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1  increase in the precursor activity of serum Gc protein.
2 -acetylgalactosaminidase that deglycosylates Gc protein.
3 ion with precursor activities of their serum Gc protein.
4                  Vitamin D3-binding protein (Gc protein), a serum glycoprotein, is the precursor for
5 8-kilobase transcript on Northern blots, and GCS protein, a approximately 38-kDa polypeptide detected
6 h was preceded by the depletion of the gamma-GCS protein and RNA and by the loss of the cellular pool
7 tudies provide information about full-length GCS protein architecture and insight into the mechanism
8                             Alphaherpesvirus gC proteins are type 1 membrane proteins and are general
9 found to be due to deglycosylation of plasma Gc protein by alpha-N-acetylgalactosaminidase detected i
10 analysis and the ER relocation of endogenous GC proteins by phospholipase A2 inhibitors demonstrate t
11            Serum vitamin D3-binding protein (Gc protein) can be converted by beta-galactosidase of B
12                               Deglycosylated Gc protein cannot be converted to macrophage activating
13                             The SFTSV Gn and Gc proteins exist as heterodimers and further assemble i
14 atients with low precursor activity of serum Gc protein had high serum alpha-N-acetylgalactosaminidas
15 idase activity and MAF precursor activity of Gc protein in patient bloodstream can serve as diagnosti
16                                        Thus, Gc protein is the precursor of the macrophage activating
17 t the essential function of alphaherpesvirus gC proteins is conserved and can be exploited during the
18 -binding protein (DBP; human DBP is known as Gc protein) is the precursor of macrophage activating fa
19 versus 298 +/- 90, respectively; P = 0.004); GC protein levels decreased with increasing severity of
20 L6, and CD10) but different from that of non-GC proteins (MUM1/IRF4 and BCL2).
21                          Mice immunized with gC protein produced antibodies that blocked C3b binding
22                                The domain of Gc protein responsible for macrophage activation was clo
23 ivating factor requires a precursor protein, Gc protein (serum vitamin D3-binding protein), as well a
24 emonstrate that Golgi-ER cycling of resident GC proteins takes place in both myoblasts and myotubes.
25 ify premature stop codons that would lead to gC proteins that lack the transmembrane domain.
26 uding CCHFV Gc and Gn antigens, we found the Gc protein to be more immunogenic compared with the Gn p
27 the MAF precursor activity of patient plasma Gc protein was found to be severely reduced in about 25%
28                         Mean serum levels of GC protein were significantly lower in patients with NAF
29 sing of BUNV GPC and found that both NSm and Gc proteins were cleaved at their own internal signal pe
30 zed beta-galactosidase or treatment of human Gc protein with immobilized beta-galactosidase and siali
31                                 Treatment of Gc protein with immobilized beta-galactosidase and siali