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1 ure (single monomer vs. tandemly repeated in Felis).
2 similar (97 to 99%) to "H. heilmannii" or H. felis.
3 eukin-10-deficient (IL-10(-/-)) mice with H. felis.
4  and the mice were orally inoculated with H. felis.
5 type (WT) C57BL/6 mice were infected with H. felis.
6 ion with Helicobacter pylori or Helicobacter felis.
7 nisms and evaluate the immune response to H. felis.
8 ainly CD4(+) for H. pylori and CD8(+) for H. felis.
9 cts mice against challenge with Helicobacter felis.
10 e to be designated a new species, Rickettsia felis.
11 nd congenic mice with a single isolate of H. felis.
12 h either Helicobacter pylori or Helicobacter felis.
13 a after infection of mice with H pylori or H felis.
14 n and IL-8Tg mice infected with Helicobacter felis.
15 urease B (ureB) gene of H. heilmannii and H. felis.
16 re positive for H. heilmannii and two for H. felis.
17 is of the sialome of cat flea Ctenocephaides felis.
18 og (2) genotypes, C. meleagridis (7), and C. felis (1).
19 re gavaged with H pylori SS1 or Helicobacter felis; 3 months later, stomachs, spleens, and sera were
20 s, 20 were qPCR positive for FHV-1, 7 for M. felis, 5 for FCV, 1 for C. felis, and 0 for B. bronchise
21 ridis (17 persons), C. canis (6 persons), C. felis (6 persons), and C. suis (1 person) infection.
22  than to that of the California strain of H. felis (84%).
23 ted to that of the Ohio-Florida strain of H. felis (89%) than to that of the California strain of H.
24             The relationship of Helicobacter felis, a bacterium observed in the stomachs of cats, to
25 ly described bacterium closely related to R. felis, a known human pathogen.
26 solates of A. fumigatus, A. viridinutans, A. felis, A. pseudoviridinutans, and A. wyomingensis but wa
27 vitro studies demonstrated that Helicobacter felis activates complement in normal mouse serum but not
28 erum from Rag2(-/-) mice, indicating that H. felis activates complement through the classical pathway
29            After treatment with Helicobacter felis Ag, DC were polarized to secrete interleukin-6 as
30 fection in a host infected with Helicobacter felis alters the natural outcome of T. gondii infection,
31 acteria were closely related to Helicobacter felis, although there was heterogeneity among the sequen
32 ri, Helicobacter heilmannii and Helicobacter felis among others.
33                                   Cytauxzoon felis, an emerging virulent protozoan parasite that infe
34 ar to those originating from C. psittaci, C. felis and C. caviae.
35  C57BL/6 (-/-)] were orally infected with H. felis and examined longitudinally using routine histolog
36 L/6-T-bet knockout (KO) litter mates with H. felis and examined the bacterial colonization, immune re
37                                Chlamydophila felis and feline herpesvirus (FHV) are pathogens commonl
38 NS-GAS mice) were infected with Helicobacter felis and given the CCK2/gastrin receptor antagonist YF4
39 ori and non-Helicobacter pylori organisms H. felis and H. heilmannii and analyzed the association bet
40  and H. pylori and 1(20%) coinfected with H. felis and H. pylori (p = 0.15).
41  and H. pylori and 3(60%) coinfected with H. felis and H. pylori (p = 0.66).
42 that the major protein bands of Helicobacter felis and Helicobacter bizzozeronii, two Helicobacter sp
43 PRIL Tg mice were infected with Helicobacter felis and Helicobacter pylori and compared with noninfec
44 uce protective immunity against Helicobacter felis and Helicobacter pylori infection in mice.
45 C57BL/6 mice were infected with Helicobacter felis and received bacterial eradication therapy after 2
46 currence of high relative copy numbers of C. felis and severe clinical signs in all cats was seen.
47  complex prevents gastric colonization by H. felis and the inflammatory response.
48 ently (e.g., Bartonella henselae, Rickettsia felis), and their mechanisms of transmission and impact
49 r FHV-1, 7 for M. felis, 5 for FCV, 1 for C. felis, and 0 for B. bronchiseptica.
50 re positive for FHV, 97 were positive for C. felis, and 16 were positive for both pathogens.
51 e orally infected with a single strain of H. felis, and 2 and 11 weeks after infection, the mice were
52 argeted mice were infected with Helicobacter felis, and a series of adoptive transfer experiments was
53 virus (FCV), Mycoplasma felis, Chlamydophila felis, and Bordetella bronchiseptica.
