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1 an increased short-term risk of epilepsy and Guillain-Barre syndrome.
2 rders, including microcephaly in infants and Guillain-Barre syndrome.
3 Cases of acute ZIKV are also associated with Guillain-Barre syndrome.
4 e care beds capacity to manage patients with Guillain-Barre syndrome.
5 5 accompanied by congenital microcephaly and Guillain-Barre syndrome.
6 e of the infection in the development of the Guillain-Barre syndrome.
7  with ZIKV infection before the onset of the Guillain-Barre syndrome.
8 an etiologic agent of fetal microcephaly and Guillain-Barre syndrome.
9  possible association between Zika virus and Guillain-Barre syndrome.
10 on has been linked to congenital defects and Guillain-Barre syndrome.
11 e patient because of multiorgan failure with Guillain-Barre syndrome.
12 ment disorder, 1 had encephalitis, and 1 had Guillain-Barre syndrome.
13 orldwide and is implicated in development of Guillain-Barre syndrome.
14 ccines used in the USA increased the risk of Guillain-Barre syndrome.
15 ng to stroke, and acute neuropathies such as Guillain-Barre syndrome.
16 eutralization of autoantibodies in the fatal Guillain-Barre syndrome.
17 orldwide and is implicated in development of Guillain-Barre syndrome.
18 possible association between vaccination and Guillain-Barre syndrome.
19 nts safety concerns about the development of Guillain-Barre syndrome.
20 as suspended because of an increased risk of Guillain-Barre syndrome.
21 ked to microcephaly, congenital disease, and Guillain-Barre syndrome.
22 ions, including irritable bowel syndrome and Guillain-Barre syndrome.
23 frequent antecedent of autoimmune neuropathy Guillain-Barre syndrome.
24 s as well as cases of the autoimmune disease Guillain-Barre syndrome.
25 nal neuropathy that closely resembled axonal Guillain-Barre syndrome.
26 nstem encephalitis or those with overlapping Guillain-Barre syndrome.
27 ical features in paralytic rabies and axonal Guillain-Barre syndrome.
28  in some cases of Miller Fisher syndrome and Guillain-Barre syndrome.
29  corticosteroids, in hastening recovery from Guillain-Barre syndrome.
30 es mapping their relationship to subtypes of Guillain-Barre syndrome.
31 its characteristic of multiple sclerosis and Guillain-Barre syndrome.
32 o a form of neuromuscular paralysis known as Guillain-Barre syndrome.
33 ecovery and impact the functional outcome of Guillain-Barre syndrome.
34 on in acute peripheral neuropathies, such as Guillain-Barre syndrome.
35  lipopolysaccharide has been associated with Guillain-Barre syndrome.
36 malformations (congenital Zika syndrome) and Guillain-Barre syndrome.
37 oimmune neuritis (EAN) is an animal model of Guillain-Barre syndrome.
38 nfectious sequelae of reactive arthritis and Guillain-Barre syndrome.
39 utoimmune neuritis (EAN), an animal model of Guillain-Barre syndrome.
40 e compatible with a role in the causation of Guillain-Barre syndrome.
41  onset of a syndrome diagnosed clinically as Guillain-Barre syndrome.
42           IVGG is a recognised treatment for Guillain-Barre syndrome.
43  disease, motor neuron disease, epilepsy and Guillain-Barre syndrome.
44 atments are needed to improve the outcome of Guillain-Barre syndrome.
45 an increased short-term risk of epilepsy and Guillain-Barre syndrome.
46 disease, motor neuron disease, epilepsy, and Guillain-Barre syndrome.
47  microcephaly, congenital malformations, and Guillain-Barre syndrome.
48 er from acute demyelinating diseases such as Guillain-Barre syndrome.
49 s human diseases, including microcephaly and Guillain-Barre syndrome.
50 as is linked to congenital birth defects and Guillain-Barre syndrome.
51  microcephaly, congenital malformations, and Guillain-Barre syndrome.
