ライフサイエンスコーパス: FPD

1 FPD did correlate with changes in salivary protein outpu

2 FPD did not correlate with intensity rating for any tast

3 FPD, diameter and position were measured and participant

4 FPD, diameter and position were unchanged at six months.

5 FPD/AML is a familial platelet disorder characterized by

6 FPD/AML patients have a bleeding disorder characterized

7 We corrected the RUNX1 mutation in 1 FPD iPSC line through gene targeting, which led to norma

8 e studied the in vitro megakaryopoiesis of 3 FPD/AML pedigrees.

9 Informative recombination events in 6 FPD/AML pedigrees with evidence of linkage to markers on

10 rrow or peripheral blood cells from affected FPD/AML individuals showed a decrement in megakaryocyte

11 let disorder with propensity to develop AML (FPD/AML).

12 Outcome measures were implant and FPD failures, biologic and prosthetic complications, and

13 milar to other viral FPDs, the putative ASLV FPD has been modeled as an amphipathic helix where most

14 Both FPD mutations accelerate formation of protofibrils, sugg

15 correlates with fungiform papillae density (FPD).

16 o 4.5 mm) supporting fixed partial dentures (FPDs) in posterior mandibular and maxillary jaws.

17 Fracture of ceramic fixed-partial dentures (FPDs) tends to occur in the connector area because of st

18 Footpad dermatitis (FPD) is used in the poultry industry as an animal welfar

19 EFPA could accurately detect FPD prolongation due to drug (moxifloxacin) or pathogeni

20 an because of FPD are more likely to develop FPD in their own childbirth compared with women born vag

21 The 41 x 41-cm digital FPD is constructed on a single monolithic glass substrat

22 with autosomal-dominant Parkinson's disease (FPD).

23 Familial platelet disorder (FPD) is associated with germline RUNX1 mutations, establ

24 om patients with familial platelet disorder (FPD), or normal untransformed HPCs.

25 high incidences of fetopelvic disproportion (FPD) in human childbirth.

26 gions referred to as fusion peptide domains (FPDs) at or near the amino terminus of the membrane-anch

27 prediction of the frictional pressure drop (FPD) in the helically coiled tubes at different conditio

28 ating period (BP), field potential duration (FPD), spike slope, and amplitude, which were consistent

29 tervals, including Field Potential Duration (FPD).

30 tion of the familial paroxysmal dyskinesias (FPD) recognizes several distinct, although overlapping,

31 a nonfunctional version of the FP enhancer (FPD) that does not bind SF2/ASF also fails to block spli

32 In addition, an explicit FPD model was developed by the genetic programming (GP),

33 nd the mononitrosyl adduct of the flavinated FPD (FDP(NO)) show nu(NO) at 1681 cm(-1), which is also

34 tal practitioners, and OPG was advocated for FPD planning, whereas CBCT was advocated for implant pla

35 No suitable animal models exist for FPD/AML, as Runx11/2 mice and zebra fish do not develop

36 Our findings support a model for FPD/AML in which haploinsufficiency of CBFA2 causes an a

37 that co-segregated with the disease in four FPD/AML pedigrees.

38 tion to hematologic malignancies (RUNX1-FPD, FPD/AML, FPDMM); ~44% of affected individuals progress t

39 The same extraction procedure and a GC-FPD analysis were used to determine nitrile metabolites

40 The compounds were analysed by GC-FPD, GC-muECD or LC-MS/MS, with LOQs from 1 to 8 mug/kg.

41 tography with flame photometric detector (GC-FPD), respectively; was carried out.

42 toward the development of a miniaturized GC-FPD capable of ultrafast detection of low levels of OP a

43 In FPD/AML MKs, expression of MYL9 and MYH9 was decreased,

44 responsible for megakaryopoietic defects in FPD patients.

45 the defects in megakaryopoiesis observed in FPD/AML are, in part, related to a deregulation of myosi

46 tosomal dominant allele for non-kinesiogenic FPD.

47 edisposition to acute myelogenous leukaemia (FPD/AML, MIM 601399) is an autosomal dominant disorder c

48 edisposition for acute myelogenous leukemia (FPD/AML).

