ライフサイエンスコーパス: Fusobacterium nucleatum

1 bserved for Bacteroides thetaiotaomicron and Fusobacterium nucleatum.

2 llowing stimulation of epithelial cells with Fusobacterium nucleatum.

3 Actinomyces israelii with the coisolation of Fusobacterium nucleatum.

4 adA adhesin from the Gram-negative bacterium Fusobacterium nucleatum.

5 olymicrobial oral infections with or without Fusobacterium nucleatum.

6 cus gordonii and the opportunistic commensal Fusobacterium nucleatum.

7 symporter (NSS) family has been cloned from Fusobacterium nucleatum.

8 olic analysis of the dominant oral bacterium Fusobacterium nucleatum.

9 ggregation with the anaerobic oral bacterium Fusobacterium nucleatum.

10 mediated adherence to a peridontal pathogen, Fusobacterium nucleatum.

11 ella parvula, Peptostreptococcus micros, and Fusobacterium nucleatum.

12 occus mutans, Lactobacillus acidophilus, and Fusobacterium nucleatum.

13 , Aggregatibacter actinomycetemcomitans, and Fusobacterium nucleatum.

14 xed infection with the otherwise stimulatory Fusobacterium nucleatum.

15 activation by another periodontal bacterium, Fusobacterium nucleatum.

16 For Fusobacterium nucleatum (100%/100%), there was no differ

17 7.4%), Porphyromonas gingivalis (15.1%), and Fusobacterium nucleatum (14.2%).

18 inations that resulted in tailing endpoints (Fusobacterium nucleatum, 86% agreement) or in cases of l

19 Further specificity was seen when Fusobacterium nucleatum (a middle colonizer), Aggregatib

20 be colonized by potential pathogens such as Fusobacterium nucleatum, a bacterium linked with intraut

21 production of CCL20 and hBDs in response to Fusobacterium nucleatum, a commensal bacterium of the or

22 Using Fusobacterium nucleatum, a Gram-negative anaerobe freque

23 entified a cell wall-associated protein from Fusobacterium nucleatum, a Gram-negative bacterium of th

24 t evidence for the immunosuppressive role of Fusobacterium nucleatum, a gram-negative oral bacterium

25 We previously reported that Fusobacterium nucleatum, a ubiquitous gram-negative bact

26 Key quorum-sensing plaque bacteria, such as Fusobacterium nucleatum, act as bridging species between

27 microbial species (Porphyromonas gingivalis, Fusobacterium nucleatum, Actinomyces naeslundii, Tannere

28 monas gingivalis, Prevotella intermedia, and Fusobacterium nucleatum activated both TLRs, but TLR4 pl

29 helial cell response to the common bacterium Fusobacterium nucleatum, an important bridging species t

30 It can be coisolated with Fusobacterium nucleatum, an opportunistic bacterial path

31 Fusobacterium nucleatum, an opportunistic pathogen, is t

32 D-2 mRNA was induced by cell wall extract of Fusobacterium nucleatum, an oral commensal microorganism

33 eration sequencing implicated coinfection of Fusobacterium nucleatum and Actinomyces israelii, resolv

34 also significantly associated with pathogens Fusobacterium nucleatum and Aggregatibacter actinomycete

35 A higher abundance of Fusobacterium nucleatum and an increased LPS pathway wer

36 odels and statistical methods, we identified Fusobacterium nucleatum and Anaerostipes hadrus with the

37 teroides fragilis, Enterococcus faecalis and Fusobacterium nucleatum and one probiotic species, Esche

38 contribute to carcinogenesis, in particular, Fusobacterium nucleatum and Porphyromonas gingivalis, ba

39 mixed infection with the periodontopathogens Fusobacterium nucleatum and Porphyromonas gingivalis.

40 The most frequently found bacteria were Fusobacterium nucleatum and Prevotella denticola.

41 n the biofilm, and reduced the expression of Fusobacterium nucleatum and Prevotella intermedia in the

42 , Tannerella forsythia, Treponema denticola, Fusobacterium nucleatum and Prevotella intermedia) in sa

43 acterial pathogens Enterococcus faecalis and Fusobacterium nucleatum and remains catalytically active

44 ycetemcomitans, Streptococcus parasanguinis, Fusobacterium nucleatum and several species belonging to

45 he opportunistic pathogens Escherichia coli, Fusobacterium nucleatum and Streptococcus agalactiae to