52  other severe neurological diseases, such as Guillain-Barre syndrome.
53  demyelinating polyneuropathy subtype of the Guillain-Barre syndrome.
54 ng evidence for Zika virus infection causing Guillain-Barre syndrome.
55 irus (ZIKV) infection has been linked to the Guillain-Barre syndrome.
56 rus and dengue virus infection in developing Guillain-Barre syndrome.
57 gical complications of the Miller-Fisher and Guillain-Barre syndromes.
58 mboembolic events, autoimmune disorders, and Guillain-Barre syndrome; 0.1 for anaphylaxis and death;
59    We interviewed 180 of 273 adults with the Guillain-Barre syndrome; 15 declined to participate, and
60 e Americas has unveiled microcephaly (1) and Guillain-Barre syndrome(2,3) as ZIKV-associated neurolog
61 kungunya infection; p<=0.0001), particularly Guillain-Barre syndrome (25 [61%] patients).
62 2 patients) or acute polyradiculoneuropathy (Guillain-Barre syndrome) (4/12).
63               Among the 68 patients with the Guillain-Barre syndrome, 50% were found to have bilatera
64 vritis with cranial nerve involvement (16%), Guillain-Barre syndrome (8%) and mixed motor and sensory
65 esses on the physician differential included Guillain-Barre syndrome (99 cases) and myasthenia gravis
66  evidence now exists that axonal subtypes of Guillain-Barre syndrome, acute motor axonal neuropathy (
67 rs found no evidence of an increased risk of Guillain-Barre syndrome after seasonal influenza vaccine
68 ring project identified an increased risk of Guillain-Barre syndrome after vaccination; however, some
69             Surges in congenital defects and Guillain-Barre syndrome alerted the world to the danger
70 immunoglobulin is an effective treatment for Guillain-Barre syndrome, although the mechanism of actio
71 uals was vaccinated in the UK, 21.5 cases of Guillain-Barre syndrome and 5.75 cases of sudden death w
72                       The reporting rates of Guillain-Barre syndrome and anaphylaxis after TIV-HD rec
73 porting rates were calculated for reports of Guillain-Barre syndrome and anaphylaxis.
74  and unexpected clinical outcomes, including Guillain-Barre syndrome and birth defects, has brought a
75 s (ZIKV) to cause serious disease, including Guillain-Barre syndrome and birth defects, was not recog
76 matory CD4(+) T cells that is reminiscent of Guillain-Barre syndrome and chronic inflammatory demyeli
77 ral neuropathy that resembles human diseases Guillain-Barre syndrome and chronic inflammatory demyeli
78 i-tubulin autoantibodies are associated with Guillain-Barre syndrome and chronic inflammatory demyeli
79                                           In Guillain-Barre syndrome and CIDP intravenous immunoglobu
80 e Zika virus (ZIKV) has been associated with Guillain-Barre syndrome and fetal abnormalities, but the
81 ction of the link between ZIKV infection and Guillain-Barre syndrome and fetal neurological defects,
82 eports of a preceding respiratory illness in Guillain-Barre syndrome and has important implications f
83 view the occurrence and clinical features of Guillain-Barre syndrome and its variant, the Miller Fish
84  sequelae such as hemolytic uremic syndrome, Guillain-Barre syndrome and malnutrition.
85  treatment is beneficial in CIDP, but not in Guillain-Barre syndrome and may worsen MMNCB.
86  of recent outbreaks and concerning links to Guillain-Barre syndrome and microcephaly are incompletel
87 d complications of ZIKV infection, including Guillain-Barre syndrome and microcephaly, potential inte
88  with serious neurological anomalies such as Guillain-Barre syndrome and microcephaly.
89 ditions with a primary immune basis, such as Guillain-Barre syndrome and multiple sclerosis.
90  of therapeutic plasma exchange for managing Guillain-Barre syndrome and myasthenia gravis were publi
91 uch as acute disseminated encephalomyelitis, Guillain-Barre syndrome and neuromyelitis optica.