54  diarrhea, only infections with C. canis, C. felis, and subtype family Id of C. hominis were associat
55  of the H. muris strains, four strains of H. felis, and two strains of Eperythrozoon suis were sequen
56 atusRickettsia amblyommii,"R. montanensis,R. felis, andR. belliiwere frequently identified species, a
57 ins of H. pylori, as well as in Helicobacter felis, another member of the same genus.
58              Sera were also evaluated for H. felis antibody by ELISA.
59                                           H. felis antral colonization remained stable over time amon
60 th C. hominis and C. parvum; C. canis and C. felis are responsible for only a small number of cases.
61 of recent reports have implicated Rickettsia felis as a human pathogen, paralleling the increasing de
62 d-type mice were colonized with Helicobacter felis, as a model of human H. pylori infection.
63  after challenge with either H. pylori or H. felis, as confirmed by the complete absence of any bacte
64 ygous mice were inoculated with Helicobacter felis at 6 weeks of age and compared at various time poi
65 e-linked immunosorbent assay (ELISA) with H. felis ATCC 49179 antigen were performed with 101 serum s
66 nd for 1 year after oral inoculation with H. felis (ATCC 49179).
67                                           C. felis became undetectable in some of the cats during or
68 etection of two genotypes of Ctenocephalides felis biting humans in New Jersey, USA.
69 le when paired with the MAT1-1 isolate of A. felis but not with any of the other species.
70 d IL-10(-/-) mice infected with Helicobacter felis by gastric lavage.
71 L/6 x 129SvEv)F1] mice were infected with H. felis by oral gavage and were assessed histologically an
72                          A unique strain (S. felis C4) was isolated from feline skin that inhibited M
73                                           H. felis can induce a hypertrophic gastropathy in the C57BL
74 obacter pylori (H. pylori) in domestic cats (Felis cattus) less than 2 years of age has been well des
75 his method to the under-annotated genomes of Felis catus (domestic cat) and Bos taurus (cow).
76 mental and theoretical analysis reveals that Felis catus exploits fluid inertia to defeat gravity and
77  present predominantly in one felid species: Felis catus GHV 1 (FcaGHV1) in domestic cats, Lynx rufus
78 sent the earliest evidence for domestic cat (Felis catus L., 1758) from Kazakhstan, found as a well p
79 ary fibroblasts isolated from domestic cats (Felis catus) and pumas (Puma concolor) with FeLV and qua
80 e disease and mortality in the domestic cat (Felis catus) and serves as a natural model for HIV infec
81               It is known that domestic cat (Felis catus) APOBEC3Z3 (A3Z3), the ortholog of human APO
82  direction and retinal disparity in the cat (Felis catus) are all strongly related to the organizatio
83                               Domestic cats (Felis catus) are known to develop cognitive impairment,
84 nce spanning 758,291 bp of the domestic cat (Felis catus) extended and classical class II region.
85  characterized the proteome of domestic cat (Felis catus) follicular fluid EVs (ffEV).
86               We show that the domestic cat (Felis catus) laps by a subtle mechanism based on water a
87                          Urban domestic cat (Felis catus) populations can attain exceedingly high den
88 ated the susceptibility of the domestic cat (Felis catus) to CWD infection experimentally.
89 nal referentiality in a common domestic cat (Felis catus) vocalization, the authors conducted 2 exper
90 and DRB gene homologues of the domestic cat (Felis catus) were cloned and sequenced to compare the pa
91 stic felids, vocalizations by domestic cats (Felis catus) were compared with cries by their closest w
92 entify felid GHVs, we screened domestic cat (Felis catus), bobcat (Lynx rufus), and puma (Puma concol
93  GHVs present in the blood of domestic cats (Felis catus), bobcats (Lynx rufus), and pumas (Puma conc
94 uenced the red and green opsin cDNAs of cat (Felis catus), horse (Equus caballus), gray squirrel (Sci
95 of FIV have been described for domestic cat (Felis catus), puma (Puma concolor), lion (Panthera leo),
96 several closely related to the domestic cat (Felis catus).
97 auses an AIDS-like disease in domestic cats (Felis catus).