46 omonas gingivalis and its consortium members Fusobacterium nucleatum and Streptococcus gordonii confi

47 treptococcus mitis, Veillonella parvula, and Fusobacterium nucleatum) and the same biofilm plus the p

48 ophilus aphrophilus, Actinomyces naeslundii, Fusobacterium nucleatum, and A. actinomycetemcomitans, a

49 as Prevotella intermedia, Selenomonas noxia, Fusobacterium nucleatum, and Actinobacillus actinomycete

50 odontal pathogens (Porphyromonas gingivalis, Fusobacterium nucleatum, and Aggregatibacter actinomycet

51 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, and Aggregatibacter actinomycet

52 lls stimulated with a periodontal bacterium, Fusobacterium nucleatum, and other stimulants.

53 inae, Mobiluncus mulieris, Prevotella bivia, Fusobacterium nucleatum, and Peptoniphilus species.

54 tella intermedia, Streptococcus intermedius, Fusobacterium nucleatum, and Peptostreptococcus micros,

55 ates were Prevotella sp., Porphyromonas sp., Fusobacterium nucleatum, and Peptostreptococcus sp.

56 Streptococcus mutans, Streptococcus sanguis, Fusobacterium nucleatum, and Porphyromonas gingivalis wi

57 eptococcus mutans, Porphyromonas gingivalis, Fusobacterium nucleatum, and Pseudomonas aeruginosa.

58 Of those, Bergeyella, Fusobacterium nucleatum, and Sneathia sanguinegens had n

59 Aggregatibacter aphrophilus, Fusobacterium nucleatum, and Streptococcus intermedius o

60 pecies biofilms of Porphyromonas gingivalis, Fusobacterium nucleatum, and Streptococcus oralis were f

61 gar Candida medium, coaggregation assay with Fusobacterium nucleatum, and sugar assimilation profiles

62 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, and Tannerella forsythia.

63 Prevotella intermedia, Tannerella forsythia, Fusobacterium nucleatum, and total bacteria count.

64 pathogens, such as Porphyromonas gingivalis, Fusobacterium nucleatum, and Treponema denticola, are am

65 Streptococcus mutans, Enterococcus faecalis, Fusobacterium nucleatum, and Veillonella dispar was used

66 ivalis, anti-Prevotella intermedia, and anti-Fusobacterium nucleatum antibody concentrations with ant

67 Fusobacterium nucleatum appears to play a role in colore

68 Candida albicans and Fusobacterium nucleatum are well-studied oral commensal

69 Bacteria, such as Fusobacterium nucleatum, are present in the tumor microe

70 these findings by identifying the bacterium Fusobacterium nucleatum as a previously unrecognized che

71 cus anginosus, Porphyromonas gingivalis, and Fusobacterium nucleatum, as well as Campylobacter rectus

72 gregatibacter actinomycetemcomitans JP2, and Fusobacterium nucleatum ATCC 10953 were unable to grow a

73 s biofilm (Streptococcus sanguinis DSM20068, Fusobacterium nucleatum ATCC10953, and Porphyromonas gin

74 Attachment of Fusobacterium nucleatum ATCC25586 and Prevotella interme

75 The extensive search was performed using "Fusobacterium nucleatum", "Bacteroides fragilis", "Color

76 i, Actinobacillus actinomycetemcomitans, and Fusobacterium nucleatum) biofilm formation under anaerob

77 , Aggregatibacter actinomycetemcomitans, and Fusobacterium nucleatum by 3 to 5-log (P < 0.001).

78 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, Campylobacter rectus, and Trepo

79 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, Campylobacter rectus, Eikenella

80 Opportunistic pathogenic bacteria, such as Fusobacterium nucleatum, can enrich within certain tumor

81 h extraction followed by oral infection with Fusobacterium nucleatum caused BONJ-like lesions and del

82 vivo experiments, the combination of Pam and Fusobacterium nucleatum caused the death of gingival fib

83 ous studies showed that hBD-2 was induced by Fusobacterium nucleatum cell wall extract without the in

84 on of 19 newly profiled species and distinct Fusobacterium nucleatum clades.