92 severe neuropathology in neonates as well as Guillain-Barre syndrome and other neurologic disorders i
93                           ZIKV is associated Guillain-Barre syndrome and other neurological complicat
94 were evidences of two severe adverse events (Guillain-Barre Syndrome and paralysis) present in TIV.
95 casionally are similar to those described in Guillain-Barre syndrome and suggest a possible relations
96 t differ significantly between patients with Guillain-Barre syndrome and those in the two control gro
97                       Two patients developed Guillain-Barre syndrome, and 3 of 4 pregnancies had adve
98 n a proportion of patients with motor axonal Guillain-Barre syndrome, and also provide an explanation
99 e (MFS) variant of the paralytic neuropathy, Guillain-Barre syndrome, and are believed to be the prin
100  (ZIKV) is an emerging virus associated with Guillain-Barre syndrome, and fetal microcephaly as well
101 patients with chronic motor neuropathies and Guillain-Barre syndrome, and GQ1b/ disialosyl antisera a
102 ction of smell and taste, muscle injury, the Guillain-Barre syndrome, and its variants.
103 ion that mimic botulism, such as stroke, the Guillain-Barre syndrome, and myasthenia gravis.
104 MENTARY ON THIS ARTICLE : In axonal forms of Guillain-Barre syndrome, anti-ganglioside antibodies bin
105 de, collaborative studies of the spectrum of Guillain-Barre syndrome are accruing data for clinical a
106      Diseases such as multiple sclerosis and Guillain-Barre syndrome are characterized not only by wi
107                   Under the umbrella term of Guillain-Barre syndrome are several recognisable variant
108 europathy syndromes, some of which may mimic Guillain-Barre syndrome, as many will have a metabolic a
109                     The relative risk of the Guillain-Barre syndrome associated with vaccination, adj
110 ratory failure is a frequent complication of Guillain-Barre syndrome, associated with high morbidity
111 ith a specific Penner serotype, and 14 of 16 Guillain-Barre syndrome-associated isolates tested in th
112              A total of 68 patients with the Guillain-Barre syndrome at six Colombian hospitals were
113                           The modest risk of Guillain-Barre syndrome attributed to vaccination is con
114  of serotype O:19 strains from patients with Guillain-Barre syndrome but not in cores of nonneuropath
115 ture confirms the use of plasma exchange for Guillain-Barre syndrome but suggests that inadequate evi
116                 Features initially suggested Guillain-Barre syndrome, but botulinum toxin was identif
117 e in access to nutrients; an outbreak of the Guillain-Barre syndrome caused by lack of chlorination c
118 er Fisher syndrome is a localized variant of Guillain-Barre syndrome, characterized by ophthalmoplegi
119 , nonlength-dependent neuropathies including Guillain-Barre syndrome, chronic inflammatory demyelinat
120 iffuse, nonlength-dependent neuropathies are Guillain-Barre syndrome, chronic inflammatory demyelinat
121 2 g/kg is effective as first-line therapy in Guillain-Barre syndrome, chronic inflammatory demyelinat
122 rts the use of intravenous immunoglobulin in Guillain-Barre syndrome, chronic inflammatory demyelinat
123 in the western hemisphere has been linked to Guillain-Barre syndrome, congenital microcephaly, and de
124                                              Guillain-Barre syndrome consists of at least four subtyp
125                                              Guillain-Barre syndrome developed following infliximab t
126             In addition, 15 patients in whom Guillain-Barre syndrome developed following TNFalpha ant
127 om the Miller Fisher syndrome variant of the Guillain-Barre syndrome developed while he was receiving
128 case-control study, cases were patients with Guillain-Barre syndrome diagnosed at the Centre Hospital
129 lmonella, Yersinia, and Shigella organisms), Guillain-Barre syndrome (due to Campylobacter organisms)
130 documented by RT-PCR among patients with the Guillain-Barre syndrome during the outbreak of ZIKV infe
131 stem represents a useful means of monitoring Guillain-Barre syndrome during the pandemic.
