ライフサイエンスコーパス: Fallot

1 ptal defect, atrioventricular septal defect, Fallot, and truncus arteriosus operations were 1.1%, 0.6

2 found the following anomalies: tetralogy of Fallot (15 patients, 25%), tricuspid atresia (12 patient

3 Most patients had tetralogy of Fallot (18/31).

4 The most common diagnoses were tetralogy of Fallot (19%) and hypertrophic cardiomyopathy (14%).

5 The 3 most common CHDs were tetralogy of Fallot (20%), isolated ventricular septal defect (14%),

6 ion (25%) or control cases with tetralogy of Fallot (30%).

7 Diagnoses included tetralogy of Fallot (51%), septal defects (20%), (congenitally correc

8 Cardiac diagnoses were tetralogy of Fallot (71%), valvular pulmonary stenosis (21%), and oth

9 ), pulmonary atresia (11%), and tetralogy of Fallot (8%).

10 prevalent CHD anatomy included tetralogy of Fallot (883 participants [22%]), transposition of great

11 ty-seven patients with repaired tetralogy of Fallot (age >/=16 y; mean age, 35.8+/-10.1 y; 38 male) u

12 92) was associated with ALL and tetralogy of Fallot (aHR: 2.40, 95% CI: 1.27-4.55) was associated wit

13 eurological outcomes, including tetralogy of Fallot (AOR 2.4, 95% CI: 1.1, 5.4), anencephaly (AOR 2.9

14 1.19; 95% CI: 1.00, 1.43), and tetralogy of Fallot (aOR = 1.44; 95% CI: 1.01, 2.19) in the various o

15 Correction of tetralogy of Fallot (cTOF) often results in pulmonary valve pathology

16 ricular septal defect (n = 11), tetralogy of Fallot (n = 3), atrioventricular canal (n = 3), and othe

17 great arteries (TGA) (n = 69), tetralogy of Fallot (n = 66), hypoplastic left heart syndrome (n = 51

18 /hypoplastic aortic arch (n=5), tetralogy of Fallot (n=1), hypoplastic right heart (n=1), and common

19 ension (PAH; n=10) and repaired tetralogy of Fallot (n=10) underwent MR-augmented cardiopulmonary exe

20 46), tricuspid atresia (n=103), tetralogy of Fallot (n=127), pulmonary atresia (n=177), heterotaxy sy

21 Patients with repaired tetralogy of Fallot (n=143; 12.5+/-3.2 years) had an echocardiogram a

22 es after Mustard/Senning (n=2), tetralogy of Fallot (n=2), aortic valve disease (n=2), and other bive

23 The majority had a variant of tetralogy of Fallot (n=36), or transposition of the great arteries, v

24 Fifty-four patients with tetralogy of Fallot (n=39), valvar pulmonary stenosis (n=10), or pulm

25 omalous pulmonary veins (n=13), tetralogy of Fallot (n=8), cor triatriatum (n=7), single ventricle ph

26 ons between carbon monoxide and tetralogy of Fallot (odds ratio = 2.04, 95% confidence interval: 1.26

27 to coarctation of the aorta and tetralogy of Fallot (P=0.002; Fisher exact test).

28 In patients with repaired tetralogy of Fallot (rTOF) and spontaneous ventricular tachycardia (V

29 comes in patients with repaired tetralogy of Fallot (rTOF) is unknown.

30 e replacement (PVR) in repaired tetralogy of Fallot (rTOF) rely on cardiovascular magnetic resonance

31 ardia (VT) late after repair of tetralogy of Fallot (rTOF).

32 Neonates with symptomatic tetralogy of Fallot (sTOF) with hypoplastic pulmonary arteries (hPA)

33 nt of neonates with symptomatic tetralogy of Fallot (sTOF).

34 alifornia carrying infants with tetralogy of Fallot (TOF) (n = 55), dextrotransposition of the great

35 congenital heart diseases like tetralogy of Fallot (TOF) and pulmonary atresia (PA).