98 roduced species, particularly the feral cat, Felis catus, and European red fox, Vulpes vulpes, and ch
99 bp mitochondrial genome of the domestic cat, Felis catus, has been sequenced and conforms largely to
100  comparative gene maps, the feline gene map (Felis catus, Order Carnivora, 2N = 38) displays the high
101                                 Following H. felis challenge, addition of the adjuvant CpG ODN provid
102 FHV-1), feline calicivirus (FCV), Mycoplasma felis, Chlamydophila felis, and Bordetella bronchiseptic
103                                           H. felis colonization also was increased in Duoxa(+/-) mice
104 atment did not alter the overall level of H. felis colonization but did result in significant down-re
105                                           H. felis colonization was significantly greater in the iNOS
106 onounced gastric atrophy after short-term H. felis colonization with a similar extent of preneoplasia
107                                           H. felis colonized the mucus layer in the stomachs of Duoxa
108     A high percentage of free-ranging pumas (Felis concolor) are infected with feline lentiviruses (p
109 rresponding transmembrane segments of the H. felis CopA pump were identified by hydrophobicity analys
110 ch single-nucleotide polymorphisms in the C. felis cytb gene had been identified.
111                                       The C. felis cytb genotype cytb1 is associated with increased s
112 69 cats with cytauxzoonosis for which the C. felis cytb genotypes had been characterized previously.
113 design a PCR panel that could distinguish C. felis cytb1 from other cytochrome b genotypes.
114                  The PCR panel identified C. felis cytb1 with 100% sensitivity and 98.2% specificity.
115 tovaquone, a ubiquinone analogue, targets C. felis cytochrome b (cytb), of which 30 unique genotypes
116                      We characterized the C. felis cytochrome b gene (cytb) in cats with cytauxzoonos
117    C57BL/6 mice inoculated with Helicobacter felis develop gastritis by 4 weeks.
118  felis were monitored for the presence of C. felis DNA on ocular swabs by using real-time PCR and for
119               Since felids outside the genus Felis do not harbor enFeLV genomes, we hypothesized abse
120 otor task and sitting quietly in adult cats (Felis domestica).
121 um pratense 1 (Phl p 1) and the cat allergen Felis domesticus 1 (Fel d 1) greater than 3.5 Immuno Sol
122 st mite (Dermatophagoides farinae), and cat (Felis domesticus) allergens in home dust samples, and sp
123                           The cat isocortex (Felis domesticus) shows a similar structure.
124  now we add the successful cloning of a cat (Felis domesticus) to this list.
125         Infection of IL-10(-/-) mice with H. felis elicited a severe chronic gastritis and a greatly
126         Infection of IL-10(-/-) mice with H. felis elicited severe gastritis.
127       The data suggest that H. pylori and H. felis employ conserved mechanisms of ATPase-dependent co
128  vitro, induction of oxidative defense by H. felis failed to prevent a direct bacteriostatic effect a
129 tion to its annoyance to pets and humans, C. felis felis is responsible for flea bite allergy dermati
130                The cat flea, Ctenocephalides felis felis, is the most important ectoparasite of domes
131  by experimental infection with Helicobacter felis, followed by antibiotic eradication therapy and su
132  mice chronically infected with Helicobacter felis for 2 mo with the SOM analogue octreotide resolved
133 eporter mice were infected with Helicobacter felis for 3 and 8 weeks.
134 ions from male C57BL/6 mice infected with H. felis for 6 months.
135 el(-/-) mice were infected with Helicobacter felis for 6 weeks or 12 months.
136 C57BL/6 wild-type mice were infected with H. felis for either 12 or 18 months.
137           The sequences of two strains of H. felis from cats in California were identical, as were th
138  enzymes DdeI and MnlI for distinguishing H. felis from closely related bacteria was examined.
139 6-bp DNA fragment of the 16S rRNA gene of H. felis from each of four experimentally infected cats at
140 rs from the numt previously described in the Felis genus in: (1) chromosomal location (F2-telomeric r
141 und in the genomes of related species of the Felis genus, previously shown to harbor enFeLVs.
142 elicobacter spp. revealed that all except H. felis grew in serum-free, unsupplemented F-12.
143 ld-type mice were infected with Helicobacter felis (H. felis) to induce gastritis.
144 ther helicobacters (H. canis, H. cineadi, H. felis, H. mustelae, H. nemestrinae, H. pullorum, H. pylo
145 te substantial antigenic homology between H. felis, H. pylori, and H. bizzozeronii.
146 the "H. heilmannii"-like organisms (HHLO) H. felis, H. salomonis, and H. bizzozeronii.