132              42 patients were diagnosed with Guillain-Barre syndrome during the study period.
133 peripheral neuropathy for the development of Guillain-Barre syndrome during Zika virus outbreaks.
134 n, and 3 patients had complete resolution of Guillain-Barre syndrome following therapy.
135 oisson-based analysis to compare the risk of Guillain-Barre syndrome following vaccination in each se
136 ination in the six weeks before the onset of Guillain-Barre syndrome for 19 patients.
137 ed risk interval analysis of chart-confirmed Guillain-Barre syndrome found an elevated but not statis
138 o increase in the risk of vaccine-associated Guillain-Barre syndrome from 1992-1993 to 1993-1994.
139 6 acute flaccid paralysis cases diagnosed as Guillain-Barre syndrome from 2000 through 2008 in childr
140                                              Guillain Barre syndrome (GBS) is the most common cause o
141 are strongly associated with axonal forms of Guillain Barre syndrome (GBS).
142                             Surveillance for Guillain-Barre syndrome (GBS) after receipt of this vacc
143                                              Guillain-Barre syndrome (GBS) and chronic inflammatory d
144                                              Guillain-Barre syndrome (GBS) and Fisher syndrome (FS) a
145 tis (EAN) is considered the in vivo model of Guillain-Barre syndrome (GBS) and has been extensively s
146 lammatory demyelinating neuropathies such as Guillain-Barre syndrome (GBS) and its animal model, expe
147  and consequences of adult infection include Guillain-Barre syndrome (GBS) and meningoencephalitis.
148  thought to be involved in the triggering of Guillain-Barre syndrome (GBS) and Miller-Fisher syndrome
149 rus was a cause of the neurological disorder Guillain-Barre syndrome (GBS) and of microcephaly and ot
150 1 vaccine) and the prior association between Guillain-Barre syndrome (GBS) and the 1976 H1N1 influenz
151                              We diagnosed 11 Guillain-Barre syndrome (GBS) cases among 71,904 COVID p
152 Americas has been associated with a surge in Guillain-Barre syndrome (GBS) cases.
153 ylobacter jejuni isolated from patients with Guillain-Barre syndrome (GBS) differ from isolates isola
154  case-control study where 24 cases developed Guillain-Barre syndrome (GBS) during the 2014 chikunguny
155 5 days of symptom onset on 369 patients with Guillain-Barre Syndrome (GBS) enrolled in a trial compar
156                         An increased risk of Guillain-Barre syndrome (GBS) following administration o
157                  Given the increased risk of Guillain-Barre Syndrome (GBS) found with the 1976 swine
158                                              Guillain-Barre syndrome (GBS) has been associated with s
159                                              Guillain-Barre syndrome (GBS) has been reported to be as
160 tween Campylobacter infection and subsequent Guillain-Barre syndrome (GBS) has been well documented.