36 late after total correction of tetralogy of Fallot (TOF) are at risk for major complications.

37 rly primary repair of acyanotic tetralogy of Fallot (ToF) can be performed safely with low requiremen

38 Patients after repair of tetralogy of Fallot (ToF) frequently have right ventricular (RV) dysf

39 Mortality in patients with tetralogy of Fallot (TOF) has decreased substantially since the start

40 ge versus "two-stage" repair of tetralogy of Fallot (TOF) in infants younger than 1 year of age.

41 ation in patients with repaired tetralogy of Fallot (TOF) is a risk factor for malignant ventricular

42 cal technique for correction of tetralogy of Fallot (TOF) is contentious for patients with a moderate

43 Tetralogy of Fallot (TOF) is the commonest cyanotic form of congenita

44 Tetralogy of Fallot (TOF) is the most common cyanotic congenital hear

45 Tetralogy of Fallot (TOF) is the most common cyanotic congenital hear

46 Tetralogy of Fallot (TOF) is the most common cyanotic congenital hear

47 Tetralogy of Fallot (ToF) is the most common form of complex congenit

48 apt to hypoxia in children with tetralogy of Fallot (TOF) is unknown.

49 Individuals with tetralogy of Fallot (TOF) now routinely survive to reproductive age a

50 mon among adults with corrected tetralogy of Fallot (TOF) or pulmonary stenosis (PS) referred for pul

51 rtic root is a known feature in tetralogy of Fallot (TOF) patients with pulmonary stenosis (PS) or pu

52 e replacement (PVR) in repaired tetralogy of Fallot (TOF) reduces pulmonary regurgitation and decreas

53 The timing of repair of tetralogy of Fallot (TOF) remains controversial.

54 aemodynamic abnormalities after tetralogy of Fallot (TOF) repair, who had undergone cardiovascular ma

55 cal status in late survivors of tetralogy of Fallot (TOF) repair.

56 Tetralogy of Fallot (TOF) typically results in clinical cyanosis or v

57 Diagnoses included tetralogy of Fallot (TOF) with pulmonary atresia (n=13), TOF with pul

58 om 16 infants with nonsyndromic tetralogy of Fallot (TOF) without a 22q11.2 deletion, 3 fetal heart s

59 21.1 locus in 948 patients with tetralogy of Fallot (TOF), 1488 patients with other forms of CHD and

60 age, ventricular septal defect, tetralogy of Fallot (TOF), and an aortic arch abnormality.

61 e and 36% (n=326) of these have tetralogy of Fallot (TOF), comprising the largest subset of severe co

62 Disease entities included were tetralogy of Fallot (TOF), double outlet right ventricle (DORV), tran

63 ial recurrence of non-syndromic Tetralogy of Fallot (TOF), implicating genetic factors that remain la

64 Tetralogy of Fallot (TOF), the most common severe congenital heart ma

65 or risk alleles associated with Tetralogy of Fallot (TOF), using a northern European discovery set of

66 Tetralogy of Fallot (TOF)-the most common cyanotic heart defect in ne

67 ohort of patients with repaired tetralogy of Fallot (TOF).

68 ain problematic after repair of tetralogy of Fallot (TOF).

69 nancy outcomes in patients with tetralogy of Fallot (TOF).

70 e complications after repair of Tetralogy of Fallot (ToF).

71 outflow tract of postoperative tetralogy of Fallot (TOF).

72 ion factor NKX2.5 as a cause of tetralogy of Fallot (TOF).

73 p of preoperative patients with tetralogy of Fallot (TOF).

74 atients after primary repair of tetralogy of Fallot (TOF).

75 arteriosus (TA), and 15.9% with tetralogy of Fallot (TOF).

76 to be a single-gene defect and tetralogy of Fallot a polygenic disorder with a small number of inter

77 ed pilot study in patients with tetralogy of Fallot aged 10 to 25 years.

78 lot with pulmonary atresia than tetralogy of Fallot alone.

79 agille syndrome, which includes tetralogy of Fallot among its more severe cardiac pathologies.