147 , or both, produced a specific fecal anti-H. felis IgA response, with the highest IgA levels occurrin
148 ection with T. gondii alters the specific H. felis immune response, converting a previously resistant
149 ombination developed increased serum anti-H. felis immunoglobulin G (IgG).
150 0 (57%), H. heilmannii in 17/250 (6%) and H. felis in 10/250 (4%), respectively.
151 , paralleling the increasing detection of R. felis in arthropod hosts across the globe, primarily in
152  whole-cell sonicate vaccine of Helicobacter felis in conjunction with cholera toxin as a mucosal adj
153    Cellular stress was induced with IR and H felis in Ikkbeta(Deltastom), Ikkbeta(F/F), and cis-NF-ka
154        The acquisition and persistence of R. felis in mosquitoes was demonstrated by quantitative PCR
155 ed with a Florida isolate of Haemobartonella felis in order to collect organisms and evaluate the imm
156                            Challenge with H. felis increased the inflammatory response in the gastric
157                            Infection with H. felis induced expression of Duox2 and Duoxa2 in the stom
158                            The suppressed H. felis-induced gastric phenotype of Stat3(SA/SA) mice was
159  the role of CD73 in regulating Helicobacter felis-induced gastritis and colonization.
160 out mice have increased susceptibility to H. felis-induced gastritis, with enhanced gastric inflammat
161 d p53 hemizygous mouse model of Helicobacter felis-induced gastritis.
162 ced peritonitis, DSS-induced colitis, and H. felis-induced gastritis.
163 n of noggin in mice increased H pylori- or H felis-induced inflammation and epithelial cell prolifera
164 ative of an impaired Th1 component of the H. felis-induced inflammatory response when the influence o
165  of SPEM to the neoplastic process in the H. felis -infected C57BL/6 mouse, we have now studied the a
166                        Natural bites from R. felis-infected An. gambiae were able to cause transient
167 lesterol levels were observed between the H. felis-infected and -uninfected iNOS-/- mice in this stud
168 ddition, a group of mosquitoes was fed on R. felis-infected BALB/c mice.
169                              Gastritis in H. felis-infected CD73-/- mice was significantly worse than
170                              In contrast, H. felis-infected IL-10(-/-) mice develop a severe hyperpla
171            In contrast to wild-type mice, H. felis-infected IL-10(-/-) mice had a marked increase in
172                                           H. felis-infected IL-10(-/-) mice may provide a model with
173 cter-associated gastric carcinogenesis in H. felis-infected mice on a uniform C57BL/6 background hous
174 ic Pdx1 expression was observed in either H. felis-infected or DMP777-treated mice.
175 nths of age, in comparison with Helicobacter felis-infected wild-type littermates.
176 us metaplasia in the fundic mucosa, while H. felis-infected wild-type mice had severe atrophic and me
177 cant reduction of gastric inflammation in H. felis-infected, as well as immunized/challenged, mice.
178 itude between 12 and 15 months of chronic H. felis infection (P = 0.167).
179 jective of this study was to determine if H. felis infection alters gastric histopathology, proinflam
180                                      Both H. felis infection and K-ras mutation induced upregulation
181 adjuvant and examined for the presence of H. felis infection and leukocyte infiltration into the gast
182 R is a sensitive technique for monitoring C. felis infection and the response to antibiotic treatment
183 Ure produces a long-lasting inhibition of H. felis infection but that residual bacteria may produce a
184                                   Chronic H. felis infection did not alter these proportions, but ora
185  infection, but in GECs by IR or long-term H felis infection during progression to dysplasia.
186 s indicate that, in the absence of MyD88, H. felis infection enhances the activation of non-canonical
187             Nlrc5(mo-KO) mice with chronic H felis infection had increased numbers of gastric B-cell
188                      Persistent Helicobacter felis infection in (C57BL/6 x 129SvEv)F1 mice induces ch
189              These findings indicate that H. felis infection in cats induces lymphoid follicular hype
190 cilitated diagnosis and discrimination of H. felis infection in cats.
191 n the recent report of high prevalence of R. felis infection in patients with "fever of unknown origi
192 y regulate the epithelial consequences of H. felis infection in the stomach, while c-Rel-mediated sig
193                                           H. felis infection leads to increased apoptosis and altered
194 ogenesis, and novel experiments involving H. felis infection of bone marrow transplanted irradiated m
195 veloped to detect and quantify Chlamydophila felis infection of cats.