161 e of neutralizing antibodies in Zika-induced Guillain-Barre syndrome (GBS) has not yet been investiga
162  national morbidity and mortality burden for Guillain-Barre syndrome (GBS) in the United States, a na
163                                              Guillain-Barre syndrome (GBS) is a postinfectious autoim
164                                              Guillain-Barre syndrome (GBS) is a postinfectious neurop
165                                              Guillain-Barre syndrome (GBS) is a rare, but potentially
166                                              Guillain-Barre syndrome (GBS) is an acute inflammatory d
167                                              Guillain-Barre syndrome (GBS) is an acute polyradiculone
168                                              Guillain-Barre syndrome (GBS) is an acute polyradiculopa
169                                              Guillain-Barre syndrome (GBS) is an acute postinfectious
170                                              Guillain-Barre syndrome (GBS) is an autoimmune disease c
171                                              Guillain-Barre syndrome (GBS) is an immune-mediated peri
172 te motor axonal neuropathy (AMAN) variant of Guillain-Barre syndrome (GBS) is associated with anti-GD
173                                              Guillain-Barre Syndrome (GBS) is believed to be caused b
174 asonal variation in incidence and subtype of Guillain-Barre syndrome (GBS) is contradictory, but has
175                                              Guillain-Barre syndrome (GBS) is the most common cause o
176       An unexplained increase in the risk of Guillain-Barre syndrome (GBS) occurred among recipients
177  are specific to O serotypes associated with Guillain-Barre syndrome (GBS) or whether they are freque
178                                  Clinically, Guillain-Barre syndrome (GBS) predominated among regions
179 a exchanges and intravenous immunoglobulins, Guillain-Barre syndrome (GBS) still carries non-negligib
180 tigenic targets of autoantibodies present in Guillain-Barre syndrome (GBS), a group of neuropathies w
181 oenteritis and often precedes development of Guillain-Barre syndrome (GBS), a life-threatening paraly
182                                              Guillain-Barre syndrome (GBS), a neurologic disease char
183                                              Guillain-Barre syndrome (GBS), a neurologic disease that
184                                              Guillain-Barre syndrome (GBS), an acute, immune-mediated
185                                              Guillain-Barre syndrome (GBS), an autoimmune disorder of
186 resent in patients with the motor variant of Guillain-Barre syndrome (GBS), and their pathological ro
187  been shown to be an antecedent infection in Guillain-Barre syndrome (GBS), but the clinical spectrum
188                       Data on AFP, including Guillain-Barre syndrome (GBS), could be analyzed from a
189                          The pathogenesis of Guillain-Barre syndrome (GBS), especially the classic in
190 V infection among patients hospitalized with Guillain-Barre syndrome (GBS), meningoencephalitis, or t
191 ested as essential for accurate diagnosis in Guillain-Barre syndrome (GBS).
192 ediate the postinfectious paralytic disease, Guillain-Barre syndrome (GBS).
193 sociated with acute immune neuropathy called Guillain-Barre syndrome (GBS).
194  are being explored in myasthenia gravis and Guillain-Barre syndrome (GBS).
195 h the development of the neurologic disorder Guillain-Barre syndrome (GBS).
196 ated in the pathogenesis of some variants of Guillain-Barre syndrome (GBS).
197 in the pathogenesis of Campylobacter-induced Guillain-Barre syndrome (GBS).
198  HS:19 is associated with the development of Guillain-Barre syndrome (GBS).
199 mmune attack is different in the subtypes of Guillain-Barre syndrome (GBS).
200 ysiologic features similar to those of human Guillain-Barre syndrome (GBS).
201 ns that include congenital abnormalities and Guillain-Barre syndrome (GBS).
202 cacy and anticipate outcome in patients with Guillain-Barre syndrome (GBS).
203 developmental disorders; (3) vaccine-induced Guillain-Barre syndrome (GBS); (4) vaccine-induced autoi
204 upport the hypothesis that C. jejuni induces Guillain-Barre syndrome (GBS); however, many questions a
205 d chronic autoimmune neuropathies, including Guillain-Barre syndromes (GBS) are often characterized b
206  risk of neurologic complications (including Guillain-Barre syndrome [GBS]) with ZIKV infection.
207        In 20 of these 42 patients (48%), the Guillain-Barre syndrome had a parainfectious onset.
208                       41 (98%) patients with Guillain-Barre syndrome had anti-Zika virus IgM or IgG,
209                                Patients with Guillain-Barre syndrome had electrophysiological finding
210                       39 (93%) patients with Guillain-Barre syndrome had Zika virus IgM and 37 (88%)
211 ni infections with subsequent development of Guillain-Barre syndrome has increased concerns from a pa
212 ed by an immune attack on the axon ("axonal" Guillain-Barre syndrome) has been advanced primarily bas
213 de of action, and roles of complement in the Guillain-Barre syndrome have been controversial.
214                              The concepts of Guillain-Barre syndrome have changed substantially over
215             About a quarter of patients with Guillain-Barre syndrome have had a recent Campylobacter
216 een described in eight patients to date, and Guillain-Barre syndrome in 19 patients.
