h before FBS addition restored the FBS response in propofol treat

91 consistent effect of cAMP was to block both FBS- and DeltaRaf-1:ER-induced expression of Cdc25A and

92 house screening programs that performed both FBS of 8800 fragments and screens of the full library.

93 sCAR) were similar with and without FBS, but FBS amplified the catalytic effect of 100 nM sCAR nearly

94 nterference and misinterpretations caused by FBS-derived RNA.

95 Smaller fragments identified by FBS occupy both these pockets independently and suggest

96 etic correlations between measured IOP, CCT, FBS, fasting insulin, and hemoglobin A1c were null.

97 e development of a potential fourth chelator FBS 0701 and the combined use of oral chelators may furt

98 Many evolutionally conserved FBS-derived RNA species can be falsely annotated as huma

99 dependent fashion and binds to the conserved FBS.

100 ycated FBS (experimental) or RPMI-1% control FBS, and cells were incubated for 1 or 4 d.

101 w)) of the chemicals was used to calculate D(FBS/w) and D(cell/w).

102 hemicals, the mass balance model predicted D(FBS/w) and D(cell/w) within a factor of 3 and 3.4, respe

103 s study, medium-water distribution ratios (D(FBS/w)) and cell-water distribution ratios (D(cell/w)) f

104 ation of a new shoulder site in deafferented FBS antidromically-activated a cell in the former forepa

105 that the new shoulder input in deafferented FBS is relayed from cells in the former forepaw region i

106 d to a new shoulder site in the deafferented FBS, we examined the thalamocortical pathway in 2 foreli

107 an L-type Ca(2+) channel blocker, decreased FBS-induced Ca(2+) response of control cells to a level

108 reporter plasmids by both estrogen-deficient FBS (ED-FBS) and EGF.

109 tion in the preparations of vesicle-depleted FBS (vdFBS) commonly utilized in the studies of extracel

110 growing them in culture media with different FBS content.

111 culture supplement), or heat-denatured DMEM-FBS.

112 MEM-AH differed from those incubated in DMEM-FBS.

113 the more spindle-shaped cells grown in DMEM-FBS.

114 atured DMEM-AH, 10% fetal bovine serum (DMEM-FBS, the standard culture supplement), or heat-denatured

115 d cell proliferation when compared with DMEM-FBS (11% vs. 141%).

116 medium isolated from cells treated with DMEM-FBS (442%), but only a 10% increase in myocilin was obse

117 same degree as the wild-type receptor by ED-FBS and EGF in both HSVSMC and PVEC.

118 plasmids by both estrogen-deficient FBS (ED-FBS) and EGF.

119 ion of primary cultures stimulated by either FBS or angiotensin II.

120 for in this study (heritability estimate for FBS is 68%).

121 tiple significant epistatic interactions for FBS, which accounts for half (14.6%) of F2 variance comp

122 ltures can be continually passaged in fresh, FBS-free F-12 medium at an initial inoculum of only appr

123 Nanobacteria were previously isolated from FBS after prolonged incubation in DMEM.

124 ntified putative nanobacteria, not only from FBS, but also from human saliva and dental plaque after

125 Furthermore, FBS transcripts can be taken up by cultured cells and af

126 No differences in fasting blood glucose (FBS), random blood glucose (RBS), or glycated haemoglobi

127 Glycated FBS resulted in a 1.8-fold increase in 3H-thymidine upta

128 The medium was changed to RPMI-1% glycated FBS (experimental) or RPMI-1% control FBS, and cells wer

129 d by 550% and to albumin by 320% in glycated FBS-treated monolayers compared with controls.

130 The media of glycated FBS-treated cells contained 45% lower collagenase activi

131 Exposing cells to glycated FBS changed the distribution of cadherin from a linear t

132 However, FBS and RD hydrolysates protected HepG2 cells against te

133 Pepsin-hydrolysed FBS, at a 5% degree of hydrolysis (DH), showed the highe

134 ty lipoprotein secretion, which is absent in FBS-grown cells, is restored in Huh7.5 cells that are cu

135 Notably, specific miRNAs abundant in FBS, such as miR-122, miR-451a and miR-1246, have been p

136 city of S. intermedius toward HepG2 cells in FBS, and a higher concentration of human plasma was nece

137 The mean within-group change in FBS from baseline to 12 months was ~0.20 points in all g

138 e intervention exhibited a greater change in FBS than their low-adherence counterparts: 0.30 points (

139 ified secondary outcomes included changes in FBS subcomponents and the effect of the knowledge of pre

140 , addition of human plasma to the culture in FBS appeared to inhibit the stimulatory effect of FBS on

141 e widely used, the use of (19)F detection in FBS has been only recently introduced with the aim of ta

142 activity, although overproduction of ILY in FBS was undetectable in mutants nanA-null and msgA-null.