196                                 Helicobacter felis infection of INS-GAS mice led to accelerated (< or
197 s of the antrum and pyloric junction, but H. felis infection of the Apc mutant mouse does not lead to
198 on between hypergastrinemia and Helicobacter felis infection on gastric corpus carcinogenesis in FVB/
199 emia in mice can synergize with Helicobacter felis infection to induce gastric carcinoma.
200                A mouse model of Helicobacter felis infection was used to study possible genetic deter
201                  T-bet KO mice respond to H. felis infection with a markedly blunted IL-1beta and TNF
202 as upregulated in myeloid cells with acute H felis infection, but in GECs by IR or long-term H felis
203 ted development of gastric pathology upon H. felis infection, but the mechanisms leading to this phen
204                         Within 4 weeks of H. felis infection, there are striking alterations in the c
205 emia, and possible synergy with Helicobacter felis infection, were investigated in insulin-gastrin (I
206  was observed in BALB/c and C3H/HeJ after H. felis infection, whereas sPLA2 expression was absent in
207 -deficient Stat3(SA/SA) mice with chronic H. felis infection, which mimics human H. pylori infection
208                         Within 4 weeks of H. felis infection, wild-type mice develop a mild, focal ch
209  by DMP-777 treatment, L-635 treatment, or H felis infection.
210  to ionizing radiation (IR) and Helicobacter felis infection.
211 the cytokine environment during Helicobacter felis infection.
212  with oral doxycycline will not eliminate C. felis infection.
213  a competent immune response to Helicobacter felis infection.
214 odel of Helicobacter pylori and Helicobacter felis infection.
215  protection of the gastric mucosa against H. felis infection.
216 ion-mediated protection against Helicobacter felis infection.
217 une-mediated gastric pathology seen after H. felis infection.
218 iency and STAT3 activation in response to H. felis infection.
219 evaluated histologically for magnitude of H. felis infection.
220 ecies has the potential to be a vector of R. felis infection.
221 epithelial pathology even 12 months after H. felis infection.
222 not increase the risk of H. heilmannii or H. felis infection.
223  be useful for the serologic diagnosis of H. felis infections in cats.
224  characterized the cat flea (Ctenocephalides felis) innate immune response to R. typhi Initially, we
225                  The stomachs of all five H. felis-inoculated cats became colonized, as determined by
226 elopment of gastric atrophy and cancer in H. felis/INS-GAS mice, while the proton pump inhibitor show
227                                   Cytauxzoon felis is a virulent, tick-transmitted, protozoan parasit
228 t was concluded that gastritis induced by H. felis is associated with increased HbA(1c) levels in the
229 d Helicobacter that is closely related to H. felis, is associated with little or no gastritis, and sh
230                       Using the Helicobacter felis mouse model or H. pylori mouse model, both prophyl
231    We have utilized the C57BL/6 Helicobacter felis mouse model to critically analyze the role of the
232 employed the well-characterized Helicobacter felis-mouse model.
233                   African black-footed cats (Felis nigripes) are endangered wild felids.
234           After 3 months of infection with H felis, Nlrc5(mo-KO) mice developed gastric hyperplasia (
235 , and four gastric ulcer samples), 10 for H. felis (one gastritis, three duodenal ulcer, and six gast
236                     Three were strains of H. felis, one was H. bilis, and one was a novel helicobacte
237 own that infection of mice with Helicobacter felis or induction of acute parietal cell loss with the
238  fed with either blood meal infected with R. felis or infected cellular media administered in membran
239  could be specifically attributed to FCV, M. felis, or C. felis were seen, although interpretation in
240 ples from 8 children with C. meleagridis, C. felis, or C. parvum dog genotype were tested for anti-hu
241 R assay, the minimum detectable number of H. felis organisms was determined to be between 50 and 704.
242 tly up-regulated in WT mice infected with H. felis (P < 0.05) but were slightly elevated or were at b
243 h1-associated IgG2c antibody responses to H. felis (P <0.0003); the Th2-associated IgG1 responses wer
244                 All the H. heilmannii and H. felis PCR positive patients were also positive for H. py
245 l role throughout the evolution of the genus Felis, predating cat exposure to feline retroviruses.
246                                 Blood for H. felis purification was repeatedly collected from splenec
247 r to Gastrospirillum hominis or Helicobacter felis (referred to as gastrospirilla).
248 th 7 and 14 days of doxycycline decreased C. felis relative copy numbers and clinical signs rapidly.
249  are caused by host-specific C. canis and C. felis, respectively.
250 ed with Helicobacter pylori and Helicobacter felis, respectively.