80 Eight patients with tetralogy of Fallot and 7 patients with transposition of the great ar

81 Of the 34 patients with tetralogy of Fallot and abnormal CA, 7 (21%) demonstrated CA compress

82 death remain common in repaired tetralogy of Fallot and affect even those with excellent anatomic rep

83 Patients with tetralogy of Fallot and an ICD experience high rates of appropriate t

84 ain variant co-segregating with tetralogy of Fallot and bicuspid aortic valve in maternal relatives (

85 ation in patients with repaired tetralogy of Fallot and chronic pulmonary regurgitation (PR).

86 tients (40.3+/-19.2 years) with tetralogy of Fallot and CRT were enrolled.

87 onary artery anomalies, such as tetralogy of Fallot and double outlet right ventricle.

88 uman congenital heart diseases, tetralogy of Fallot and double outlet right ventricle.

89 inically significant portion of tetralogy of Fallot and idiopathic AV block.

90 rwent surgical intervention for tetralogy of Fallot and major aortopulmonary collateral arteries at L

91 native and previously operated tetralogy of Fallot and major aortopulmonary collateral arteries.

92 laterals (APCs) in infants with tetralogy of Fallot and pulmonary atresia (TOF/PA) and to prospective

93 in 12 of 25 patients (48%) with tetralogy of Fallot and pulmonary atresia, and 4 of 54 (8%) with pros

94 which predominantly consist of tetralogy of Fallot and related variants.

95 Adults with repaired tetralogy of Fallot and RV dysfunction (RV ejection fraction [EF] <50

96 dian 12.0 years after repair of tetralogy of Fallot and similar lesions were studied echocardiographi

97 Neonates with tetralogy of Fallot and symptomatic cyanosis (sTOF) require early int

98 f abnormalities associated with tetralogy of Fallot and the DiGeorge syndrome.

99 We detected associations for tetralogy of Fallot and the upper exposure categories for TCAA, DCAA,

100 of PR in patients with repaired tetralogy of Fallot and warrants further investigation.

101 Patients with tetralogy of Fallot are at risk for ventricular arrhythmias and sudde

102 diac death late after repair of tetralogy of Fallot are devastating complications in adult survivors

103 ction in patients with repaired tetralogy of Fallot are incompletely understood.

104 Patients with repaired tetralogy of Fallot are monitored for pulmonary regurgitation (PR) an

105 rs after surgical correction of tetralogy of Fallot are nonexistent.

106 h clinical outcomes in repaired tetralogy of Fallot are unknown.

107 tcomes of transatrial repair of tetralogy of Fallot are unknown.

108 with Abernethy malformation and tetralogy of Fallot associated with nodular regenerative hyperplasia

109 f PVR in adults after repair of tetralogy of Fallot at a single tertiary center were retrospectively

110 ergoing a transatrial repair of tetralogy of Fallot between 1980 and 2005 were reviewed, their follow

111 p 2 (n = 22) received repair of tetralogy of Fallot by the traditional technique with ventricular sep

112 r diagnosis and first repair of Tetralogy of Fallot compared to natural progression.

113 nd adult patients with repaired tetralogy of Fallot continues to grow as a result of excellent surviv

114 llar infarct and a history of a tetralogy of Fallot corrected during childhood.

115 growing number of patients with tetralogy of Fallot develop left ventricular systolic dysfunction and

116 Adults with repaired tetralogy of Fallot die prematurely from ventricular tachycardia (VT)

117 Patients with repaired tetralogy of Fallot experience variable outcomes for reasons that are

118 ar tachycardia (VT) in repaired Tetralogy of Fallot focuses on isthmuses in the right ventricle but m

119 and trended toward lower in the tetralogy of Fallot group (13.5+/-1.29 mL/kg per minute; P=0.06) comp

120 PVR after repair of tetralogy of Fallot has a low and improving mortality, with a low nee

121 e third of adults with repaired tetralogy of Fallot have an aortic root diameter >/=40 mm, the preval

122 r events (MACE) after repair of tetralogy of Fallot have been limited by modest predictive capacity a

123 placement (PVR) after repair of tetralogy of Fallot have recently been broadened to include asymptoma

124 ied (atrial septal defect in 6, tetralogy of Fallot in 4, and Fontan procedure in 6).

