コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 t T cells, which lack endogenously expressed Fc gamma receptors.
2 on recognized to improve Fc interaction with Fc gamma receptors.
3 coy and as a modulator of leukocytes bearing Fc gamma receptors.
4 njury was dependent on neutrophils and their Fc gamma receptors.
5 of HA responses, relied on signaling through Fc-gamma receptors.
6 ector functions, through engagement of their Fc-gamma receptors.
7 es, as manifested by increased expression of Fc-gamma receptors.
8 mice and cells with deficiencies of specific Fc-gamma receptors.
10 agD-2 had significantly more C1q binding and Fc gamma receptor activation, a surrogate for ADCC funct
13 mportant PTMs impacting the interaction with Fc gamma receptors and hence effector functions such as
14 functionally significant mutations in human Fc gamma receptors and possible novel mechanisms for inh
15 at ANCA-mediated activation occurred through Fc gamma receptors and that neutrophil immobilization wa
16 pression of other surface receptors, such as FC-gamma receptor and tumor necrosis factor receptors (T
18 e interaction of human antibodies with human Fc-gamma receptors, and it remains largely unknown how s
20 tibodies to DV that enhance the infection of Fc gamma receptor-bearing cells have been implicated in
21 ivation of complement and ability to mediate FC-gamma-receptor binding, for immunotherapeutically enh
24 ns that not only silence unwanted binding to Fc-gamma receptors but also could bring resistance to Id
25 modulate the binding affinity of IgG1 Fc to Fc gamma receptors, but it is unclear how the structural
30 iated enhancement of IFN production required Fc gamma receptor engagement, bypassed fusion, and initi
31 ies did not enhance the infection of DENV in Fc gamma receptor-expressing cells, offsetting the conce
32 s generated in response to engagement of the Fc gamma receptor (Fc gamma R) and potently contributes
34 om maternal to fetal circulation, we studied Fc gamma receptor (Fc gamma R) expression by immunohisto
37 agocytosis of IgG-opsonized microbes via the Fc gamma receptor (Fc gammaR) requires the precise coord
38 es to demonstrate a role for PKC- epsilon in Fc gamma receptor (Fc gammaR)-dependent phagocytosis.
43 n neutrophil expression of the high-affinity Fc gamma-receptor (Fc gammaRI) was observed that peaked
44 d the requirement for both Cdc42 and WASP in Fc(gamma) receptor (Fc(gamma)R)-mediated phagocytosis, t
52 he periphery, upregulation of the inhibitory Fc gamma receptor (FcgammaR) IIb at the tumor site preve
54 sstalk between Toll-like receptor (TLR)4 and Fc gamma receptor (FcgammaR) is well documented, the det
55 ment-inactive" Fc modifications that engaged Fc gamma receptor (FcgammaR) rendered 2C7 ineffective, n
56 infected patients enhanced ZIKV infection of Fc gamma receptor (FcgammaR)-bearing cells in vitro.
58 ate the loss of virus-infected cells through Fc gamma receptor (FcgammaR)-mediated effector functions
61 and a subset of antibody therapeutics engage Fc gamma receptor (FcgammaR)IIIa/CD16a to stimulate a pr
63 n to neutralization, interaction of IgG with Fc gamma receptors (FcgammaR) may play an important role
64 engaging complement components and distinct Fc gamma receptors (FcgammaR) on different host immune c
65 hrough endosomal Toll-like receptors (TLRs), Fc gamma receptors (FcgammaR), and antigen receptors in
67 ular determinant of fragment crystallizable (Fc) gamma receptor (FcgammaR) binding, are exceedingly r
68 triggering cellular fragment crystallizable (Fc)gamma receptors (FcgammaR), resulting in the release
69 nd IgG2 and mutated variants thereof lacking Fc-gamma receptor (FcgammaR) binding in human cells expr
70 of the inhibitory immunoglobulin G receptor, Fc-gamma receptor (FcgammaR) IIB, maintains peripheral i
71 n in vivo model that revealed that human IgG-Fc-gamma receptor (FcgammaR) interactions could regulate
72 reexisting heterotypic antibodies, via their Fc-gamma receptor (FcgammaR) interactions, may increase
73 es, macrophages, and dendritic cells via the Fc-gamma receptor (FcgammaR), a process termed antibody-
75 hox on Ser304 to Ser328, suggesting that IgG Fc-gamma receptors (FcgammaR) and complement receptor 3
76 adioimmunoconjugates by immune cells bearing Fc-gamma-receptors (FcgammaR) that bind to the Fc region
77 ate HAR that is dependent on complement, the Fc-gamma receptors FcgammaRII/III (CD32/CD16), and NK ce
81 ed through immune complexes interacting with Fc gamma receptors (FcgammaRs) expressed by sensory neur
82 tic monoclonal antibodies require binding to Fc gamma receptors (FcgammaRs) for full effect and incre
86 bound immunoglobulin G (IgG) to cell surface Fc gamma receptors (FcgammaRs) triggers a wide variety o
87 immune complex formation and the activating Fc gamma receptors (FcgammaRs) were involved in the anti
