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1   KLF5's promoter lacks a TATA box and has a GC-rich region.
2 ificantly from the mechanism mediated by the GC-rich region.
3  between positions +52 and +93 base pairs, a GC-rich region.
4 smid pBR322, which contains an AT-rich and a GC-rich region.
5 tential Sp1 binding sites within this highly GC-rich region.
6 box and has several GC boxes within a highly GC-rich region.
7 g more methyl induced CpG-->TpG mutations in GC rich regions.
8 h and a portion of the guanine and cytosine (GC)-rich region.
9 ing four hypoxia-response elements and three GC-rich regions.
10 gion lacks canonical TATA and CAAT boxes and GC-rich regions.
11 tate replication through telomeres and other GC-rich regions.
12 topology, RNA stability, and the presence of GC-rich regions.
13 lysis of the VEGF gene promoter identified a GC-rich region (-66 to -47) which was required for E2-in
14         The rdLRP contains no DNA repeats or GC rich regions and 30% less positively charged amino-ac
15 tream of exon 1 consists of a promoter-like, GC-rich region and a number of putative cis active eleme
16 '-upstream region was sequenced, revealing a GC-rich region and TATA-less sequence containing several
17 ve significant enrichment of heritability in GC-rich regions and in higher-frequency SNPs for both sc
18    IRs are AT-rich sequences flanked by more GC-rich regions and located predominantly in intergenic
19 that NRF2 prefers binding to AREs flanked by GC-rich regions and that NRF1 prefers AT-rich flanking r
20                The SEGS are characterized by GC-rich regions and the absence of long open reading fra
21 man PGHS-1 gene lacks a TATA box, has a very GC-rich region, and contains multiple transcription star
22  consensus element around -45 bp and several GC-rich regions around -60, each of which is responsible
23                      RNAcompete identifies a GC-rich region as SERBP1-binding motif; subsequent genom
24 s stretch of 78 base pairs, which contains a GC-rich region as well as the TATA box.
25 ion site and another laboratory identified a GC-rich region between the TATA box and transcription in
26 thod not only for amplification of extremely GC-rich regions, but also for routine DNA diagnostics an
27                             This contained a GC-rich region composed of two tandemly arrayed Sp-1 sit
28 egion between -62 and -55, which contained a GC-rich region consistent with a consensus sequence for
29     This region contains no TATA box but has GC-rich regions, consistent with the ubiquitous expressi
30                                         This GC-rich region constitutes a "GC box" capable both of bi
31 ed the essential promoter region to a highly GC-rich region containing four Sp-1 binding sites.
32 ses of the VEGF promoter demonstrated that a GC-rich region containing four Sp1 response elements, lo
33 f the 2.2 kb TF 5' promoter indicated that a GC-rich region containing three copies each of the EGR-1
34                A guanine- and cytosine-rich (GC-rich) region directly upstream of the P1 site has bee
35               Furthermore, disruption of the GC-rich region dramatically decreases transcription fact
36 found that Polkappa activity was enhanced in GC-rich regions, euchromatin regions, the promoter of ge
37                                              GC-rich regions flanked by a sharp GC-to-AT transition d
38 ow-coverage ( < 15), 67% were located within GC-rich regions ( &gt; 60%).
39                    A nuclease-hypersensitive GC-rich region in KRAS promoter can fold into a four-str
40   The nuclear factors Sp1 and Sp3 bound this GC-rich region in N2A, H19-7, and HRP.1 cells.
41  EKLF site within a previously characterized GC-rich region in the p21 proximal promoter but also by
42 family of transcription factors that bind to GC-rich regions in gene promoters.
43 thermophilic organisms: simply screening for GC-rich regions in the AT-rich Methanococcus jannaschii
44 on of this promoter is mediated equally by a GC-rich region located between bp -303 and -271 and by t
45 ugments promoter activity of p21 through the GC-rich region located between nucleotides -84 and -74 w
46 of the p21(WAF1/CIP1) promoter showed that a GC-rich region located between positions -83 and -74, wh
47                              CpG islands are GC-rich regions located in the promoter regions of house
48 lated promoter activity occurred through two GC-rich regions located within 633 bp of the transcripti
49 urther point mutation studies found that two GC-rich region mutations disrupted the Satb2 130bp promo
50                 The HSK2 promoter contains a GC-rich region, not present in the mouse promoter, and h
51 mplex bound to the proximal germinal center (GC)-rich region of the PD-L1 gene promoter.
52                                 A relatively GC- rich region of the genome 5' of the alternatives to
53 re CCAAT(N9)CCACG, with N being a strikingly GC-rich region of 9 bp.
54 ags with 100% heavy nucleosides to examine a GC-rich region of a polycytidine string with an unknown
55 howed that WP631 binds preferentially to the GC-rich region of the DNA.
56                                 A relatively GC-rich region of the genome 5' of exon 1 was distinctly
57                                 A relatively GC-rich region of the genome just 5' of exon 1 as well a
58        These mutations, clustered around the GC-rich region of the human MnSOD promoter, change the b
59 Sp1-p300 DNA binding complex on the proximal GC-rich region of the survivin promoter.
60  proteins from ZR-75 cells with the proximal GC-rich region of the VEGF gene promoter were investigat
61 e with worker-biased expression are found in GC-rich regions of the bee genome and also experience hi
62 selective manner, methylation alterations at GC-rich regions of the genome in metachronous tumors and
63 ions for the accumulation of Alu elements in GC-rich regions of the human genome.
64                              CpG islands are GC-rich regions often located in the 5' end of genes and
65 effects are mediated by interaction with the GC-rich region on the promoter.
66 he promoter of the rat pgp2/mdr1b gene has a GC-rich region (pgp2GC) that is highly conserved in mdr
67 iments demonstrate that the proximal, highly GC-rich region (positions -165 to -139) of the human PDG
68                   The binding of EGR1 to the GC-rich region prevented TBP binding to the AT-rich regi
69 moter and that targeted deletion of a single GC-rich region spanning -93 to -58 interrupts Sp1- and D
70 nsity of Alu is two to three times higher in GC-rich regions than in AT-rich regions, while the oppos
71 taining a half-estrogen response element and GC-rich region that interact with ER and Sp1 proteins.
72  these sites shows that RNA Pol II pauses at GC-rich regions that are marked by a sequence motif.
73             These genes tended to cluster in GC-rich regions that have poor coverage in genome sequen
74 onal point mutants were used to identify two GC-rich regions that were responsible for VEGF promoter
75 vealed no classical TATA or CCAAT box in the GC-rich region upstream of cap site.
76   We localized promoter activity to a 452-bp GC-rich region upstream of noncoding exon A, including a
77                                     A highly GC-rich region upstream of the P1 promoter plays an impo
78        The human BCL-2 gene contains a 39-bp GC-rich region upstream of the P1 promoter that has been
79              The human bcl-2 gene contains a GC-rich region upstream of the P1 promoter that has been
80 ns and on the short arms of the chromosomes, GC-rich regions were associated with heterochromatic reg
81 nd to lack TATA or CAAT boxes and to contain GC-rich regions, which are features typical of promoters
82 particular: structural variants, variants in GC-rich regions, which have significantly improved cover
83 ellifera, represents a mosaic of GC-poor and GC-rich regions with rates of recombination an order of
84 nitiation sites in brain and thymus within a GC-rich region, with multiple Sp1-binding motifs located