125 patient died after surgery for tetralogy of Fallot in situs inversus.

126 sex-specific controls, repaired tetralogy of Fallot in women had larger right ventricular end-systoli

127 Tetralogy of Fallot is a congenital heart disease affecting newborns

128 Tetralogy of Fallot is a congenital heart disease that requires surgi

129 rgical repair for neonates with tetralogy of Fallot is associated with a significantly higher risk fo

130 placement (PVR) after repair of tetralogy of Fallot is commonly required and is burdensome.

131 linical outcome after repair of tetralogy of Fallot is determined by the adaptation of the right vent

132 atrioventricular canal, and the tetralogy of Fallot malformation.

133 ection." Patients with repaired tetralogy of Fallot often have pulmonary regurgitation, which is freq

134 ptal defect was associated with tetralogy of Fallot or double-outlet right ventricle in 3 individuals

135 years) with surgically repaired tetralogy of Fallot or pulmonary atresia with ventricular septal defe

136 rome and some cases of isolated tetralogy of Fallot or pulmonic stenosis.

137 my, especially in patients with tetralogy of Fallot or transposition of the great arteries.

138 Sixty-nine consecutive repaired tetralogy of Fallot patients (43 male, mean 40+/-15 years) clinically

139 We included 413 tetralogy of Fallot patients (age, 36 +/- 13 years; QRS duration, 148

140 e (ACHD) centres that follow up Tetralogy of Fallot patients and Great Ormond Street Hospital (GOSH),

141 , this physiology is present in tetralogy of Fallot patients at mid-term follow-up and whether it is

142 ds from 28 consecutive repaired Tetralogy of Fallot patients from 2 centers who underwent VT ablation

143 Both the PAH and tetralogy of Fallot patients had blunted exercise-induced increases i

144 Although tetralogy of Fallot patients had the largest increase in cardiac outp

145 Tetralogy of Fallot patients predicted to be at high risk of ventricu

146 hich, when defective, cause the tetralogy of Fallot phenotype.

147 (PVR) in patients with repaired tetralogy of Fallot provides symptomatic benefit and right ventricula

148 ar stimulation in patients with tetralogy of Fallot referred for PVR from January 2020 to December 20

149 ar stimulation in patients with tetralogy of Fallot referred for PVR yields a high rate of inducible

150 undergoing 5 common operations: tetralogy of Fallot repair (n = 250), complete atrioventricular septa

151 In the first 24 hours after tetralogy of Fallot repair (n=11 patients), serial prospective measur

152 inical outcomes in adults after tetralogy of Fallot repair (rTOF).

153 V) dysfunction are common after tetralogy of Fallot repair (rTOF).

154 al cavopulmonary connection and tetralogy of Fallot repair and may prove to be an important therapeut

155 ive patients receiving standard tetralogy of Fallot repair in a single institution from 1964 to 2009.

156 n patients, 3 to 35 years after tetralogy of Fallot repair or pulmonary valvotomy, had PR measured by

157 ho underwent complete or staged tetralogy of Fallot repair prior to 30 days of age.

158 V) restrictive physiology after tetralogy of Fallot repair results in low cardiac output and a prolon

159 RLS alone explained 5% (tetralogy of Fallot repair) to 20% (Norwood) of variation in the prim

160 After tetralogy of Fallot repair, acute restrictive RV physiology is associ

161 ate the long-term outcome after tetralogy of Fallot repair, yet the diagnostic and predictive value o

162 g a nontransannular approach to tetralogy of Fallot repair.

163 ion and exercise capacity after tetralogy of Fallot repair.

164 oing Fontan-type operations and tetralogy of Fallot repair.

165 be present early and late after tetralogy of Fallot repair.

166 patients require PVR late after tetralogy of Fallot repair.