143 those of the wild type; however, survival in FBS was reduced but not to the levels in CSF.

144 x-loaded TSL (TSLDox) was stable in vitro in FBS, BALB/c-nu plasma and human plasma, although release

145 dy directed against integrin beta1 inhibited FBS-, Fn-, and Lm-mediated invasion but did not abrogate

146 Ca2+ chelator BAPTA only partially inhibited FBS-stimulated LC20 phosphorylation and did not signific

147 LXA4 also significantly inhibited FBS-dependent proliferation and TGF-beta1-dependent coll

148 ked recombinant proteins in a complex media (FBS) and measured a limit of detection, 0.004 pg/mL (300

149 These results suggest that monomeric FBS AChE is trimmed at the C-terminus, and the results a

150 Moreover, FBS are very sensitive to strain, which induces an impor

151 ased >2.5 times when grown in the absence of FBS but not in the presence of 1% FBS.

152 hat oscillatory fluid flow in the absence of FBS failed to increase [Ca(2+)](i) in MC3T3-E1 cells.

153 reased in SK-Mel-110 cells in the absence of FBS.

154 n turn depends on the presence or absence of FBS.

155 rvention, this study evaluated the change of FBS at 24 months.

156 AM) in DMEM with different concentrations of FBS.

157 gical effects associated with RNA content of FBS on cell cultures.

158 ppeared to inhibit the stimulatory effect of FBS on ILY, MsgA, and NanA, although there were individu

159 We studied the effect of FBS substitutes on both human BMSC proliferation in vitr

160 did not significantly alter the magnitude of FBS-stimulated isometric contraction.

161 ilable exRNA datasets supports the notion of FBS contamination.

162 One mutation altered the upstream portion of FBS 1, whereas the other, originally created to improve

163 dvertently altered the downstream portion of FBS 2.

164 Analysis of purified preparations of FBS AChE by gel permeation chromatography revealed the p

165 in SK-Mel-110 in the absence or presence of FBS, respectively.

166 ing their differentiation in the presence of FBS.

167 increases in [Ca(2+)](i) in the presence of FBS.

168 d ERK1/2 dephosphorylation and repression of FBS-induced cell proliferation.

169 The use of FBS in hybridoma culture media is examined here, with re

170 This is the first study on FBS showing this behaviour in the full temperature, fiel

171 Of the stimuli tested only FBS and thrombin were able to stimulate a sustained acti

172 growth enhancement in F-12 containing BSA or FBS.

173 h recovery of Hg(2)(+) spiked in the BSA- or FBS-containing medium, and high stability of fluorescenc

174 In the presence of apoptotic cells or FBS, elicited expression of IL-1 beta by NOD macrophages

175 ytes to protein coronas preformed of EfCP or FBS.

176 , NGF, PDGF-BB, bovine pituitary extract, or FBS; or a combination of factors.

177 further show that epidermal growth factor or FBS stimulation induces association of endogenous RSK2 w

178 lth based on the number of favorable ICHS or FBS.

179 g/ml), beta-cyclodextrin (200 microg/ml), or FBS (2 to 4%) was added; addition of urea and phenol all

180 21 to 0.11) and greater efficacy in reducing FBS level by 0.24 mmol/l (95% CI: -0.48 to -0.01).

181 Serial fetal blood sampling (FBS) and intrauterine platelet transfusions (IUPT), as w

182 ], alimentation [A], and tobacco [T]) score (FBS), are also available.

183 imentation, and tobacco (Fuster-BEWAT Score [FBS]), ranging from 0 to 15 (ideal health = 15).

184 ton-based NMR methods of fragment screening (FBS) have been well documented and are widely used, the

185 hich insulin-like growth factor I and serum (FBS) activate NADPH oxidase in pancreatic cancer (PaCa)

186 inesterase isolated from fetal bovine serum (FBS AChE) was previously characterized as a globular tet

187 viated in AM without 10% fetal bovine serum (FBS) [AM(-S)].

188 growth factor (EGF) and fetal bovine serum (FBS) also increased Src activity in Vector cells, but no

189 in media containing 0.1% fetal bovine serum (FBS) and 1 of 5 concentrations of PDGF-BB.

190 ured in media containing fetal bovine serum (FBS) and a glycogen synthase kinase-3 (GSK3) inhibitor,

191 ut cross-talk in buffer, fetal bovine serum (FBS) and whole blood samples, the latter being possible

192 a native repertoire and fetal bovine serum (FBS) as a non-native reference.

193 membrane inserts and 20% fetal bovine serum (FBS) as chemoattractant.