217 IKV) and neurologic complications, including Guillain-Barre syndrome in adults and CNS abnormalities
218 quito-borne pathogen that has been linked to Guillain-Barre syndrome in adults and congenital microce
219 associated with congenital malformations and Guillain-Barre syndrome in adults have placed public hea
220 mans, including microcephaly in newborns and Guillain-Barre syndrome in adults.
221 mans, including microcephaly in newborns and Guillain-Barre syndrome in adults.
222 d other neurological defects in neonates and Guillain-Barre syndrome in adults.
223 use Congenital Zika Syndrome in neonates and Guillain-Barre syndrome in adults.
224        Zika virus infection could also cause Guillain-Barre syndrome in adults.
225 ital malformations, and its association with Guillain-Barre syndrome in adults.
226 s ability to cause microcephaly in utero and Guillain-Barre syndrome in adults.
227  to devastating congenital birth defects and Guillain-Barre syndrome in adults.
228 ngenital malformations in infants as well as Guillain-Barre syndrome in adults.
229 e occurrence of microcephaly in neonates and Guillain-Barre syndrome in adults.
230                                              Guillain-Barre syndrome in northern China occurs in two
231            Patients given a diagnosis of the Guillain-Barre syndrome in the 1992-1993 and 1993-1994 i
232 racterized the clinical features of cases of Guillain-Barre syndrome in the context of this ZIKV infe
233 nown as congenital Zika syndrome, as well as Guillain-Barre syndrome, in ZIKV-infected adults.
234 rican and the Caribbean be used to establish Guillain-Barre syndrome incidence rates.
235 re associated with a small increased risk of Guillain-Barre syndrome (incidence rate ratio 2.35, 95%
236 neuropathies include sensory variants of the Guillain-Barre syndrome, including Miller-Fisher syndrom
237                                              Guillain-Barre syndrome is a rare but serious health dis
238                  The motor axonal variant of Guillain-Barre syndrome is associated with anti-GD1a imm
239                                              Guillain-Barre syndrome is now recognized as a heterogen
240                                              Guillain-Barre syndrome is often caused by Campylobacter
241                                              Guillain-Barre syndrome is the most common and most seve
242       Inflammatory demyelination of nerve in Guillain-Barre syndrome is triggered in most patients by
243 il and its association with microcephaly and Guillain-Barre syndrome led to accelerated vaccine devel
244 its likely association with microcephaly and Guillain-Barre syndrome make Zika an urgent public healt
245 bstantial mortality and morbidity, including Guillain-Barre syndrome, microcephaly and other fetal de
246 als, IVIg has been effective in treating the Guillain-Barre syndrome, multifocal motor neuropathy, ch
247 yelinating polyradiculoneuropathy (n=53) and Guillain-Barre syndrome (n=21).
248 res (n = 44), meningitis (n = 3), and other (Guillain-Barre Syndrome) (n = 1).
249 f human immunoglobulin in an animal model of Guillain-Barre syndrome, namely experimental autoimmune
250            ZIKV infection might also trigger Guillain-Barre syndrome, neuropathy, and myelitis.
251                                           In Guillain-Barre syndrome, newly described antibodies bind
252  Possible associations with microcephaly and Guillain-Barre syndrome observed in this outbreak have r
253 l neurological disorders (n = 8), seven with Guillain-Barre syndrome, one with brachial plexopathy, s
254 in a demyelinating neuropathy that may mimic Guillain-Barre syndrome or chronic inflammatory demyelin
255 an sural nerves and those from patients with Guillain-Barre syndrome or chronic inflammatory demyelin
256  invasion), neuromuscular complications (eg, Guillain-Barre syndrome or transient muscle dysfunctions
257 o pneumococcal infection), PCV13 and PPSV23 (Guillain-Barre syndrome), or PPSV23 (cellulitis).
258 ctic serious allergic reaction, anaphylaxis, Guillain-Barre syndrome, or invasive Hib disease.