85 Gnotobiotic studies revealed that while Fusobacterium nucleatum clinical isolates with FadA and

86 Obligate anaerobes other than Fusobacterium nucleatum coaggregated only poorly with ox

87 ve enhanced proliferation in the presence of Fusobacterium nucleatum compared to HPV+ cells, suggesti

88 dance was positively correlated with that of Fusobacterium nucleatum, consistent with hypothesized pr

89 carcinoma (PDAC), high intratumoral loads of Fusobacterium nucleatum correlate with shorter survival

90 omonas gingivalis, Campylobacter rectus, and Fusobacterium nucleatum, could cause localized bone reso

91 Co-culture studies of HNSCC cell lines with Fusobacterium nucleatum demonstrated that HPV-negative c

92 In vitro, Fusobacterium nucleatum disrupts epithelial contacts, in

93 for the presence and amount of EBV, CMV, and Fusobacterium nucleatum DNA using real-time polymerase c

94 F together with either Escherichia coli DNA, Fusobacterium nucleatum DNA, or Porphyromonas gingivalis

95 Fusobacterium nucleatum, Eikenella corrodens, Actinobaci

96 The gram-negative opportunistic pathogen Fusobacterium nucleatum encodes an electron transfer fla

97 s as potentiators of tumorigenesis-including Fusobacterium nucleatum, enterotoxigenic Bacteroides fra

98 wall extracts of Porphyromonas gingivalis or Fusobacterium nucleatum, Escherichia coli lipopolysaccha

99 The presence of Fusobacterium nucleatum (F. nucleatum) and Bacteroides f

100 sought to determine the performance of fecal Fusobacterium nucleatum (F. nucleatum) and Streptococcus

101 Effects of Fusobacterium nucleatum (F. nucleatum) biofilm on epithe

102 eviously demonstrated that sonic extracts of Fusobacterium nucleatum FDC 364 were capable of inhibiti

103 pic dento-epithelial (OD-E) model exposed to Fusobacterium nucleatum (Fn) biofilm.

104 Fusobacterium nucleatum (Fn) has been associated with co

105 Fusobacterium nucleatum (Fn) is one of the most frequent

106 he ubiquitous inflammophilic oral pathobiont Fusobacterium nucleatum (Fn) is widely recognized for it

107 enol red indicator (QLAMP-PhR) for detecting Fusobacterium nucleatum (Fn) levels in colorectal cancer

108 Fusobacterium nucleatum (Fn), a bacterium present in the

109 Fusobacterium nucleatum (Fn), a commensal in the human o

110 ction was used for detecting and quantifying Fusobacterium nucleatum (Fn), Aggregatibacter actinomyce

111 Aggregatibacter actinomycetemcomitans (Aa), Fusobacterium nucleatum (Fn), and Prevotella intermedia

112 ivalis (Pg), Eubacterium saburreum (Es), and Fusobacterium nucleatum (Fn).

113 ermedia (Pi), Tannerella forsythia (Tf), and Fusobacterium nucleatum (Fn).

114 factor in coaggregated mixed infection with Fusobacterium nucleatum (Fn).

115 (Sg)/S. oralis (So)/S. sanguinis (Ss) and Sg/Fusobacterium nucleatum (Fn)/Porphyromonas gingivalis (P

116 employed by the Gram-negative oral pathogen Fusobacterium nucleatum for cell death induction of huma

117 s were significantly higher for P. micra and Fusobacterium nucleatum for the screw-retained group.

118 i, enterotoxigenic Bacteroides fragilis, and Fusobacterium nucleatum, for their virulence features re

119 omonas gingivalis, Tannerella forsythia, and Fusobacterium nucleatum from subgingival biofilm were de

120 tella intermedia, Peptostreptococcus micros, Fusobacterium nucleatum, Fusobacterium polymorphum, Eike

121 t substrate, full-length Vibrio cholerae and Fusobacterium nucleatum glycine riboswitch aptamers with

122 nfection was community acquired in 48 (84%); Fusobacterium nucleatum group and/or Streptococcus inter

123 found that the most common detection was the Fusobacterium nucleatum group and/or Streptococcus inter

124 We propose the term e-FuSion (effusion with Fusobacterium nucleatum group, Streptococcus intermedius

125 ontal pathogens Porphyromonas gingivalis and Fusobacterium nucleatum growth and attachment to human g

126 In previous studies Fusobacterium nucleatum has been shown to induce either

127 Fusobacterium nucleatum has long been found to cause opp

128 monas gingivalis, Prevotella intermedia, and Fusobacterium nucleatum, have recently been shown to sec

129 10-1.56, p=2.64 x 10(-3)) and infection with Fusobacterium nucleatum (HR 1.63, 95% CI 1.08-2.45, p=1.