194 n presence or absence of fetal bovine serum (FBS) can provide reliable cryopreservation of various ki

195 days in media containing fetal bovine serum (FBS) concentrations (0, 0.1, 1, 5, 10, and 20%) as appro

196 eraction was enhanced by fetal bovine serum (FBS) exposure, and TROY knockdown also led to down-regul

197 Fetal bovine serum (FBS) has been used in eukaryotic cell cultures for decad

198 sitive to a component in fetal bovine serum (FBS) identified as serum albumin.

199 tion, we have shown that fetal bovine serum (FBS) induces Yes auto-phosphorylation and activation.

200 , 6, and 10 days in 0.2% fetal bovine serum (FBS) media containing different concentrations of either

201 on and quantification of fetal bovine serum (FBS) N-linked sialylglycan isomers, during which many br

202 the media containing 1% fetal bovine serum (FBS) on the 4 DIV, surface galC could be reexpressed in

203 's medium (DMEM) plus 2% fetal bovine serum (FBS) or 2% FBS plus EMD (100 microg/ml), with and withou

204 in medium containing 8% fetal bovine serum (FBS) plus additional growth factors.

205 tarved ME-180 cells with fetal bovine serum (FBS) resulted in a rapid increase in steady-state levels

206 Cells incubated with 10% fetal bovine serum (FBS) served as positive controls.

207 d mammalian cell growth, fetal bovine serum (FBS) supplemented media are still commonly used: a pract

208 treated (TCT) plastic in fetal bovine serum (FBS) supplemented medium.

209 us PC574 was cultured in fetal bovine serum (FBS) than when it was grown in the standard culture medi

210 of human ferritin in 10% fetal bovine serum (FBS) to mimic a real detection environment in complex me

211 n antagonized effects of fetal bovine serum (FBS) to stimulate cell proliferation, whereas siRNA-medi

212 harcoal-dextran-stripped fetal bovine serum (FBS) was found to be more efficient for serum supplement

213 r lavage (BAL) fluid and fetal bovine serum (FBS), (ii) survival in macrophages, and (iii) virulence

214 5, and 7 days using 0.1% fetal bovine serum (FBS), 10% FBS +/- 10 microM SB, or 20 ng/ml PDGF +/- 10

215 e reports the effects of fetal bovine serum (FBS), a physiologically relevant mixture of proteins, on

216 ure condition containing fetal bovine serum (FBS), Cdc25C protein levels were similar in these PCa ce

217 covered during growth in fetal bovine serum (FBS), elicit a robust increase in the amount of ACT, and

218 Fetal bovine serum (FBS), fibronectin (Fn), the extracellular matrix protein

219 ia supplemented with 10% fetal bovine serum (FBS), to media supplemented with 2% HS.

220 1) in the presence of 5% fetal bovine serum (FBS), whereas XMP.Z enhanced BRP growth by 27% +/- 7% (P

221 ffects of humic acid and fetal bovine serum (FBS), which are ubiquitous in aquatic environments and r

222 ulture medium containing fetal bovine serum (FBS), which forms a protein corona on the surface of the

223 is a major component of fetal bovine serum (FBS), which is commonly used as a culture medium during

224 eletion of Mcl-1 reduces fetal bovine serum (FBS)-, VEGF-, and IL-6-induced proliferation.

225 cAMP blocked the fetal bovine serum (FBS)-induced degradation of p27(KIP1); however, loss of

226 ative role of [Ca2+]i in fetal bovine serum (FBS)-stimulated LC20 phosphorylation and force developme

227 te (HS)-, TGF-beta1-, or fetal bovine serum (FBS)-supplemented DMEM/F12 medium.

228 upon incubation in pure fetal bovine serum (FBS).

229 days in the presence of fetal bovine serum (FBS).

230 the G6P concentration in fetal bovine serum (FBS).

231 butylxanthine (IBMX) and fetal bovine serum (FBS).

232 (2+) response induced by fetal bovine serum (FBS).

233 ay in the presence of 2% fetal bovine serum (FBS).

234 ining either 0.1% or 10% fetal bovine serum (FBS).

235 fied Eagle's Medium with fetal bovine serum (FBS).

236 d ex vivo in medium with fetal bovine serum (FBS).

237 bsence or presence of 1% fetal bovine serum (FBS).

238 (DMEM), with and without fetal bovine serum (FBS).

239 PMI media containing 10% fetal bovine serum (FBS).

240 medium with and without fetal bovine serum (FBS).

241 (F-12) in the absence of fetal bovine serum (FBS); this represents a breakthrough for studies in whic

242 defined (D) medium (+/- fetal bovine serum, FBS) under normoxic (N, p(O(2)) = 20%) or hypoxic (H, p(

243 ays in growth media (20% fetal bovine serum, FBS), myoblasts from IUGR fetuses had 34% fewer (P < 0.0

244 uture eCO2 using global food balance sheets (FBS).

245 volutionary conserved forkhead binding site (FBS).

246 erichia coli contains two Fur-binding sites (FBS) offset by 6bp.