167 Most patients with repaired tetralogy of Fallot require pulmonary valve replacement (PVR), but th

168 rmed a cross-sectional study of tetralogy of Fallot subjects who were tested for 22q11.2 deletion, an

169 rd deviation scores in repaired tetralogy of Fallot suggest that women perform poorer than men in ter

170 f PVR in patients with repaired tetralogy of Fallot that developed pulmonary insufficiency, until Dec

171 tory follow-up of patients with Tetralogy of Fallot to model the health-related costs and outcomes ov

172 to cardiac defects ranging from tetralogy of Fallot to transposition of the great arteries and that d

173 In patients with tetralogy of Fallot treated with CRT consistent improvement in QRS du

174 Infants with tetralogy of Fallot undergo surgical repair, which requires the remov

175 t of 252 patients with repaired tetralogy of Fallot undergoing programmed ventricular stimulation was

176 d on 205 patients with repaired tetralogy of Fallot undergoing PVR at our institution between 1988 an

177 Patients with repaired tetralogy of Fallot undergoing PVR with history of ventricular tachyc

178 nge, 8-20 years]) with repaired tetralogy of Fallot underwent cardiac magnetic resonance for ventricu

179 ollow-up, transatrial repair of tetralogy of Fallot was associated with a minimal risk of sudden deat

180 significantly increased risk of tetralogy of Fallot was observed (7 cases in fluconazole-exposed preg

181 ost per patient of a repair for Tetralogy of Fallot was pound26,938 (SE = pound4,140).

182 Tetralogy of Fallot was present in 23 subjects and was accompanied by

183 Tetralogy of Fallot was present in 55.2%, with native/patched right v

184 lation was 21.5+/-11 years, and tetralogy of Fallot was the cardiac condition in 59%.

185 secutive patients with repaired tetralogy of Fallot were analyzed (median age at follow-up 23 years [

186 gurgitation following repair of tetralogy of Fallot were considered benign.

187 Autopsy hearts with tetralogy of Fallot were evaluated to clarify the pathological substr

188 en hearts with the diagnosis of tetralogy of Fallot were examined.

189 es such as QRS fragmentation in tetralogy of Fallot were identified.

190 Infants with tetralogy of Fallot were smaller in all measured dimensions, but they

191 ior history was significant for tetralogy of Fallot which was repaired at nine months of age.

192 strategy to treat patients with tetralogy of Fallot who are symptomatic in the neonatal period.

193 (PVR) in patients with repaired tetralogy of Fallot who develop pulmonary insufficiency remains uncle

194 Patients with repaired tetralogy of Fallot who had a CMR before the occurrence of a composit

195 nsists of the 144 patients with tetralogy of Fallot who underwent surgical repair at <15 years of age

196 1G>C p.R184P, in a patient with tetralogy of Fallot with absent pulmonary valve and extra-cardiac phe

197 try including all patients with tetralogy of Fallot with an ICD was initiated in 2010 by the French I

198 the human birth defect syndrome tetralogy of Fallot with complete pulmonary artery atresia.

199 Four groups of tetralogy of Fallot with confluent central pulmonary arteries were st

200 Tetralogy of Fallot with cyanosis requiring surgical repair in early

201 Tetralogy of Fallot with diminutive pulmonary arteries and severe pul

202 catheter management approach in tetralogy of Fallot with diminutive pulmonary arteries.

203 Tetralogy of Fallot with major aortopulmonary collateral arteries is

204 w tract stenting of symptomatic tetralogy of Fallot with poor anatomy (small pulmonary arteries) and

205 lagille syndrome suffering from tetralogy of Fallot with pulmonary atresia and multiple aortopulmonar

206 ic features, and more common in tetralogy of Fallot with pulmonary atresia than tetralogy of Fallot a

207 stenosis is rare and resembles tetralogy of Fallot with pulmonary atresia: There is a high incidence

208 cribed as a phenotypic mimic of Tetralogy of Fallot with pulmonary atresia; however, subsequent repor

209 p 1 (n = 20) received repair of tetralogy of Fallot with the modified technique with transatrial vent

210 s had undergone total repair of tetralogy of Fallot within 2 wks after admission for RSV infection.