130 report a case of Lemierre's syndrome due to Fusobacterium nucleatum in a previously healthy 19-year-

131 2019) observe detrimental overpopulation of Fusobacterium nucleatum in mice and patients, suppressin

132 infection with Porphyromonas gingivalis and Fusobacterium nucleatum in mice.

133 inoculation of Porphyromonas gingivalis and Fusobacterium nucleatum in young (4 to 5 mo) and aged (1

134 dentify an anaerobic Gram-negative bacillus, Fusobacterium nucleatum, in a patient with "culture-nega

135 otella intermedia, Campylobacter rectus, and Fusobacterium nucleatum, in subgingival dental plaque of

136 /+) mouse model of intestinal tumorigenesis, Fusobacterium nucleatum increases tumor multiplicity and

137 ed replication plan of key experiments from 'Fusobacterium nucleatum infection is prevalent in human

138 thelial cells reached its peak 2 h following Fusobacterium nucleatum infection whereas it rapidly dec

139 Fusobacterium nucleatum is a common oral anaerobe involv

140 Fusobacterium nucleatum is a Gram-negative anaerobe asso

141 Fusobacterium nucleatum is a gram-negative anaerobe that

142 Fusobacterium nucleatum is a gram-negative anaerobe ubiq

143 Fusobacterium nucleatum is a gram-negative oral anaerobe

144 Fusobacterium nucleatum is a Gram-negative oral anaerobe

145 Fusobacterium nucleatum is among the most prevalent bact

146 Fusobacterium nucleatum is an opportunistic oral pathoge

147 Fusobacterium nucleatum is an oral bacterium associated

148 Fusobacterium nucleatum is an oral commensal bacterium t

149 Fusobacterium nucleatum is an oral pathogen that is link

150 Fusobacterium nucleatum is associated with colorectal ca

151 Fusobacterium nucleatum is capable of binding to and inv

152 Fusobacterium nucleatum is implicated in accelerating co

153 ristic of the suspected periodontal pathogen Fusobacterium nucleatum is its ability to adhere to a pl

154 The oral bacterium Fusobacterium nucleatum is often found in colorectal can

155 Fusobacterium nucleatum is the most commonly observed sp

156 and difficult-to-cultivate species, such as Fusobacterium nucleatum, Leptotrichia (Sneathia) spp., a

157 Fusobacterium nucleatum, long known as a common oral mic

158 agonize TLR4-specific activation by agonist, Fusobacterium nucleatum LPS.

159 l as with Veillonella sp. (early colonizer), Fusobacterium nucleatum (middle colonizer), and Aggregat

160 Fusobacterium nucleatum might be the cause or consequenc

161 Streptococcus intermedius and Fusobacterium nucleatum most likely represent key pathog

162 ly significant, Porphyromonas gingivalis and Fusobacterium nucleatum occur in higher concentrations m

163 ere incubated with Porphyromonas gingivalis, Fusobacterium nucleatum, P. gingivalis lipopolysaccharid

164 ella forsythia [previously T. forsythensis], Fusobacterium nucleatum, Parvimonas micra [previously Pe

165 elevated in AgP in comparison with CP, while Fusobacterium nucleatum, Parvimonas micra, and Campyloba

166 pathogens, including Prevotella intermedia, Fusobacterium nucleatum, Peptostreptococcus micros, and

167 ection protocol using Prevotella intermedia, Fusobacterium nucleatum, Peptostreptococcus micros, and

168 their ability to coaggregate with strains of Fusobacterium nucleatum, Peptostreptococcus micros, Pept

169 inomycetemcomitans, Eikenella corrodens, and Fusobacterium nucleatum/periodonticum were statistically

170 alis (Pg); 4) group G-PgFn: oral gavage with Fusobacterium nucleatum + Pg; 5) group I-Pg: heat-killed

171 , Campylobacter curvus, Eikenella corrodens, Fusobacterium nucleatum, Porphyromonas gingivalis, and P

172 nisms (Actinobacillus actinomycetemcomitans, Fusobacterium nucleatum, Porphyromonas gingivalis, Pepto

173 ctinomycetemcomitans), Campylobacter rectus, Fusobacterium nucleatum, Porphyromonas gingivalis, Prevo

174 ntified virulence mechanisms of oral species Fusobacterium nucleatum, Porphyromonas gingivalis, Strep

175 imens yielded pathogenic bacteria, including Fusobacterium nucleatum, Prevotella heparinolytica, Prev