247 Genetic analysis suggests that, of these six FBS QTL, three influence BMD, two influence bone quality

248 rimi wastes, including frame, bone and skin (FBS) and refiner discharge (RD), were investigated.

249 cells are grown in HS, compared to standard FBS culture conditions.

250 ic loci influencing femur-breaking strength (FBS), which was measured by three-point bending using an

251 e reorganization in forepaw barrel subfield (FBS) cortex.

252 organization in the forepaw barrel subfield (FBS) of primary somatosensory cortex (SI) that follows f

253 tes-related traits like fasting blood sugar (FBS) and type 2 diabetes (T2D) and glaucoma-related trai

254 c), and mean changes in fasting blood sugar (FBS) level, body weight (BW) and homeostasis model asses

255 ost tunable of the Fe-based superconductors (FBS) in terms of acceptance of high densities of self-as

256 Fe-based superconductors (FBS) present a large variety of compounds whose properti

257 d significantly higher than for cells in TCT/FBS condition.

258 expansion medium supplementation (bFGF, TFP, FBS) and self-assembled construct seeding density (2, 3,

259 Addition of human aqueous humor rather than FBS to trabecular monolayer cell cultures triggers signi

260 e, using RNA sequencing, we demonstrate that FBS contains a diverse repertoire of protein-coding and

261 stimulation on TFPI secretion and found that FBS induced a 5-fold increase in TFPI antigen and activi

262 Eliminating the FBS-stimulated rise in [Ca2+]i with the Ca2+ chelator BA

263 input from the shoulder first appears in the FBS 4 weeks after amputation, and by 6 weeks, the new sh

264 strate for large-scale reorganization in the FBS.

265 ilar accuracy, highlighting the value of the FBS as a simpler and more affordable score for evaluatin

266 ossibly due to the confounding effect of the FBS.

267 w shoulder input comes to occupy most of the FBS.

268 istered 1 h before FBS addition restored the FBS response in propofol treated cells to a level simila

269 he shoulder becomes expressed throughout the FBS that quite likely has a subcortical origin.

270 Addition of purified MsgA and NanA to the FBS resulted in a release of 2.8 mM galactose and 4.3 mM

271 Therefore, FBS and RD hydrolysates have a potential as antioxidativ

272 ted Fbs, regardless of the HLA type of these FBS: CD8(+) T cell stimulation was inhibited by pretreat

273 Here we show that in contrast to FBS, addition of increasing amounts of human serum from

274 ely robust proliferative response of HCEC to FBS may involve stimulation of multiple downstream signa

275 shorter lasting rise in [Ca2+]i relative to FBS.

276 ration/proliferation in vitro in response to FBS or BDNF compared with WT astrocytes.

277 unique concentration-dependent responses to FBS (P<0.0025).

278 However, a comparison of responses to FBS, thrombin, TRPs, LPA and EGF revealed that Fra-1 and

279 Identification of the genes unique to FBS may have an impact on prediction of osteoporosis in

280 an HDL and lower mean T.chol, Triglycerides, FBS and LDL levels.

281 supplementation than dialyzed or undialyzed FBS, due to lower contaminating unlabeled pantothenate c

282 rb any detectable serum protein in undiluted FBS.

283 In solutions of 10% (vol/vol) FBS, we find rapid endonuclease hydrolysis at specific s

284 % confidence interval [CI]: 0.31 to 0.55 vs. FBS OR: 0.49; 95% CI: 0.36 to 0.66), coronary artery cal

285 applications, however, contact of BMSCs with FBS should be minimized.

286 crease by incubation of the COS-7 cells with FBS and to decrease upon adding the ERK pathway inhibito

287 e formed by BMSCs cultured continuously with FBS, bone formed by cells cultured with HS, or with FBS

288 ve, while bone formed by cells cultured with FBS switched to serum-free medium (SFM) was considerably

289 x vivo expansion of human BMSCs, medium with FBS remains most effective.

290 Medium with FBS was more effective in stimulating BMSC proliferation

291 In medium with FBS, colistin was less efficacious in the 3-D cell model

292 ne formed by cells cultured with HS, or with FBS switched to HS, was considerably less extensive, whi

293 med in human cell cultures supplemented with FBS instead of human serum (HS).

294 al cells cultured in media supplemented with FBS, bFGF/HS, or TGF-beta1.

295 an virus produced in media supplemented with FBS.

296 eceptor (sCAR) were similar with and without FBS, but FBS amplified the catalytic effect of 100 nM sC

297 ization under oscillatory fluid flow without FBS.

298 ed to the supernatant, unlike growth without FBS, in which >/=90% is associated with the bacterium.

299 M/Ham's Nutrient Mixture F-12 medium without FBS and allowed to grow for one week.

300 Adding ATP or UTP to flow medium without FBS restored the ability of fluid flow to increase [Ca(2