211 eripheral pulmonic stenosis and tetralogy of Fallot) in the absence of liver dysfunction.

212 years; range, 8.5 to 64.9; 72% tetralogy of Fallot) underwent PVR for severe pulmonary regurgitation

213 nd heart defects (predominantly tetralogy of Fallot).

214 maller placental size at birth: tetralogy of Fallot, -0.45 (95% confidence interval, -0.58 to -0.31);

215 Four patients had tetralogy of Fallot, 1 had pulmonary atresia, and 1 had rheumatic val

216 tion of the great arteries, 123 tetralogy of Fallot, 132 single ventricle, and 130 other CHD).

217 ctive endocarditis was 1.3% for tetralogy of Fallot, 2.7% for isolated ventricular septal defect, 3.5

218 ), including 9 of 201 (4%) with tetralogy of Fallot, 3 of 71 (4%) with a secundum ASD, one each with

219 7.8%; 2) paternal anesthesia in tetralogy of Fallot, 3.6%; 3) painting in atrioventricular septal def

220 during S-phase, in infants with tetralogy of Fallot, a common form of congenital heart disease.

221 MD identified in a patient with Tetralogy of Fallot, a serious heart valve defect, affects the substr

222 sition of the great arteries or tetralogy of Fallot, an event rate of 1/454 patients-years.

223 atent ductus arteriosus, 2 with tetralogy of Fallot, and 1 with pulmonary valve dysplasia.

224 position of the great arteries, tetralogy of Fallot, and anomalous pulmonary venous return, were also

225 ect, ventricular septal defect, tetralogy of Fallot, and transposition of the great arteries decrease

226 ing ventricular septal defects, tetralogy of Fallot, and tricuspid atresia, defects that resemble tho

227 right shunts, valvular disease, tetralogy of Fallot, and truncus arteriosus.

228 nes in mice are involved in the tetralogy of Fallot, and, using zebrafish phenotypes, propose the hyp

229 ly 20 years after operation for tetralogy of Fallot, aortic stenosis and coarctation.

230 n of the great arteries, and 81 tetralogy of Fallot, as well as their unaffected parents.

231 l patients after correction for tetralogy of Fallot, BNP levels were elevated and correlated signific

232 l defect, atrial septal defect, tetralogy of Fallot, coarctation of the aorta, cleft lip, cleft palat

233 y to escape clinical diagnosis (tetralogy of Fallot, coarctation of the aorta, transposition of the g

234 e most common reintervention in tetralogy of Fallot, could potentially inform therapy to improve arrh

235 aberrant subclavian artery and Tetralogy of Fallot, demonstrating that Hoxa1 is required for pattern

236 position of the great arteries, tetralogy of Fallot, double-chamber right ventricle, interrupted aort

237 r conotruncal anomalies include tetralogy of Fallot, double-outlet right ventricle, transposition of

238 ect, ventricular septal defect, tetralogy of Fallot, Ebstein anomaly, transposition of the great vess

239 abnormalities, five with severe tetralogy of Fallot, four with Ebstein's anomaly and two with single

240 or =10 mm, diagnosis other than tetralogy of Fallot, Genesis stent, higher prestent RV:aortic pressur

241 In tetralogy of Fallot, heart defects occurred in seven (3.1%) of 223 of

242 ified included heart failure in tetralogy of Fallot, mechanical circulatory support/transplantation,

243 fic malformations that resemble tetralogy of Fallot, overriding aorta with ventricular septal defect,

244 ersal in patients with repaired tetralogy of Fallot, precipitating a complex pathophysiological casca

245 ventricular septal defects, and tetralogy of Fallot, resembling some of the most common congenital ma

246 After repair of tetralogy of Fallot, right ventricular (RV) dilation has been associa

247 In patients with tetralogy of Fallot, severe pulmonary stenosis and diminutive pulmona

248 nts with atrial septal defects, tetralogy of Fallot, single ventricle physiology, and following cardi

249 ongenital heart defects such as tetralogy of Fallot, systemic right ventricle, and univentricular hea

250 phic ventricular tachycardia in tetralogy of Fallot, the anatomy and histology of these regions have

251 art disease, such as those with tetralogy of Fallot, the right ventricle (RV) is subject to pressure

252 been described in patients with tetralogy of Fallot, the scope of the problem remains poorly defined.