88 ression of the two uniquely human neutrophil Fc gamma receptors (FcgammaRs), FcgammaRIIA and FcgammaR
89 The FcRgamma subunit is a key component of Fc gamma receptors (FcgammaRs), which are activated by I
91 he antibody-binding crystallizable fragment (Fc) gamma receptors (FcgammaRs) are expressed by leukocy
93 he balance between activating and inhibitory Fc-gamma receptors (FcgammaRs) by inducing upregulation
94 rus neutralization, whereas Fc engagement of Fc-gamma receptors (FcgammaRs) could mediate an array of
95 tate its uptake into target cells expressing Fc gamma receptors (FcgR)-a process known as antibody-de
96 icrobiota-specific antibodies and binding to Fc-gamma-receptors (FcgR), with the strongest relationsh
98 report that the signaling crosstalk between Fc gamma receptor (FcyR) and Toll-like receptor (TLR)2/1
100 variants: G236R/L328R (GRLR) that abrogates Fc-gamma receptor (FcyR) binding, and two variants that
101 ediated immune control by counteracting host Fc-gamma receptor (FcyR) mediated immune control mechani
103 t deficiencies and an allele of a particular Fc gamma receptor gene (FCGR2A) also have been described
106 These results provide further evidence that Fc gamma receptor genetic variations may modulate HIV va
108 n, we found that host genetics, specifically Fc gamma receptor genetic variations, influenced whether
111 expressed less Fc gammaRIIb, the inhibitory Fc gamma receptor; however, this did not account for enh
112 lso inhibits murine lung carcinoma growth in Fc gamma receptor-humanized mice, and such effect is med
114 enhanced via an opsonizing antibody through Fc gamma receptor I (FcgammaRI)-mediated endocytosis.
115 ly to prostate-specific membrane antigen and Fc gamma receptor I, thus eliciting highly selective can
116 especially the integrin receptor Mac-1, the Fc-gamma receptor I (FcgammaRI), and the transcription f
118 aired AM phagocytosis through C5a receptor 1/Fc-gamma receptor I inhibition or by stealth NP surfaces
119 ecognize cell surface-associated NS1 trigger Fc-gamma receptor I- and/or IV-mediated phagocytosis and
120 nd IgG2a, but not mouse IgG3, and by a mouse Fc gamma receptor II and III (FcgammaRII/III)-specific r
123 n to be mediated by crystallizable fragment (Fc)-gamma receptor IIa-dependent platelet activation.
124 rotein engineering to decrease Fc binding to Fc-gamma receptor IIa while retaining certain other effe
130 h CD16b, the neutrophil-specific form of the Fc gamma receptor III, whereas the transmembrane glycopr
132 e of a pathway involving anti-ASNase IgG and Fc-gamma receptor III (Fc-gammaRIII) implies that IgG an
133 s applied to examine the interaction between Fc gamma receptor IIIA (CD16A) expressed on Chinese hams
136 ial cell tethering requires the low-affinity Fc gamma receptor IIIB (Fc gamma RIIIB), and the beta(2)
137 Immunoglobulin G (IgG) cross-linking with Fc gamma receptor IIIB (FcgammaRIIIB) triggers neutrophi
139 we demonstrate that signaling via activating Fc gamma receptors in conjunction with Toll-like recepto
140 dels supporting the importance of inhibitory Fc gamma receptors in modulating immune-complex-mediated
141 te mucosal tolerance, the role of activating Fc gamma receptors in modulating T helper type (Th)2-dep
142 icient mice were used to examine the role of Fc gamma receptors in the induction of peripheral tolera
145 that broadly reactive antibodies require Fc-Fc gamma receptor interactions for optimal protection; h
147 nd that binding of the immune complex to the Fc gamma receptor is required for TNF-alpha and IL-6 mRN
151 ptome differential expression (DE) analysis, Fc gamma receptor-mediated phagocytosis and axon guidanc
152 ated that they are involved in the lysosome, Fc gamma receptor-mediated phagocytosis, regulation of a
155 ortion of opsonizing anti-Gal interacts with Fc gamma receptors on APC and induces effective uptake o
156 nteractions between host immunoglobulins and Fc gamma receptors on effector cells, in addition to the
157 Leishmania amastigotes allows them to ligate Fc gamma receptors on inflammatory macrophages to prefer
159 ector functions through either complement or Fc gamma receptors, providing the bacteria with a surviv
160 ive efforts to "silence" unwanted binding to Fc-gamma receptors, resulting in at least 45 different v
164 herapy, and matched control individuals were Fc gamma-receptor-stimulated with/without priming with P
165 To determine the role of the GPI anchor in Fc gamma receptor synergy, we have developed a model sys
168 o target cytotoxic effector cells expressing Fc gamma receptor type I (Fc gammaRI, CD64) to HER2/neu-
169 ils with blocking antibodies directed toward Fc gamma receptor type IIA or the integrin chain CD11b c
172 an cytomegalovirus (HCMV) encodes four viral Fc-gamma receptors (vFcgammaRs) that counteract antibody