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1 SP55 (GORASP2) proteins function in stacking Golgi cisternae.
2  per Golgi stack, suggesting a loss of trans-Golgi cisternae.
3 le in calcium homeostasis in the CGN and cis-Golgi cisternae.
4 e arranged in successive concentric rings in Golgi cisternae.
5  the structural organization and function of Golgi cisternae.
6 cleate) from both the centrosome and the cis-Golgi cisternae.
7 ype of secretory vesicles derived from trans-Golgi cisternae.
8 ed in understanding tubules that connect the Golgi cisternae.
9 iffer in 3 amino acids and localize to trans Golgi cisternae.
10 SP-dependent membrane tethering of analogous Golgi cisternae.
11 se two GRASP proteins work together to stack Golgi cisternae.
12 imeras localize at the interior of the trans-Golgi cisternae.
13 cles were recruited to the region of nascent Golgi cisternae.
14 th the loss of membrane tethers on the trans-Golgi cisternae.
15 localized with Sec7-DsRed, a marker of trans-Golgi cisternae.
16  close proximity to ER exit sites and to cis Golgi cisternae.
17 rather than a direct role in the stacking of Golgi cisternae.
18 eticulum (ER)-derived membranes nucleate new Golgi cisternae.
19 all of the buds that lack ER also lack early Golgi cisternae.
20 ts in the inheritance of both early and late Golgi cisternae.
21  existing Golgi compartments to generate new Golgi cisternae.
22 hat GRASP65 is directly involved in stacking Golgi cisternae.
23  regulate assembly of COPI coat complexes on Golgi cisternae.
24 ickness and alignment with the other stacked Golgi cisternae.
25 ocalization, and in marked disruption of the Golgi cisternae.
26 nt (ERGIC), and ERGIC elements then generate Golgi cisternae.
27 y of SNARE proteins, Sed5p, present in early Golgi cisternae.
28 onation indicate that it is present in early Golgi cisternae.
29 lpha-factor and the accumulation of abnormal Golgi cisternae.
30  reticulum before being transported into the Golgi cisternae.
31 n the p115-mediated docking of vesicles with Golgi cisternae [12].
32 proportional increase in the number of trans-Golgi cisternae, a switch to larger-sized secretory vesi
33                             Margins of trans-Golgi cisternae accumulate the LM8 xylogalacturonan (XGA
34 down or in ARCL2 cells resulted in distended Golgi cisternae, accumulation of abnormal lysosomes and
35 tion of the golgin GMAP-210 causes a loss of Golgi cisternae and accumulation of numerous vesicles.
36 icles bud exclusively from medial- and trans-Golgi cisternae and are confined to the space around the
37 alization away from the ER to the downstream Golgi cisternae and blocked the RESET pathway.
38 in flattened, disc-shaped organelles such as Golgi cisternae and ER sheets.
39  BP-80 protein is present in dilated ends of Golgi cisternae and in "prevacuoles," which are small va
40  GRASP65 have been implicated in stacking of Golgi cisternae and lateral linking of stacks within the
41    Coat protein I (COPI) vesicles arise from Golgi cisternae and mediate the recycling of proteins fr
42 es are segregated from each other within the Golgi cisternae and packaged into separate vesicles.
43 al endocytosis and retrograde transport into Golgi cisternae and perhaps ER.
44 infected cells, F13L-GFP was associated with Golgi cisternae and post-Golgi vesicles containing the L
45 s in transit resulted in condensation of the Golgi cisternae and stacks.
46 nalysis revealed that Cdc42 is active at all Golgi cisternae and that this activity is controlled by
47 ctron microscopy, CALNUC is localized to cis-Golgi cisternae and the cis-Golgi network (CGN).
48 amily has been implicated in the stacking of Golgi cisternae and the regulation of Golgi disassembly/
49 ed with the plasma membrane and occasionally Golgi cisternae and tubulovesicles.
50  were continuously concentrated over stacked Golgi cisternae and tubulovesicular Golgi structures sim
51 level, Golgi fragments showed swollen distal Golgi cisternae and vesicular structures.
52  analogous to early (RER and ERGIC), middle (Golgi cisternae), and late (TGN) secretory pathway compa
53 d secretory vesicles that bud from the trans-Golgi cisternae, and an increase in osmium staining of t
54 e contacts with mitochondria, multiple trans Golgi cisternae, and compartments of the endo-lysosomal
55 Protein I)-coated vesicles and buds, but not Golgi cisternae, and it specifically activates Rab1.
56 34 expressed in HeLa cells was restricted to Golgi cisternae, and its membrane association was sensit
57 ulum and the Golgi complex, between adjacent Golgi cisternae, and possibly in the endocytic pathway.
58 rgo sequential enzymatic modification in the Golgi cisternae are displayed at the plasma membrane as
59                            During telophase, Golgi cisternae are regenerated and stacked from a heter
60  and transport continue-even when individual Golgi cisternae are separated and "land-locked" between
61   Before entering mitosis, the stacks of the Golgi cisternae are separated from each other, and inhib
62 localizes with RabE(RAB11), arriving at late Golgi cisternae as they dissipate into exocytic carriers
63 bute to stacking and lateral interactions of Golgi cisternae as well as help it function as a vesicle
64 on probably does not occur at the surface of Golgi cisternae but likely at post-Golgi stages of the v
65 isiae, in which the maturation of individual Golgi cisternae can be visualized.