259  the paralytic form, which closely resembles Guillain-Barre syndrome, or when a patient is comatose a
260 ected in a cohort of mechanically ventilated Guillain-Barre syndrome patients.
261  with dysglycemia in mechanically ventilated Guillain-Barre syndrome patients.
262 between dysglycemia and neurologic status in Guillain-Barre syndrome patients.
263 ding translated to about 1.6 excess cases of Guillain-Barre syndrome per million people vaccinated.
264 ts slightly more than one additional case of Guillain-Barre syndrome per million persons vaccinated a
265 os and attributable risks of excess cases of Guillain-Barre syndrome per million vaccinations.
266                      The long-term sequelae (Guillain-Barre syndrome, reactive arthritis, and postinf
267 l might be an adjuvant therapy for improving Guillain-Barre syndrome recovery.
268 f glycosylated flagellin with development of Guillain-Barre syndrome remains speculative, but the pos
269                                              Guillain-Barre syndrome remains the most frequent neurol
270  virus (ZIKV), which causes microcephaly and Guillain-Barre syndrome, signals an urgency to identify
271 emic lupus erythematosus, myasthenia gravis, Guillain-Barre syndrome, skin blistering syndromes, and
272    Highly visible health conditions, such as Guillain-Barre syndrome, spontaneous abortion, or even d
273                                       In the Guillain-Barre syndrome subform acute motor axonal neuro
274  site-specific injury in acute AGAb-mediated Guillain-Barre syndrome subforms and their chronic count
275 te motor axonal neuropathy (AMAN), a form of Guillain-Barre syndrome that selectively affects motor n
276 re frequently than expected in patients with Guillain-Barre syndrome, this association is extremely r
277 eports of neurological sequelae ranging from Guillain Barre syndrome to microcephaly.
278 r of reports of influenza-vaccine-associated Guillain-Barre syndrome to the national Vaccine Adverse
279 ipid reactivity was studied in patients with Guillain-Barre syndrome using both ELISA and combinatori
280 mptoms of ZIKV infection and symptoms of the Guillain-Barre syndrome was 7 days (interquartile range,
281 ecember 1976 after an increased incidence of Guillain-Barre syndrome was attributed to the vaccine.
282 smitted, and an increase in the incidence of Guillain-Barre syndrome was reported in some countries d
283       During the same period, an increase in Guillain-Barre syndrome was reported, suggesting a possi
284 ocedures employed in people to help diagnose Guillain-Barre syndrome, we determined that nerve conduc
285 through March 2016, clusters of cases of the Guillain-Barre syndrome were observed during the outbrea
286 mbers of neurological complications, such as Guillain-Barre syndrome, were reported(13,14) together w
287 s mounting that Zika virus can contribute to Guillain-Barre syndrome which causes temporary paralysis
288 study did not control for infection prior to Guillain-Barre syndrome, which may have been a confounde
289 oneuropathy and in none of the patients with Guillain-Barre syndrome, which was confirmed by cell bin
290 nant women, their infants, and patients with Guillain-Barre syndrome who had traveled to areas with Z
291                                Patients with Guillain-Barre syndrome who had Zika and chikungunya dua
292          In 18 of 42 patients (43%) with the Guillain-Barre syndrome who underwent laboratory testing
293 series method to investigate the relation of Guillain-Barre syndrome with influenza vaccine and influ
294     The severe, generalised manifestation of Guillain-Barre syndrome with respiratory failure affects
295                            An association of Guillain-Barre syndrome with TNFalpha antagonist therapy
296       In contrast, the relative incidence of Guillain-Barre syndrome within 90 days of an influenzali
297                    The relative incidence of Guillain-Barre syndrome within 90 days of vaccination wa
298 roup, 11 (0.5%) patients were diagnosed with Guillain-Barre syndrome within the first year after LNB
299 group 11 (0.5%) patients were diagnosed with Guillain-Barre syndrome within the first year after LNB
300 g microcephaly, congenital malformation, and Guillain-Barre syndrome, Zika virus (ZIKV) remains a per

 
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