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1 neuron loss, the same as with NT-3 given via gelfoam.
2 splants of unmodified primary fibroblasts or Gelfoam alone did not elicit regeneration.
3                           Ad5-PSA priming in Gelfoam also abrogated the inhibitory effects of adenovi
4                 The nude mice with implanted Gelfoam and OSPDCs also were divided into the four thera
5 nesis were investigated by use of an in vivo Gelfoam angiogenesis assay in which IL-1beta produced a
6 on angiogenesis were studied with an in vivo Gelfoam angiogenesis assay.
7 e of Src inhibition to angiogenesis, in vivo gelfoam assays were done.
8                   Endothelial cells grown on Gelfoam blocks exhibited bactericidal activity towards S
9 in the bactericidal activity demonstrated by Gelfoam blocks laden with endothelial cells.
10                            Implantation of a Gelfoam/EC block in rats nearly abrogated the immune res
11 on of free porcine aortic ECs (PAEs) or of a Gelfoam/EC block, or after concomitant injection of PAEs
12                                    Moreover, gelfoams embedded with growth factors failed to induce f
13                                     Finally, Gelfoam enhanced immunization in a self-Ag model using t
14                                        Thus, Gelfoam enhances CTL activation by recombinant viral vec
15                       Aortic ECs embedded in Gelfoam expressed lower levels of MHC class II, costimul
16                                  The in vivo Gelfoam fluorescence angiogenesis assay was performed wi
17 combination on angiogenesis using an in vivo Gelfoam fluorescence angiogenesis mouse model implanted
18 eased the vessel length ratio in the in vivo Gelfoam fluorescent angiogenesis model, compared with al
19 re cultured on the three-dimensional matrix, Gelfoam, for 8-10 days.
20 pension, or seeded onto (2) collagen-matrix (Gelfoam [GF];), (3) GF/Matrigel (GF/MG), (4) GF/MG/VEGF
21  In the angiogenesis assay, vessel counts in Gelfoam implants were significantly decreased by the add
22 er concomitant injection of PAEs adjacent to Gelfoam in rats.
23                   Injecting PAEs adjacent to Gelfoam induced a significant response comparable to tha
24 At the completion of PIT, thrombin-saturated Gelfoam (Johnson and Johnson, Summerville, NJ) was embol
25 ortic endothelial cells were cultured within Gelfoam matrices and implanted in the perivascular space
26 ecules by free aortic ECs or ECs embedded in Gelfoam matrices by flow-cytometry.
27 ated the ability of a collagen-based matrix (Gelfoam; Pharmacia and Upjohn, Kalamazoo, MI) to improve
28 bust infiltration of vessels was observed in gelfoam saturated with conditioned medium from pancreati
29    This angiogenesis was nearly abrogated in gelfoams saturated with conditioned medium from cells tr
30                              Bioassay of the gelfoam showed that NT-3 bioactivity remained at 5 days
31                  The tract was plugged using Gelfoam slurry.
32                                 A pledget of Gelfoam soaked in Fluoro-Gold was placed at the thoracol
33                        Animals that received gelfoam soaked in nerve growth factor, brain derived neu
34 n into subcutaneously implanted Matrigel and Gelfoam sponge implants and the growth of orthotopically
35                                              Gelfoam sponges containing 0.4% agarose and IL-6 or basi
36 gy were only seen in follicles maintained on gelfoam supports and moreover, hair follicle size appear
37 then maintained these follicles in vitro, on Gelfoam supports, for up to 23 d (35 d of age) and compa
38              By day 10, endothelial cells on Gelfoam were found to secrete NO* at a rate exceeding 1.
39            The data show that coinjection of Gelfoam with type 5 adenovirus recombinant for prostate-