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1 ld be triggered in part through ligand-gated Glu receptors.
2 ctivates a plethora of pre- and postsynaptic Glu receptors.
3 ent antagonists of ionotropic L-glutamate (L-Glu) receptors.
4 he membrane trafficking of the AMPA receptor Glu receptor 1 (GluR1) subunit, but the role of direct p
5 tamate receptor subunits NMDA receptor 1 and Glu receptor 1 are selectively reduced in LRP1 forebrain
6 ta colocalize with the AMPA receptor subunit Glu receptor 1 but not with the GABAA receptor subunits
7 eurons consistently expressed high levels of Glu receptor 2 (GluR2) mRNA and AMPA receptors with low
8 hibit properties of Ca(2+)/Zn(2+)-permeable, Glu receptor 2 (GluR2)-lacking AMPARs before the rise in
9 r relative lack of the AMPA receptor subunit Glu receptor 2 (GluR2).
10 antages in applications where pre-photolysis Glu receptor activation and desensitization must be mini
11 loxyaspartic acid, but not by the ionotropic Glu receptor agonists, alpha-2-amino-3-(5-methyl-3-oxo-1
12 titute for L-glutamate (L-Glu) at excitatory Glu receptors, and occurs as free D-Asp in the mammalian
13                    A GABAA receptor agonist, Glu receptor antagonists, or a GABA-elevating agent were
14 y was designed to determine which glutamate (Glu) receptors are involved in excitatory neurotransmiss
15                                         Both GluD receptors are expressed in wider brain regions than
16                             Glutamate delta (GluD) receptors belong to the ionotropic glutamate recep
17 hibition of Asso signaling to Fyn, Pyk2, and Glu receptors by AZD0530 was tested by brain slice assay
18 deficient in either vesicular Glu release or Glu receptor expression and could be phenocopied by muta
19 minants of Ca(2+)-modulated signaling in the Glu receptor family remain elusive.
20  fact, we have now studied the properties of Glu receptors (GluRs) from the cerebral cortices of AD a
21                   The fast acting ionotropic Glu receptors (iGluRs) are ligand gated ion channels and
22 f the AD risk gene product Pyk2, and of NR2B Glu receptors in brain slices.
23                        Ionotropic glutamate (Glu) receptors in the central nervous system of animals
24 sp, which is transported but does not act at Glu receptors, induced [H+]i and [Na+]i changes that wer
25  pmol), an antagonist of the AMPA subtype of Glu receptors, into MD.
26 nce indicates that at least one of the plant Glu receptor-like molecules, GLR3.4, functions as an ami
27                      Metabotropic glutamate (Glu) receptors (mGlu receptors) play a key role in modul
28            Within NTS, multiple metabotropic Glu receptors (mGluRs) are present, but their roles are
29                  The metabotropic glutamate (Glu) receptors (mGluRs) play key roles in modulating exc
30 ad no effect, suggesting that the functional Glu receptors on the somata may be the same as those at
31 work for establishing a comprehensive map of Glu receptor populations within this major subdivision o
32                                          The Glu receptors present at the membrane level were investi
33 escribed phospholipase D-linked metabotropic Glu receptor to maintain the excitability of the sensory
34  ability to inhibit constitutive currents of GluD receptor variants and found that pentamidine potent
35                   Activation of postsynaptic Glu receptors was neither sufficient nor necessary to in
36 uron was stimulated during a period when the Glu receptors were blocked with the antagonist DNQX.