66                     In mammalian cells, flat Golgi cisternae closely arrange together to form stacks.
67 ipogenesis and is primarily localized to the Golgi cisternae distinct from the trans-Golgi network (T
68 t mediate both formation and stacking of the Golgi cisternae during post-mitotic reassembly.
69         This juxtaposition may indicate that Golgi cisternae form at tER sites.
70  are most readily explained by assuming that Golgi cisternae form at the cis face of the stack, progr
71                                        Early Golgi cisternae form near the ER and become loaded with
72 an NSF function during cell-free assembly of Golgi cisternae from mitotic Golgi fragments revealed th
73 s essential for the p97-mediated regrowth of Golgi cisternae from mitotic Golgi fragments, a process
74 mammalian cells.The different composition of Golgi cisternae gave rise to two different models for in
75 ter incubation demonstrated that the stacked Golgi cisternae generated a heterogeneous population of
76 sicle trafficking between the cis and medial Golgi cisternae has been used previously to identify sev
77            However, an inability to separate Golgi cisternae has meant that the cisternal distributio
78 d that CS2 antiserum stains medial and trans-Golgi cisternae in all cell types investigated.
79                 EM data showed a dilation of Golgi cisternae in cells expressing the M2 ion channel p
80 the cell, and PtdIns4P is elevated on medial Golgi cisternae in cells lacking Vps74 or Sac1, suggesti
81                Although the dilation of some Golgi cisternae in Cog5-deficient cells resembled that o
82 s coexpressed, but was largely restricted to Golgi cisternae in the absence of F13L-GFP or when the F
83 been shown to play a role in stacking of the Golgi cisternae in vitro.
84  Ypt/Rab family, Ypt1 and Ypt31, to specific Golgi cisternae interferes with addressing this question
85                              The stacking of Golgi cisternae involves GRASP65 and GRASP55.
86                        This behavior of late Golgi cisternae is consistent with the cisternal maturat
87       The order of enzymatic activity across Golgi cisternae is essential for complex molecule biosyn
88 he TGN is disrupted and vesicle budding from Golgi cisternae is reduced in the tno1 mutant, and these
89  Golgi has been explained by the movement of Golgi cisternae, known as cisternal maturation.
90 importance of retrieval-based targeting, few Golgi cisternae-localized proteins have been demonstrate
91 f eicosanoid products, while the location in Golgi cisternae may also reflect its action as a secrete
92 lized to vesicular tubular complexes and cis-Golgi cisternae, mostly in brain, atlastin-2 and -3 are
93 s correlate strongly with a proliferation of Golgi cisternae observed in earlier electron microscopy.
94 f Ena increases the number of cis- and trans-Golgi cisternae per cell, and Ena overexpression also re
95             APM1 localizes at the margins of Golgi cisternae, plasma membrane, select multivesicular
96 d a mouse lacking both GORASPs and find that Golgi cisternae remained stacked.
97               Restacking of the newly formed Golgi cisternae requires dephosphorylation of Golgi stac
98          We are prone to thinking by analogy-Golgi cisternae stack like pancakes, red blood cells loo
99  elevates C1P steady-state levels and alters Golgi cisternae stack morphology.
100 ow three types of direct connections between Golgi cisternae that are normally distinct from one anot
101 ps74 is a sensor of PtdIns4P level on medial Golgi cisternae that directs Sac1-mediated dephosphospho
102 ese tomograms revealed structures within the Golgi cisternae that have not been seen before.
103 Staples and soluble aggregates placed in cis-Golgi cisternae therefore have different fates.
104 mine the narrow central spacing of the trans-Golgi cisternae through zipper-like interactions, thereb
105 anes have been implicated in the adhesion of Golgi cisternae to each other to form their characterist
106 SP domain and mediate homotypic tethering of Golgi cisternae to form extended Golgi ribbons.
107 stacking of Golgi by the ectopic adhesion of Golgi cisternae to mitochondria.
108 e different types of substrates in different Golgi cisternae to retrieve some proteins back to the ER
109 pergillus nidulans hyphae, we show that late Golgi cisternae undergo changes in composition to gradua
110                 The pericentriolar stacks of Golgi cisternae undergo extensive reorganization during
111  and have been implicated in the stacking of Golgi cisternae, vesicle tethering, and mitotic progress
112 ate that GRASP55 and GRASP65 stack mammalian Golgi cisternae via a common mechanism.
113                            GRASP65 links cis-Golgi cisternae via a homotypic, N-terminal PDZ interact
114 C17 knockdown, the polarized organization of Golgi cisternae was altered, creating glycosylation defe
115 ess response, a significant expansion of the Golgi cisternae was observed in BHK-21, Vero E6 and mosq
116 rane protein complex localized on cis/medial Golgi cisternae where it may participate in tethering in
117 t intermediate production and consumption by Golgi cisternae, with only a minor contribution of pre-e
118 nsferase was mapped to the medial- and trans-Golgi cisternae, with some being present in the TGN.
119 hondria to invade the stack and even replace Golgi cisternae within a few hours.

 
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