251  cell-deficient TCRbetadelta-/- mice with H. felis resulted in high levels of colonization, but no de
252 on in the body of the stomach, lower anti-H. felis serum IgG antibody responses (although both the wi
253 nfected animalls developed sustained anti-H. felis serum immunoglobulin G antibody responses.
254          The co-occurrence of domestic cats (Felis silvestris catus) and wild felids in rural landsca
255                    Worldwide, domestic cats (Felis silvestris catus) outnumber domestic dogs (Canis f
256 st-virus evolution.IMPORTANCE Domestic cats (Felis silvestris catus) were domesticated from wildcats
257 enetic linkage (GL) map of the domestic cat (Felis silvestris catus).
258 youngest feline ERV groups in domestic cats (Felis silvestris catus); some members are replication co
259 ment of paw preferences in the domestic cat, Felis silvestris catus, is explored.
260 closest wild relative, the African wild cat (Felis silvestris lybica).
261 979 domestic cats and their wild progenitors-Felis silvestris silvestris (European wildcat), F. s. ly
262     Here, we detected several ERV-DC loci in Felis silvestris silvestris Notably, a species-specific
263 we investigated ERV-DC in European wildcats (Felis silvestris silvestris) and detected four loci: ERV
264 The survival of indigenous European wildcat (Felis silvestris silvestris) populations can be locally
265 group represents a distinctive subspecies of Felis silvestris.
266 ed by 16S rRNA gene sequencing as Mycoplasma felis (six cases) and Mycoplasma gateae (one case).
267 ed controls (P = 0.0008 and P = 0.002 for H. felis sonicate and CT, respectively).
268 tection induced by oral immunization with H. felis sonicate and CT.
269 e proportions, but oral immunization with H. felis sonicate plus cholera toxin (CT) or with CT alone
270  this sequence data, we designed a set of H. felis-specific primers.
271 E. suis being most similar to that of the H. felis strain from California.
272 ession of the H2O2-inducible katA gene in H. felis that colonized Duoxa(-/-) mice, compared with that
273             One year after infection with H. felis, the wild-type and p53 hemizygous mice showed seve
274  critical regulator of R. typhi burden in C. felis These data suggest that targeting the IMD pathway
275  The untreated cats remained positive for C. felis throughout the trial; clinical signs were most sev
276 control mice were infected with Helicobacter felis to create a model of Helicobacter pylori infection
277 ce were infected with Helicobacter felis (H. felis) to induce gastritis.
278      While bioinformatics analysis of the C. felis transcriptome identified homologs to all of the Dr
279                                  Four SPF H. felis-uninfected cats served as controls.
280 ori and H. heilmannii was 17(6%) and with H. felis was 10(4%), respectively.
281                                           R. felis was also found in the cotton used for sucrose feed
282         The 16S rRNA gene of Haemobartonella felis was amplified by using universal eubacterial prime
283                                           R. felis was detected in mosquito feces up to day 14.
284 cing of PCR products of H. heilmannii and H. felis was done.
285 y cytokine expression, or colonization of H. felis was evaluated in CD73-deficient (CD73-/-) mice or
286                                           H. felis was inoculated by gastric intubation into SPF C57B
287       The prevalence of H. heilmannii and H. felis was low in our patients with dyspepsia.
288                              Furthermore, R. felis was visualized by immunofluorescence in salivary g
289 er bacteria (H. pylori, H. hepaticus, and H. felis) was mediated not by TLR4 but rather by TLR2.
290 SvEv (B6129) mice infected with Helicobacter felis, we conducted a study to characterize H. pylori in
291 rons of Helicobacter pylori and Helicobacter felis were cloned by gene library screening.
292 ), IgG2a, and IgG2c antibody responses to H. felis were determined.
293 . pylori, an H. pylori cagE(-) mutant, or H. felis were killed 2-24 weeks postchallenge.
294     Fifteen cats infected with Chlamydophila felis were monitored for the presence of C. felis DNA on
295 cifically attributed to FCV, M. felis, or C. felis were seen, although interpretation in this cohort
296 rgastrinemic mice infected with Helicobacter felis were studied at multiple stages of gastric dysplas
297 l disease (colitis), and antibody levels (H. felis) were examined.
298                                 Helicobacter felis, which does not express CagA or VacA, causes chron
299 als immunized intranasally with sonicated H. felis with CT and CpG.
300 e were immunized with sonicated Helicobacter felis with CT and/or CpG ODN adjuvants.

 
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