253 In patients with tetralogy of Fallot, the strongest correlations were observed with le

254 included complex heart defects (tetralogy of Fallot, transposition of the great arteries, atrial sept

255 higher-risk substrates include tetralogy of Fallot, transposition of the great arteries, cyanotic he

256 sus, valvar pulmonary stenosis, tetralogy of Fallot, transposition of the great arteries, muscular ve

257 rdiovascular disease, including tetralogy of Fallot, truncus arteriosus and interruption of the aorti

258 Severe CHD was defined as tetralogy of Fallot, truncus arteriosus, transposition complexes, end

259 left heart syndrome (HLHS) and tetralogy of Fallot, two common forms of cyanotic CHD lesions, as wel

260 Tetralogy of Fallot, univentricular heart, and dextro-transposition o

261 may confer an increased risk of tetralogy of Fallot.

262 art defects such as in repaired tetralogy of Fallot.

263 in adult patients with repaired tetralogy of Fallot.

264 ted cardiomyopathy and repaired tetralogy of Fallot.

265 RVOT-sparing strategy to treat tetralogy of Fallot.

266 dic, nonsyndromic patients with Tetralogy of Fallot.

267 tenosis, pulmonic stenosis, and tetralogy of Fallot.

268 atifying patients with repaired tetralogy of Fallot.

269 ongenital cardiac defect termed tetralogy of Fallot.

270 ae for patients after repair of tetralogy of Fallot.

271 block, idiopathic AV block, or tetralogy of Fallot.

272 ntricular septal defect or with tetralogy of Fallot.

273 outcome parameters in repaired tetralogy of Fallot.

274 ly, of patients after repair of tetralogy of Fallot.

275 nant of outcome after repair of tetralogy of Fallot.

276 ive failure than in hearts with tetralogy of Fallot.

277 well as in surgically repaired tetralogy of Fallot.

278 comes in patients with repaired tetralogy of Fallot.

279 fication in adults with repared tetralogy of Fallot.

280 utations accounting for 2.3% of Tetralogy of Fallot.

281 tment of arrhythmia in repaired tetralogy of Fallot.

282 nction in adults after repaired tetralogy of Fallot.

283 er transannular patch repair of tetralogy of Fallot.

284 ition of the great arteries and tetralogy of Fallot.

285 al heart defects in general, or tetralogy of Fallot.

286 he clinical variability seen in tetralogy of Fallot.

287 ance in patients with corrected tetralogy of Fallot.

288 and young adults with repaired tetralogy of Fallot.

289 -D deletions had CTDs including tetralogy of Fallot.

290 tcome in patients with repaired tetralogy of Fallot.

291 es ablation outcome in repaired Tetralogy of Fallot.

292 nd among patients with repaired tetralogy of Fallot.

293 f the great arteries; and 8 for tetralogy of Fallot.

294 nt class to have offspring with tetralogy of Fallot.

295 atation in adults with repaired tetralogy of Fallot.

296 onary artery stenosis (n = 94), tetralogy of Fallot/pulmonary atresia (n = 72), congenital branch pul

297 tabases of adults with repaired tetralogy of Fallot: (1) for model development, a prospectively const

298 ight Ventricular Dysfunction in Tetralogy of Fallot: Inhibition of the Renin-Angiotensin-Aldosterone

299 ed by supplement use except for tetralogy of Fallot; among supplement users, those in the Western cla

300 osis; coarctation of the aorta; tetralogy of Fallot; and D-transposition of the great arteries.