49 th predisposition to acute myeloid leukemia (FPD/AML) is an autosomal dominant disease of the hematop

50 rcurrent flame photometric detector (microcc-FPD) was adapted and optimized for ultrafast gas chromat

51 r and hydrogen are introduced to the microcc-FPD from opposite directions, creating a hydrogen-rich f

52 The microcc-FPD is capable of detecting very narrow (13 ms) chromato

53 In this microcc-FPD, combustion takes place between the burner tips with

54 , 71% birds in all treatments developed mild FPD and pens were top-dressed with dry litter to promote

55 of a microfabricated column and a miniature FPD is an important step toward the development of a min

56 scores during d21-42, HTM reduced the AUC of FPD lesion scores during d7-21 and d21-42.

57 M, LTM reduced area under the curve (AUC) of FPD lesion scores during d21-42, HTM reduced the AUC of

58 lies that women born by Caesarean because of FPD are more likely to develop FPD in their own childbir

59 FPD for mothers born by Caesarean because of FPD is 2.8 times the risk for mothers born vaginally.

60 by blebbistatin rescued the ploidy defect of FPD/AML MKs.

61 , which, in turn, has inflated incidences of FPD.

62 demonstrate successful in vitro modeling of FPD with patient-specific iPSCs and confirm that RUNX1 m

63 which we show here predicts that the risk of FPD for mothers born by Caesarean because of FPD is 2.8

64 strongly affects the fracture resistance of FPDs.

65 This study evaluated the impact of TM on FPD and consisted of 3 treatments supplemented with 0 (N

66 One FPD failed in the NDI group versus two FPDs in the SDI g

67 al types across the synchondrosis, and FH or FPD measurements (p > 0.05).

68 stance between the fragment and the patella (FPD), and signal characteristics within the synchondrosi

69 nd universal models for estimating two-phase FPD in smooth coiled tubes with different orientations w

70 trogen phosphorous (NPD), flame photometric (FPD) detectors, as well as gas chromatography-olfactomet

71 greater for fixed partial denture planning (FPD) 59%, whereas CBCT was highly preferred for implant

72 were top-dressed with dry litter to promote FPD healing.

73 ut also reduced FPD development by promoting FPD wound healing.

74 mutations were introduced into the putative FPD.

75 improved growth performance but also reduced FPD development by promoting FPD wound healing.

76 RUNX1-FPD HSPCs were myeloid biased, had increased self-renewa

77 -mediated signaling was exaggerated in RUNX1-FPD HSPCs compared with healthy controls, leading to the

78 that inflammation is an early event in RUNX1-FPD pathogenesis, and CD74 signaling is one of the drive

79 sposition to hematologic malignancies (RUNX1-FPD, FPD/AML, FPDMM); ~44% of affected individuals progr

80 tream targets JAK1/2 and mTOR reversed RUNX1-FPD differentiation defects in vitro and in vivo and red

81 analysis of samples from patients with RUNX1-FPD (n > 75) revealed that FPD hematopoietic stem and pr

82 improve the phenotype of patients with RUNX1-FPD and prevent myeloid progression.

83 Bone marrow from patients with RUNX1-FPD contained an elevated cytokine milieu, exerting chro

84 The bone marrow from patients with RUNX1-FPD showed high transcript and protein expression of CD7

85 Five-year data indicate that FPD treatment in posterior mandibular and maxillary jaws

86 tients with RUNX1-FPD (n > 75) revealed that FPD hematopoietic stem and progenitor cells (HSPCs) disp

87 The FPD provides radiographic images with excellent inherent

88 elope glycoprotein (EnvA) proposed to be the FPD is internal and contains a centrally located proline

89 Basic imaging characteristics of the FPD and associated image processing system were assessed

90 f curvature at the gingival embrasure of the FPD connector significantly affects the fracture resista

91 in a family with FPD/AML, and found that the FPD iPSCs display defects in megakaryocytic differentiat

92 Images obtained with the FPD demonstrated excellent uniformity, repeatability, an

93 Results with the FPD system were compared to those with a storage phospho

94 scatter content of images acquired with the FPD were equivalent to those acquired with the storage p

95 tangent and threshold measurements for these FPD ranged between r: 0.93-1.00.

96 egated with the disease in the remaining two FPD/AML pedigrees at phylogenetically conserved amino ac

97 nite element models (FEMs), representing two FPD connector designs, were created in a manner correspo

98 One FPD failed in the NDI group versus two FPDs in the SDI group (P >0.99).

99 ffects the fracture resistance of three-unit FPDs.

100 Similar to other viral FPDs, the putative ASLV FPD has been modeled as an amphi

101 lls (iPSCs) from 2 patients in a family with FPD/AML, and found that the FPD iPSCs display defects in