176 three orange-complex periodontal pathogens (Fusobacterium nucleatum, Prevotella intermedia, and Camp

177 phyromonas gingivalis, Tannerella forsythia, Fusobacterium nucleatum, Prevotella intermedia, and Camp

178 phyromonas gingivalis, Tannerella forsythia, Fusobacterium nucleatum, Prevotella intermedia, and tota

179 outcomes and sustained decreased numbers of Fusobacterium nucleatum, Prevotella intermedia, Campylob

180 h a re-application of MM 3 months after SRP, Fusobacterium nucleatum, Prevotella intermedia, Campylob

181 same double-labeling techniques to identify Fusobacterium nucleatum, Prevotella intermedia, oral Cam

182 n Tannerella forsythia, Treponema denticola, Fusobacterium nucleatum, Prevotella intermedia, Parvimon

183 thogens such as Porphyromonas gingivalis and Fusobacterium nucleatum produce five different short-cha

184 ere we present the crystal structures of the Fusobacterium nucleatum riboswitch bound to FMN, ribofla

185 Chemical mapping on the Fusobacterium nucleatum riboswitch with N-methylisatoic

186 Here, we report the structures of Fusobacterium nucleatum SiaQM with and without Neu5Ac.

187 odels colonized by tumor-promoting bacteria (Fusobacterium nucleatum spp.) or probiotics (Escherichia

188 re stimulated with Porphyromonas gingivalis, Fusobacterium nucleatum, Staphylococcus epidermidis, or

189 Endothelial cells were infected with Fusobacterium nucleatum (strain 25586) for periods of 4,

190 anaerobic pathogens, Prevotella intermedia, Fusobacterium nucleatum, Streptococcus intermedius, and

191 nfections (endodontic pathogens [EP]), i.e., Fusobacterium nucleatum, Streptococcus intermedius, Parv

192 lms and a three-species synthetic community (Fusobacterium nucleatum, Streptococcus mutans, and Strep

193 e draft genome sequence and its analysis for Fusobacterium nucleatum sub spp. vincentii (FNV), and co

194 is issue of Cell Host & Microbe reveals that Fusobacterium nucleatum subsp.

195 ges the long-held notion of the dominance of Fusobacterium nucleatum subsp.

196 Fusobacterium nucleatum subsp. polymorphum was the most

197 ing infection with Porphyromonas gingivalis, Fusobacterium nucleatum subspecies (ssp) nucleatum, ssp

198 rometry (MALDI-TOF MS) for identification of Fusobacterium nucleatum subspecies.

199 e in corncob formation between S. crista and Fusobacterium nucleatum, this property was examined.

200 eponema denticola, Tannerella forsythia, and Fusobacterium nucleatum to colonize the periodontium and

201 Reassignment of two subspecies of Fusobacterium nucleatum to species designations (Fusobac

202 omonas gingivalis, Tannerella forsythia, and Fusobacterium nucleatum using real time polymerase chain

203 ms enumerated were Porphyromonas gingivalis, Fusobacterium nucleatum, Veillonella sp., and total anae

204 givalis, whereas phagocytosis of heat-killed Fusobacterium nucleatum was augmented compared with that

205 es naeslundii, Porphyromonas gingivalis, and Fusobacterium nucleatum was grown on sandblasted and aci

206 Fusobacterium nucleatum was present in 17 patients prior

207 Fusobacterium nucleatum was prevalent in the subgingival

208 omonas gingivalis, Tannerella forsythia, and Fusobacterium nucleatum were analyzed for prediction of

209 , Aggregatibacter actinomycetemcomitans, and Fusobacterium nucleatum were assessed in anaerobic condi

210 Three native plasmids of Fusobacterium nucleatum were characterized, including DN

211 votella intermedia, Eikenella corrodens, and Fusobacterium nucleatum were determined by real-time pol

212 Streptococcus intermedius and/or Fusobacterium nucleatum were identified as a dominant co

213 the bacterium Helicobacter pylori, and later Fusobacterium nucleatum, were implicated in the developm

214 lla parahaemolysans, Lactococcus lactis, and Fusobacterium nucleatum, were significantly more abundan

215 nd humans increases the amount of intestinal Fusobacterium nucleatum, which releases SFCAs.

216 lla, Salmonella, Haemophilus influenzae, and Fusobacterium nucleatum, which share structural and func

217 irocin to successfully target tumor-residing Fusobacterium nucleatum without an immediate effect on t